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populations at risk for TBI
- young children- child abuse
- adolescents to young adult- risky activities
- older adults- falls
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risk factors of TBI
- MVA
- bicycling without helmet
- alcohol use
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closed injury
- force drives brain into contact with skull
- no skull fracture
- can occur with severe neck injury without direct trauma to skull (whip lash)
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open injury
- skull fractures
- meninges are penetrated, brain exposed
- impact, gun shot, explosives
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primary injury
- due to forces exerted at time of injury
- coup: site of initial contact
- counter-coup: impact on opposite sd due to recoil (contusion, swelling, blood clots)
- diffuse axonal injury- shearing and tensile forces; white matter injury greater predictor of mortality and disability
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secondary injury
- swelling/edema causes inc ICP
- can also have hemorrhage which inc ICP
- vascular damage from contusion
- excitotoxicity from inc Ca
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Wallerian degeneration
when axon is damaged, distal segement degenerates due to loss of axonal transport
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concussion
mild TBI; difficulties with cognition or balance following direct/indirect head trauma
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evaluating athlete w/ concussion off-site
- neurophyschological assessment
- postural stability testing
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concussion sequelae
post concussion syndrome: headache, fatigue, dizziness, personality changes, dec control of emotions
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cerebral perfusion pressures
- difference bewtween MABRP and ICP
- if too low, risk of ischemia
- normal is 70-85
- inc by using IV fluid to inc blood volume
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managing ICP
- normal is 15 mmHg
- step 1: hyperventilation
- step 2: manitol or hyperosmolar saline
- step 3: barbituate therapy (medically induced coma)
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retrograde amnesia
loss of recal of events immediately preceding injury
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posttraumatic amnesia
- loss of recall of period between injury and time of functional recovery
- indication of injury severity
- no carryover of tasks requiring learning during this period
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anterograde amnesia
- difficulty/inability to form new memory
- dec attention abilities
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poorer prognosis of TBI
- 90% of pts who die do so within 48 hrs
- uncontrolled ICP is primary cause of death
- injury severity
- long duration of LOC
- loss of papillary reflexes
- acute hemispheric swelling
- mid-line shift
- epilepsy developing within 7 days
- older age
- lower education
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Rancho Level I-V
no ability to learn new info
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Rancho level IV
confused and agitated; unable to live in community, need structured non-distracting environment
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Rancho level VI
can have carry over for re-learned tasks (previously known skills, but won't acquire new skills)
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Rancho level VII
will have carry over for new skills, but takes some time
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Rancho level VIII
can become independent
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factors affecting safety for TBI pt
- ext environment
- cognitive function (inc level of agitation/impulsiveness)
- balance (fall risk)
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poor learning ability post TBI
closed skills, discrete tasks, frequent feedback
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good learning ability post TBI
open skills (variable environment, serial or continuous tasks, variable feedback)
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