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What is the formula for cardiac output?
Fick Principal
- Cardia Output = __Oxygen Consumption
- Arterial - Venou O2 Diff
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What chamber do we take O2 stat levels?
- Right Atrium 75% +/-
- Right Ventrical 75% +/-
- Pulmonary Artery 75%
- Aorta 95% - 100%
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Supraventricular Tachycardia (SVT)
- -Atria abnormally excited
- -Causes ventricles to contract at high rate
- -Low blood pressure and dizziness may occur
- -Begins abrubtly from few seconds to few hours
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Atrial Fibrillation (Afib)
- -Cells in different areas of atria depolorize, repolorize; no Pwaves
- -Ventricular contraction - irregular
- -Not life threatening as long as ventricular rate supports cardiac output
- -Thrombi is common side effect
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Bundle Branch Block (BBB)
- -Can occur in right or left purkinje fibers as a result of MI
- -Verntricular deporalization less synchronous
- -Wideneing of QRS complex
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Ventricular Tachycardia
- -Pulse orginate from ecotpic focus
- -Ventricle driven at high rate diastolic filling time limited
- -Can be life threatning
- -Often precedes Vfib
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Prolonged Q-T Intervals
- -Prolonged refractory period
- -Maybe caused by genetic cardiac excitablity mutations, electrolyte disturbances, pharmaclogoical agents
- -Vulnerable to Torsades De Pointes
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Aortic Stenosis
- Narrow opening
- -Resistance flow is increased
- -Increase ventricular pressure required to eject blood
- -Intraventricular pressure increases to high levels at systole
- -Aortic pressure rides more slowly than normal
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Mitral Stenosis
- -Narrow valve opening
- -Increases pressure gradients LA to LV
- -Resistance to flow causes high LA pressure to left atrial enlargments
- -Elevated left atrial pressures are reflected back to pulmonary bed, cause pulmonary congestions, SOB, Pulmonary HTN
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Aortice Insufficiency
- (AO Regurgitation)
- -Cusps (leaflets) do not seal properly
- -AO pressure falls fast and further than normal
- -Causes a low diastolic pressure
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Mitral Insufficiency
- (Mitral Regurgitation)
- -Leaflets to not seal properly
- -Part of left ventrical volume to LA during Systole
- -Mitral valve prolapse (MVP) is common cause
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Homeostasis
balance in the human body system and primary role of cardiovascular system
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Pulmonary circulation
circulation of the deoygenated blood comprised of the right heart system & the lungs
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System circulation
composed of left heart system and sytemic organs (except gas exchange of lungs)
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Plasma
responsible for the continual process of supplying fresh nutrients and waste disposal
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Action Potential
a rapid voltage change triggered by contraction that occures in cell membrane (electromechanical change)
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Chronotrope
Increase or decrease of heart rate and impacted by concentraion of Ca(+)
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Inotrope
force of contraction
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Dromotrope
Conduction speed (related to AV node)
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Preload
end diastolic ventricular pressure and amount of blood filling (EDV and EDP)
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Afterload
systemic atrial pressure; blood that heart pumps against
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Parasympathetic
decrease heart rate; releases ACH; stimulation of vagus nerve
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Sympathetic
increases heart rate; influences all portions of the heart; release NE on myocytes
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Viscosity
Thickness of fluid
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Stenosis
A narrowing or stricture of vessel/valve
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Insufficiency
Regurgitation; Leaflets do not seal
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Pulmonary capillary Wedge (PCWP)
Pressure from balloon wedged into pulmonary artery back flow is recorded and should be equal to left atrial pressure
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Beta Blockers
Block action of Epinepherine at all beta receptors to decrease heart rate and dilate blood vessels
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ACE Inhibitors
Drugs that block angiotensin I and II from converting; results in vasodilation and decrease BP
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ARB
Block angigtensin II from binding to and activating receptors on the smooth muscle of the blood vessel; Results in Vasodilation and decrease in BP
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