-
with this resp. disease early stages are usually symptom free
TB
-
S/S of the resp disease are fatigue, anorexia, weight loss, low-grade fever, night sweats
-cough becomes freq. produces white frothy sputum
TB
-
this form of TB spreads to other organ systems
Miliary
-
TB pneumonias manifertations include
- leukocytosis
- pleuric pain
- cough
- chills
- fever
-
A chest x-ray of TB will show ____ _____ infiltrates, ______ infiltrates, and _____ ______ involvement
- Upper lobe
- cavitary
- lymph node
-
Patients with TB are treated with ____ for ___ to ____ months
-
this vaccine is currently used to prevent TB in many parts of the world; can result in positive PPD reaction
Bacille Calmette-Guerin (BCG)
-
in a patient with TB you need to assess for:
- productive cough
- night sweats
- afternoon temp ^
- weight loss
-
causes airway hyperresponsiveness leading to wheezing, breathlessness, chest tightness and cough
asthma
-
allergens, exercise, air pollutants, and nose and sinus problems are all triggers of ____
asthma
-
the asthma triad includes:
- nasal polyps
- asthma
- sensitivity to aspirin and NSAIDS
-
early phase responses to asthma include production of thick mucous, .....
- vascular congestion
- edema formation
- bronchial spasm
- thickening of the airway walls
-
with asthma expiration my be ______ and air takes _____ to move out
-
an acute attack of asthma usually reveals these physical signs
- restlessness
- increases anxiety
- increased pulse and B/P
- pulsus paradoxus
-
clinical manifestations of this disease include edema, mucous plugging, bronchospasm, resp. acidosis
status asthmaticus
-
the causes of this disease are viral illnesses, ingestion of aspirin or other NSAIDS, allergens, emotional stress, Beta Blockers
status asthmaticus
-
Tx for acute astham attacks include
Beta adrenergic agonists by metered dose inhaler
-
a pt with asthma should have a fluid intake of ___ to ____L a day
2-3
-
4th leading cause of death
airflow limitation not reversible
includes chronic bronchitis and emphysema
COPD
-
abnormal permanent enlargement of the air space distal to the terminal bronchioles w/o obvious fibosis
COPD
-
the air in your lungs can not get out with this disease
COPD
-
pulmonary vascular changes for COPD include
- blood vessels thicken
- surface area for diffusion of 02 decreases
-
common characteristics of COPD include
- mucous hypersecretion
- dysfunctional cilia
- hyperinflaton of lungs
-
the physical findings of COPD include
- prolonged expiratory phase
- wheezes
- becreased breath counds
- bluish-red color of skin
-
complications of this disease include cor pulmonale (RHF), acute resp. failure, peptic ulcer disease, depression/anxiety
COPD
-
ABG typical findings for this disease include:
Low PaO2
^ PaCO2
Low pH
^HCO3
COPD
-
drug therapy for this disease includes bronchodilators and inhaled corticosteroids therapy
COPD
-
gravity assists in bronchial drainage; techniques individualized according to patients pulmonary condition and response to intial treatment
postural drainage
-
facilitates movement of secretions to larger airways
vibration
-
produces vibration in lungs to loosen mucus for expectoration
flutter mucus clearance device
-
inflatable vest that vibrates the chest, works on all lobes, more effective then CPT
High freq. chest compression
-
vibrates lungs to shake free mucus plugs; improves clearance of secretions; faster and more tolerable then CPT
Acapella
-
Patients with COPD should have a fluid intake of
3L
-
this type of histamine blocker is used to reduce gastric acid in peptic ulcer disease
- H2 blockers/antagonists
- -tagamet
- -zantac
- -pepcid
-
these histamine blockers are referred to as antihistimines and are also anticholinergic(drying), and sedatives
H1 antagonists
-
-prevents adverse conseq. of histimine stimulation.
-these compete with histimine at the unoccupied receptors; cannot push histimine off receptor is already bound
antihistimines
-
what effects does histimine have on the cardiovascular system?
dilation and increased permeability
-
what effects does antihistimine have on the cardiovascular system?
- reduce dilation of blood vessels
- reduce increase permeability of blood vessels
-
what effects does histimine have on smooth muscles (endocrine glands)
stimulate salivary, gastric, lacrimal and bronchial secretions
-
what effects does antihistimine have on the smooth muscles (endocrine glands)
reduce salivary, gastric, lacrimal and bronchial secretions
-
what are the histimine effects of the immune system
mast cells release histimine and other substances, resulting in allergic reaction
-
what are the effects of antihistimine on the immune system
binds to histimine receptors, preventing histimine from causin gresponse
-
the most common adverse effects of antihistimines are
- dry mouth
- difficulty urinating
- constipation
- changes in vision
-
these antihistimines work both peripherally and centrally
-have drying effect
-traditional (old)
-
newer antihistimines
-nonsedating/peripheral acting
-developed to eliminate unwanted adverse effects, mainly sedation
-
antihistimes should be used with caution in cases of a patient having:
- -cardiac or renal disease
- -HTN
- -asthma
- -COPD
- -peptic ulcer disease
-
the largest group of decongestants
-sympathomimetics
adrenergics
-
less commonly used decongestants
-parasympatholytics
anticholinergics
-
topical or intranasal steroids used as decongestants
corticosteroids
-
this dosage type of decongestants have a delayed onset but prolonged effects
-exclusively adrenergics
-Sudafed
Oral decongestants
-
these decongestants have a prompt onset, are potent
-can not use for more then 3 days
topical nasal adrenergics
-
these work to turn off the immune system cells involved in the inflammatory response; include flonase and nasalide
intranasal steroids
-
this type of nasal decongestant constricts small blood vessels that supply URI structure, resulting in the tissue to shrink
-swollen membranes better to drain
-nasal stiffness relieved
nasal adrenergics
-
what are the adverse effects of nasal adrenergics
- nervousness
- insomnia
- palpitations
- tremors
-
what are the adverse effects of nasal steroids
local mucosal dryness and irritation
-
decongestants should be avoided in cases where patients have:
- HTN
- palpitations
- CNS stimulation
-
these are used ONLY for nonproductive coughs
antitussives
-
this form of antitussive suppresses the cough reflex by direct action on the cough center of the medulla
-cause sedation, N/V, lightheadedness
- opiods
- -codeine
- -hydrocodone
-
these antitussives suppress the cough reflex by numbing the stretch receptors in the resp. tract and preventing the cough reflex
-dizziness, headache, sedation, nausea
- non-opiods
- -tesslon perles
- -vicks 44
- -robitussin DM
-
drugs that aid in the expectoration of mucus; reduce the viscosity of secretions and disintegrate and thin secretions
expectorants
-
this drug causes irritation of the GI tract; loosening and thinning of repiratory tract secretions occur in response to this irritation
expectorants
-
patients taking this type of drug should increase thier fluids to helps loosen and liquefy secretions
expectorants
-
used during acute phase of asthmatic attacks
-quickly reduce airway constriction and restore nomal airflow
bronchodilators
-
stimulate only B2 receptors
-relaxes smooth muscles of the airway and results in bronchial dilation and ^ airflow
Albuterol
-
if this is used too frequently is loses its B2 specific actions at larger doses and then B1 is stimulated, causing nausea, ^ anxiety, palpitations, tremors, ^ HR
Albuterol
-
causes bronchial constriction and narrowing of the airways
-bronchoconstriction is prevented, airways dilate
- acetylcholine
- -atrovent
- -spiriva
-
causes relief of bronchospasm and greater airflow into and out of the lungs, smooth muscle relaxation
-^ levels of energy producing cAMP
-CNS/cardio stimulation
- Xanthine Derivatives
- -caffine
- -theobromine
- -theophylline
-
adjunct drug in the management of copd
-caution use with cardiac disease, PUD
- xanthine derivatives
- -theophlline
- -caffine
-
prevent s leukotrienes from attaching to receptors on cells in the lungs and in circulation
-inflamm. of the lungs in blocked
- antileukotrienes
- -singular
- -accolate
- -zyflo
-
this medication should be taken every night on a continuous schedule, even if asthma symptoms improve
-used for chronic asthma
antileukotrienes
-
NOT considered first line drugs for management of acute asthmatic attacks or status asthmaticus
- inhaled corticosteroids
- -azmacort
- -flovent
- -flonase
-
teach patients to gargle and rinse the mouth with luke warm water afterward to prevent the development of oral fungal infections
- inhaled corticosteroids
- -azmacort
- -flovent
- -flonase
-
the adverse effects of inhaled corticosteroids is:
- -pharyngeal irritation
- -coughing
- -dry mouth
- -oral funal infections
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