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defined as clinical attachment loss with subsequent bone loss
periodontitis
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What is the diagnosis of periodontitis based on?
clinical and radiographic findings
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periodontitis is catoragized as
chronic and aggressive
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Primary risk factor for periodontitis
dental biofilms
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last structures to be lost in PD
periodontal ligament fibers
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plasma cell/antibody mediated
advanced
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occurs with clinical connective tissue attachment loss to the root surface and the apical migration of the apical aspect of the JE along the root surface taht was previously occupied by connective tissue
Periodontal pocket
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What are developed in PD pockets between the root surface and the gingiva
rete pegs and microulcerations
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__and__migration of the junctional epithelium continues and as this epithelium separates from the root surface
apical and lateral
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Two types of periodontal pockets
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What distinguishes the two types of pockets
relationship of the base of the pocket or the coronal extent of the JE to the alveolar crest and type of bone destruction
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If the base is coronal to the crest of bone
suprabony
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base is apical to the crest of bone
infrabony
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Where is the JE in gingivitis and healthy gingiva
CEJ
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how is teh texture of cementum without PDL fibers
rough
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Is it necessary to remove the roughness of cementum in order to remove the endotoxins, bacteria, and other byproducts?
No, rootplanning is not needed bc the endotoxins are found to be very superficia and not embedded deeply into the cementum.
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Bone loss involves inflammatory cells including
PMNs and macrophages
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Substances involved in bone resorption (4)
- prostaglandins
- endotoxins
- cytokines
- b-cells
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what release prostoglandins?
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What do prostaglandins do
- activate osteoclasts
- increase # of osteoclasts
- increase # of macrophages
- inhibit bone collagen formation
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most important in periodontal distruction
IL-1
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what do endotoxin do
activate inflammatory cells such as macrophages
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What do macrophages release
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What can IL-1 stimulate the production of
PGE's
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cytokines and prostaglandis stimulate collagenase which does what?
breaks down collagen
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Does pocket formation and bone loss occur in all areas of the dentition at the same time?
no: site specific
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Is the degree of bone loss aloways correlated with the depth of periodontal pockets
no; a person could have changed and now has healthy gingiva but lots of bone resorption
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They types of pockets that form and the pattern of bone loss depend on what?
the rout the inflammation takes from the gingiva to the underlying supporting sturcutures
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Where is bone most vascular?
interdentally (so it has the most resorption)
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two patterns of bone loss
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Bone loss that occurs from its outer aspect buccal and lingual walls. lost equally on the surfaces of two adjacent teeth. Deepest portion of the pocket is located coronal to the alveolar crest
Horizontal bone loss
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Bone loss that occurs when teh inflammation travels directly formt eh gingiva into the peridontal ligament and then the bone. occurs at different rates aroun the tooth...more rapid ont eh side of the tooth than the other. the deepest poriton of the bony defect is apical to the alveolar bone crest
Vertical /angular bone loss
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A three wall defect that wraps around the tooth and involves two or more adjacent root surfaces is referred to as
circumferential defect
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classified according to the number of osseous walls surrounding the pocket
infrabony defects
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three bony walls remaining interproximally or facially or lingually and the tooth forming the fourth wall
three wall bony defect
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Infrabony defects are usually composed of what bone
what about intrabony
cortical
cancellous
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how do you determine if it is intra or infra
surgically
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a three wall defect that wraps around the tooth and involves two or more adjacent root surfaces is referred to as
circumferential
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has two bony walls remaining
two wall bony defect
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the most common angular bony defect
an interdental crater (two wall bony defect with buccal and lingual remaining)
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has one wall remaining
one wall bony defect
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usually occurs interdentally
one wall defect
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if teh remaining wall is the proximal wall the defect is referred to as a
hemiseptum
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outside a __mm circle, bone loss occurs
2mm
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Bone destruction occuring where thiere is a wide interdental septume and more thatn 2 mm of with is lost will be verticl or horizontal?
horizontal
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bone destruction occuring where the interdental septum is narrow, less than 2 mm, results in vertical or horizontal
vertical
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Teeth in anterior are usually
horizontal
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molars are usually
vertically
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most common form of PD
chronic PD
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has a slow rate of progression
chronic PD
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in order to develope chronic pd what must be present first
gingivitis
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is chronic pd consisered a specific bacterial infection?
no, its a nonspecific bacterial infection
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Presence of what bacteria indicates a high probability that pd is presence
Pg
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Local risk factors for pd
- calculus
- overhang restorations
- retentive conditions
- smoking
- systemic conditions
- hormonal facots
- stress
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chronic Pd is classified according to
extent and severity of disease
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defined as the number of sites affected
extent
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extent is divided into
- localized less than 30%
- generalized..more than 30%
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how much disease has occured
severity
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severity is calculated from the
CAL
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features of chronic periodiontisi include
- pocket formation
- alveolar and supporitng bone destruciton
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is mobiity always evident in CHronic PD
no
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established risk factors for PD
- bacteria in dental plaque
- smoking
- diabetes mellitus
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nicotine in tobacco causes
constriction of blood vessels
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smokers appear to have decreased..
PMNs or macrophages..not both
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decreasedPMN migration into the oral cavity leads to
depressed phagocytic fxn and impaired healing response
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rapidly progressive form of periodontitis
Aggressive periodontitis (AgP)
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common features of localized and generalized aggressive periodontisi
- except for the presence of pd, patient are otherwise clincally healthy
- rapid attachment loss and bone destruction
- familial disposition
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secondary features that may be present include the following
- amts of microbial depositis are inconsistent with teh seerity of the periodontal tissue destruction
- elevated levels of Aa and in some populations, Pg
- progressiong of attachment loss and bone loss may be self-arresting
- pahgocyte abnormlities
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features of localized aggressive pd
- circumpubertal onset
- serum antibody response to the bacteria
- localized first molar/incisor presentaion with interproximal atachemnt loss on at least two permanent teeth, one of which is a first molar and involving no moreht than two teeth other than first molars adn incisors
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features of gineralized aggressive periodontitis
- usuaally affecting individuals under 30 years of age but can be older
- pronounced episodic nature of the destruction of attachment oand bone
- poor antibody response to the bacteria
- generalized interproximal attachment looss and bone destructionaffecting at least three permanent teeth other than the first molars and inisiors
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a distinguishing feature found in AfP that is not seen in chronic PD is the presence of
PMN an macrophage defects
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Periodontisis can be linked to
- heart attacks
- diabetes melitus
- lowbirth weight babies
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children with GAgP are more pron to
- ear
- skin
- upper respiratory tract infections
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Plaque accumulation is minimal
bone los is not super rapid
usually no accompanying infection
slight gingival inflammation
LAgP
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there is a defect in PMN chemotaxis and impaired phagocytosis in what percent of patients
70-80%
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Predominant bacteria in LAP
- Aa
- Prevotella intermedia
- Kikenella corrodens
- Campylobacter rectus
- Capnocytophaga
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high numbers of what are in the subgingvial pocke associate with LAP which invade the soft tissue
Aa
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what personce of LAP have hgih counts of Aa
90% must be on antiobiotics bc it's reaching the soft tissue
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patient previously treated conventionally and do not respond favorable to therapy and are concidered to be resistant to tx
refractory Periodontitis
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patient with poor compliance are termed
recurrent periodontitis
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high numbers of which bacterias are found in refractory pd
- prevotella intermedia
- tannerella forsythensis
- fusobacterium nucleatum
- Porphyromonas gingivalis
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Lesions of the PDL and adjacent alveolar bone may originate from interactions of the periodontium or tissues of the dental pulp. This is periodontitis associate with what?
Endodontic lesions
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Which systemic diseases contribute to periodontitis
- familial or cyclic neutropenia
- down syndrome
- leukocyte adhesion deficiency syndromes
- papillon-Lefevre syndrome
- chediak-higashi syndrom
- histiocytosis syndromes
- glycogen storage disease
- infantile genetic agranulocytosis
- cohen syndrome
- ehlers-danlos syndrome
- hypophosphatasia
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collective term for soft-tissue inflammation surrounding an inplant
peri-implant disease
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term used to describe reversible inflammation in the gingiva around a functioning implant
peri-implant mucosititis
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Tx of all types of periodontal diseases begins with
- oral hygiene care
- nonsurgical therapy
- smoking cessation
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may be required for pocket elimination or reduction
periodontal sugery
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Do patients with chronic periodontitis require systemic antibiotics?
no
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In selected cases if the patient is having a difficult time with homecare, what may be implicated
mouthrinses (chlorhexadine)
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do patients with aggressive periodontits require systemic antibiotic during scaling and root planing and periodontal surgery
yes
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why do patients with aggressive periodontitis require systemic antibiotic require med
bc 90% LAP paitients have so much Aa
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What do to with diabetic patients who have PD
placed in a monitored oral health program that will take care of bothe the paitent's dental and general health.
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