-
exacerbation
to make worse
-
remission
laying dormant, not active
-
Avonex
which disease
- (Interferon beta-1a)
- Given IM. 1-3xa week, store in refridgerator,teach
- family to give. .5 ml dose. Before bed time.let it warm up to room temp before giving to prevent burning and irritation. Prefilled syringe. May have to put ice on site for sore and redness.
- M.S.
-
sandimune
which disease
- has to be in a glass container
- M.G.
-
-
Parkinsons INTV of ambulation
When walking hands folded behind back.
-
Dysphagia NSG INTV
chin to chest, double swallow
-
giving immunosuppressive drugs INTV
-
IV tensilion test
which disease
- short acting cholinesterase inhibitor, makes acetylcholine available to muscles, patient response evaluated.
- M.G.
-
Rilutek
which disease
- protects damaged motor neurons. Huge side effects. Adds 3 mo to life expectancy. Take with no food.
- A.L.S.
-
EMG
what to do
prep
post
intv
-
Baclofen
which disease
- decreased muscle spacity
- M.S.
-
valium
which disease
- decrease mucle spacity
- M.S.
-
huntingtons criteria for dx
DNA, family hx, clinical hx
-
parkinsons criteria for dx
rule things out, 2/3 signs tremor rigidity, bradykinesia, positive response to antiparkinsons meds
-
MG criteria for dx
Tensilon test, look up for 2-3 min, serum testing, EMG
-
early S&S of MS
change in vision=double vision or black spots, complete loss in one eye.
-
early S&S of Parkinsons
mild tremor, change in handwriting, slight limp, decrease arm swinging
-
early S&S of MG
- ptosis=eyelid drooping
- Diplopia= double vision.
-
early S&S of ALS
- muscle stiffness of cramping, speech problems,
- fatigue, tripping , difficulty ambulating, twitching and spasms. Emotional incontinence- can’t stop crying or laughing.
-
early S&S of huntingtons
often mood swings, depression, irritability
-
def of MS
Chronic, progressive, noncontiguous(flairs up and goes away) degenerative disease of the CNS
-
def of parkinsons
depletion of dopamine that interferes with the inhibition of excitatory muscles
-
def of MG
Neuromuscular disease characterized by marked weakness and abnormal fatigue of voluntary muscles(autoimmune disease)
-
def of ALS
Progressive loss of motor neurons in the spinal cord and brain
-
def of huntingtons
Over activity of the dopamine pathway
-
triggers of MS
fatigue, extreme heat or cold, emotional stress, physical illnesses.
-
-
triggers of MG
URI, emotional tension, and menstruation
-
triggers of ALS
stress, fatigue
-
triggers of hungtintons
stress, fatigue
-
tx of MS
Corticosteroids,Immunosuppressive, antibiotics, anti-seizure meds, muscle relaxants
-
tx of parkinsons
- Symmetrel (amantadine hydrochloride)
- Artane (trihexphendidyl hydrochloride)
- Cogentin (benztropine mesylate)
- Larodopa, Dopar (levodopa)-lot of drug interactions, may become aggressive.
- Sinemet (carbidopa/levodopa)-tardive dyskinesia, extrapyramidals.
- -Pallidotomy- destroys area
- of brain that controls tremors. Helps ambulate better. Use if drugs no longer work.
- -Fetal dopamine cell
- transplant- help increase dopamine.
-
tx of MG
Anticholinesterase medications, Corticosteroids Immunosuppressive medications
-
tx of ALS
rilutek, or specialized hospital
-
tx of huntingtons
Palliative, treat physical symptoms, higher nutrition requirements
-
-
-
nystagmus
involuntary mvt of the eye
-
-
vertigo
sensation of room moving around, dizzy
-
muscle spacity
uncontrolled spasm of a broup of muscles
-
-
dysphagia
trouble swallowing
-
dysarthria
difficult speech(brain, jaw and tongue, brocas area)
-
neurogenic bladder
problem with bladder caused by the nervous sys
-
plasmapheresis
get rid of the antibidies in M.G. short term improvement.
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