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Appendicitis
inflammation of the appendix R lower q of abdomen.
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Diverticulitis
manfestations
- small pouches that bulge outward through the colon/large intense
- main cause is low fiber diet
- Ss: pain usually L, constipation or inc frq of defecation
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1. enteral nutrition
2. parenteral nutrition
- 1. ensure tube feeding
- 2. TPN: nutrition deivered in the form of specialized intravenous fluids.
- calories: glucose and lipids (dextose)
- protein: amino acid (goal is to stop catbolism (bkdn))
- usually pt that are: major surgery, trauma, seriously undernourised.
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GERD (upper GI)
how do you developed?
- acid reflux
- developed:
- lower esophageal sphincter does not work
- dec esophageal motility
- detay of acid emptying
- inc pressure w/in stomach
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Hiatal Hernia
sliding and rolling
development
- stomach protrudes up through an opening in the diaphragm into the thoracic cavity
- Sliding: most common. entire lower esophageal sphincter slides up
- devep: weakening of themuscle of the diagram or inc intra-abdominal pressure (preg, obesity, heavy lifting)
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GERD/ Hital Hernia:
SS
complications
- SS:
- heartburn (frm morphine, chololate, alcohol, l meals, smoking)
- diff of swallowing or a hump feeling
- reguruation of gastric fluid up to mouth
- Complications
- chronic esophagitis w scarring
- barretts esophagus
- ulcer
- micro aspiration
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GERD/ Hital Hernia:
medical tx
nr
- Medical tx:
- nexium, prevacid, prilosec, protanix
- NR:
- taking meds at the right time
- shouldn't stop taking meds just bc they feel better
- HOB >30
- avoid high fat meals
- avoid thight clothing
- shouldn't eat so late
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Peptic Ulcer Disease
cause
patho
ss
medical m
nc
- open lesion on the mucosa of the esophagus, stomach or dudoenum (usually where there's gastric acid)
- cause: h. pylori bacteria or use of NSAIDS (advil..)
patho: disruption in mucosa barriers
SS: pain (buring, aching, gawing) **can be relieve by - eating, heart burncomplications: hemorrhage, scarring will cause
- obsrution, perforation
- Medical m: focus is to (relieve pain, treat infection, slow production of and neutralize
- HCI acid, restore integrity of mucosa)
- Diet: avoid caffine, high fat, alcohol
- pharm: antibiotics, proton pump inhibitors, antiacids, h2 blockers
- ND (NOT had surgery):
- Pain: give meds on time
- nutritional needs: monitor lab, meal planning
- complication: focus on abd
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Types of gastric surgeries?
1. Subtotal gastrectomy ( Billroth I n II)
2. Total gastrectomy
- 1. Billroth I (gastrodudoenostomy, 1/2 stomach removed and connected to duodenum)
- Billroth II (gastrojejunostom, "" connected to jejunum)
2. total removal of the stomach usually only if its cancer.
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Dumping syndrome (complication of gastric surgery)
patho
ss
ns
- complication to the billroth II surgery used in tx of peptic ulcer disease
- patho: when pylrus has been resected, undigested food bolus rapidly enter the duo or jejunum
- SS: (5-30mins after eating), nausea, vomiting, diarrhea, cramping, tachycarida
- NC: low fat and no sugear! low carb, small freq meals, meals w/out fluid, lay down after eating.
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Ulcerative Colitis n Crohn's Disease (lower GI)
ss, medical tx
- inflammatory and ulceration of colon and rectum
- ss:
- diarrhea! (w bl and mucous), fatigue, anorexia, weakness
- Medical tx:
- Control/dec inflammation: *antidiarrheals: only use Mild colitis
- Nuritional support: NPO to rest the bowel, food that does not stimulate the bowel, provide fluid/elec replacement
- Surgical tx: total colectomy (surgical resection and removal of the colon), ileostomy is usually for colitis
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Intestinal Obstruction
- Blockage of the flow of intestinal contents
- Types:
- Mechanical: physical (outside the intestine, ex: tumors or inflammatory bowel disease, high pitch sounds)
- Functional: change in parastalsis
- SS:
- SBO: quicker onset of sx, abd pain, cramping distention, bowl sounds.
- as the obstruction cont = dehydration, hypovolemia, electroyte imblances.
- LBO: slower onset, deep cramping pian, constipation, vomitng (very late sign)
- ND:
- Deficient Fluid vol
- Pain
- other nursing care: focused abd ass, monitoring respiratory (distended abd cannot breath as well).
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Peritonitis
Inflammatory of gastro fluids and bacteria into the abdominal cavity/peritoneal space
Cause: anything that ruptures or proferiated in GI
- SS: what happens if gastric and bacteria leaks somewhere it shouldn't be?
- diminshed/absent bowel sounds, pain, anorexia, n&v, rigid abdomen, infections,pale
- ND:
- fluid vol deficit
- risk for injury
- pain
- Alt in nutrition: less than body requirements (use TPN)
- Anxiety
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What are the manifestations for DMI?
result of a lack of insulin to transport glucose across the cell membrane
- -Hyperglycemia
- -polyuria (lots of peeing), polydipsia (thirst)
- polyshagia (hungry), ketosis, weight loss
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what are the ss for DM2
- hyperglycemia
- slow wound healing
- blurred version
- fatigue
- skin infections
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Rapid Acting insulin
- onset (5-15mins
- peak: 45-75mins
- dur: 3-5hrs
lispro, aspart, glulisine
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Short acting insulin
- onset: 30-60 min
- peak: 2-4 hr
- dur: 6-8 h
regular (R)
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Intermediate acting insulin
- NPH: O: 2-4hr, P 6-10hr, D 12-18 hr
- Lente: 3-4 hr, 8-12hr, 12-18hr
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DKA (diabetic ketoacidosis)
- -occurs more in type 1
- - severe lack of insulin which causes
- hyperglycemia
- dehydration
- ketosis
- acidosis
- fruity oder to breath
- renal failure
- tx:
- resp first! then fluid resuscitation, K replacment
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long acting insulin
latus: 1-2hr, no peak, 24h
....more
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insluin shock
- -severe hypoglycemia
- tachycardia, LOC, pale, cold skin, hunger, tremors
"pale and cammy give me candy"
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GallBladder
store and concentrate bile that was made in the liver --> release bile into the duodenum to digest fat.
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Cholecystits
Inflammation of the gallbladder (commonly assoicated w/ tumor, obsrtution causes the bile remine in the gallbladder)
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Cholelithiasis
gall stone
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Cholecystectomy
removal of the gallbladder
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fn of the liver?
- makes bile
- makes proteins
- detoxify bl
- clotting factors
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Cirrhosis
scarring of the liver
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what are the later symptoms of cirrhosis?
- 1. Portal hypertension:
- when bl can't get into the congested scarred liver--> venous inc pressure
- (enlargement of the speen, dilation of veins in the gast tract of abd)
- 2. Esophageal varices:
- back up bl extends up to the small vessels/veins ---> inc pressure --> ruptures of the veins
- (devlp of hemorrhoids, dilated, congested vessels in the esophageals)
- 3. Hepatic Encephalopathy:
- Liver is NOT detoxifying ammonia from teh intestines
- (asterixis (flappy tamers due to inc ammonia), fetor hepaticus (bad breath))
- 4. Ascities:
- third spacing of fluid into the abdominal cavity.
.....
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Bronchoscopy
- Fiberoptic direct examination of airways
- Indications for Bronchoscopy:
- -direct visualization of tumors, inflammation or structures
- - for aspiration of sputum
- - removal of foreign bodies/object
- NI:
- remove dentures
- keep pt NPO 8 to 12 hrs
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Pneumothorax
- -acummulation of air in the pleural space
- - ruptured bleb
- - blunt force trauma
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Hemothorax
- -bleeding in the pleural space
- - piercing chest trauma (mwa, gun shot wounds, stabbing)
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Pleural effusion
- -collection of excess fluid in the pleural space
- -high protein fluid
- -often r/t chf, cancer pheumonia, tb
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What are some Nconsiderations for chest tube?
- -keep drainage system BELOW level of chest
- -check suction q2hrs
- - sterile irrigation fluid and vaseline gauze kept at beside at all times
- - chest wall dressing at tube exit site must be airtight
- - check thoracic wall for crepitus
- - position pt for optimal lung expansion
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Thoracentesis
- -invasive procedure in which fluid/air is removed from the pleural space w a needle.
- - to obtain specimens, instill medications, remove fluid or air for relief of inc pleural pressures
- **limit to 1L of fluid removal bc it causes pain, reduction in lung capacity and inc respiratory rate and effort
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4 diff type of tracheotomy?
- Emergent: acute closure of upper airways
- scheduled: chronic supported needed
- temporary: short term survival until
- permanent: long term
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Emphysema (COPD)
- -loss of elasticity and hyperinflation of lung. Destroys alveoli, dec surface area available for gas exchange.
- *** 1st is getting airout first!!
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Chronic bronchitis (COPD)
- inflammation of the bronchi and bronchioles due to chronic exposure to irritants
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diagnostic test for COPD
- PFT
- CXR (flattend diaphragm)
- hgb and Hct (monitor for polycythemia)
- alpha antitrypsin 1 level
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assessment/ss for COPD?
- - chronic dyspnea
- - rapid shallow respirations
- - thin extremities, enlarged neck muscles
- - hypoxemia
- - hypercarbia
- - resp acidosis and metabolic alkalosis
- - r sided heart failure
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Atelectasis
airway inflammation and edema leads to alveolar collapse w resultant inc risk for hypoxemia
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Three major injuries affecting older adults
- Falls
- •Fires
- •Motor vehicle accidents
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Health Risks for Older Adults
- Drug toxicities/ side effects
- Due to changes in liver and kidney function
- •Due to altered nutritional status
- •Polypharmacy
- •Longer exposure to environmental hazards
- •Sun
- •Smoking
- •Pollutants
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Cardiovascular Changes in Elders
- •Reduced cardiac output
- •Reduced arterial elasticity
- •Increase in systolic blood pressure; peripheral resistance•
- Orthostatic hypertension
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Three Basic Components of chest tube
- •Protection from atmospheric air by means of a “water seal” chamber
- •Collection of drainage, either air or fluid
- .•Suction chamber, to pull negative pressure and so facilitate lung reexpansion
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Chest Tubes—Complications
- •Air leaks
- –From pleural space to external chest around tube
- –Into subcutaneous tissue
- •Accidental disconnection
- –For accidental removal
- —Vaseline gauze @ bedside
- –Sterile water
- —if tube cut, submerge end
- •Tension pneumothorax
- —may occur if evacuation of air or fluid is not allowed
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