Mechanism of action

Increases heart rate, Competes reversibly with acetylcholine.

Slows the conduction of electrical impulses at the AV node.

Stimulates Beta 2 adrenergic receptors (relaxes bronchial smooth muscles)

Delays repolarziation and increases the duration of the action potential. Slows sinus rate, increases PR and QT intervals, decreases peripheral vascular resistance.

Prevents formation of thromboxane A2 (platelet inhibitor) Anti-inflammatory.

Increases cardiac contractility.

Prolongs conduction of electrical impulses through the AV node. Causes vasodilation. Decreases rate of ventricular response. Decreases myocardial oxygen demand.

Increases blood glucose concentrations.

Enhances the effects of GABA. Can produce any level of CNS depression (including sedation, muscle relaxation, anticonvulsant activity)