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Basal Metabolic Rate + Factors
- resting need for energy, just what it takes to keep body functioning
- Factors: age, gender, activity, infections, endocrine status
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What is the normal range for BMI?
Body Mass: 18.5-24 is normal
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Calorie Content of:
Fat, Protein, Carb, One lb body fat
- Fat: 9 kcal/g
- Protein: 4 kcal/g
- Carb: 4 kcal/g
- 1lb: 3500 kcal/g
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Which 3 supply energy and build tissue?
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Which 3 regulate/control body processes?
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Fat Soluble Vitamins
- ADEK
- absorbed into lymphatic
- not needed daily
- excessive A&D can be toxic
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Non-fat Soluble Vitamins
- (water-soluble)
- Vit C & B-complexes
- absorbed in intenstinal wall
- directly into bloodstream
- daily intake
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Carbohydrate
- sugar, starch
- framework of plants
- animals-- lactose
- 90% is digested
- stored in liver as glucose
- oxidized into energy, CO2, water
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Fat
- insoluble triglycerides
- bile digests it/emulsifies it
- pancreatics break down even further
- absorbed in lymphatic
- Saturated/Unsaturated/Transfats
- Cholesterol:LDL blocks arteries HDL clears arteries. Total <200 mg/dl
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Protein
- pancreatic enzymes breakdown into amino acids
- absorbed into intestinal mucosa-->liver
- recombined to new
- released to bloodstream to tissues and cells
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Vitamin
- Water Soluble: absorbed directly into bloodstream by intestinal wall, not stored
- Fat Soluble: must be attached to fat, absorbed into lymphatic, stored in livver and adipose
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Mineral
- Not broken down in body
- Remains in ash after digestion
- Calcium: 99 % bones and teeth
- Macrominerals
- Microminerals
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Water
- Not Stored
- Needed for everything50-60% of total body weight
- Infants have more water
- 2/3 ICF, 1/3 ECF
- necessary for all chemical reactions, solvent to many solutes
- Intake 2000-3000mL/day, but 1500mL avg
- Input = Output
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Digestive Process
- mouth: enzymes begin breakdown
- stomach: acidic pH (HCl) to help food breakdown
- duodenum: sodium bicarbonate neutralizes acid from stomach [basic] gallbladder: release bile (emulsifies fats)
- pancreas: releasing enzymes (amylase) to break down proteins to amino acids
- carbohydrates broken down to glucose (enzyme sucrose)
- Small Intestine: absorption of nutrients
- Large intestine: reabsorbing water
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Glycogenesis
making glycogen from glucose, then stored in liver until needed
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Glycogenolysis
glycogen is broken down and stored into glucose for energy
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Gluoneogenesis
making glucose from some other form of nutrient (ex. amino acid) when glucose is not available
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Infant Needs
- breastfeed 6-12 mo
- solids at 4-6 mo (periods of 5-7 days to test allergies)
- 1 yr: table food and cows milk
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Toddler Needs
- 3-5 yrs
- develops attitude toward food
- increased appetite and becomes eratic
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School Age Needs
- 6-12 yr
- body accumulates reserves
- Health Promo: intake balance w/requirements
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Adolescent Considerations
- increased nutrient needs to support growth
- anorexia nervosa & bulimia
- eat rapidly--> overconsumption
- unhealthy meals aways from home
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Adult Considerations
- Growth ceases
- BMR decreases
- If calorie change not met, weight gain occurs
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Pregnancy Considerations
- increase to support growth
- especially in 2-3 trimester
- extra 300 kcal/day
- *protein, cals, iron, folic acid, calicium, iodine
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Older Adult Considerations
- decreased need for calories
- same need for nutrients
- BMR decrease
- Decreased peristalsis
- Decreased taste sensation
- prone to dehydration
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Complete Diet
- Animal (plus soy)
- sufficient amounts of all essential amin acids to support growth
- eggs, dairy, meat
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Incomplete Diet
- Plant (except soy)
- deficient in one or more essential amino acids
- grains, legumes, veggies
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Complementary Proteins
- combine to make up for where the other is deficient
- corn torilla + refried beans
- lentil + rice soup
- cereal + milk
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Essential Fats/Amino Acids
they are not synthesized within the body and must be supplied through diet
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Goals of Food Pyramid
- Increase vitamin, mineral, fiber
- Decrease sat fat, trans fat, cholesterol
- Increase fruit, veggie, whole grain
- *support calor intake based on energy needs to promote a healthy weight
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Clear Liquid Diet
- clear at room temp
- gelatin
- fat free broth
- bouillon
- ice pop
- clear juice
- carbonated beverage
- coffee
- tea
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Full Fluid Diet
- Clear, Plus Others:
- milk/milk drinks
- plain frozen desserts
- pudding, custard
- eggs, cereal
- veggie juice
- milk & egg substitutes
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Soft Diet
- decreased fiber, unseasoned
- decrease GI distress
- soy, yogurt
- cooked fruit/veggies
- banana
- avacado
- melon
- lean, tender meat
- potatos, rice, pasta
- egg, cheese, peanut butter
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Pureed Diet
- blenderized to liquid form
- all foods unless contraindicated
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Mechanical Soft Diet
- modification for texture
- (difficulty chewing/swallowing)
- chopped, ground, mashed, pureed
- ripened fruits
- cooked veggies
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Methods for Improving Appetite/Nutritional Status:
- small, frequent meals
- solicit food preferences
- encouragement
- pleasant environemnt
- loos attractive
- procedured dont interfere
- control pain, nausea, depresson
- offer alternatives
- good oral hygiene
- free from odors
- easily reach food
- comfortable position
- any rituals during mealtime
- if patient is absent, order late food try
- dont interrupt meal time
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Purpose of Enteral Feeding Tubes
- in NPO, next best method
- pass tube into GI tract to admin a formula of adequate nutrients
- stomach acts as reservoir
- regulates amt of food/liquid into small intestine
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Methods to Check Placement of Feeding Tube
- Nasogastric: ensure tip of tube is in stomach or intestine
- Radiographic: verify initial placement
- pH of Aspirate: stomach (<5.5), intestine (7.0+), respiratory (6.0+)
- Color of Aspirate: Stomach (grass-green, tan, off-white, bloody, brown), Intestines (medium to deep golden yellow), Respiratory (off white, tinged with mucous)
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Safety Considerations w/Feeding Tube
- Aspiration
- Clogged Tube
- Nasal Erosion
- Diarrhea
- GI Distress
- Unplanned Extubation
- Stoma Infection
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HMG COA Enzyme Reducers
(statins) used in synthesis of cholesterol
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Micturation
- voiding/urination
- brain + spinal cord
- largely involuntary
- 150-250mL/day
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Urinary Incontinence
involuntary loss of urine
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Autonomic Bladder
- not brain-controlled
- reflex only
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Urinary Retention
urine produced normally but not excreted completely from bladder
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Enuresis
involuntary/unintentional urinary into bed or clothes
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Anuria
- 24 hr rine output less than 50mL
- kidney shutdown/renal failure
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Dysuria
painful or difficult urination
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Glycosuria
presence of sugar in urine
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Nocturia
awakening at night to urinate
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Oliguria
- scanty or greatly diminished amount of urine voided in a given time
- 24 hr urine output less than 400 mL
- less than 30mL/hr
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Polyuria
- excessive output of urine
- diuresis
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Proteinuria
- protein in urine
- indicates kidney disease
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Pyuria
- pus in urine
- urine appears cloudy
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Suppression
- stoppage of urine production
- normally produce 60-120 mL/h
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Urgency
strong desire to void
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Hematuria
- blood in urine
- pink or red color
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Nephrotoxic drugs
capable of causing kidney damage
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Ureters
transport urine fro kidney to bladder by rhythmic peristalsis
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Bladder
- smooth muscle
- temporary reservoir for urin
- 3 layers
- Detrusor: inner longitudinal layer, middle circular layer, outer longitudinal layer
- Internal/Involuntary Sphincter: guards opening between urinary bladder and urethra
- SNS: retains urine
- PNS: releases urine
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Urethra
- bladder to exterior
- Male: 5 1/2"-6 1/4"
- Female: 1 1/2"-2 1/2"
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Nephrons
- remove end product of metabolism from blood plasma and form urine
- maintain and regulate fluid balance through selective reasborption and secreation of water, electrolytes, and others
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Elimination Changes in LifeSpan
- Infant: no voluntary control, 6-10 wet diapers/day
- 18-24 mo: voluntary control of urethral sphincters
- 2-5 yrs: develop urinary control
- Aging: decreased ability for kidney to concentrate urine, nocturia, decr. bladder muscle tone & contractility, incr. frequency, urine retention/stasis, incr. risk UTI
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Factors for Urinary Output
- Developmental
- Food/Fluid intake
- Psychological
- Activity/Muscle Tone
- Pathologic Conditions
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Urinary Diversion
surgical creation of alternative routh for excretion
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Specific Gravity
density of urin compared w/density of water
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Routine Urinalysis
- urine collected and measured
- avoid contaminated
- aseptic technique
- pt name/date/time to lab soon
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Clean Catch Specimen
- 1st flush
- Catch midstream
- Discard the rest
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Specimen from Indwelling Catheter
- with needle and syringe
- directly from specimen port
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24 hr Urine Specimen
- initiate
- discard first void
- all other voids are save
- last 24 hr void added to previous
- entire specimen sent to lab
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Post Void Residual
- traditional catherization or portable ultrasound device to scan bladder
- supine position
- <50mL is adequate
- >150mL recommend cath= risk for UTI
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Transient Incontinence
- appear suddenly
- lasts for 6mo/less
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Functional Incontinence
- altered environment, sensory, cognitive, mobility deficits
- used to be able to, but change occur
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Reflex Incontinence
- neurologic
- cant sense filling
- no urge to void
- no feeling of bladder fullness
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Stress Incontinence
- overdistension
- weakened supportive muscles
- leakage w/cough, sneeze, laugh
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Total Incontinence
- neurologic/trauma
- constant/unpredictable flow
- no distention
- nocturia
- unawareness of incontinence
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Mixed Incontinence
urine loss w/features of 2+ types of incontinence
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Urge Incontinence
involuntary loss of urine soon after feeling urgent need to void
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Overflow Incontinence
overdistention/overflow of bladder
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Kegal Exercises
- improve voluntary control of urination
- eliminate/reduce stress incontinence by increase muscle tone
- targets inner muscles that lie under and support bladder
- contract 10 sec, relax 10 sec, 30-80x/day for 6+ wks
- (don't use abs, inner thigh, buttocks)
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Course of Bowel Elimination
- Small intestine
- Ileocecal valve
- Cecum (veriform process attached)
- Ascending colon
- Hepatic Flexure
- Transverse Colone
- Splenic Flexure
- Descending Colon
- Sigmoid Colon (contains feces)
- empties into rectum
- excreted through anal sphincter
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Nervous Regulation of Bowel Elimination
- PNS: stims movement, craniosacral inhibitory impulses
- SNS: inhibits movement, thoracolumbar motor impulses
- Act of Defactation: medulla, spinal cord, PNS stims to relax sphincter
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Valsalva Maneuver
- bearing down
- cuts of vagus nerve=hypertension
- may be contraindicated in those w/cardiovascular problems
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Factors of Bowel Eliminiation
- Developmental
- Daily Patterns vary
- Food/Fluid intake
- Activity/Muscle Tone
- Lifestyle (acceptance, preoccupation, "dirty")
- Psychological (anxiety, worry)
- Pathologoical
- Medications
- Diagnostic Studies
- Surgery/Anesthesia
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Medications affecting Bowel Elim
- Cathartics/Laxative: promotes peristalsis
- Antidiarrheal: inhibit peristalsis
- Inhaled General Anesthetic: block impulses
- Narcotics: paralytic ileus
- Constipation: opoid, antacid (aluminum), iron sulfate, anticholinergic drugs
- Diarrhea: amoxicillin, antibiotics
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Medications and Appearance of Stool:
Red/Black, Black, Speckled/White, Green/Gray
- Red-Black: anticoagulant, aspirin
- Black: iron oxidation/salts
- Speckled-White: antacids
- Green-Gray: antibiotics
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Fecal Elimination Problems:
Constipation, Diarrhea, Incontinence, Flatulence
- Constipation: dry, hard stool
- Diarrhea: excessively liquid stool
- Incontinence: inability of anal sphincter to control discharge of fecal & gaseous matter (usually organic disease)
- Flatulence: excessive formation of gases in stomach/int; can lead to distention if not expelled
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Areas of Bowel Assessment
- Abdomen: inspection, auscultation, percussion, palpation
- Anus/Rectum: inspection, palpation
- Stool Characteristics: color, form, etc.
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Bowel Tones:
Hyperactive, Hypoactive, Decreased/Absent
- Hyperactive: incr. motility (diarrhea, gastroenteritis, early bowel obstr)
- Hypoactive: diminished motility (abd srgry, late bowel obstr)
- Decr/Absent: absence of motility (peritonitis/parlytic ileus)
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Bulk-Forming Laxatives
- psyllium/grain/synthetic
- stool absorbs water, swells, stimulates peristalsis
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Emollient/Stool Softener
agents w/detergent activity allow water and fat to penetrate and lubricate stool
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Stimulant Laxative
irritating intestinal mucosa or stimulates nerve endings in intestinal wall
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Saline Osmotic Laxative
draws water into intestine to stimulate peristalsis
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Cleansing Enema
- (tap, normal saline, soap solution, hypertonic solution)
- relieve constipation/fecal impaction
- prevent involuntary escape during surgical procedures
- promote visualization of int tract by radiography/intrument
- help establish regular bowl fxn during bowel training program
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Retention Enema:
Oil, Carminative, Nutritive, Medicated
- Oil: lubricates
- Carminative: helps expel flatus/relief from gaseous distention
- Nutritive: admin of fluid/nutrients
- Medicated: for parasites
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Return Flow Enema
- expel flatus
- alternate solution in and out
- promote peristalsis
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Bowl Training Program
manipulate factors within pt control (intake, exercise, time for defacation) to produce the elimination of a soft, formed stool at regular intervals w/out a laxative
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LDL: Bad Cholesterol
delivery of cholesterol to nonhepatic tissue, greatest contribution to atherosclerosis
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HDL: Good Cholesterol
cholesterol from peripheral back to liver to be removed
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Atherosclerosis:
process of plaque formation
- LCL into arterial subendothelial space and oxidizes
- Attracts monocytes/macrophages
- Inhibited mobility so macrophages accumulate and increase uptake, enlarge, and become foam cells
- Foam cells can rupture
- Platelets aggregate and collagen forms a fatty streak
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How do HMG-CoA enzyme lowering meds (statins) lower lipids? How are they effective for treatment?
- inhibition of HMG-CoA Reducatse inhibits cholesterol production
- this stimulates incr. LDL receptor formation
- this allows more LDL to be drawn from blood into liver
- (thus lowering LDL level)
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How is atherosclerotic plaque r/t the inflammatory response?
Atherogenesis: when LDLs penetrate wall, they cause mild injury that trigguers the inflamm. response, which can cause plaque to rupture, leading to thrombosis
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What is a CRP level?
- CRP: C-Reactive Protein
- compount produced when inflammation occurs
- biomaker for ongoing inflammatory process
- elevated CRP is an independent risk factor of CV events
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What does ARP III stand for?
- Adult Treatment Panel
- Executive Summary of the 3rd Report of the Natn'l Cholesterol Educ. Program Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults
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Normal Levels:
LDL, HDL, Total
- LDL: <100 mg/dL
- HDL: <40 mg/dL
- Total: <200 mg/dL
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Risk Factors for CV disease?
- Age: men >45yr, women >55yr
- Family Hx or premature CHD
- Hypertension: >140/90 mmHG or taking antihypertensives
- Current cigarette smoking (at least 1 in last month)
- Low HDL: <40 mg/dL
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Lifestyle Changes to Lower Cholesterol
- Diet: incr. soluble fiber, incr. plant stanols/sterols
- Weight Control: obesity is a major risk
- Exercise: sedentary incr. risk
- Smoking Cessation: incr. LDL, decr. HDL
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Statins:
Major Adver Effects & Common Side Effects
- Major: (1) Myopathy/Rhabdomyolysis, (2) Hepatotoxicity
- Common: (side effects uncommon) mild/transient HA, rash, GI disturbance
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When is the best time of day to administer statins?
- evening is best time of day
- endogensou cholesterol synthesis increases during the night
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