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CLASSIFICATION of WOUNDS
- OPEN
- involves disriuption or break in the skin
- CLOSED
- no break in skin (bruise)
- INTENTIONAL
- occurs during treatment (i.e. surgery, radiation burn, venipuncture, drawing blood)
- UNINTENTIONAL
- accidental (i.e. fracture caused by a fall, stabwound)
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WOUNDS: DESCRIBED BY HOW ACQUIRED
- INCISION
- caused by sharp instrument (surgical)
- CONTUSION
- blow from blunt force (bruise)
- ABRASION
- surface scrape (intentional or accidental)
- PUNCTURE
- penetration of skin & possibly underlying tissues from sharp instrument
- LACERATION
- open wound with jagged edges
- PENETRATING
- goes through skin, into body cavity or organ
- PRESSURE ULCER
- related to excessive pressure on body site
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WOUNDS ARE DESCRIBED BY DEGREE OF WOUND CONTAMINATION
- CLEAN
- free of infectious organisms
- CONTAMINATED
- containing microorganisms and includes:
- -open, fresh accidental wound
- -surgical wound involving major break in sterile technique
- -surgical wound where there is spillage from GI tract
- DIRTY OR INFECTED
- -old, accidental wound containing dead tissue
- -wound with evidence of infection (i.e. purulent drainage)
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PHYSIOLOGY OF WOUND HEALING: PHASES
- INFLAMMATORY PHASE
- time of injury - day 3 or 4
- PROLIFERATIVE OR RECONSTRUCTIVE PHASE
- day 4-21
MATURATION OR REMODELING PHASE - day 21-2 years
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PHYSIOLOGY OF WOUND HEALING: INFLAMMATORY PHASE
- first 3-4 days of injury
- -blood supply to wound increases, bringing nutrients and substances necessary for healing process
- -area appears red and edematous (signs of infection, but normal in this phase)
- PAIN is experienced
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PHYSIOLOGY OF WOUND HEALING: INFLAMMATORY PHASE
2 major processes
- HEMOSTASIS (clotting)
- -vasoconstriction for control of bleeding
- -platelet accumulation
- -deposition of fibrin and formation of blood clots to provide a fibrin matrix
- -scab formation on surface of wound aids hemostasis & inhibits wound contamination
- PHAGOCYTOSIS
- -leukocytes (neutrophils) move in
- -macrophages engulf, digest microorganisms to clean debris from wound bed
- -shedding of dead tissue occurs
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PHYSIOLOGY OF WOUND HEALING: PROLIFERATIVE PHASE
- GRANULATION
- -new capillaries grow across wound, increasing blood supply
- -tissue becomes translucent red, fragile, bleeds easily
- EPITHELIALIZATION
- -when granulation tissue matures, epithelial cells migrate to it to fill wound bed.
- SCAR FORMATION
- -if wound has greater tissue loss, healing occurs from inside out.
- -fibroblasts migrate to wound & initiate collagen synthesis (protein substance that adds strength)
- -progressive accumulation of collagen forms basic structure of scar.
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PHYSIOLOGY OF WOUND HEALING: MATURATION OR REMODELING PHASE
- -fibroblasts continue to synthesize collagen
- -maturation of scar occurs
**scar tissue never exceeds 80% of pre-injury strength**
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TYPES OF WOUND HEALING
- PRIMARY INTENTION
- -occurs when tissue surfaces are approximated (brought together or closed)
- -minimal or no tissue loss
- -heals with minimal granulation tissue and scarring (scar is thin, flat)
- --->examples: minor clean cut; surgical incision with surgical closure
- SECONDARY INTENTION
- -wound edges cannot be approximated due to tissue loss and type of wound
- -repair time is longer
- -epithelialization occurs from wound margins
- -scarring is greater
- -allowed to heal and close on own
- -greater susceptibility to infection (i.e. pressure ulcer; large, irregular, or infected wounds)
- TERTIARY INTENTION
- delayed closure
- -occurs in wounds which may be contaminated, infected, or draining excessive exudate
- -wound is intentionally left open to permit drainage of contaminated material
- -wound is surgically closed when infection has cleared & initial granulation has taken place
- i.e. contaminated abdominal wound
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FACTORS AFFECTING WOUND HEALING
- HYGIENE
- NUTRITIONAL/FLUID STATUS
- AGE
- OXYGEN STATUS
- IMMUNE STATUS
- NEUROLOGICAL STATUS
- MEDICATIONS
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FACTORS AFFECTING WOUND HEALING:
in detail
- HYGIENE
- risks for wound infection
- -Poor personal hygiene
- -diaphoresis, incontinence
- NUTRITIONAL/FLUID STATUS
- -Protein, Zinc, Vitamins A & C necessary (vital) for tissue repair
- -Obesity--adipose tissue has poor blood supply (diabetes)
- -Emaciation (wasted condition of body; thin)--O2 transport is deficient
- -Metabolic demand--increases for wound healing
- -Hydration--promotes cellular function
- AGE
- -Degenerative changes in blood vessels, immune system and respiratory system slow healing time
- -Inflammatory response is delayed in elderly
OXYGEN STATUS - -impaired blood flow to wound or tissues delays wound healing
- -smoking constricts blood vessels, reduces tissue oxygenation; also impairs clotting (hemostasis)
- IMMUNE STATUS
- immunosuppressed patients have:
- ---slower inflammatory response,
- ---slower wound epithelialization,
- ---decrease in leukocytes
- NEUROLOGICAL STATUS
- -Impaired sensation or sensory awareness (LOC) increase risk for skin breakdown
- MEDICATIONS
- meds which inhibit inflammation can alter cell growth and inflammatory phase of healing
- i.e. steroids, NSAIDS, chemotherapy drugs
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WOUND DRAINAGE
- EXUDATESEROUS DRAINAGE
- SANGUINOUS (HEMORRHAGIC) DRAINAGE
- SEROSANGUINOUS
- PURULENT DRAINAGE
- SLOUGH
- ESCHAR
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PRESSURE ULCERS
aka DECUBITOUS ULCERS
- A CHALLENGE TO NURSING CARE
- AREA of LOCALIZED ISCHEMIA
- -deficiency of blood to tissues results in deprivation of oxygen and nutrients (ischemia)
- -tissue death (necrosis) may occur
- TYPICALLY LOCATED OVER BONY PROMINENCE
- -caused by constant pressure and moisture
- SHEARING AND FRICTION OFTEN ACT IN CONJUNCTION WITH PRESSURE TO PRODUCE AN ULCER
- -can put powder on bedpan rim to prevent rubbing
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STAGES OF ULCER FORMATION (NPUAP 2/07)
- STAGE I
- localized, non-blanchable redness of intact skin
- in dark-skinned persons--area may be warm, cool, discolored or bluish
- STAGE II
- partial-thickness skin loss of dermis and epidermis (involves first 2 layers of skin)
- appears as shallow, open ulcer with red pink wound bed
- does not have slough or bruising
- may also present as an intact or open/ruptured serum-filled blister
- STAGE III
- full thickness tissue loss
- subcutaneous fat may be visible (3 layers of skin)
- slough may be present, but does not obscure the depth of tissue loss
- may include undermining or tunneling
- STAGE IVfull thickness tissue loss with exposed bone, tendon, or muscle (4 layers of skin)
- slough or eschar may be present on some parts of the wound bed
- often includes undermining, tunneling, or sinus tracts may form
- UNSTAGEABLE
- full thickness tissue loss
- base of ulcer is covered by slough (yellow, tan, gray, green, brown) and/or eschar (tan brown, black) in the wound bed
- SUSPECTED DEEP TISSUE INJURY
- -purple or maroon localized area of discolored intact skin or blood-filled blister due to damage of underlying tissue from pressure and/or shear
- -may be preceded by tissue which is painful, mushy, boggy, warmer, or cooler in comparison to adjacent tissue.
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VALIDATED RISK ASSESSMENT TOOLS
- NORTON SCALE
- 5 categories of risk:
- -general physical condition
- --mental state
- ---activity
- ----mobility
- -----incontinence
- maximum score = 20
- score 5 of 12 or < indicates R/F development of ulcer
- BRADEN SCALE
- consists of 6 subscales
- maximum score = 23
- score of 16 or < indicates risk
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PREVENTION OF PRESSURE ULCERS
- Perform risk assessment on admission and daily
- --lift folds of skin: breasts, wrinkles, fat excess skin...
- Provide adequate nutrition
- Maintain skin hygiene: use mild cleansing agents; minimize friction and force when cleaning; don't use same washcloth for opposite folds; pat dryDo not massage over bony prominences
- Reduce friction by use of cornstarch or powder
- Pad bony prominences esp. heels and elbows
- Provide smooth, firm, wrinkle-free foundation
- Provide special mattress/specialty bed per risk assessment score and facility protocol
- Reposition, turn q 2 hours or more; consider use of trapeze bar.
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COMPLICATIONS OF WOUND HEALING
- THE CHRONIC WOUND
- non-healing/slow-healing
- does not progress from Inflammatory Phase
- causes: repetitive chemical and/or mechanical trauma
- Signs/Symptoms:
- --Delayed healing despite intervention
- --change in color of wound bed
- --friable (dry, crumbly) granulation tissue
- --new or increased pain, drainage, or odor
- INFECTION
- wound may be infected at time of injury, during surgery, or post-op
- signs/symptoms (post-op on day 2-7)
- --fever, elevated WBC's
- --Purulent drainage
- --swelling, tenderness
- --erythema at wound edges
- --general malaise
- HEMORRHAGE
- possible causes:
- dislodged clot
- slipped ligature during surgery
- erosion or cut of blood vessel
- internal hemorrhage
- --may occur without evidence of external bleeding
- --Signs/Symptoms:
- swelling in wound area
- change in general condition of pt
- --Hematoma = localized collection of blood inside tissues; forms in early post-op period
- External Hemorrhage
- greatest risk = first 48 hours
- ID'd by bloody drainage at site or on surgical dressing
- --mark with date, time, initials and check back with patient to view progression of strikethrough.
- FISTULA FORMATION
- fistula = abnormal passage between 2 internal organs or between an organ and the external skin surface.
- May result from:
- Abscess or infection
- Injury, inflammation
- Disease such as cancer
- DEHISCENCE
- partial or total rupture of wound
- may occur post-op day 4-5
- factors include:
- --obesity
- --poor nutrition; dehydration
- --excessive coughing; vomiting
- --suture failure; multiple trauma
- Evisceration = dehiscence with protrusion of internal viscera
- Interventions:
- --support wound with large sterile dressing soaked in sterile normal saline
- --notify M.D. STAT!
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ASSESSMENT OF WOUNDS
LSD SD STP WDD
- LOCATION
- document anatomical size
- SIZE
- measure in cm: length, width, depth
- COLORof wound bed (red, yellow, black)SURROUNDING SKIN
- color of wound edges
- erythema of wound edge
- DRAINAGEamount, color, consistency, odordegree of saturation of dressing (i.e. how many gauzes)SWELLING
- use gloves; palpate for tenseness around wound
- minimal to moderate swelling normal in early stages of healing process
- TEMPERATURE
- palpate wound and surrounding tissues
- cold = hypoxia, possible tissue necrosis (not enough O2 to tissues
- increased warmth = infection
- PAIN
- expect severe-moderate pain for 3-5 days post-op
- suddens onset of severe pain = possible hemorrhage or infection
- WOUND CLOSUREdescribe type of closure (staples, suture...)note if intactwell-approximated?DRAINS
- type of drain used
- inspect placement
- note/measure amount, character of drainage (purulent, sanguinous, serosanguinous...)
- DIAGNOSTICS/LAB TESTS USED IN ASSESSMENT
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DIAGNOSTIC/ LAB TESTS USED IN WOUND ASSESSMENT
- WBCS
- increase = infection
- decrease = delayed wound healing
- Hgb
- low = impaired O2 transport to tissues
- Erythrocyte Sed. Rate (ESR)high = general indication of inflammation/infectionALBUMIN
- overall nutritional status; indicates body's reserves for rebuilding cells
- low = hemodilution (increase in plasma volume = reduced RBC concentration) (i.e. with blood loss)
- Wound Culture
- r/o (rule out) or confirm presence of infectious organism
- Diagnostic Imaging
- to rule out or diagnose infection of bone
- --X-Ray
- --Bone Scan
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COMMON NURSING DIAGNOSIS
Impaired Skin Integrity
Risk For Impaired Skin Integrity
Risk for Infection
Acute Pain
Body Image Disturbance
Imbalanced Nutrition
Impaired Mobility
Anxiety, Hopelessness
Fear
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GOALS/OUTCOMES FOR WOUNDS
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Goals must be realistic: for example:
Can a serious, infected, chronic wound heal completely by client’s discharge from acute care?
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INTERVENTIONS TO PROMOTE WOUND HEALING
- IN ORDER TO EFFECTIVELY TREAT A WOUND, THE NURSE MUST TREAT
- “THE WHOLE PERSON.”
Referral to wound care specialist is necessary for all complicated or chronic wounds, including Stage 3-4 pressure ulcers.
Medications:
- o Treat systemic or local infection:
- antibiotics, topical antiinfectives
- o Treat pain:
- analgesics, topical local anesthetics
o Nutritional supplements
Dietary Interventions
o Provide adequate Nutrition/Calories
Vitamin C (citrus, green vegs.)
Vitamin A (green/yellow fruits & vegs. dairy, fish-liver oil, egg yolk)
Protein (meat, fish, eggs, dairy, legumes)
Zinc (dairy, eggs, legumes, liver, whole grains)
Hygiene
o Keep skin clean and dry
Clean the Wound With Cleaning Agents
o Removes bacteria, slough and necrotic tissue
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DEBRIDEMENT OF A WOUND
- DEBRIS IN A WOUND PREVENTS THE WOUND FROM HEALING
- Wound Debridement
- – Removal of dirt, foreign matter or necrotic tissue (e.g. eschar) from a wound that cannot be removed via normal cleaning.
- Allows thorough examination of the extent of wound
- o Types of Debridement
- Sharp – use of a scalpel, scissors, or laser
- Mechanical – use of mechanical force, (e.g. whirlpool, scrubbing, or wet-to-moist dressing)
- Enzymatic – chemical breakdown of necrosis
- Autolytic – moisture-retentive dressing with occlusive seal allows body’s own macrophages and neutrophils to destroy necrotic tissue
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WOUND DRESSINGS: PURPOSE
Purposes of Dressings
Protect wound from mechanical injury
Protect wound from microbial contamination
- Provide high humidity
- a moist wound bed promotes re-epithelialization of the wound
Absorb drainage and/or debride a wound
Prevent hemorrhage ( when applied as a pressure dressing)
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WOUND DRAINS: PURPOSES
- Permit drainage of excessive fluid from surgical
- incisions
Drain purulent material from infected wounds
Promote healing
Assist in wound closure/ approximation
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OTHER INTERVENTIONS FOR WOUND HEALING
Hyperbaric therapy
Light therapy
Ultrasound, Electrotherapy
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CLIENT TEACHING
- Self-care includes
- maintaining hygiene (handwashing)
- understanding correct wound care supplies and techniques
- signs of infection
- Family teaching includes:
- positioning
- protective devices
- hygiene and nutrition
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EVALUATION OF INTERVENTIONS may include
Does wound show signs of healing?
Does wound remain free from signs of infection?
Does skin of Stage 1 pressure ulcer remain intact?
Is client verbalizing adaptation to changes in body image?
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