CH7 Innate Immunity: Inflammation

  1. Which action is a purpose of the inflammatory process?



    B.
  2. How do surfactant proteins A through D provide innate resistance?



    • B.
    • The lung produces and secretes a family of glycoproteins, collectins, which includes surfactant proteins A through D and mannose-binding lectin. Collectin binding facilitates macrophages to recognize the microorganism, enhancing macrophage attachment, phagocytosis, and killing.
  3. Which secretion is a first line of defense against pathogen invasion that involves
    antibacterial and antifungal fatty acids, as well as lactic acid?



    B.
  4. What causes the edema that occurs during the inflammatory process?



    D.
  5. Activation of the classical pathway begins with:



    A.
  6. What plasma protein system forms a fibrinous meshwork at an inflamed site?



    • D.
    • The coagulation (clotting) system is a group of plasma proteins that form a fibrinous meshwork at an injured or inflamed site. This protein system (1) prevents the spread of infection to adjacent tissues, (2) traps microorganisms and foreign bodies at the site of inflammation for removal by infiltrating cells (e.g., neutrophils and macrophages), (3) forms a clot that stops the bleeding, and (4) provides a framework for future repair and healing.
  7. Which component of the plasma protein system tags pathogenic microorganisms for
    destruction by neutrophils and macrophages?



    • C.
    • C3b (a component of the complement cascade) adheres to the surface of a pathogenic microorganism and serves as an efficient opsonin. Opsonins are molecules that tag
    • microorganisms for destruction by cells of the inflammatory system, primarily neutrophils and macrophages.
  8. _______ are molecules that tag
    microorganisms for destruction by cells of the inflammatory system, primarily neutrophils and macrophages.
    Opsonins
  9. What is the vascular effect of histamine released from mast cells?



    • A.
    • mast cell degranulation  and the release of histamine causes vasodilation and increased capillary permeability.
  10. \What is an outcome of the complement cascade?



    • A.
    • (1) anaphylatoxic activity, resulting in mast cell
    • degranulation
    • (2) leukocyte chemotaxis
    • (3) opsonization
    • (4) cell lysis.
  11. The function of opsonization related to the complement cascade is to:
    a. Tag of pathogenic microorganisms for destruction by neutrophils and
    macrophages.
    b. Process pathogenic microorganisms so that activated lymphocytes can be created
    for acquired immunity.
    c. Destroy glycoprotein cell membranes of pathogenic microorganisms.
    d. Promote anaphylatoxic activity, resulting in mast cell degranulation.
    a.
  12. In the coagulation (clotting) cascade, the intrinsic and the extrinsic pathways converge at
    which factor?



    C. X
  13. Which chemical interacts among all plasma protein systems by degrading blood clots,
    activating complement, and activating the Hageman factor?



    • C.
    • Only plasmin regulates clot formation by degrading fibrin 

    Plasmin can activate the plasma kinin cascade by activating the Hageman factor (factor XII) and producing prekallikrein activator
  14. Plasmin can activate the plasma kinin cascade by activating the ______________ and producing prekallikrein activator
    kinin; Hageman factor (factor XII)
  15. The chemotactic factor affects the inflammatory process by:



    B.
  16. What affect does the process of histamine binding to the histamine-2 (H2) receptor have on
    inflammation?



    D.
  17. Binding of histamine to the __________ is essentially proinflammatory; that is, it promotes inflammation
    H1 receptor
  18. Some older adults have impaired inflammation and wound healing because of which
    problem?



    B.
  19. What is the inflammatory effect of nitric oxide (NO)?



    • B.
    • Effects of NO on inflammation include vasodilation by inducing relaxation of vascular smooth muscle, a response that is local and short lived, and by suppressing mast cell function, as well as platelet adhesion and aggregation.
  20. When considering white blood cell differentials, acute inflammatory reactions are related to elevations of which leukocyte?



    • C.
    • Only neutrophils are the predominant phagocytes in the early inflammatory site, arriving within 6 to 12 hours after the initial injury, they ingest (phagocytose) bacteria, dead cells,and cellular debris at the inflammatory site.
  21. In the later stages of an inflammatory response, which phagocytic cell is predominant?



    • A.
    • Only monocytes and macrophages perform many of the same functions as neutrophils but for a longer time and in a later stage of the inflammatory response.
  22. In regulating vascular mediators released from mast cells, the role of eosinophils is to
    release:



    • D.  
    • Eosinophil lysosomes contain several enzymes that degrade vasoactive molecules, thereby controlling the vascular effects of inflammation. These enzymes include histaminase, which mediates the degradation of histamine, and arylsulfatase B, which mediates the degradation of some of the lipid-derived mediators produced by mast cells.
  23. histaminase mediates the degradation of histamine, and _____________, which mediates the degradation of some of the lipid-derived mediators produced by mast cells.
    arylsulfatase B
  24. What is the role of a natural killer (NK) cells?



    C.
  25. Which cytokine is produced and released from virally infected host cells?



    A.
  26. What occurs during the process of repair after tissue damage?



    B.
  27. The role of fibroblasts during the reconstructive phase of wound healing is to:



    A.
  28. transforming growth factor–beta (TGF-ß) stimulates _______.
    fibroblasts
  29. A keloid is the result of which dysfunctional wound healing response?



    D.
  30. Many neonates have a transient depressed inflammatory response as a result of which
    condition?



    C.
  31. During phagocytosis, what is occurring during the step referred to as opsonization?



    B.
  32. Fusion is the step in phagocytosis during which:



    A.
  33. During the process of endocytosis, the phagosome step results in:



    C.
  34. When cellular damage occurs and regeneration is minor with no significant complications,
    the process of returning the cells to preinjury function is referred to as:



    A.
  35. Newborns often have deficiencies in collectin-like proteins, making them more susceptible
    to what type of infection?



    D.
  36. Which cell is the body’s primary defense against parasite invasion?



    • A.
    • Eosinophils serve as the body’s primary defense against parasites. T lymphocytes and B lymphocytes are involved in acquired immunity. Neutrophils are the predominant
    • phagocytes in the early inflammatory site
  37. Which chemical mediators induce pain during an inflammatory response? (Select all that
    apply.)
    a. Prostaglandins
    b. Leukotrienes
    c. Tryptase
    d. Phospholipase
    e. Bradykinin
    a, e
  38. Sebaceous glands protect the body from infection by secreting the following except for which one?



    B.
  39. The main function of NK cells does not include:



    B.
  40. Normal bacterial flora found in the intestines produce vitamin K to assist in the absorption of the following except for which one?



    A.
  41. An individual’s acquired immunity is dependent on the function of the following cells except for which one? 



    A.
  42. Which is NOT an example of a pathogen capable of surviving and even multiplying inside a macrophage?




    A.
  43. An older adult is particularly susceptible to infections of the following body parts except for which one? 



    B.
  44. collectins
    The lung produces and secretes a family of glycoproteins, collectins, which includes surfactant proteins
  45. 3 plasma protein systems
    • the complement system
    • the clotting system
    • the kinin system
  46. complement cascade activation: 3 types
    classical, lectin, alternative
  47. _________ pathway of complement cascade is activated by antigen-antibody complexes
    classical
  48. ________ pathway of complement cascade is activated by mannose-binding lectin (MLB)
    lectin
  49. _________ pathway of the complement cascade is activated by gram-negative bacterial polysaccharides and fungal cell wall polysaccharides
    Alternative
  50. The main substance in this fibrinous mesh is an insoluble protein called ______ that is the end product of the coagulation cascade
    fibrin
  51. extrinsic pathway reacts with ____ factor
    VII
  52. intrinsic pathway interacts with ___ factor
    XII, the Hageman Factor
  53. the coagulation system produces
    fibrin
  54. the kinin system produces
    bradykinin
  55. plasmin
    degrade fibrin polymers in clots
Author
BodeS
ID
362430
Card Set
CH7 Innate Immunity: Inflammation
Description
Innate Immunity: Inflammation
Updated