Purple Notes (De Lisa)

  1. Root level of biceps




    A) C5
  2. Root level of brachioradialis




    C) C6
  3. Root level of pronator teres




    A) C6
  4. Sensation over the lateral (radial) side of antecubital fossa




    B) C5

    Sensation over the medial (ulnar) side of antecubital fossa: T1
  5. Sensation over the thumb




    D) C6
  6. Sensation over the middle finger




    A) C7
  7. C6/C7




    B) Both
  8. C5/C6





    D) All of these
  9. Amount of movement that the humeral head moves beyond the rim of the glenoid fossa: >1/2-1




    A) Grade II

    Grade Movement
    I <1/2
    II >1/2-1
    III Subluxation
  10. L1,2,3, except:




    D) Abductors

    It should be adductors.
  11. L2,3,4, except:





    E) None of these
  12. L3,4,5





    E) All of these
  13. S1,2, except:





    E) Gluteus medius

    It should be gluteus maximus. L4,5 is extensor hallucis longus and gluteus medius.
  14. Reflex testing: patellar tendon




    A) L4
  15. Reflex testing: medial hamstring




    A) L5
  16. Reflex testing: Achilles tendon




    C) S1
  17. Sensation testing of the hip: L2





    A) Midanterior thigh

    Root Sensory Area
    L2 Midanterior thigh
    L3 Medial femoral condyle
    L4 Medial heel
    L5 Dorsum of the foot at the third MTP joint
    S1 Lateral heel
  18. Sensation testing of the knee: superior and medial to the patella




    C) L3

    Root Location
    L3 Superior and medial to the patella
    L4 Medial side of the knee and anteromedial shin
    L5 Anterolateral shin and dorsum of the foot between the 2nd and 3rd web space
    S2 Popliteal fossa
  19. Sensation testing of the knee: L5, except:




    B) Dorsum of the foot between 1st and 2nd web space

    It should be dorsum of the foot between the 2nd and 3rd web space.

    Root Location
    L3 Superior and medial to the patella
    L4 Medial side of the knee and anteromedial shin
    L5 Anterolateral shin and dorsum of the foot between the 2nd and 3rd web space
    S2 Popliteal fossa
  20. MMT: Examiner can overcome





    B) 4
  21. Fibrillation and Positive Sharp Wave Grading: continuous discharges in all areas of the muscle; baseline often obscured





    B) Grade 4

    Grade Characteristics
    0 None
    1+ Persistent single runs >1s in two areas
    2+ Moderate runs >1s in three or more areas
    3+ Many discharges in most muscle regions
    4+ Continuous discharges in all areas of the muscle; baseline often obscured
  22. Fibrillation and Positive Sharp Wave Grading: persistent single runs >1s in two areas





    C) Grade 1+

    Grade Characteristics
    0 None
    1+ Persistent single runs >1s in two areas
    2+ Moderate runs >1s in three or more areas
    3+ Many discharges in most muscle regions
    4+ Continuous discharges in all areas of the muscle; baseline often obscured
  23. Strength requirements classification: occasional lifting/carrying: ≤10 lbs





    A) Sedentary work

    Degree of Strength Occasional Frequent
    Sedentary work ≤10 lbs None
    Light work ≤20 lbs ≤10 lbs
    Medium work ≤50 lbs ≤20 lbs
    Heavy work ≤100 lbs ≤50 lbs
    Very heavy work >100 lbs ≥50 lbs
  24. Strength requirements classification: occasional lifting/carrying: ≤50 lbs





    C) Medium work

    Degree of Strength Occasional Frequent
    Sedentary work ≤10 lbs None
    Light work ≤20 lbs ≤10 lbs
    Medium work ≤50 lbs ≤20 lbs
    Heavy work ≤100 lbs ≤50 lbs
    Very heavy work >100 lbs ≥50 lbs
  25. Not an example of hyperkinetic (quick) dysarthria





    E) Dyskinesia

    Dyskinesia is an example of hyperkinetic (slow) dysarthria.
  26. Flaccid dysarthria





    A) Bulbar palsy

    Type of Dysarthria Example/s
    Flaccid Bulbar palsy
    Spastic Pseudobulbar palsy
    Ataxic Cerebellar ataxia
    Hypokinetic Parkinsonism
    Hyperkinetic (quick) Chorea, myoclonus, Gilles de la Tourette syndrome
    Hyperkinetic (slow) Athetosis, dyskinesia, dystonia
    Hyperkinetic (tremor) Organic voice tremor
    Mixed ALS, MS, Wilson's disease
  27. Mixed dysarthria, except:




    C) Parkinsonism

    Parkinsonism is an example of hypokinetic dysarthria.
  28. Location of neuropathology: extrapyramidal system, except:





    C) None of these

    Location of Neuropathology Type of Dysarthria
    Lower motor neuron Flaccid
    Upper motor neuron Spastic
    Cerebellum (or tracts) Ataxic
    Extrapyramidal system Hypokinetic, hyperkinetic (quick, slow, tremor)
    Multiple motor systems Mixed
  29. Type of dysarthria: reduced range, force, speed; hypertonia





    E) Spastic

    Type Neuromuscular Deficit
    Flaccid Muscular weakness; hypotonia
    Spastic Reduced range, force, and speed; hypertonia
    Ataxic Hypotonia; reduced speed; inaccurate range, timing, direction
    Hypokinetic Markedly reduced range; variable speed of repetitive movements; movement arresting rigidity
    Hyperkinetic (quick) Quick unsustained, random, involuntary movements
    Hyperkinetic (slow) Sustained, distorted movements and postures; slowness; variable hypertonus
    Hyperkinetic (tremor) Involuntary, rhythmic, purposeless, oscillatory movements
    Mixed Muscular weakness, limited range and speed
  30. Type of dysarthria: Quick, unsustained, random, involuntary movements





    E) Hyperkinetic (quick)

    Type Neuromuscular Deficit
    Flaccid Muscular weakness; hypotonia
    Spastic Reduced range, force, and speed; hypertonia
    Ataxic Hypotonia; reduced speed; inaccurate range, timing, direction
    Hypokinetic Markedly reduced range; variable speed of repetitive movements; movement arresting rigidity
    Hyperkinetic (quick) Quick unsustained, random, involuntary movements
    Hyperkinetic (slow) Sustained, distorted movements and postures; slowness; variable hypertonus
    Hyperkinetic (tremor) Involuntary, rhythmic, purposeless, oscillatory movements
    Mixed Muscular weakness, limited range and speed
  31. Type of Dysarthria: Involuntary, rhythmic, purposeless, oscillatory movements





    E) Hyperkinetic (tremor)

    Type Neuromuscular Deficit
    Flaccid Muscular weakness; hypotonia
    Spastic Reduced range, force, and speed; hypertonia
    Ataxic Hypotonia; reduced speed; inaccurate range, timing, direction
    Hypokinetic Markedly reduced range; variable speed of repetitive movements; movement arresting rigidity
    Hyperkinetic (quick) Quick unsustained, random, involuntary movements
    Hyperkinetic (slow) Sustained, distorted movements and postures; slowness; variable hypertonus
    Hyperkinetic (tremor) Involuntary, rhythmic, purposeless, oscillatory movements
    Mixed Muscular weakness, limited range and speed
  32. Type of dysarthria: hypotonia; reduced speed; inaccurate range, timing, direction





    A) Ataxic

    Type Neuromuscular Deficit
    Flaccid Muscular weakness; hypotonia
    Spastic Reduced range, force, and speed; hypertonia
    Ataxic Hypotonia; reduced speed; inaccurate range, timing, direction
    Hypokinetic Markedly reduced range; variable speed of repetitive movements; movement arresting rigidity
    Hyperkinetic (quick) Quick unsustained, random, involuntary movements
    Hyperkinetic (slow) Sustained, distorted movements and postures; slowness; variable hypertonus
    Hyperkinetic (tremor) Involuntary, rhythmic, purposeless, oscillatory movements
    Mixed Muscular weakness, limited range and speed
  33. The absence of recognized grammatical elements during speech attempts





    A) Agrammatism
  34. Difficulty producing nouns




    A) Anomia
  35. Attempts at word retrieval end in descriptions or associations related  to the word




    A) Circumlocution
  36. An accurate repetition of a preceding utterance when repetition is not required




    B) Echolalia
  37. A fluent utterance that lacks substantive parts of the language, such as nouns and verbs




    C) Empty speech
  38. Mostly incomprehensible but well-articulated language




    C) Jargon
  39. Mostly incomprehensible, some words are partially recognizable, others are contrived or "new"




    B) Neologistic jargon
  40. Misuse of grammatical elements, usually during fluent utterances




    C) Paragrammatism
  41. Flair for chair, also called literal paraphasia




    B) Phonemic paraphasia
  42. Excessively lengthy, often incomprehensible, well-articulated language




    B) Press for speech
  43. A combination of unrelated semantic and phonemic paraphasia, together with recognizable words




    C) Semantic jargon
  44. Table for chair, also called nominal paraphasia




    D) Semantic paraphasia
  45. Nonsensical repetition of similar syllables for all communicative attempts, such as dee dee dee




    C) Stereotypes
  46. Language similar to a telegram, mostly nouns and verbs




    A) Telegraphic speech
  47. Nonfluent aphasias, except:





    D) Conduction

    Conduction aphasia is a fluent aphasia.
  48. Aphasia: telegraphic, agrammatic expression often associated with apraxia; good comprehension except on more abstract tasks




    B) Broca's
  49. Aphasia: limited language output; fair naming; intact repetition; fair comprehension




    C) Transcortical motor
  50. Aphasia: severe expressive and receptive reduction in language




    A) Global
  51. Aphasia: severe reduction in expression and reception; repetition intact




    C) Mixed transcortical
  52. Aphasia: word-finding difficulty without other serious linguistic deficits




    A) Anomic
  53. Aphasia: phonemic paraphasic errors; good comprehension; fluency in bursts; deficits in repetition of low-probability phrases




    D) Conduction
  54. Aphasia: phonemic and semantic paraphasia; poor comprehension




    D) Wernicke's
  55. Aphasia: fluent neologistic language; poor comprehension; intact repetition




    C) Transcortical sensory
  56. Therapeutic and Compensatory Techniques for Managing Patients with Dysphagia: improve bolus flow through the pharynx by improving tongue base-to-pharyngeal wall contact





    C) Effortful or hard swallow
  57. Therapeutic and Compensatory Techniques for Managing Patients with Dysphagia: prevent advancement of the bolus beyond the valleculae; reduce distance of tongue base to-pharyngeal wall contact




    C) Chin down/chin tuck
  58. Therapeutic and Compensatory Techniques for Managing Patients with Dysphagia: head is typically turned to the ________ side in an effort to direct the bolus to the stronger side of the pharynx allowing for better clearance of the bolus




    B) Weak

    This is the definition of head turn.
  59. Therapeutic and Compensatory Techniques for Managing Patients with Dysphagia: head is tilted toward the _______ side to improve oral control of the bolus




    A) Both

    This is the definition of head tilt.
  60. Therapeutic and Compensatory Techniques for Managing Patients with Dysphagia: improve vocal fold closure to prevent aspiration




    A) Supraglottic swallow
  61. Therapeutic and Compensatory Techniques for Managing Patients with Dysphagia: improve vocal fold closure and arytenoids-to-epiglottic petiole contact to improve airway closure during the swallow




    C) Super-supraglottic swallow
  62. Therapeutic and Compensatory Techniques for Managing Patients with Dysphagia: improve bolus control by slowing the bolus




    B) Thickened liquids
  63. Therapeutic and Compensatory Techniques for Managing Patients with Dysphagia: reduced friction; improve bolus passage to esophagus




    D) Thin liquids
  64. Therapeutic and Compensatory Techniques for Managing Patients with Dysphagia: indicated for tight UES




    D) Thin liquids
  65. Therapeutic and Compensatory Techniques for Managing Patients with Dysphagia: indicated for impaired/delayed vocal fold closure




    A) Supraglottic swallow
  66. Therapeutic and Compensatory Techniques for Managing Patients with Dysphagia: indicated for impaired/reduced vocal fold and laryngeal closure




    D) Super-supraglottic swallow
  67. Therapeutic and Compensatory Techniques for Managing Patients with Dysphagia: indicated for pharyngeal weakness, except




    D) Head tilt

    Head tilt is indicated for oral weakness and reduced sensation.
  68. Therapeutic and Compensatory Techniques for Managing Patients with Dysphagia: indicated for oral weakness and reduced sensation




    B) Head tilt
  69. Therapeutic and Compensatory Techniques for Managing Patients with Dysphagia: indicated for reduced tongue base retraction




    D) Effortful/hard swallow
  70. Therapeutic and Compensatory Techniques for Managing Patients with Dysphagia: indicated for tongue base retraction




    A) Chin down/chin tuck
  71. Therapeutic and Compensatory Techniques for Managing Patients with Dysphagia: indicated for bolus control




    B) Thickened liquids
  72. Therapeutic and Compensatory Techniques for Managing Patients with Dysphagia: indicated for delayed initiation of pharyngeal swallow




    D) Chin down/chin tuck
  73. Classification System for Degree of Hearing Loss: 71-90 dB HL




    A) Severe

    Pure Tone Average Classification
    0-15 dB HL Normal
    16-25 dB HL Slight
    26-40 dB HL Mild
    41-55 dB HL Moderate
    56-70 dB HL Moderately severe
    71-90 dB HL Severe
    91 dB HL and above Profound
  74. Classification System for Degree of Hearing Loss: 26-40 dB HL





    D) Mild

    Pure Tone Average Classification
    0-15 dB HL Normal
    16-25 dB HL Slight
    26-40 dB HL Mild
    41-55 dB HL Moderate
    56-70 dB HL Moderately severe
    71-90 dB HL Severe
    91 dB HL and above Profound
  75. Classification System for Degree of Hearing Loss: moderately severe




    D) 56-70 dB HL

    Pure Tone Average Classification
    0-15 dB HL Normal
    16-25 dB HL Slight
    26-40 dB HL Mild
    41-55 dB HL Moderate
    56-70 dB HL Moderately severe
    71-90 dB HL Severe
    91 dB HL and above Profound
  76. Rehabilitation Medications Affecting Sexual Functioning: all of the following medications cause decreased sex drive, arousal dysfunction, and ejaculatory or orgasmic dysfunction, except:





    C) Anticonvulsants

    Anticonvulsants cause decreased sex drive and arousal dysfunction.
  77. How does digoxin, H2-blockers, and anticonvulsants affect sexual functioning?




    A) Decreased sex drive and arousal dysfunction
  78. All of the following drugs cause arousal dysfunction and ejaculatory or orgasmic dysfunction, except:





    E) Antihypertensives

    Antihypertensives cause decreased sex drive, arousal dysfunction, ejaculatory or orgasmic dysfunction.
  79. Sex suppressant, especially orgasm




    B) SSRIs
  80. Central sympatholytic/peripheral a-blocker




    D) Antihypertensives
  81. ↓Testosterone levels and/or ↑prolactin levels: with chronic use, sex suppressant, except:




    B) Anticonvulsants

    Anticonvulsants decrease arousal.
  82. May suppress sexual reflexes




    D) Antispasmodics
  83. ↓Testosterone and LH and ↑estrogen




    C) Digoxin

    Indomethacin causes fertility problems; gabapentin causes variable effects; and marijuana decreases testosterone and increases prolactin levels.
  84. Can cause reversible spermatogenic suppression




    A) H2 blockers
  85. All of the following are modifiable risk factors for stroke, except:





    A) Decreased fibrinogen

    It should be elevated fibrinogen.
  86. Least common cause of stroke





    C) Subarachnoid hemorrhage

    Cause %
    Large vessel occlusion, infarction 32%
    Embolism 32%
    Small vessel occlusion, lacunar 18%
    Intracerebral hemorrhage 11%
    Subarachnoid hemorrhage 7%
  87. Which of the following is not a possible source of cerebral embolism?





    D) Bacterial valve vegetations

    It should be nonbacterial valve vegetations. 

    • Cardiac sources:
    •  1.) Atrial fibrillation, other arrhythmias
    •  2.) Mural thrombus - recent MI, hypokinesis, cardiomyopathy
    •  3.) Bacterial endocarditis
    •  4.) Valve prosthesis 
    •  5.) Nonbacterial valve vegetations
    •  6.) Atrial myxoma

    Large artery source: atherosclerosis of aorta and carotid arteries

    Paradoxical cause: peripheral venous embolism with R-L cardiac shunt
  88. Which of the following is not a cause of stroke in children and young adults?





    E) Trauma to intracranial arteries

    It should be trauma to extra cranial arteries. 

    • Causes of stroke in children and young adults:
    •  1.) Cerebral embolism
    •  2.) Trauma to extracranial arteries - e.g thromboembolic occlusion, dissection
    •  3.) Subarachnoid hemorrhage - aneurysm, arteriovenous malformation
    •  4.) Sickle cell anemia
    •  5.) Vasculopathy - Moyamoya disease, SLE, drug-induced, vasculitis
    •  6.) Coagulopathy - deficiencies in antithrombin III, protein C, and protein S
    •  7.) Homocystinuria
    •  8.) Oral contraceptives
    •  9.) Postpartum
    •  10.) Drug-induced
  89. This aphasia test produces a classification of the aphasic features observed in a patient. Besides classifying the aphasia, it also provides a score of severity of the aphasia, which can be compared to aphasic patients in general.




    B) Boston Diagnostic Aphasia Examination
  90. This aphasia test is somewhat similar to Boston. It measures various parameters of spontaneous speech and examines comprehension, fluency, object naming, and repetition.




    C) Western Aphasia Battery
  91. This aphasia test is different from the other tests in that it evaluates verbal, gestural, and graphic responses. It is very structured in its format and must be given by a trained professional. It provides a useful statistical summary of the details of the language impairments and offers outcome prediction.




    A) Porch Index of Communication Ability
  92. This aphasia test is not diagnostic; the score indicates the severity and can be useful indicator of recovery.




    A) Functional Communication Profile
  93. Brunnstrom Stages of Motor Recovery: no activation of the limb




    C) Stage 1
  94. Brunnstrom Stages of Motor Recovery: spasticity appears, and weak basic flexor and extensor synergies are present




    D) Stage 2
  95. Brunnstrom Stages of Motor Recovery: spasticity is prominent; the patient voluntarily moves the limb, but muscle activation is all within the synergy patterns




    A) Stage 3
  96. Brunnstrom Stages of Motor Recovery: the patient begins to activate muscles selectively outside the flexor and extensor synergies




    C) Grade 4
  97. Brunnstrom Stages of Motor Recovery: Spastic decreases, most muscle activation is selective and independent from the limb synergies




    B) Grade 5
  98. Brunnstrom Stages of Motor Recovery: isolated movements are performed in a smooth, phasic, well-coordinated manner




    B) Stage 6
  99. Most common medical complication during post acute stroke rehabilitation





    E) All of these

    Least common medical complication is pressure ulcer.
  100. Least common neurologic complication during post acute stroke rehabilitation




    B) Seizure

    Most common neurologic complication is toxic or metabolic encephalopathy.
  101. Glasgow Outcome Scale-Extended: no cerebral function that can be judged by behavior (not able to follow simple commands or communicate)





    A) 2

    2 is vegetative state.
  102. Glasgow Outcome Scale-Extended: 5




    A) Independent in ADLs; can shop and travel independently on public transportation, but has not returned to previous position or lifestyle

    Grade Description
    1 Dead
    2 Vegetative state: no cerebral function can be judged by behavior (unable to follow simple commands or communicate
    3 Lower severe disability: needs full assistance throughout the day
    4 Upper severe disability: needs assistance/supervision in ADLs, but can be alone for >8 h/d
    5 Lower moderate disability: independent in ADLs, can shop and travel independently on public transportation, but unable to return to previous position or lifestyle
    6 Upper moderate disability: able to return to previous position or lifestyle with alternative/modified duties or part-time due to injury
    7 Lower good recovery: able to return to previous position or lifestyle (may be modified), but reporting some problems
    8 Upper good recovery: able to return to previous position or lifestyle with no reported problems
  103. Not a positive phenomenon in PD:





    C) Bradykinesia

    Bradykinesia is a negative phenomenon in PD.
  104. Not a negative phenomenon in PD:





    B) Rigidity

    Rigidity is a positive phenomenon.
  105. Most common symptom in PD




    D) Tremor
  106. Not true about tremor in PD




    B) Proximal involvement

    It should be distal involvement.
  107. Not true about rigidity in PD:




    C) Decreases if contralateral limb is engaged in volition

    Rigidity in PD increases if the contralateral limb is engaged in volition.
  108. On EMG, bradykinesia will manifest as:





    B) All of these
  109. Loss of postural reflexes in PD will result in:





    E) All of these
  110. Not true about shuffling gait pattern in patients with PD





    E) None of these

    Shuffling gait is associated with bradykinesia, hypokinesia, and movement initiation and execution impairment.
  111. Festination is associated with all of the following impairments, except:




    C) Rigidity

    Rigidity is associated with stooped (flexed) posture and cautious gait (fear of falling).
  112. Which of the following impairments is not associated with freezing or "start hesitation"?




    C) Impaired postural responses
  113. Which of the following PD medications causes St. Anthony's fire?





    D) Bromocriptine

    St. Anthony's fire refers to red, inflamed skin which is reversible on drug discontinuation.
  114. Which of the following areas should not be stimulated during DBS procedures in patients with PD?




    D) Globus pallidus pars externa

    It should be globus pallidus pars interna.
  115. Brief, rapid, forceful, dysrhythmic, discrete, purposeless, flinging of the limb




    B) Chorea
  116. Slow, writhing movements and inability to maintain position of limb or body part




    B) Athetosis
  117. Large amplitude, flinging movement of limb (usually proximal)




    A) Hemiballismus
  118. Sustained muscle contraction that leads to repetitive twisting movement of variable speed and abnormal posture




    B) Dystonia
  119. Rhythmic, oscillatory movements of a body part




    D) Tremor
  120. Intermittent, repetitive, stereotypical, abrupt, jerky, typically affecting the head and face




    D) Tic
  121. Purposeless, uniformly repetitive, voluntary movement of the whole body areas




    B) Stereotypy
  122. Subjective restlessness, compulsion to move about




    B) Akathisia
  123. Sudden, brief, irregular contraction of a group of muscles




    B) Myoclonus
  124. The level at which, by radiological examination, the greatest vertebral damage is found




    A) Skeletal level

    Sensory level is the most caudal dermatome to have normal sensation (2/2) for both pinprick /dull and light touch on both sides. 

    Motor level is the most caudal key muscle group that is graded 3/5 or greater with the segments cephalad graded 5/5. 

    Neurological level of injury is the most caudal level at which both sensory and motor modalities are intact.
  125. Suggested equipment for complete tetraplegic: BFO (mobile arm support)





    E) C1-C4
  126. Suggested equipment for complete tetraplegic: resting hand splint, except




    C) C7
  127. Suggested equipment for complete tetraplegic: long opponens splint




    D) C5

    Long opponens splint is indicated for C1-C5 SCI.
  128. Suggested equipment for complete tetraplegic: spiral splint




    B) Both
  129. Suggested equipment for complete tetraplegic: powered tenodesis splint





    C) C5
  130. Suggested equipment for complete tetraplegic: wrist-driven tenodesis splint





    C) C6
  131. Suggested equipment for complete tetraplegic: ratchet tenodesis splint





    E) C5
  132. Suggested equipment for complete tetraplegic: short opponens splint




    C) C6

    Short opponens splint is indicated for C6-C7 SCI.
  133. Suggested equipment for complete tetraplegic: universal cuff, except




    B) C8-T1
  134. Suggested equipment for complete tetraplegic: lumbrical bar





    D) C8-T1
  135. Suggested equipment for complete tetraplegic: mouth stick





    E) C1-C4
  136. Suggested equipment for complete tetraplegic: power/mechanical lift, except:




    C) C7
  137. Suggested equipment for complete tetraplegic: transfer board






    C) All of these
  138. Suggested equipment for complete tetraplegic: manual wheelchair






    D) All of these
  139. Suggested equipment for complete tetraplegic: power wheelchair






    C) C6
  140. Suggested equipment for complete tetraplegic: power wheelchair with tilt/recline




    A) Both
  141. Suggested equipment for complete tetraplegic: power wheelchair assist






    D) C5

    Power wheelchair assist is indicated in C5-C6 SCI.
  142. Suggested equipment for complete tetraplegic: utensils with built-up handles




    B) Both
  143. Suggested equipment for complete tetraplegic: ADL splints (wash mitt, razor holders)




    C) Both
  144. Suggested equipment for complete tetraplegic: dressing equipment (pant loops, sock aide, dressing stick, long shoe horn, etc.)






    C) C6

    Dressing equipment is indicated for C6-C7 SCI.
  145. Suggested equipment for complete tetraplegic: gooseneck mirror






    B) All of these
  146. Suggested equipment for complete tetraplegic: computer, except:





    B) C8-T1
  147. Suggested equipment for complete tetraplegic: book holder, except:





    B) C8-T1
  148. Suggested equipment for complete tetraplegic: grab bars, except:




    C) C5
  149. Suggested equipment for complete tetraplegic: reci shower or commode chair





    A) C1-C4
  150. Suggested equipment for complete tetraplegic: tub seat/shower chair (padded), except





    D) C1-C4
  151. Suggested equipment for complete tetraplegic: handheld spray attachment






    F) All of these
  152. Suggested equipment for complete tetraplegic: full electric hospital bed, except:




    B) C7
  153. Suggested equipment for complete tetraplegic: full specialized mattress




    C) Both
  154. Suggested equipment for complete tetraplegic: overlay mattress 




    C) Neither

    Overlay mattress is indicated for C6-C7 SCI.
  155. Modified International Classification for Individuals with Tetraplegia: in motor group 5, all of the following muscles are functional (grade of 4 or 5), except:





    D) Finger extensors

    Motor Group Functional Muscles
    0 Weak or absent BR (grade 3 or less)
    1 BR
    2 BR, ECRL
    3 BR, ECRL, ECRB
    4 BR, ECRL, ECRB, PT
    5 BR, ECRL, ECRB, PT, FCR
    6 BR, ECRL, ECRB, PT, FCR, finger extensors
    7 BR, ECRL, ECRB, PT, FCR, finger extensors, thumb extensors
    8 BR, ECRL, ECRB, PT, FCR, finger extensors, thumb extensors, finger flexors
    9 Lacks intrinsics only
  156. Correlation of Peripheral Nerve Anatomy and Pathology: rate limit to healing





    A) Axon
  157. Correlation of Peripheral Nerve Anatomy and Pathology: susceptibility to lipid lowering medication





    B) Schwann cell
  158. Correlation of Peripheral Nerve Anatomy and Pathology: ischemia





    C) Mesoneurium
  159. Correlation of Peripheral Nerve Anatomy and Pathology: axonal swelling





    E) Perineurium
  160. Correlation of Peripheral Nerve Anatomy and Pathology: vulnerability to compression injury





    C) Epineurium
  161. Correlation of Peripheral Nerve Anatomy and Pathology: acts as capillary support





    D) Mesoneurium
  162. Correlation of Peripheral Nerve Anatomy and Pathology: acts as blood-nerve barrier





    B) Perineurium
  163. Correlation of Peripheral Nerve Anatomy and Pathology: provides resistance to stretch





    D) Epineurium
  164. Not an etiology of peripheral neuropathy in which the most commonly affected structure is the axon.




    A) Nutritional

    In nutritional peripheral neuropathy, the most commonly affected structure is the myelin.
  165. Etiology of peripheral neuropathy in which the most commonly affected structure is the axon and possibly myelin




    A) Toxic or metabolic

    In hereditary/genetic, traumatic, and thermal etiologies, the most commonly affected structures are the myelin and axon.
  166. Seddon Classification of Peripheral Nerve Pathology: caused by compression



    A) Neurapraxia
  167. Seddon Classification of Peripheral Nerve Pathology: caused by traction or severe compression



    C) Axonotmesis
  168. Seddon Classification of Peripheral Nerve Pathology: caused by trauma or infection



    B) Neurotmesis
  169. Seddon Classification of Peripheral Nerve Pathology: consequence is conduction block



    C) Neurapraxia
  170. Seddon Classification of Peripheral Nerve Pathology: consequence is Wallerian degeneration



    A) Axonotmesis
  171. Seddon Classification of Peripheral Nerve Pathology: consequence is complete disruption of nerve continuity



    A) Neurotmesis
  172. Seddon Classification of Peripheral Nerve Pathology: affects thick, myelinated motor nerves



    C) Neurapraxia
  173. Seddon Classification of Peripheral Nerve Pathology: affects myelinated motor or sensory nerves



    B) Axonotmesis
  174. Seddon Classification of Peripheral Nerve Pathology: affects any nerve



    B) Neurotmesis
  175. Seddon Classification of Peripheral Nerve Pathology: healing in 1-2 months



    A) Neurapraxia
  176. Seddon Classification of Peripheral Nerve Pathology: good prognosis for healing if short or distal motor or sensory segments are affected



    B) Axonotmesis
  177. Seddon Classification of Peripheral Nerve Pathology: poor prognosis for healing even with surgery



    B) Neurotmesis
  178. HSMN type I





    A) Charcot-Marie Tooth disease

    Type Name
    I Charcot-Marie-Tooth disease
    II Charcot-Marie-Tooth disease
    III Dejerine Sottas disease
    IV Refsum's disease
    V With spastic paraplegia
    VI With optic atrophy
    VII -
  179. Which of the following is not associated with paraneoplastic neuropathies?





    C) Prostate cancer
  180. Site of entrapment: lancinate ligament





    A) Tibial nerve

    Nerve Site of Entrapment
    Cervical/thoracic neurovascular bundle Scalene muscles
    Thoracic outlet syndrome Accessory rib, pancoast tumor
    Median nerve Flexor retinaculum (carpal tunnel)
    Ulnar nerve Elbow (ulnar sulcus, heads of FCU, cubital tunnel), Guyon's canal, hypothenar eminence
    Radial nerve Circumflex portion at midhumerus; proximal to supinator margin, arcade of Frohse
    Lateral femoral cutaneous nerve Along pelvis, exiting pelvis, iliac crest, and ASIS
    Peroneal nerve Fibular head
    Tibial nerve Lancinate ligament (tarsal tunnel)
  181. Site of entrapment: pancoast tumor





    C) Thoracic outlet syndrome

    Nerve Site of Entrapment
    Cervical/thoracic neuromuscular bundle Scalene muscles
    Thoracic outlet syndrome Accessory rib, pancoast tumor
    Median nerve Flexor retinaculum (carpal tunnel)
    Ulnar nerve Elbow (ulnar sulcus, heads of FCU, cubital tunnel), Guyon's canal, hypothenar eminence
    Radial nerve Circumflex portion at midhumerus; proximal to supinator margin, arcade of Frohse
    Lateral femoral cutaneous nerve Along pelvis, exiting pelvis, iliac crest, and ASIS
    Peroneal nerve Fibular head
    Tibial nerve Lancinate ligament (tarsal tunnel)
  182. Electrodiagnostic Findings After Nerve Injury: increased insertional activity; increased activity at rest; (+) fibrillation potentials; (+) positive sharp waves; large, long duration polyphasic




    D) Both
  183. Electrodiagnostic Findings After Nerve Injury: normal insertional activity; silent activity at rest; (-) fibrillation potentials; (-) positive sharp wave




    B) Both

    The difference is that in normal, the shape of the waveform is biphasic or triphasic.
  184. Electrodiagnostic Findings After Nerve Injury: increased insertional activity, except:





    D) Neurapraxia

    There is normal insertional activity in neurapraxia.
  185. Electrodiagnostic Findings After Nerve Injury: silent activity at rest, except:





    C) Axonal neuropathy

    In axonal neuropathy, there is increased activity at rest.
  186. Electrodiagnostic Findings After Nerve Injury: no interference pattern, except:




    D) Axonal neuropathy

    Axonal neuropathy has incomplete interference pattern.
  187. Inheritance Patterns and Gene Mutations of Muscular Dystrophies: X-linked, except:





    D) EDMD 2

    The inheritance pattern of EDMD 2 is autosomal dominant.
  188. Inheritance Patterns and Gene Mutations of Muscular Dystrophies: autosomal recessive, except:




    C) Limb girdle muscular dystrophy (type 1s)

    LGMD (type 1s) is autosomal dominant, while LGMD (type 2s) is autosomal recessive. OPMD can either be autosomal dominant or recessive.
  189. Inheritance Patterns and Gene Mutations of Muscular Dystrophies: autosomal dominant, except:





    A) Emery-Dreifuss muscular dystrophy (type 1)

    EDMD (type 1) is X-linked, while EDMD (type 2) is autosomal dominant.
  190. Inheritance Patterns and Gene Mutations of Muscular Dystrophies: gene loci of DMD




    D) xp21

    Muscular Dystrophies Gene Loci
    DMD Xp21
    BMD Xp21
    DM1 19q13.3
    DM2 3q21
    FSHD 4q35
    LGMD (type 1s) Multiple loci
    LGMD (type 2s) Multiple loci
    OPMD 14q11.2-q13
    EDMD 1 Xq28
    EDMD 2 1q21.2
    CMD 5q22
  191. Inheritance Patterns and Gene Mutations of Muscular Dystrophies: gene loci of EDMD 1




    C) Xq28

    Muscular Dystrophies Gene Loci
    DMD Xp21
    BMD Xp21
    DM1 19q13.3
    DM2 3q21
    FSHD 4q35
    LGMD (type 1s) Multiple loci
    LGMD (type 2s) Multiple loci
    OPMD 14q11.2-q13
    EDMD 1 Xq28
    EDMD 2 1q21.2
    CMD 5q22
  192. Inheritance Patterns and Gene Mutations of Muscular Dystrophies: dystrophia myotonia protein kinase (DMPK)





    C) DM1

    Muscular Dystrophies Gene (Mutations)
    DMD Dsytrophin
    BMD Dystrophin
    DM1 DMPK (expansion of CTG repeat)
    DM2 ZNF9 (expansion of CCTG repeat)
    FSHD DUX4 (deletion of D4Z4 repeats)
    LGMD (type 1s) Myotilin, laminin A/C, caveolin-3
    LGMD (type 2s) Calpain-3, dysferlin, sarcoglycans, FKRP
    OPMD PABN1 and PABP2 (expansion of GCG repeat)
    EDMD 1 Emerin
    EDMD 2 Laminin A/C
    CMD Merosin
  193. Inheritance Patterns and Gene Mutations of Muscular Dystrophies: DUX4




    A) FSHD

    Muscular Dystrophies Gene (Mutations)
    DMD Dsytrophin
    BMD Dystrophin
    DM1 DMPK (expansion of CTG repeat)
    DM2 ZNF9 (expansion of CCTG repeat)
    FSHD DUX4 (deletion of D4Z4 repeats)
    LGMD (type 1s) Myotilin, laminin A/C, caveolin-3
    LGMD (type 2s) Calpain-3, dysferlin, sarcoglycans, FKRP
    OPMD PABN1 and PABP2 (expansion of GCG repeat)
    EDMD 1 Emerin
    EDMD 2 Laminin A/C
    CMD Merosin
  194. Which of the following findings is not suggestive of myopathies?





    E) Abnormal nerve conduction studies

    It should be normal nerve conduction studies. 

    • Clinical Features and Laboratory Findings Suggestive of Myopathies:
    •  1.) Proximal symmetric weakness
    •  2.) Normal sensation
    •  3.) Normal or mildly diminished tendon reflexes
    •  4.) Elevation of serum CK
    •  5.) EMG demonstrating brief, low-voltage, polyphasic potentials
    •  6.) Normal nerve conduction studies
    •  7.) Muscle biopsy with muscle fiber necrosis and regeneration with central nuclei
  195. Age of onset: childhood or early adolescence




    D) BMD

    Age of onset in DMD is childhood <age 5.
  196. Pattern of weakness: proximal




    B) Both
  197. Wheelchair dependence: late childhood/early adolescence




    B) DMD

    Wheelchair dependence in BMD occurs during late adolescence or later.
  198. Scoliosis: severe and progressive




    C) DMD

    Scoliosis in BMD is mild.
  199. Cardiac involvement: significant




    C) Both
  200. Pulmonary dysfunction: usually mild




    A) BMD

    Pulmonary dysfunction in DMD is severe and progressive
  201. Cognitive involvement: unusual 




    D) BMD

    Cognitive involvement in DMD is frequent.
  202. Which of the following is not a biological effect of viscosupplements?






    A) Immediate restoration and maintenance of synovial fluid rheologic properties

    Immediate restoration and maintenance of synovial fluid rheologic properties (viscosity and elasticity) and antinociceptive effects are rheological effects of viscosupplements. Cartilage and chondrocyte anabolic effects include increased biosynthesis and proliferation, as well as decreased degradation and apoptosis.
  203. Types of Scoliosis and Typical Ages of Onset: idiopathic





    D) Infancy to adolescence

    Pathophysiological Type of Scoliosis Typical Age of Onset
    Idiopathic Infancy to adolescence
    Neuromuscular Childhood to adolescence
    Bone or ligamentous dysfunction Childhood
    Traumatic/posttraumatic All ages
    Infectious or neoplastic All ages
    Degenerative Middle age to seniors
  204. Types of Scoliosis and Typical Ages of Onset: all ages





    E) All of these

    Pathophysiological Type of Scoliosis Typical Age of Onset
    Idiopathic Infancy to adolescence
    Neuromuscular Childhood to adolescence
    Bone or ligamentous dysfunction Childhood
    Traumatic/posttraumatic All ages
    Infectious or neoplastic All ages
    Degenerative Middle age to seniors
  205. Moe-Nash Classification of Spinal Rotation: pedicles are seen symmetrically positioned at the lateral border of the vertebral bodies





    D) Grade 0
  206. Moe-Nash Classification of Spinal Rotation: slight asymmetry





    C) Grade 1
  207. Moe-Nash Classification of Spinal Rotation: one pedicle is almost out of view





    A) Grade 2
  208. Moe-Nash Classification of Spinal Rotation: one pedicle is seen, positioned at the center of the vertebral body 





    D) Grade 3
  209. Moe-Nash Classification of Spinal Rotation: only one pedicle is seen, positioned lateral to the center of the vertebral body





    B) Grade 4
  210. Types of Idiopathic Scoliosis: age of onset is 10 y-skeletal maturity



    A) Adolescent
  211. Types of Idiopathic Scoliosis: age of onset is <3 y



    C) Infantile
  212. Types of Idiopathic Scoliosis: age of onset is 3-10 y



    B) Juvenile
  213. Types of Idiopathic Scoliosis: 1% of all idiopathic scoliosis



    B) Infantile
  214. Types of Idiopathic Scoliosis: 12%-21% of all idiopathic scoliosis



    C) Juvenile
  215. Types of Idiopathic Scoliosis: 80%-90% of all idiopathic scoliosis



    A) Adolescent
  216. Types of Idiopathic Scoliosis: male:female ratio is 1:5



    C) Adolescent
  217. Types of Idiopathic Scoliosis: male:female ratio is equal until 6 y then 1:8



    A) Juvenile
  218. Types of Idiopathic Scoliosis: male:female ratio is male > female



    B) Infantile
  219. Types of Idiopathic Scoliosis: common curve pattern is T or T-L



    C) Infantile
  220. Types of Idiopathic Scoliosis: common curve pattern is T, double major, T-L




    B) Both
  221. Types of Idiopathic Scoliosis: usual side of curve vertex is L



    A) Infantile
  222. Types of Idiopathic Scoliosis: usual side of curve vertex is R




    B) Juvenile

    The usual side of curve vertex in infantile scoliosis is L, while the usual side of curve vertex in juvenile and adolescent scolioses is R.
  223. Types of Idiopathic Scoliosis: may spontaneously resolve



    C) Infantile
  224. Types of Idiopathic Scoliosis: usually will not spontaneously resolve



    B) Juvenile
  225. Types of Idiopathic Scoliosis: dependent on age of onset and size of curve 



    B) Adolescent
  226. Role of Orthotic Management in Spinal Deformity of Different Etiologies: role of orthotic management is to correct and prevent progression





    C) Idiopathic
  227. Role of Orthotic Management in Spinal Deformity of Different Etiologies: role of orthotic management is limited since it cannot stop progression





    D) Myopathic/lower motor neuron
  228. Role of Orthotic Management in Spinal Deformity of Different Etiologies: role of orthotic management is for comfort and support but will not likely affect curve





    D) Degenerative
  229. Role of Orthotic Management in Spinal Deformity of Different Etiologies: role of orthotic management is to slow or stop progression in long flexible curves





    A) Congenital bony dysfunction
  230. Role of Orthotic Management in Spinal Deformity of Different Etiologies: role of orthotic management is to slow progression in seated patients





    C) Upper motor neuron
  231. Role of Orthotic Management in Spinal Deformity of Different Etiologies: rigid orthosis is used in the following types of scoliosis, except:





    B) Ligamentous dysfunction

    Soft orthosis is used in scoliosis secondary to ligamentous dysfunction.
  232. Role of Orthotic Management in Spinal Deformity of Different Etiologies: rigid or semirigid orthosis





    D) Degenerative
  233. Ligament Sprains: minimal pain and swelling



    B) First degree
  234. Ligament Sprains: no ligamentous instability



    B) First degree
  235. Ligament Sprains: minimal localized tenderness



    A) First degree
  236. Ligament Sprains: significant pain and swelling



    B) Second degree
  237. Ligament Sprains: subtle structural instability



    C) Second degree
  238. Ligament Sprains: occasional joint effusion



    B) Second degree
  239. Ligament Sprains: marked swelling and hemoarthrosis



    B) Third degree
  240. Ligament Sprains: structural instability



    A) Third degree
  241. Ligament Sprains: no significant loss of function



    A) First degree
  242. Ligament Sprains: bracing often unnecessary



    B) First degree
  243. Ligament Sprains: rapid return to activity



    C) First degree
  244. Ligament Sprains: protective bracing indicated



    A) Second degree
  245. Ligament Sprains: significant activity limitations



    B) Second degree
  246. Ligament Sprains: susceptible to recurrence



    C) Second degree
  247. Ligament Sprains: immobilization is indicated



    C) Third degree
  248. Ligament Sprains: surgery may be required



    A) Third degree
  249. Ligament Sprains: prolonged functional limitation



    C) Third degree
  250. Classification of Tendon Injuries: inflammation of paratenon with associated pain, swelling, and tenderness




    C) Tenosynovitis

    Tenosynovitis is also known as paratenonitis.
  251. Classification of Tendon Injuries: inflammation of tendon with associated vascular disruption and inflammation




    D) Tendinitis
  252. Classification of Tendon Injuries: intratendinous atrophy and degeneration with a relative absence of inflammation; a palpable nodule may be present over a tendon




    C) Tendinosis
  253. Classification of Tendon Injuries: acute inflammation superimposed on chronic tendinitis





    B) Paratenonitis with tendinosis
  254. Classification of Tendon Injuries: acute inflammation superimposed on chronic inflammation with tendinosis





    E) Partial or complete rupture
  255. Which of the following is not a goal in rehabilitation of soft tissue injuries?





    D) None of these

    • Goals of Rehabilitation of Soft Tissue Injuries:
    •  1.) Prescribe activity-specific training
    •  2.) Correct functional deficiencies
    •  3.) Improve strength, flexibility, and general conditioning
    •  4.) Address structural deficiencies
    •  5.) Promote healing
    •  6.) Control pain and inflammation
    •  7.) Establish correct diagnosis
  256. Tendon Injuries (Functional Scale): pain after exercise, subsiding within 24h





    A) Grade 1
  257. Tendon Injuries (Functional Scale): minimal discomfort during exercise; without activity limitation





    D) Grade 2
  258. Tendon Injuries (Functional Scale): pain interferes with exercise





    A) Grade 3
  259. Tendon Injuries (Functional Scale): pain interferes with activities of daily living





    C) Grade 4
  260. Tendon Injuries (Functional Scale): rest pain interfering with sleep





    A) Grade 5
  261. All of the following are intrinsic variables in overuse injuries, except:





    E) Training errors/poor. technique

    • Intrinsic variables:
    •  1.) Age
    •  2.) Flexibility imbalance
    •  3.) Muscle imbalance/weakness
    •  4.) Anatomic malalignment
    •  5) Genetic predisposition

    • Extrinsic variables:
    •  1.) Training error/poor technique
    •  2.) Environmental factors
    •  3.) Equipment factors
  262. Tests Used in the Diagnosis of Shoulder pain: inferior translation force at the humerus with retraction of soft tissue above the humeral head




    D) Sulcus sign
  263. Tests Used in the Diagnosis of Shoulder pain: laxity in inferior capsule




    C) Sulcus sign
  264. Not true about apprehension and relocation test





    E) None of these
  265. Tests Used in the Diagnosis of Shoulder pain: elbow is flexed and forearm supinated




    B) Yergason's test

    If Yergason's test is positive, it is indicative of bicipital tendonitis.
  266. Tests Used in the Diagnosis of Shoulder pain: arm is flexed with elbow straight and supinated; resistance is applied to arm flexors




    A) Speed's

    If Speed's test is positive, it is indicative of bicipital tendonitis.
  267. Tests Used in the Diagnosis of Shoulder pain: arm is abducted to 90 degrees and internally rotated, causing pain





    B) None of these

    This is Hawkin's test and if it is positive, it is indicative of impingement of supraspinatus tendon.
  268. Tests Used in the Diagnosis of Shoulder pain: arm is flexed to 90 degrees and internally rotated, patient resists downward force





    E) Neer's
  269. Tests Used in the Diagnosis of Shoulder pain: pain noted on abduction from 60 to 120 degrees




    B) Painful arc

    If painful arc is positive, it is indicative of supraspinatus tendonitis.
  270. Tests Used in the Diagnosis of Shoulder pain: arm placed in full abduction and patient is asked to lower arm slowly; arm drops from midabduction





    E) Drop arm 

    If drop arm test is positive, it is indicative of supraspinatus full-thickness tear.
  271. Tests Used in the Diagnosis of Shoulder pain: translation of humerus over the edge of labrum





    E) Clunk

    If clunk test is positive, it is indicative of glenohumeral instability or possibly labral tear.
  272. Tests Used in the Diagnosis of Shoulder pain: pulse is palpated, and a drop in the pulse may be noted during inhalation, coupled with rotation of the head activating the scalene muscles




    C) Adson's

    If Adson's test is positive, it is indicative of thoracic outlet syndrome.
  273. Tests Used in the Diagnosis of Shoulder pain: hands repetitively opened and closed with arms abducted and elbows bent increasing vascular demand




    C) Roos'

    If Roos' test is positive, it is indicative of thoracic outlet syndrome.
  274. Differential Diagnosis of Lateral Elbow Pain: articular, except:





    B) Annular and lateral ligament sprains

    Annular and lateral ligament sprains affect soft tissues.

    • Articular:
    •  1.) Radiohumeral joint disease
    •  2.) Radioulnar joint disease
    •  3.) Ectopic calcification
    •  4.) Periostitis
    •  5.) Panner's osteochondrosis
    •  6.) Fibrillation of the radial head
  275. Differential Diagnosis of Lateral Elbow Pain: soft tissue, except:




    D) Rupture of common flexor origin

    It should be rupture of common extensor origin.
  276. Differential Diagnosis of Lateral Elbow Pain: soft tissue, except:




    B) Anterior interosseous nerve compression

    It should be posterior interosseous nerve compression.
  277. Special Diagnostics Tests Associated with CTD: pain with full passive shoulder flexion




    C) Neer's
  278. Special Diagnostics Tests Associated with CTD: tapping of ulnar nerve at cubital tunnel in flexed elbow




    D) Tinel's
  279. Special Diagnostics Tests Associated with CTD: internal rotation and abduction of shoulder with elbow flexed at 90 degrees




    D) Hawkin's
  280. Special Diagnostics Tests Associated with CTD: resistance to shoulder extension with the subject's elbow fully extended and the fist directed such that the thumb is pointing toward the ground




    C) Empty can
  281. Special Diagnostics Tests Associated with CTD: shoulder flexion with the arm in supinated position and the elbow flexed to 15 degrees




    C) Speed's
  282. Special Diagnostics Tests Associated with CTD: passive ulnar deviation of the hand with the thumb fully flexed in a closed fist




    C) Finkelstein's
  283. Special Diagnostics Tests Associated with CTD: resistance of wrist extension and radial deviation with the subject's arm in pronated position




    A) Cozen's
  284. EMG findings in carpal tunnel syndrome, except:




    A) Prolonged distal latency of median motor, possible sensory

    It should be prolonged distal latency of median sensory, possible motor.
  285. Not a cause of carpal tunnel syndrome




    C) Indirect trauma

    It should be direct trauma.
  286. Diagnosis and Treatment of Hand Disorders: locking of digits in flexion, snapping, popping, tender nodules, and crepitus




    C) Trigger finger
  287. Diagnosis and Treatment of Hand Disorders: radial wrist pain, positive Finkelstein's




    D) de Quervain's
  288. Diagnosis and Treatment of Hand Disorders: superficial palmar nodules; painless flexion contractures




    B) Dupuytren's
  289. Diagnosis and Treatment of Hand Disorders: nighttime hand paresthesias, positive Tinel's, carpal tunnel compression, and Phalen's





    A) None these

    These are signs and symptoms of carpal tunnel syndrome.
  290. Diagnosis and Treatment of Hand Disorders: ulnar hand pain and paresthesias, positive Tinel's at elbow, possible interosseous weakness





    E) Cubital tunnel syndrome
  291. Diagnosis and Treatment of Hand Disorders: pain at base of the thumb, positive grind maneuver




    B) Basilar thumb degenerative disease
  292. Not included in the prescription for trigger finger





    B) Splint

    Splint is used in de Quervain's, carpal tunnel syndrome, and basilar thumb degenerative disease.
  293. Not included in the prescription for de Quervain's tenosynovitis





    C) Rest

    Rest is indicated for trigger finger.
  294. Dupuytren's contracture prescription, except:




    D) Cortisone injection

    It should be collagenase injection.
  295. Carpal tunnel syndrome prescription, except:





    A) Rest

    Rest is indicated for trigger finger.
  296. Not a part of prescription for cubital tunnel syndrome





    D) Cortisone injection

    Cortisone injection is indicated for trigger finger, de Quervain's tenosynovitis, and carpal tunnel syndrome.
  297. Basilar thumb degenerative joint disease prescription, except:





    E) Surgical release

    Surgical reconstruction is indicated for basilar thumb degenerative joint disease, while surgical release is indicated for carpal and cubital tunnel syndromes, as well as Dupuytren's contracture.
  298. Diagnosis and Treatment of Hand Disorders: cortisone injection, except:





    E) Cubital tunne syndrome
  299. Diagnosis and Treatment of Hand Disorders: activity modification, except:




    D) de Quervain's tenosynovitis
  300. Lisfranc's Dislocation Classification: all metatarsals are displaced in the same direction




    A) Homolateral
  301. Lisfranc's Dislocation Classification: metatarsals are displaced in multiple planes and differing directions




    B) Divergent
  302. Lisfranc's Dislocation Classification: one or more metatarsals displaced and separated




    B) Isolated
  303. Lisfranc's Dislocation Classification: total incongruity of entire Lisfranc's complex in sagittal or transverse planes





    A) Type A total
  304. Lisfranc's Dislocation Classification: medial displacement of 1st metatarsal in isolation or in combination with one or more of the adjacent metatarsals





    B) Type B partial (medial)
  305. Lisfranc's Dislocation Classification: lateral displacement of one or more of the lateral metatarsals with the 1st metatarsal unaffected 





    A) Type B partial (lateral)
  306. Lisfranc's Dislocation Classification: 1st metatarsal displaced medially, with one or more of the lateral 4 metatarsals displaced laterally in any number of planes





    E) Type C divergent (partial)
  307. Lisfranc's Dislocation Classification: 1st metatarsal displaced medially, with all lateral 4 metatarsals displaced laterally in any number of planes





    C) Type C divergent (total)
  308. Midfoot Sprain Classification: pain at Lisfranc's ligament complex, no displacement on weight-bearing anteroposterior x-ray plus bone




    A) Stage I
  309. Midfoot Sprain Classification: 1st and 2nd metatarsal bone diastasis of 1-5 mm on weight-bearing anteroposterior x-ray; no evidence of loss of arch height on lateral x-ray




    C) Stage II
  310. Midfoot Sprain Classification: 1st and 2nd metatarsal bone diastasic >5 mm on weight-bearing anteroposterior x-ray; loss of arch height on lateral x-ray




    A) Stage III
  311. Ulcer Classification: intact skin may have bony deformity or limited skin mobility






    E) Grade 0
  312. Ulcer Classification: localized superficial ulcer of the skin






    C) Grade 1
  313. Ulcer Classification: deep ulcer extending to bone, tendon, ligament, or joint






    E) Grade 2
  314. Ulcer Classification: deep ulcer with abscess or osteomyelitis






    B) Grade 3
  315. Ulcer Classification: forefoot gangrene






    C) Grade 4
  316. Ulcer Classification: gangrene of the entire foot






    A) Grade 5
  317. Defining Bone Loss by BMD: BMD is within 1 SD of a “young normal” adult (T-score ≥ −1)




    C) Normal
  318. Defining Bone Loss by BMD: BMD is within 1  and 2.5 SDs below that of a "young normal" adult (T-score between -1.0 and -2.5)




    A) Osteopenia
  319. Defining Bone Loss by BMD: BMD is 2.5 SDs or more below that of a "young normal" adult (T-score ≤ 2.5)




    C) Osteoporosis
  320. Which of the following events has the greatest loss of quality of life years in osteoporosis (hip fracture)?





    B) Nonmedical home care (6 mo)
  321. Which of the following events has the greatest loss of quality of life years in osteoporosis?




    C) Hip fracture

    Meanwhile, vertebral fracture has the least loss of QALYs.
  322. All of the following medications can cause secondary osteoporosis, except:






    B) Antipsychotics
  323. Which of the following malignancies is not associated with secondary osteoporosis?




    C) Lung cancer
  324. Which of the following is not a risk factor for osteoporotic fractures?






    D) None of these

    • Other risk factors for osteoporotic fractures are:
    •  1.) Personal history of low-impact fracture
    •  2.) Current low BMD
    •  3.) Caucasian race
    •  4.) Advanced age
    •  5.) Female sex
    •  6.) Recurrent falls
    •  7.) Inadequate physical activity
    •  8.) Current smoker
    •  9.) Low body weight
    •  10.) Estrogen deficiency
    •  11.) Corticosteroid use
    •  12.) Vitamin D deficiency
    •  13.) Low lifetime calcium intake
    •  14.) Impaired eyesight despite correction
  325. Laboratory Tests in Disorders of Calcium Metabolism: increased serum calcium, except:




    D) Renal osteodystrophy

    Renal osteodystrophy, vitamin D deficiency, and primary hypoparathyroidism have decreased serum calcium.
  326. Laboratory Tests in Disorders of Calcium Metabolism: increased PTH, except:





    C) Hypercalcemia of malignancy

    Hypercalcemia of malignancy and primary hypoparathyroidism have decreased PTH.
  327. Laboratory Tests in Disorders of Calcium Metabolism: normal vitamin D, except:




    D) Primary hyperparathyroidism

    Primary hyperparathyroidism has increased vitamin D, while renal osteodystrophy and primary hypoparathyroidism have decreased vitamin D.
  328. Laboratory Tests in Disorders of Calcium Metabolism: increased urine calcium





    E) Hypercalcemia of malignancy

    Primary hyperparathyroidism has normal urine calcium, while the other choices have decreased calcium urine.
  329. Laboratory Tests in Disorders of Calcium Metabolism: decreased renal function





    A) None of these

    Renal osteodystrophy has decreased renal function.
  330. Recommended Daily Calcium Intake: 0-6 months




    C) 210 mg/d
  331. Recommended Daily Calcium Intake: 7-12 months




    B) 270 mg/d
  332. Recommended Daily Calcium Intake: 1-3 years




    C) 500 mg/d
  333. Recommended Daily Calcium Intake: 4-8 years




    B) 800 mg/d
  334. Recommended Daily Calcium Intake: 9-18 years




    C) 1300 mg/d
  335. Recommended Daily Calcium Intake: 19-50 years




    C) 1000 mg/d
  336. Recommended Daily Calcium Intake: >50 years




    B) 1200 mg/d
  337. Recommended Daily Calcium Intake: pregnant or lactating women ≤18 years




    B) 1300 mg/d
  338. Recommended Daily Calcium Intake: pregnant or lactating women 19-50 years




    A) 1000 mg/d
  339. Which of the following food has the highest amount of calcium?





    B) Plain yogurt, nonfat

    Vanilla ice cream has the least amount of calcium. 

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  340. Which of the following biphosphonates decreases spine and hip fractures?




    B) Alendronate
  341. Which of the following biphosphonates does not decrease spine, non spine, and hip fractures




    B) Ibandronate

    Ibandronate only decreases spine fractures.
  342. Which of the following biphosphonates is not used for treatment of glucocorticoid-induced osteoporosis?





    E) Ibandronate

    Ibandronate is used for prevention and treatment of postmenopausal osteoporosis.
  343. Which of the following biphosphonates cannot be used for treatment of men with osteoporosis? 





    D) Ibandronate
  344. Which of the following biphosphonates is used for post-hip fragility fracture?





    C) Zoledronic acid
  345. Which of the following biphosphonates cannot be used to prevent postmenopausal osteoporosis?





    C) Zoledronic acid
  346. Nutritional and Pharmacologic Treatments for Osteoporosis: decreased bone resorption, except:





    B) Teriparatide

    Teriparatide increases bone mineralization.
  347. Nutritional and Pharmacologic Treatments for Osteoporosis: side effects are esophageal irritation, jaw osteonecrosis, and atypical femoral fractures





    A) Biphosphonates
  348. Nutritional and Pharmacologic Treatments for Osteoporosis: side effects are possible increased risk of cancer, high blood pressure, deep vein thrombosis, stroke, heart disease, and thromboembolic disease





    A) Estrogen with or without progesterone
  349. Nutritional and Pharmacologic Treatments for Osteoporosis: side effects are hot flashes, leg cramps, and deep vein thrombosis





    B) Estrogen agonist/antagonist
  350. Nutritional and Pharmacologic Treatments for Osteoporosis: side effects are musculoskeletal pain, elevated cholesterol, and cystitis pancreatitis. 





    D) RANKL inhibition
  351. Nutritional and Pharmacologic Treatments for Osteoporosis: side effect is nasal irritation





    D) Calcitonin
  352. Synovial Fluid Analysis: pale yellow, transparent, high viscosity, and good mucin clot





    A) Normal
  353. Synovial Fluid Analysis: <25% cells/mm3





    E) Normal
  354. Synovial Fluid Analysis: <10% polymorphonuclear leukocytes





    E) Normal
  355. Synovial Fluid Analysis: straw or yellow, transparent, high viscosity, and good mucin clot





    B) Noninflammatory
  356. Synovial Fluid Analysis: <2000 cells/mm3





    B) Noninflammatory
  357. Synovial Fluid Analysis: <25% polymorphonuclear leukocytes





    C) Noninflammatory
  358. Synovial Fluid Analysis: straw or yellow; transparent to opaque slightly cloudy, variably decreased viscosity, and fair to poor mucin clot





    C) Moderately inflammatory
  359. Synovial Fluid Analysis: 3,000-5,000 cells/mm3





    C) Moderately inflammatory
  360. Synovial Fluid Analysis: >70% polymorphonuclear leukocytes





    A) Moderately inflammatory
  361. Synovial Fluid Analysis: yellow-gray, purulent; opaque, cloudy; low viscosity, and poor mucin clot





    D) Highly inflammatory, septic
  362. Synovial Fluid Analysis: 50,000-100,000 cells/mm3 (usually 100,000 or more)





    B) Highly inflammatory, septic
  363. Synovial Fluid Analysis: >75%, usually close to 100% polymorphonuclear leukocytes





    E) Highly inflammatory, septic
  364. Synovial Fluid Analysis: red, opaque, high viscosity, and good mucin clot

    A) Noninflammatory
    B) Moderately inflammatory
    C) Highly inflammatory, septic
    D) Normal
    E) Hemorrhagic
    E) Hemorrhagic
  365. Synovial Fluid Analysis: cells/mm3 is up to normal count in blood





    C) Hemorrhagic
  366. Synovial Fluid Analysis: good mucin clot, except:





    D) None of these
    B) Highly inflammatory, septic

    Moderately inflammatory group has fair to poor mucin clot.
  367. Synovial Fluid Analysis: high viscosity, except:





    D) None of these
    D) Highly inflammatory, septic

    Highly inflammatory, septic group has low viscosity.
  368. All of the following rheumatic diseases are symmetric, except:





    E) AS

    Disease Anatomic Distribution
    Rheumatoid arthritis Symmetric
    Spondyloathropathies (e.g. ankylosing spondylitis, Reiter's syndrome, and psoriatic arthritis) Asymmetric
    Septic Asymmetric
    Gout Asymmetric
    Pseudogout Symmetric
    Systemic Lupus Erythematosus Symmetric  
    Progressive Systemic Sclerosis Symmetric
  369. All of the following joints are most frequently affected in RA, except:





    D) Ankle

    Ankle, knee, hip, shoulder, and cervical spine are often affected in RA.
  370. All of the following joints are most frequently affected in AS, except:




    A) Cervical spine
  371. All of the following joints are most frequently affected in Reiter's, except:




    D) Knee
  372. Radiographic Findings in RDs: juxtaarticular osteoporosis, fusiform soft-tissue swelling, marginal erosion, bony cysts





    B) RA
  373. Radiographic Findings in RDs: subluxation (swan-neck/Boutonniere/ulnar deviation)





    B) RA
  374. Radiographic Findings in RDs: late: bony eburnation, compressive erosions, surface resorption





    E) RA
  375. Radiographic Findings in RDs: sausage fingers, except:




    B) AS
  376. Radiographic changes seen in spondyloarthropathies, except:





    A) Soft-tissue speckled calcification

    Soft-tissue speckled calcification is seen in gout.
  377. Radiographic changes seen in septic arthritis, except:




    D) Subchondral cysts

    Subchondrl cysts are seen in pseudogout.
  378. Radiographic Findings in RDs: soft-tissue speckled calcification, gouty tophi, erosion of bone with marginal overhangs





    A) Gout
  379. Radiographic Findings in RDs: chondrocalcinosis and subchondral cysts





    D) Pseudogout
  380. Radiographic Findings in RDs: calcific deposits, subchondral lucency, subchondral sclerosis, subchondral collapse and remodeling of bone/tuft resorption





    E) SLE
  381. Radiographic Findings in RDs: acroosteolysis, soft tissue calcification, sausage digits





    A) PSS
  382. Which of the following is not a seronegative spondyloarthropathy?





    B) None of these
  383. Which of the following is not an inflammatory connective-tissue disease?





    A) None of these

    Other examples of inflammatory connective-tissue diseases are JIA, PM-DM. and MCTD.
  384. Which of the following is not a noninflammatory arthritis?





    C) None of these

    • Noninflammatory arthritis may be classified as: 
    •  1.) Degenerative, posttraumatic, or overuse (e.g. osteoarthritis, posttraumatic. aseptic necrosis)
    •  2.) Inherited or metabolic (e.g. lipid storage disease, hemochromatosis, ochronosis, hypogammaglobulinemia, hemoglobinopathies)
  385. Which of the following rheumatic diseases does not affect the skin?





    D) Rheumatoid arthritis

    • The following rheumatic diseases affect the disease: 
    • 1.) Juvenile idiopathic arthritis
    •  2.) Reiter's syndrome
    • 3.) Psoriatic arthritis
    •  4.) Colic arthritis
    •  5.) Sarcoid arthritis
    •  6.) Septic arthritis
    •  7.) Hyperlipoproteinemia
    •  8.) Systemic lupus erythematosus
    •  9.) Amyloidosis
    •  10.) Dermatomyositis
  386. Which of the following rheumatic diseases does not affect the nasopharynx and ear?




    B) Psoriatic arthritis
  387. Which of the following rheumatic diseases does not affect the eyes?





    D) None of these
  388. Which of the following rheumatic diseases does not affect the GI tract?




    A) Systemic lupus erythematosus
  389. Which of the following diseases does not affect the heart and circulation?





    C) Dermatomyositis

    It should be polymyositis.
  390. Which of the following rheumatic diseases does not affect the respiratory tract?




    A) Scleroderma
  391. Which of the following diseases affect/s the nervous and hematologic systems?




    D) Both
  392. Which of the following rheumatic diseases does not affect the renal system?





    C) Pseudogout
  393. Epidemiology of Inflammatory Arthritis: higher in whites and Native Americans (Pima Indians)




    D) RA
  394. Epidemiology of Inflammatory Arthritis: lower in African Americans, Asian, and Japanese




    A) JIA
  395. Epidemiology of Inflammatory Arthritis: three times higher in blacks, Chinese; increased in Haida, North American Indian




    A) SLE
  396. Epidemiology of Inflammatory Arthritis: higher in Central European




    B) AS
  397. Epidemiology of Inflammatory Arthritis: three times higher in black females




    C) PM
  398. Epidemiology of Inflammatory Arthritis: increased in Southern US higher among African-Americans




    C) PSS
  399. Epidemiology of Inflammatory Arthritis: African-Americans, Asian-Pacific Islanders
     



    B) Gout
  400. Epidemiology of Inflammatory Arthritis: 10:1 male 




    C) Gout
  401. Epidemiology of Inflammatory Arthritis: 4:1 female




    D) PSS
  402. Epidemiology of Inflammatory Arthritis: 2.5:1 female




    B) Both
  403. Epidemiology of Inflammatory Arthritis: 2:1 female





    A) JIA
  404. Epidemiology of Inflammatory Arthritis: 3:1 male




    C) AS
  405. Epidemiology of Inflammatory Arthritis: 9:1 female




    A) SLE
  406. Most common onset of RA



    A) Insidious over weeks to months
  407. Type of RA which meets ACR criteria on presentation, but does not meet the criteria 2-5 years later



    B) Self-limited polyarthritis
  408. Type of RA which is managed by conservative treatment and has few significant long-term consequences



    C) Persistent disease
  409. Type of RA which has functional decline and premature morbidity



    B) Progressive disease with radiologic damage
  410. Stage/s of RA/OA in which full ROM is indicated





    A) All of these
  411. Stage/s of RA/OA in which partial ROM is indicated





    D) None of these
  412. Stage/s of RA/OA in which gentle stretch is indicated





    D) Subacute
  413. Stage/s of RA/OA in which full stretch is indicated





    A) Chronic (inactive)
  414. Stage/s of RA/OA in which submaximal isometrics is indicated





    E) Subacute
  415. Stage/s of RA/OA in which maximal isometrics is indicated





    E) Chronic (inactive)
  416. Stage/s of RA/OA in which low resistance isotonic exercises are indicated





    D) Subacute
  417. Stage/s of RA/OA in which moderate resistance isotonic exercises are indicated





    C) Chronic (inactive)
  418. Stage/s of RA/OA in which low resistance isokinetic exercises are indicated





    C) Chronic (inactive)
  419. Stage/s of RA/OA in which moderate resistance isokinetic exercises are indicated





    D) Chronic (inactive)
  420. Stage/s of RA/OA in which low intensity aerobics is indicated





    D) Subacute and chronic (inactive)
  421. Stage/s of RA/OA in which moderate-intensity aerobics is indicated





    D) Chronic (inactive)
  422. Functional level/s in which partial ROM is indicated




    C) Both

    Full ROM is indicated for functional levels I, II, and III.
  423. Functional level/s in which maximal isometrics is indicated




    D) Neither

    Maximal isometrics is indicated for functional levels I and II, while sub maximal isometrics is indicated for all functional levels.
  424. Functional level/s in which isotonic exercises are indicated




    B) Both

    Low- and moderate resistance exercises are indicated for functional levels I and II.
  425. Functional level/s in which isokinetic exercises are indicated




    C) II

    Low- and moderate-resistance isokinetic exercises are indicated for functional levels I and II.
  426. Functional level/s in which moderate intensity aerobics is indicated




    C) II

    Moderate-intensity aerobics is indicated in functional levels I and II, while low-intensity aerobics is indicated in functional levels I, II, and III.
  427. Functional level in which gentle stretch is not indicated




    A) II

    Meanwhile, full stretch is not indicated in functional levels I and IV.
  428. Which of the following is not indicated in patients with joint replacement?





    B) Low-intensity isokinetic exercises

    It should be moderate-resistance isokinetic exercises.
  429. Which of the following is not a branch of the right coronary artery?




    D) Circumflex artery

    Circumflex artery and anterior intraventricular (descending) branch come from left coronary artery.
  430. Coronary arteries: supplies the right atrium and SA node





    D) Nodal branch
  431. Coronary arteries: supplies the right ventricle to apex





    D) Right marginal branch
  432. Coronary arteries: supplies the AV node, posterior third of septum, and right bundle of His





    B) Posterior interventricular (descending) branch
  433. Coronary arteries: supplies the anterior left and right ventricles, anterior two-thirds of septum, left bundle of His, and AV node





    D) Anterior interventricular (descending) branch
  434. Coronary arteries: supplies the left atrium and superior portions of left ventricle





    C) Circumflex artery
  435. All of the following may result from occlusion of left anterior descending artery, except:





    E) Posterior wall thrombus

    It should be anterior wall thrombus.
  436. Occlusion to which of the following coronary arteries will lead to apical thrombus?




    D) Left circumflex artery
  437. Occlusion to which of the following coronary arteries will lead to massive congestive heart failure?




    A) Left main coronary artery
  438. Occlusion to the right coronary artery may result to the following, except:




    B) Conduction block

    Conduction block results from occlusion to left anterior descending and left main coronary arteries.
  439. Occlusion to the left main coronary artery may result to the following, except:





    E) None of these
  440. Occlusion to which of the following coronary arteries will lead to papillary muscle necrosis?




    D) Left anterior descending artery
  441. Occlusion to this coronary artery will result in infarction of anterior wall and septum




    B) Left anterior descending artery
  442. Occlusion to this coronary artery will result in infarction of apex and lateral wall




    B) Left circumflex artery
  443. Occlusion to this coronary artery will result in infarction of anterior and lateral walls, as well as apex




    B) Left main coronary artery
  444. Occlusion to this coronary artery will result in infarction of inferior wall and right ventricle




    D) Right coronary artery
  445. METS: sitting at rest
    1 MET
  446. METS: dressing
    2-3 METS
  447. METS: eating
    1-2 METS
  448. METS: hygiene (sitting)
    1-2 METS
  449. METS: hygiene (standing)
    2-3 METS
  450. METS: sexual intercourse
    3-5 METS
  451. METS: showering
    4-5 METS
  452. METS: tub bathing
    2-3 METS
  453. METS: walking at 1 mph
    1-2 METS
  454. METS: walking at 2 mph
    2-3 METS
  455. METS: walking at 3 mph
    3-3.5 METS
  456. METS: walking at 3.5 mph
    3.5-4 METS
  457. METS: walking at 4 mph
    5-6 METS
  458. METS: climbing up stairs
    4-7 METS
  459. METS: bed making
    2-6 METS
  460. METS: carrying 18 lbs upstairs
    7-8 METS
  461. METS: carrying suitcase
    6-7 METS
  462. METS: housework (general)
    3-4 METS
  463. METS: mowing lawn (push power mower)
    3-5 METS
  464. METS: ironing
    2-4 METS
  465. METS: snow shovelling
    6-7 METS
  466. METS: backpacking (45 lbs)
    6-11 METS
  467. METS: baseball (competitive)
    5-6 METS
  468. METS: baseball (noncompetitive)
    4-5 METS
  469. METS: basketball (competitive)
    7-12 METS
  470. METS: basketball (noncompetitive)
    3-9 METS
  471. METS: card playing
    1-2 METS
  472. METS: cycling at 5 mph
    2-3 METS
  473. METS: cycling at 8 mph
    4-5 METS
  474. METS: cycling at 10 mph
    5-6 METS
  475. METS: cycling at 12 mph
    7-8 METS
  476. METS: cycling at 13 mph
    8-9 METS
  477. METS: karate
    8-12 METS
  478. METS: running at 12 min/mile
    8-9 METS
  479. METS: running at 11 min/mile
    9-10 METS
  480. METS: running at 9 min/mile
    10-11 METS
  481. METS: skiing cross country at 3 mph
    6-7 METS
  482. METS: skiing cross country at 5 mph
    9-10 METS
  483. METS: skiing downhill
    5-9 METS
  484. METS: skiing water
    5-7 METS
  485. METS: swimming (backstroke)
    7-8 METS
  486. METS: swimming (breaststroke)
    8-9 METS
  487. METS: swimming (crawl)
    9-10 METS
  488. METS: television
    1-2 METS
  489. METS: tennis (singles)
    4-9 METS
  490. METS: assembly line work
    3-5 METS
  491. METS: carpentry (light)
    4-5 METS
  492. METS: carry 20-44 lb
    4-5 METS
  493. METS: carry 45-64 lb
    5-6 METS
  494. METS: carry 65-85 lb
    7-8 METS
  495. METS: chopping wood
    7-8 METS
  496. METS: desk work
    1.5-2 METS
  497. METS: digging ditches
    7-8 METS
  498. METS: handyman
    5-6 METS
  499. METS: janitorial (light)
    2-3 METS
  500. METS: lift 100 lb
    7-10 METS
  501. METS: painting
    4-5 METS
  502. METS: sawing hardwood
    6-8 METS
  503. METS: sawing softwood
    5-6 METS
  504. METS: sawing (power)
    3-4 METS
  505. METS: shoveling 10 lb, 10/min
    6-7 METS
  506. METS: shoveling 14 lb, 10/min
    7-9 METS
  507. METS: shoveling 16 lb, 10/min
    9-12 METS
  508. METS: tools (heavy)
    5-6 METS
  509. METS: typing
    1.5-2 METS
  510. METS: wood splitting
    6-7 METS
  511. Wenger Protocol: PROM, ankle pumps, introduction to program, self-feeding




    D) Step 1
  512. Wenger Protocol: PROM, ankle pumps, introduction to program, self-feeding, dangle at side of bed




    A) Step 2
  513. Wenger Protocol: AAROM, sitting upright in chair, light recreation, use of bedside commode




    B) Step 3
  514. Wenger Protocol: light activities with moderate resistance, unlimited sitting, seated ADLs




    C) Step 5
  515. Wenger Protocol: increased resistance, walking to bathroom, standing ADL, up to 1h long group meeting




    A) Step 6
  516. Wenger Protocol: walking to 100 ft, standing, warm-up exercises




    A) Step 7
  517. Wenger Protocol: increased walking, walk down stairs (not up) , continued education




    A) Step 8
  518. Wenger Protocol: increased exercise program, review energy conservation, and pacing techniques




    C) Step 9
  519. Wenger Protocol: increased exercises with light weights and ambulation, begin education on home exercise program




    B) Step 10
  520. Wenger Protocol: increased duration of activities




    A) Step 11
  521. Wenger Protocol: walk down two flights of stairs, continue to increase resistance in exercises




    D) Step 12
  522. Wenger Protocol: continue activities, education, and home exercise program teaching




    A) Step 13
  523. Wenger Protocol: walk up and down two flights of stairs, complete instruction on home exercise program and in energy conservation and pacing techniques




    A) Step 14
  524. Activity Classification: no known heart disease





    B) Class A
  525. Activity Classification: <40 years old





    E) Class A
  526. Activity Classification: no symptoms of heart disease





    A) Class A
  527. Activity Classification: normal ETT





    D) Class A
  528. Activity Classification: healthy individuals, not needing cardiac rehabilitation program





    E) Class A
  529. Activity Classification: known stable heart disease




    C) Both
  530. Activity Classification: low risk from cardiac exercise




    B) Both
  531. Activity Classification: NYHA class 1 or 2




    C) Both
  532. Activity Classification: exercise capacity >6 METS




    C) Both
  533. Activity Classification: no clinical heart failure




    D) Class B

    Classes B and C have no clinical heart failure.
  534. Activity Classification: no ischemia or angina at rest or on ETT at ≤6 METs




    B) Both
  535. Activity Classification: appropriate rise in BP with exercise




    B) Both
  536. Activity Classification: no high-grade ventricular ectopy




    C) Both
  537. Activity Classification: individualized activity guidelines with exercise prescription by qualified personnel or restricted to walking





    C) Class B
  538. Activity Classification: ECG and BP monitoring only during prescriptive exercises, usually 6-12 per session




    A) Both
  539. Activity Classification: medical supervision for prescriptive sessions; nonmedical supervision for other exercise prescriptions




    A) Class B

    Medical supervision for prescriptive sessions and nonmedical supervision for other exercise prescriptions are indicated for class B and C.
  540. Activity Classification: medical supervision for prescriptive sessions; nonmedical supervision for other exercise prescriptions




    C) Class B

    Medical supervision for prescriptive sessions and nonmedical supervision for other exercise prescriptions are indicated for classes B and C.
  541. Activity Classification: unable to understand prescribed activity levels





    D) Class C
  542. Activity Classification: individualized activity guidelines with exercise prescription by qualified personnel and supervised by nonmedical personnel trained in CPR





    C) Class C
  543. Activity Classification: moderate to high risk for complications during exercise





    A) Class D
  544. Activity Classification: ETT abnormalities not directly related to ischemia





    E) Class D
  545. Activity Classification: previous V-Fib or sudden death not related to acute ischemia or cardiac procedure





    D) Class D
  546. Activity Classification: high grade ventricular arrhythmias that are uncontrolled at mild to moderate work intensities





    A) Class D
  547. Activity Classification: left main disease or three vessel disease





    D) Class D
  548. Activity Classification: EF <30%




    D) Class D
  549. Activity Classification: two or more MI





    A) Class D
  550. Activity Classification: NYHA class 3 or greater





    D) Class D
  551. Activity Classification: exercise capacity <6 METS





    E) Class D
  552. Activity Classification: angina with exercise





    D) Class D
  553. Activity Classification: down sloping or horizontal ST depression of more than 4 mm





    C) Class D
  554. Activity Classification: fall in systolic BP with exercise





    D) Class D
  555. Activity Classification: life-threatening medical problems





    A) Class D
  556. Activity Classification: previous episodes of primary cardiac arrest





    B) Class D
  557. Activity Classification: ventricular tachycardia at loads of less than 6 METS





    B) Class D
  558. Activity Classification: ECG and BP monitoring are continuous during rehabilitation sessions until safety is established, usually 6-12 per session





    A) Class D
  559. Activity Classification: medical supervision during all rehabilitation sessions until safety is established





    C) Class D
  560. Activity Classification: unstable disease which restricts activity





    D) Class E
  561. Activity Classification: unstable ischemia





    A) Class E
  562. Activity Classification: uncontrolled heart failure





    B) Class E
  563. Activity Classification: uncontrolled arrhythmia





    A) Class E
  564. Activity Classification: severe symptomatic aortic stenosis





    B) Class E
  565. Activity Classification: other conditions that can be aggravated by exercise





    E) Class E
  566. Activity Classification: no activity is recommended for conditioning purposes





    A) Class E
  567. Self-monitoring guidelines, except:





    B) Wait 1h after meals

    It should be wait 2h after meals.
  568. Which of the following is not a benefit cardiac rehabilitation after bypass surgery?





    B) Decreased ischemic threshold

    It should be increased ischemic threshold. Cardiac rehabilitation also decreases platelet aggregation, increases fibrinolysis, and improves psychological status.
  569. Relative Cardiovascular Response to UE and LE exercise: UE<LE during maximal exercise, except:





    A) Heart rate

    In heart rate and SBP, there are equal increases in maximal UE and LE exercises.

    Image Upload 4
  570. Relative Cardiovascular Response to UE and LE exercise: UE=LE during submaximal exercise





    E) Heart rate

     Image Upload 6
  571. Relative Cardiovascular Response to UE and LE exercise: UE>LE during submaximal exercise, except:





    B) Stroke volume

    Stroke volume increases more in LE exercise at submaximal intensity. 

    Image Upload 8
  572. Management of Patients with SCI: tracheostomy intermittent positive pressure ventilation (TIPPV) for both daytime and nocturnal



    A) Above C1
  573. Management of Patients with SCI: electrophenic pacing for daytime



    B) C2-C3
  574. Management of Patients with SCI: nasal or mouthpiece intermittent positive pressure ventilation (N/MIPPV) for nocturnal





    E) C2-C3 and below C2
  575. Management of Patients with SCI: intermittent abdominal pressure ventilator (IAPV) during daytime





    C) Below C2
  576. Burn Severity Classifications: first degree




    B) Superficial thickness
  577. Burn Severity Classifications: second degree





    B) Superficial and deep partial thickness
  578. Burn Severity Classifications: third degree




    D) Full thickness
  579. Burn Severity Classifications: erythematous, dry, mildly swollen, blanches with pressure, painful





    A) First degree
  580. Burn Severity Classifications: blistering, moist, weeping, blanches with pressure, painful





    C) Second degree (superficial)
  581. Burn Severity Classifications: no blisters, wet or waxy dry, variable color, less painful, at risk for conversion to full-thickness because of marginal blood supply





    A) Second degree (deep)
  582. Burn Severity Classifications: white waxy to leathery gray to charred black, insensate to pain, does not blanch to pressure





    D) Third degree
  583. Burn Severity Classifications: black (eschar), exposed bones, ligaments, and tendons





    D) Fourth degree
  584. Burn Severity Classifications: exfoliation, heals spontaneously in 1 wk, no scarring





    E) First degree
  585. Burn Severity Classifications: reepithelialization in 7-20 days





    A) Second degree (superficial)
  586. Burn Severity Classifications: reepithelialization in weeks to months; skin grafting may speed recovery; associated with scarring





    B) Second degree (deep)
  587. Burn Severity Classifications: reepithelialization does not occur; requires skin grafting; associated with scarring





    B) Third degree
  588. Burn Severity Classifications: may require amputation or extensive deep debridement





    E) Fourth degree
  589. American Burn Association Criteria for Referral to a Burn Center, except:





    A) Partial-thickness burn greater than 20% TBSA

    It should be partial-thickness burn greater than 10% TBSA.
  590. American Burn Association Criteria for Referral to a Burn Center, except:





    C) Burned adults in hospitals without qualified personnel or equipment

    It should be burned children in hospitals without qualified personnel or equipment.
  591. Preferred position for axillary burns:




    C) Both
  592. Preferred position for elbow burns:




    B) Extension 5 degrees
  593. Preferred position for forearm burns:




    B) Supination
  594. Preferred position for wrist burns:




    C) Neutral

    The preferred position for wrist burns is neutral or slight extension.
  595. Preferred position for hand burns, except:





    C) None of these
  596. Preferred position for hip burns:




    D) Both
  597. Preferred position for knee burns:




    A) Extension
  598. Positioning devices for neck burns, except:




    C) None of these
  599. Positioning devices for axillary burns, except:





    B) Wedge positioning to adduction

    It should be wedge positioning to abduction.
  600. Positioning devices for elbow burns, except:




    B) Neither
  601. Positioning devices for hand burns, except:





    A) Intrinsic minus hand splint

    It should be intrinsic plus hand splint.
  602. Localized Neuropathies and Associated Risk Factors: not a risk factor for brachial plexus neuropathy




    D) Neither
  603. Localized Neuropathies and Associated Risk Factors: not a risk factor for ulnar nerve neuropathy




    C) Supination

    It should be pronation.
  604. Localized Neuropathies and Associated Risk Factors: tourniquet paralysis, except:




    A) Brachial plexus
  605. Localized Neuropathies and Associated Risk Factors: resting on side rails, hanging over edge of operating table, tourniquet paralysis, wrist restraints




    B) Radial nerve
  606. Localized Neuropathies and Associated Risk Factors: not a risk factor for median nerve neuropathy




    C) Prolonged or repeated wrist hyperflexion

    It should be prolonged or repeated wrist hyperextension.
  607. Localized Neuropathies and Associated Risk Factors: not a risk factor for peroneal nerve neuropathy





    E) Tourniquet paralysis

    Torniquet paralysis is a risk factor for ulnar, radial, and median nerve neuropathies.
  608. Localized Neuropathies and Associated Risk Factors: not a risk factor for femoral nerve neuropathy




    D) Neither
  609. Karnofsky Scale: normal, no complaints, no evidence of disease




    D) 10
  610. Karnofsky Scale: able to carry on normal activity; minor signs or symptoms of disease




    B) 9
  611. Karnofsky Scale: normal, no complaints, no evidence of disease




    A) 8
  612. Karnofsky Scale: able to carry on normal activity; no special care is needed, except:




    A) 7
  613. Karnofsky Scale: cares for self; unable to carry on normal activity or do active work




    C) 7
  614. Karnofsky Scale: requires occasional assistance, but is able to care for most of own needs




    C) 6
  615. Karnofsky Scale: requires considerable assistance and frequent medical care




    C) 5
  616. Karnofsky Scale: unable to work, able to live at home; cares for most personal needs; varying amounts of assistance is needed, except




    D) 4
  617. Karnofsky Scale: disabled; requires special care and assistance




    D) 4
  618. Karnofsky Scale: severely disabled; hospitalization is indicated, although death is not imminent




    D) 3
  619. Karnofsky Scale: very sick; hospitalization necessary; active supportive treatment necessary




    B) 2
  620. Karnofsky Scale: moribund; fatal process progressing rapidly




    A) 1
  621. Karnofsky Scale: dead




    A) 0
  622. Pharmacologic Management of Pain in Cancer: side effects are gastritis and tinnitus





    E) Aspirin
  623. Pharmacologic Management of Pain in Cancer: side effect is hepatotoxicity





    E) Acetaminophen
  624. Pharmacologic Management of Pain in Cancer: side effect is gastritis





    E) NSAIDs
  625. Pharmacologic Management of Pain in Cancer: side effects are sedation, nausea, and constipation





    E) Tramadol
  626. Pharmacologic Management of Pain in Cancer: side effects are sedation, respiratory depression, constipation, confusion, and pruritus





    D) Morphine
  627. Pharmacologic Management of Pain in Cancer: causes constipation (often more severe than other opioids)





    D) Hydrocodone
  628. Pharmacologic Management of Pain in Cancer: used for breakthrough pain





    A) Fentanyl
  629. Harrington's Classification of Vertebral Metastases: no significant neurological involvement





    A) I
  630. Harrington's Classification of Vertebral Metastases: involvement of bone without collapse or instability





    A) II
  631. Harrington's Classification of Vertebral Metastases: major neurological involvement without significant bone involvement





    C) III
  632. Harrington's Classification of Vertebral Metastases: vertebral collapse without neurological impairment





    A) IV
  633. Harrington's Classification of Vertebral Metastases: vertebral collapse with neurologic impairment





    B) V
  634. Fracture Risk: upper extremity



    A) 1 point
  635. Fracture Risk: lower extremity



    B) 2 points
  636. Fracture Risk: trochanter



    B) 3 points
  637. Fracture Risk: blastic



    A) 1 point
  638. Fracture Risk: blastic/lytic



    B) 2 points
  639. Fracture Risk: lytic



    A) 3 points
  640. Fracture Risk: <1/3 diameter



    C) 1 point
  641. Fracture Risk: >1/3, <2/3 diameter



    A) 2 points
  642. Fracture Risk: >2/3 diameter



    B) 3 points
  643. Fracture Risk: mild pain



    B) 1 point
  644. Fracture Risk: moderate pain



    B) 2 points
  645. Fracture Risk: severe pain



    A) 3 points
  646. Chemotherapy-Associated Polyneuropathies: Taxanes





    E) Sensory > motor
  647. Chemotherapy-Associated Polyneuropathies: Platinum





    B) Pure sensory
  648. Chemotherapy-Associated Polyneuropathies: Thalidomides





    A) Sensory > motor
  649. Chemotherapy-Associated Polyneuropathies: Vinca alkaloids





    C) Motor = sensory
  650. Chemotherapy-Associated Polyneuropathies: Taxanes are used for the following types of cancer, except:




    B) Colon

    Platinum is used for colon cancer.
  651. Chemotherapy-Associated Polyneuropathies: Platinum is used for the following types of cancer, except:




    B) Breast
  652. Chemotherapy-Associated Polyneuropathies: Vinca alkaloids are used for which type of cancer






    B) Lymphoma
  653. Chemotherapy-Associated Polyneuropathies: Thalidomides are used for which type of cancer






    A) Myeloma
  654. Not an acute side effect of radiation therapy:





    E) Lymphedema

    Lymphedema is a delayed side effect of radiation therapy.
  655. Not an acute side effect of radiation therapy:





    C) Skin atrophy

    Skin atrophy is a delayed side effect of radiation therapy.
  656. Not a delayed side effect of radiation therapy:





    A) Proctitis

    Proctitis is an acute effect of radiation therapy.
  657. Not a delayed side effect of radiation therapy:





    C) Sterility

    Sterility is an acute side effect of radiation therapy.
  658. Side effects of radiation therapy: fatigue

    A) Delayed
    B) Acute
    B) Acute
  659. Side effects of radiation therapy: nausea

    A) Delayed
    B) Acute
    B) Acute
  660. Side effects of radiation therapy: vomiting

    A) Delayed
    B) Acute
    B) Acute
  661. Side effects of radiation therapy: anorexia

    A) Delayed
    B) Acute
    B) Acute
  662. Side effects of radiation therapy: skin erythema

    A) Delayed
    B) Acute
    B) Acute
  663. Side effects of radiation therapy: desquamation

    A) Delayed
    B) Acute
    B) Acute
  664. Side effects of radiation therapy: mucositis

    A) Delayed
    B) Acute
    B) Acute
  665. Side effects of radiation therapy: xerostomia

    A) Delayed
    B) Acute
    B) Acute
  666. Side effects of radiation therapy: taste loss

    A) Delayed
    B) Acute
    B) Acute
  667. Side effects of radiation therapy: proctitis

    A) Delayed
    B) Acute
    B) Acute
  668. Side effects of radiation therapy: cystitis

    A) Delayed
    B) Acute
    B) Acute
  669. Side effects of radiation therapy: decreased libido

    A) Delayed
    B) Acute
    B) Acute
  670. Side effects of radiation therapy: amenorrhea

    A) Delayed
    B) Acute
    B) Acute
  671. Side effects of radiation therapy: sterility

    A) Delayed
    B) Acute
    B) Acute
  672. Side effects of radiation therapy: hematologic changes

    A) Delayed
    B) Acute
    B) Acute
  673. Side effects of radiation therapy: soft tissue fibrosis

    A) Delayed
    B) Acute
    A) Delayed
  674. Side effects of radiation therapy: skin atrophy

    A) Delayed
    B) Acute
    A) Delayed
  675. Side effects of radiation therapy: pulmonary fibrosis

    A) Delayed
    B) Acute
    A) Delayed
  676. Side effects of radiation therapy: GI stricture

    A) Delayed
    B) Acute
    A) Delayed
  677. Side effects of radiation therapy: auditory changes

    A) Delayed
    B) Acute
    A) Delayed
  678. Side effects of radiation therapy: thyroid dysfunction

    A) Delayed
    B) Acute
    A) Delayed
  679. Side effects of radiation therapy: brain necrosis

    A) Delayed
    B) Acute
    A) Delayed
  680. Side effects of radiation therapy: osteonecrosis

    A) Delayed
    B) Acute
    A) Delayed
  681. Side effects of radiation therapy: myelitis

    A) Delayed
    B) Acute
    A) Delayed
  682. Side effects of radiation therapy: plexopathy

    A) Delayed
    B) Acute
    A) Delayed
  683. Side effects of radiation therapy: lymphedema

    A) Delayed
    B) Acute
    A) Delayed
  684. Side effects of radiation therapy: secondary malignancies

    A) Delayed
    B) Acute
    A) Delayed
  685. Head and Neck Cancer: communication impairments and dysphagia, except:




    D) Total laryngectomy

    Total laryngectomy is used for severe communication impairments and mild dysphagia.
  686. Head and Neck Cancer: chewing impairments 





    E) Mandibulectomy
  687. Head and Neck Cancer: facial nerve palsy





    D) Salivary gland tumors/resection
  688. Head and Neck Cancer: cranial nerve abnormalities





    B) Nasopharyngeal tumors/treatment
  689. Head and Neck Cancer: shoulder depression and protraction, facial lymphedema, neck asymmetry





    E) Radial neck dissection
  690. Head and Neck Cancer: contracture (neck, jaw), osteonecrosis, dental changes, decreased salivary flow, dysphagia (usually mild), trismus, CNS complications





    E) Radiation therapy
  691. Diagnostic Arterial Testing: assesses skin perfusion, except:




    C) Ankle-bracheal index

    Ankle-bracheal index assesses patency.
  692. Diagnostic Arterial Testing: assesses patency, except:





    E) Transcutaneous oximetry
  693. Diagnostic Arterial Testing: does not evaluate aneurysm





    E) Continuous wave Doppler
  694. Diagnostic Arterial Testing: does not determine location of stenosis or occlusion, except:




    C) Duplex scan

    Duplex scan, computed tomography, magnetic resonance angiography, contrast angiography, continuous wave doppler, and pulse volume recording determine the location of stenosis or occlusion.
  695. Diagnostic Arterial Testing: does not monitor disease progression




    C) Photopletysmography
  696. Diagnostic Arterial Testing: not accurate in the presence of non compressible calcified arteries




    B) Ankle-bracheal index
  697. Diagnostic Venous Tests: assesses obstruction in acute DVT only





    C) D-Dimer
  698. Diagnostic Venous Tests: assesses insufficiency, except:





    E) MR or CT imaging

    MR or CT imaging, as well as D-Dimer, do not assess insufficiency.
  699. Diagnostic Venous Tests: does not assess location of occlusion





    C) Pletysmography

    Plethysmography and D-Dimer does not assess location of occlusion.
  700. Immunosuppressive Agents: cytokine inhibitor




    B) Steroids
  701. Immunosuppressive Agents: purine synthesis inhibitor




    A) Azathioprine
  702. Immunosuppressive Agents: inhibits IL-2, except:




    D) Mycophenolic acid

    Mycophenolic acid is a purine synthesis inhibitor.
  703. Immunosuppressive Agents: side effects are neurotoxicity, nephrotoxicity, and electrolyte imbalance




    C) Cyclosporine and Tacrolimus
  704. Immunosuppressive Agents: side effects are neutropenia, hyperlipidemia, and concerns regarding bronchial anastomosis





    C) Sirolimus
  705. Immunosuppressive Agents: side effects are muscle wasting, osteoporosis, and poor wound healing





    A) Steroids
  706. Immunosuppressive Agents: side effects are GI distress and pancytopenia





    E) Azathioprine
  707. Immunosuppressive Agents: side effects are pancytopenia and diarrhea





    A) Mycophenolic acid
  708. Rejection Symptoms: occurs in OR during initial transplantation surgery

    A) Hyperacute
    B) Acute chronic
    A) Hyperacute
  709. Rejection Symptoms: secondary to preformed antibodies

    A) Hyperacute
    B) Acute chronic
    A) Hyperacute
  710. Rejection Symptoms: graft replacement/retransplantation

    A) Hyperacute
    B) Acute chronic
    A) Hyperacute
  711. Rejection Symptoms: T-cell mediated anti graft inflammatory infiltrate

    A) Hyperacute
    B) Acute chronic
    B) Acute chronic
  712. Rejection Symptoms: organ replacement is the treatment of choice

    A) Hyperacute
    B) Acute chronic
    B) Acute chronic
  713. Rejection Symptoms: which of the following is not an acute chronic rejection symptom?





    C) None of these
  714. Which of the following is not an early cardiovascular change associated with cardiac transplantation?





    C) Heart rate increases before stroke volume increases during exercise

    Following cardiac transplantation, stroke volume increases before heart rate increases during exercise.
  715. Which of the following is not a late cardiovascular change associated with cardiac transplantation?





    B) Improvement of parasympathetic tone to transplanted organ

    It should be improvement of sympathetic tone to transplanted organ.
  716. Classification of HIV: AIDS, except:





    E) None of these

    A3, B3, C1, C2, and C3 are all considered as AIDS.
  717. Classification of HIV: acute HIV



    C) Group A
  718. Classification of HIV: asymptomatic



    B) Group A
  719. Classification of HIV: generalized lymphadenopathy



    B) Group A
  720. Classification of HIV: symptomatic



    B) Group B
  721. Classification of HIV: indicator conditions present



    A) Group C
  722. Anatomical Classification of Contractures: not a primary cause of arthrogenic contracture





    E) Immobility

    Immobility is a secondary cause of arthrogenic contracture.
  723. Anatomical Classification of Contractures: not an intrinsic cause of myogenic contracture





    C) Muscle imbalance 

    Muscle imbalance is an extrinsic cause of myogenic contracture. Other extrinsic causes are faulty positioning in bed or chair and spasticity.
  724. Primary chronic neurobiological disease with genetic psychosocial and environmental factors influencing its development and manifestations




    C) Addiction

    Addiction is also known as psychological dependence.
  725. Characterized by behaviors that include one or more of the following: impaired control over drug use, compulsive use, continued use despite armed craving




    C) Addiction

    Addiction is also known as psychological dependence.
  726. Adaptation that is manifested by a drug class-specific withdrawal syndrome that can be produced by abrupt sensation, rapid dose reduction, decreasing blood level of the drug, and/or administration of antagonist




    A) Physical dependence
  727. State of adaptation in which exposure to a drug induces change that results in diminution of one or more of the drugs effects over time




    D) Tolerance
  728. Illegal use or inappropriate use of medications




    A) Diversion
  729. Commonly Used Oral Medications for Spasticity: presynaptic inhibition of GABAB receptors.; active both presynaptically and postsynaptically; hyperpolarizes cell membrane





    B) Baclofen
  730. Commonly Used Oral Medications for Spasticity: facilitates postsynaptic effects of GABAA by opening chloride channels in membranes resulting in increased presynasptic inhibition secondary to hyperpolarzation





    C) Diazepam
  731. Commonly Used Oral Medications for Spasticity: facilitates postsynaptic effects of GABAA by opening chloride channels in membranes resulting in increased presynasptic inhibition secondary to hyperpolarzation





    E) Diazepam
  732. Commonly Used Oral Medications for Spasticity: interferes with calcium release from sarcoplasmic reticulum





    A) Dantrolene
  733. Commonly Used Oral Medications for Spasticity: decreases tonic facilitation by locus coeruleus and in spinal cord enhances presynaptic inhibition





    A) Clonidine
  734. Commonly Used Oral Medications for Spasticity: blocks release of excitatory neurotransmitters and facilitates inhibitory neurotransmitters; antinociceptive and reduces spinal reflexes





    E) Tizanidine
  735. Commonly Used Oral Medications for Spasticity: oldest class of medications used for spasticity that is still in common use





    B) Diazepam
  736. Commonly Used Oral Medications for Spasticity: only true peripherally acting oral agent





    B) Dantrolene
  737. Commonly Used Oral Medications for Spasticity: risk of withdrawal seizures and hallucinations





    C) Baclofen
  738. Commonly Used Oral Medications for Spasticity: dose must be adjusted with renal disease





    E) Baclofen
  739. Commonly Used Oral Medications for Spasticity: primary use in SCI population 





    E) Clonidine
  740. Commonly Used Oral Medications for Spasticity: slow titration reduces sedation side effect that is major limiting factor





    B) Tizanidine
  741. Comparison of Different Treatment Modalities for Spasticity: indicated for generalized tone, spasms, no focal region of spasticity




    C) Oral medications

    Botulinum toxins and phone are indicated for focal areas of spasticity.
  742. Comparison of Different Treatment Modalities for Spasticity: potential improvement in passive or active ADLs





    C) Orthopedics procedure
  743. Comparison of Different Treatment Modalities for Spasticity: indicated for significant tone not treated by other modalities





    E) ITB
  744. Comparison of Different Treatment Modalities for Spasticity: disadvantages of phenol, except:




    A) Expensive

    Phenol is cheaper and longer lasting than botulinum toxin.
  745. Not an extrinsic risk factor in pressure ulcer development





    B) Impaired mobility

    Impaired mobility is an intrinsic risk factor for pressure ulcer development.
  746. Not an intrinsic risk factor for pressure ulcer development





    B) Psychosocial

    Psychosocial is an extrinsic risk factor for pressure ulcer development.
  747. Classes of Available Dressings: indicated for scabbed over wounds





    D) Gauze

    Gauze is nonselective and may remove granulation tissue.
  748. Classes of Available Dressings: indicated for stage I, II pressure ulcers with no drainage





    A) Occlusive

    There may be excessive moisture retention if occlusive dressings are used on wounds that drain excessively.
  749. Classes of Available Dressings: indicated for stage II, III, and IV with little to no drainage





    B) Gel
  750. Classes of Available Dressings: indicated for stage II, III, and IV ulcers with little to great drainage





    C) Foam
  751. Classes of Available Dressings: indicated for stage III and IV ulcers with moderate to great drainage





    C) Alginates
  752. Classes of Available Dressings: transparent film dressing that are semipermeable





    E) Occlusive
  753. Classes of Available Dressings: hydrophilic polymer that comes in sheet, granules, or liquid gel forms





    B) Gel
  754. Classes of Available Dressings: dressings containing gel-forming agents, often combined with elastomers and adhesives





    B) Hydrocolloid
  755. Classes of Available Dressings: highly absorbent, biodegradable dressings derived from seaweed





    D) Alginates
  756. Classes of Available Dressings: polyurethane dressing that comes in sheets or fillers





    E) Foam
  757. Cushion Material Characteristics: excellent pressure relief




    D) Flotation
  758. Cushion Material Characteristics: good pressure relief




    C) Gel
  759. Cushion Material Characteristics: poor to good pressure relief




    A) Viscoelastic foam
  760. Cushion Material Characteristics: poor to medium pressure relief




    D) Foam
  761. Cushion Material Characteristics: poor postural control




    C) Flotation
  762. Cushion Material Characteristics: medium to good postural control




    C) Foam

    Viscoelastic foam and gel have good postural control.
  763. Phases of Sports Rehabilitation: resolving pain and inflammation





    A) I
  764. Phases of Sports Rehabilitation: restoring range of motion





    C) II
  765. Phases of Sports Rehabilitation: strengthening





    C) III
  766. Phases of Sports Rehabilitation: proprioceptive training





    E) IV
  767. Phases of Sports Rehabilitation: sports/task-specific activities





    C) V
  768. Which of the following is not true about concussion?





    E) It results in a graded set of clinical syndromes that involves LOC.

    Concussion results in a graded set of clinical syndromes that may or may not involve LOC. Resolution of the clinical and cognitive symptoms typically follows a sequential course.
  769. Anterior Ankle Impingement Etiology: hypertrophic scar






    F) Anterior talofibular ligament
  770. Anterior Ankle Impingement Etiology: synovitis, tears






    C) Soft tissue
  771. Anterior Ankle Impingement Etiology: osteophytes, traction spurs






    D) Anterior tibia
  772. Anterior Ankle Impingement Etiology: avulsion fracture






    A) Distal tibia
  773. Anterior Ankle Impingement Etiology: loose bodies






    C) Talar neck
  774. Anterior Ankle Impingement Etiology: calcium deposits






    C) Capsule
  775. Anterior Ankle Impingement Etiology: inversion ankle sprains






    A) Anterior talofibular ligament
  776. Anterior Ankle Impingement Etiology: plantarflexion






    F) Soft tissue
  777. Anterior Ankle Impingement Etiology: repetitive forced dorsiflexion






    F) Anterior tibia
  778. Anterior Ankle Impingement Etiology: forced plantarflexion






    A) Capsule
  779. Anterior Ankle Impingement Etiology: MOI for distal tibia and talar neck





    D) Rapid plantarflexion
  780. Gross Motor Milestones: head in midline
    2 months
  781. Gross Motor Milestones: prone prop on extended elbows
    3 months
  782. Gross Motor Milestones: rolls prone to supine
    4 months
  783. Gross Motor Milestones: rolls supine to prone
    5 months
  784. Gross Motor Milestones: infantile swimming
    5 months
  785. Gross Motor Milestones: pivot circles in prone
    5 months
  786. Gross Motor Milestones: sits with straight back
    6 months
  787. Gross Motor Milestones: crawls on hands and knees ("creeps")
    9 months
  788. Gross Motor Milestones: transitions into sit from four-point
    9 months
  789. Gross Motor Milestones: pulls to stand through half-kneel cruises
    10 months
  790. Gross Motor Milestones: independent ambulation
    12 months
  791. Gross Motor Milestones: comes to stand independently
    15 months
  792. Gross Motor Milestones: climbs into adult-sized chair
    18-19 months
  793. Gross Motor Milestones: up and down stairs with hands on rail
    21-24 months
  794. Gross Motor Milestones: jumping clearing ground and lands on feet together
    30 months
  795. Gross Motor Milestones: pedals tricycle
    3 years
  796. Gross Motor Milestones: climbs up stairs with alternating feet
    3 years
  797. Gross Motor Milestones: true run
    3 years
  798. Gross Motor Milestones: hops
    4 years
  799. Gross Motor Milestones: gallop (not true skipping)
    4 years
  800. Gross Motor Milestones: walks down stairs with alternating feet
    4 years
  801. Gross Motor Milestones: true skipping
    5 years
  802. Primitive reflexes: onset of Moro
    birth
  803. Primitive reflexes: onset of palmar grasp
    birth
  804. Primitive reflexes: onset of plantar grasp
    birth
  805. Primitive reflexes: onset of adductor spread of patellar reflex
    birth
  806. Primitive reflexes: onset of tonic neck
    2 months
  807. Primitive reflexes: onset of Landau
    3 months
  808. Primitive reflexes: onset of parachute response
    8-9 months
  809. Primitive reflexes: Moro disappears by
    6 months
  810. Primitive reflexes: palmar grasp disappears by
    6 months
  811. Primitive reflexes: plantar grasp disappears by
    9-10 months
  812. Primitive reflexes: adductor spread of patellar reflex disappears by
    7 months
  813. Primitive reflexes: tonic neck disappears by
    5 months
  814. Primitive reflexes: Landau disappears by
    24 months
  815. Primitive reflexes: parachute response disappears by
    persists
  816. Orthopedic Surgical Procedure by Joint: for ankle equinus




    C) Tendon Achilles lengthening
  817. Orthopedic Surgical Procedure by Joint: for ankle inversion and dorsiflexion




    A) Split anterior tibialis transfer
  818. Orthopedic Surgical Procedure by Joint: for ankle inversion and plantarflexion




    A) Split posterior tibialis transfer
  819. Orthopedic Surgical Procedure by Joint: for calcaneovalgus




    C) Subtalar arthrodesis
  820. Orthopedic Surgical Procedure by Joint: often combined with lateral column (fifth metatarsal ray) lengthening




    D) Subtalar arthrodesis
  821. Orthopedic Surgical Procedure by Joint: for crouch, internal rotated gait




    B) Hamstring lengthening
  822. Orthopedic Surgical Procedure by Joint: to balance hamstring weakness and prevent recurvatum




    C) Rectus transfer

    Rectus femoris is transferred to either sartorius or semitendinosus.
  823. Orthopedic Surgical Procedure by Joint: for knee internal rotation




    B) Tibial derotation osteotomy
  824. Orthopedic Surgical Procedure by Joint: for hip flexion




    C) Psoas lengthening
  825. Orthopedic Surgical Procedure by Joint: for scissored gait




    C) Adductor tenotomy
  826. Orthopedic Surgical Procedure by Joint: for early hip subluxation




    C) Adductor tenotomy
  827. Orthopedic Surgical Procedure by Joint: for hip subluxation




    B) Varus derotational osteotomy
  828. Orthopedic Surgical Procedure by Joint: for subluxation with severe acetabular dysplasia




    D) Pelvic shelf procedures

    Examples of pelvic shelf procedures are Salter and Chiari.
  829. Genetic Loci of Select Neuromuscular Diseases: Charcot-Marie Tooth, type 1B





    C) 1q21.2q23
  830. Genetic Loci of Select Neuromuscular Diseases: Charcot-Marie Tooth, type 1A





    B) 17p13.1
  831. Genetic Loci of Select Neuromuscular Diseases: Nemaline myopathy, autosomal dominant





    D) 1q21q23
  832. Genetic Loci of Select Neuromuscular Diseases: Werdnig-Hoffman (infantile) SMA





    E) 5q13
  833. Genetic Loci of Select Neuromuscular Diseases: Kugelberg-Welander (juvenile) SMA





    A) 5q13
  834. Genetic Loci of Select Neuromuscular Diseases: limb girdle muscular dystrophy





    E) 15q
  835. Genetic Loci of Select Neuromuscular Diseases: paramyotonia congenita





    D) 17q13.1q13,5
  836. Genetic Loci of Select Neuromuscular Diseases: myotonic muscular dystrophy





    B) 19q13.3
  837. Genetic Loci of Select Neuromuscular Diseases: malignant hyperthermia syndrome





    C) 19q12q13.2
  838. Genetic Loci of Select Neuromuscular Diseases: central core disease





    A) 19q13.1
  839. Genetic Loci of Select Neuromuscular Diseases: Duchenne muscular dystrophy




    D) Xp21.2
  840. Genetic Loci of Select Neuromuscular Diseases: Becker muscular dystrophy




    D) Xp21.2
  841. Genetic Loci of Select Neuromuscular Diseases: Charcot-Marie Tooth, X-linked




    C) Xq13
  842. Characteristics of the Three Metabolic Systems: substrate is stored phosphagens



    C) ATP-CP system
  843. Characteristics of the Three Metabolic Systems: substrate is glycogen/glucose



    B) Glycolysis
  844. Characteristics of the Three Metabolic Systems: substrate is glycogen/glucose, fats



    B) Aerobic metabolism
  845. Characteristics of the Three Metabolic Systems: very fast ATP mobilization 



    B) ATP-CP system
  846. Characteristics of the Three Metabolic Systems: fast ATP mobilization



    A) Glycolysis
  847. Characteristics of the Three Metabolic Systems: slow ATP mobilization



    B) Aerobic metabolism
  848. Characteristics of the Three Metabolic Systems: very limited total ATP production capacity



    B) ATP-CP system
  849. Characteristics of the Three Metabolic Systems: limited total ATP production capacity



    B) Glycolysis
  850. Characteristics of the Three Metabolic Systems: virtually unlimited total ATP production capacity



    C) Aerobic metabolism
  851. Characteristics of Different Metabolic Substrates: energy content is 4.1 kcal/g



    A) Carbohydrates
  852. Characteristics of Different Metabolic Substrates: energy content is 4.3 kcal/g



    C) Protein
  853. Characteristics of Different Metabolic Substrates: energy content is 9.3 kcal/g



    A) Fat
  854. Characteristics of Different Metabolic Substrates: oxygen equivalent is 5.0 kcal/L



    A) Carbohydrates
  855. Characteristics of Different Metabolic Substrates: oxygen equivalent is 4.7 kcal/L



    A) Fat
  856. Characteristics of Different Metabolic Substrates: oxygen equivalent is 4.4 kcal/L



    B) Protein
  857. Characteristics of Different Metabolic Substrates: respiratory quotient is 1



    C) Carbohydrates
  858. Characteristics of Different Metabolic Substrates: respiratory quotient is 0.7



    C) Fat
  859. Characteristics of Different Metabolic Substrates: respiratory quotient is 0.8



    B) Protein
  860. Coronary Artery Disease Risk Factor Thresholds: not true about family history





    D) Sudden death before 45 years of age in mother or other female first-degree relative

    It should be sudden death before 65 years of age in mother or other female first-degree relative.
  861. Coronary Artery Disease Risk Factor Thresholds: cigarette smoking




    D) Both of these
  862. Coronary Artery Disease Risk Factor Thresholds: hypertension




    A) On hypertensive medication

    It should be SBP ≥140 mm Hg or DBP ≥90 mm Hg, confirmed by measurements on at least two separate occasions.
  863. Coronary Artery Disease Risk Factor Thresholds: not true about dyslipedemia





    B) Total serum cholesterol >150 mg/dl

    Total serum cholesterol should be >200 mg/dl.
  864. Coronary Artery Disease Risk Factor Thresholds: not true about obesity





    A) Waist/hip ratio of ≥0.90 in women

    Waist/hip ratio should be ≥0.86 in women.
  865. Coronary Artery Disease Risk Factor Thresholds: not a positive risk factor for CAD





    B) High serum HDL

    High serum HDL is >60 mg/dl.
  866. Medical examination and clinical exercise testing prior to exercise participation is not necessary in the following conditions, except:




    D) Moderate risk, vigorous exercise

    Medical examination and clinical exercise testing is recommended for high risk, moderate exercise; moderate risk, vigorous exercise; and high risk, vigorous exercise.
Author
carminaorlino
ID
356662
Card Set
Purple Notes (De Lisa)
Description
Updated