Geriatric comanagement benefits (3) in pt >80 undergoing THA/TKA
Decreases complications
Decreases hospital stays
Decreases rehospitalization
Primary blood supply of proximal humerus
Posterior circumflex artery ( 64%)
2 radiographic measurements correlate with humeral head ischemia
Metaphyseal head extension <8mm
Medial hinge disruption >2mm
However does not correlate with AVN
Outcomes of ORIF vs HA comparison (2) of proximal humerus fractures
ORIF IN <65 y.o. better constant scores
ORIF more complications vs HA (AVN + cutout)
4 relative indications IM nail humerus
2 part Surgical neck
Young age
Concomittant humeral shaft
Impending pathologic
2 requirements for a succesful hemiarthroplasty
Functioning rotator cuff
Anatomically healed tuberosities
How to judge humeral head height based on anatomic landmarks
Top humeral head 5.6 cm above from upper border of pec major
4 complications of RSA
Scapular notching
Glenoid loosening
INstability
Infection
RSA vs ORIF for proximal humerus fractures
RSA less complications
ORIF better functional outcomes
RSA vs ORIF for proximal humerus fractures
RSA less complications
ORIF better functional outcomes
List anatomical risk factors for glenohumeral instability 4
Increased glenoid retroversion
Glenoid hypoplasia
Reverse hillsack
Increased humeral head retroversion
Position of dislocation of posterior instability
Flexion Adduction IR
4 static stabilizers of posterior instability shoulder
Posterior labrum: increases concavity compression
Posterior capsule: PIGHL most important in IR and flexion.
Posterior glenoid bone loss?humeral head
Rotator interval
What is the concept of shoulder circle?
If shoulder dislocates posterior à the rotator interval should be disrupted anteriorly à needs plication when stabilizing posterior
What are the four etiologies for posterior shoulder instability
Acute trauma
Repetitive microtrauma
Insidiuos onset: generalized ligamentous laxity
Voluntary
What are the physical exam signs associaed ith posterior instability 5
Jerk
Kim
Posterior stress
Load and shift
Sulcus
What are 3 the soft tissue MRI findings? of posterior instability
POsterior bankart
Enlargement of posterior capsule
Kim lesion: incomplete avulsion of the posterior labrum
2 indications for surgery in posterior shoulder instability
Failed conservative
instability from an acute traumatic etiology and identifiable soft-tissue and/or osseous pathology.
2 Options for >20% glenoid loss in posterior instability
Ilac crest or distal tibial allograft
no difference in results
Posterior instability outcome: suture anchor vs no suture anchor
Suture anchor > greater return to play
Posterior instability shoulder post surgery:
Overall return to sport
Overall return to same level
90%
64%
Distal humerus # in elderly:
Union
COmplications
Timing
3%
30%: same to younger
Improved if <3 days: decrease hospital discharge + minimize complications
Indicatios of ulnar nerve transposition in distal humerus ORIF 2
If pre op symptoms: better recovery
If nerve damaged pre op
2 indications for TEA in distal humerus #
Unable to ORIF with good stabilit
Presence of pre existence of arthritis, no union, malunion
5contraindications of TEA
Young high demand: weight restriction 10lb
Cognitive impairement
Neuro injury
Presence infection
Poor soft issue
List the complications with TEA? (5)
Osteolysis
Loosening
Failure
Infection
Fracture
Can you excise the condyles in TEA?
yes, Canadian study:
does not compromise elbow strength or motion and can decrease surgical time and complexity, as well as eliminate pain secondary to condylar nonunion
Type of elbow most used?
Semi- constraint
Sloppy hindge ofloads cement bone interface
Non constrainted- requires perfect soft tissue balenceing but preserves bone stalk
Comparing TEA to ORIF:
Surgical time
daSH score
MEPS
TEA beter time
TEA better score at 6 months
TEA better at all point
List the advantages of volar plating in elderly 4 vs dorsal for DRF
Improved radiological outcomes
Low complications
Improved function
Better clinical results vs CRPP and ex fix
Lower risk displacement and tendon irritation
List 4 graft options in DRF in elderly
Iliac crest auto graft
allograft
hydroxyapatite
calcium biphosphate
Has formal physio improved outcomes over home progam for DR #?
NO
Definition of pelvic discontinuity: definition
separation of the ilium superiorly from the ischiopubic segment inferiorly
3 risk factors pelvic discontinuity
Female
Rheumatoid
Radiation therapy
Options for pelvic discontinuity
Hemisphereic cup with posterior column plating: setting of acute fracture
Highly porous coated acetabular components: need 50% host bone contact to cup
Cup cage
Distraction method
Custom triflange
Q) What is the definition of HO?
HO is the formation of trabecular bone in soft tissues where bone does not usually occur.
How early can pain X-rays or Bone scan detect HO?
X-rays => 4wks // Bone scan can show inc. uptake before 4wks
What are the lab values in HO?
Normal Ca & PO4 levels throughout Dx process
Alk Phos inc. in early HO & plateau’s at 4wks
How do you classify HO in hip?
Brooker
1) Island of bone within the soft tissues about the hip
2) Bone spurs from pelvis or prox femur with > 1cm between bone opposing surfaces
3) Similar to Class 2 but < 1cm between ends of bone spurs
4) Ankyloses of the hip
2 surgical factors that increase risk HO
Iliofemoral > kocher langenbech > ilioinguinal
Complex exposure:
-double exposure
-trochanteric osteotomy
Soft tissue factors that decrease risk of HO
Debridement of necrotic muscle tissue
Clinical/systemic factors that increase risk HO 7
a) Male
b) TBI or thoraco-abdominal trauma
c) Sciatic nerve injury
d) femoral head injury/ intra-articular debris
e) Delay to Sx
f) Ipsilateral femur #
g) Prolonged mechanical ventilation
Early vs late THA for acetabular # risk of HO
Higher with early
What is the best Treatment/prophylaxis option for HO in Acetabular #?
XRT 600-800cGy within 72hrs post-op: better if given within 72 hrs
Better than pre op XRT
Better than NSAIS's
What is the role of NSAIDs in HO prophylaxis of Acetab #?
**Literature indicates that NSAIDs are ineffective for HO prophylaxis after Acetab Sx**
What is the surgical Rx for HO in Acetab #? (4)
- Traditionally, resection delayed until mass was “Mature” but could be active in bone scan up to 2yrs.
- The recommendation is to resect the HO if symptomatic (causing neurodeficit or significant ROM limitations).
- Anticipate significant blood loss, prepare nerve stimulators & doppler to aid in difficult locations. XRT or 2wk course of indomethacin post-op.
Risk of HO and timing of acetabular #
Increased if delayed acetabular fixation
Describe the boarders of the ulnar tunnel (guyons canal) (3)
begins at the proximal edge of the volar carpal ligament
extends to the fibrous arch of the hypothenar muscles.
borders are not constant but most notably the pisiform and the hamate
Describe the zones of the ulnar canal and their significances (3)
Zone 1:-3 cm in length - volar carpal ligament palmary and the transverse carpal ligament dorsally
-proximal to bifurcation of the ulnar nerve into its motor and sensory branches
-both paresthesia and intrinsic muscle deficit.
Zone 2
-After the ulnar nerve bifurcates
-deep motor branch goes dorsal and radial around the hamate as it dives deep to the fibrous arch of the hypothenar muscles
-deficits in motor function only
Zone 3- superficial sensory branch of the ulnar nerve as it courses palmar to the fascia of the hypothenar muscles.
Common causes of zone 3 ulnar nerve compression (2)?
ulnar artery thrombosis
ulnar artery aneurysm
What are the common causes of zone 1 ulnar nerve compression (4)
- Ganglion cysts
- hook of hamate fractures
- traumatic adhesions
- anomalous muscles
Causes of ulnar nerve Zone 2 compression (3)
ganglion cysts - 90% of cause in zone1 and 2
fractures
thickened pisohamate ligament.
Descibe the common exam findings of ulnar neuropathy (8)
Tinel test
sensory threshold testing,
interossei wasting,
inability to cross fingers
unable to abduct little finger ( Wartenberg sign)
Ulnar claw deformity-secondary to lumbrical paralysis of the little and ring fingers
-extensor tendons place MCPs in hyperextension
-long flexors place the PIP and DIP in flexed position.
Froment sign:Attempted pinch between the thumb and the index finger may lead to compensatory thumb IP flexion
Jeanne sign: hyperextension of the thumb MCP > paralysis of the adductor pollicis muscle.
What is the ddx of ulnar neuropathy and how can it be determined (4)
ulnar tunnel
cubital tunnel
thoracic outlet
cervical radiculopathy syndromes
Determine using EMG!!
2 main focus of surgical management of ulnar neuropathy
removal of mass effect lesionsdecompression of all 3 zones
antebrachial fascia,
volar carpal ligament,
hypothenar fibrous arch (overlying the deep motor branch)
What is the most common cause of compression of ulnar nerve syndrome
Ganglion cysts
What are the boundaries of the radial tunnel (5)
5 cm long and begins as the radial nerve courses past the radiocapitellar joint.
Roof: brachioradialis muscle
Medially: biceps tendon and the brachialis
Lateral: ECRB and ECRL + brachioradialis muscle.
Distally ends at the fibrous arch of the proximal edge of the supinator muscle (arcade of Frohse)
What is the Hx funding of radial tunnel syndrome
proximal, lateral forearm or elbow pain that often worsens with rotational movements of the forearm
Physical exam findings of radial tunnel syndrome (3)
lateral forearm pressure over the supinator muscle, with the wrist in full supination should reproduce pain: Pronation of the wrist during this maneuver moves the radial nerve away from the thumb-directed pressure, should relieve the pain.
Pain with resisted, active extension of the wrist or the long finger.
Slight weakness of the extensors secondary to pain rather than motor nerve dysfunction.
Treatment of radial tunel syndrome: areas of decompression (3)
Arcade of froshe
Leading edge of proximal ECRB
Compressive fascia of distal supinator
Pronator syndrome defintiion:
compressive neuropathy of the median nerve at the level of the elbow
List 8 site of compression for pronator syndrome:
supracondylar process
residual osseous structure on distal humerus present in 1% of population
ligament of Struthers
travels from tip of supracondylar process to medial epicondyle
not to be confused with arcade of Struthers which is a site of ulnar compression neuropathy in cubital tunnel syndrome
bicipital aponeurosis (a.k.a. lacertus fibrosus)
between ulnar and humeral heads of pronator teres
FDS aponeurotic arch
AIN syndrome defnition
compressive neuropathy of the AIN that results in motor or sensory deficits
Physical exam findings AIN syndrome (2)
motor deficits only
no cutaneous sensory changes
List 7 sites of sites of entrapment for AIN
tendinous edge of deep head of pronator teres
FDS arcade
edge of lacertus fibrosus
accessory head of FPL (Gantzer's muscle)
accessory muscle from FDS to FDP
abberant muscles (FCRB, palmaris profundus)
thrombosed ulnar radial or ulnar artery
Difference between AIN and pronator syndrom 2
AIN syndrome: inflammatory
Pronator syndrome mechanical
AIN syndrom motor deficits only
Most common site of AIN compression
tendinous edge of deep head of pronator teres
Distinguish carpal tunnel from more proximal compression of AIN
Palmar paresthesia may help surgeons distinguish carpal tunnel compression from compression at more proximal locations
Physical exam finding in AIN compression if:
Pronator syndrome
Lacertus fibrosus
Pronator teres
FDS
Carpal tunnel
Pronator syndrome: presence of sensory deficits rules out an isolated AIN lesion but supports a diagnosis of pronator syndrome
Lacertus fibrosus: resisted elbow flexion with the forearm in a supinated position.
pronator teres : resisted forearm pronation.
FDS arch : resisted finger flexion -- PIP of long fingure.
Carpal tunnel: Tinel sign and a positive scratch collapse test may be observed in patients with proximal median neuropathy.
Mannerfelt syndrome:
attritional rupture of the FPL secondary to a carpal osteophyte
What is the function of TFCC? (2)
1) Stabilizes DRUJ
2) Cushion for force distribution btw carpus & ulna
Q) What are the structures forming TFCC? (5)
1) Articular disc
2) Volar & Dorsal DRU ligaments
3) Ulnocarpal ligaments
4) ECU sheath
5) Meniscus homolog
When is the DRU ligaments tight/lax?
A) Lax in neutral rotation & tight in Max. pro & supination
DDx of Ulnar sided wrist pain? (4 broad categories)
1) Traumatic
2) Inflammatory
3) Congenital
4) Degenerative
Ddx ulnar sided wrist pain JAAOS table
What are the provocative test to further assess Ulnar sided wrist pathology? (7)
Ulnocarpal stress test
PT grind test
Kleinman shuck
Shear test
Derby relocation
Ulnar fovea
Piano key
Describe ulnocarpal stress test
For ulnar sided wrist pain
wrist Max Uln deviation, forearm Neut, elbow 90deg then axial load on wrist while sup/pron => Pain is +ve
Describe PT grind test
For ulnar sided wrist pain
Simply grind the pisiform on the triquetrum
Describe Kleinman Shuck test
For ulnar sided wrist pain
Also simple, hold triquetrum & pisiform on one hand btw thumb & index while the other hand holds the lunate & move both hands in opposite directions.
Shear test
For ulnar sided wrist pain
Same as Shuck test but stabilize radiolunate instead of only lunate
Describe ulnar fovea sign
to assess UT ligament or foveal disruption => Pain replicated by pressing on interval btw ulnar styloid & FCU tendon btw the volar surface of ulnar head & pisiform.
Describe piano key test
for ulnar sided wrist pain
evaluates DRUJ inj => Pronate the forearm, ulna is dorsally prominent relative to radius => if minimal restraint to volar displacement of the ulna relative to radius => +ve test for instability. Recheck with forearm in Supination.
What radiological images required to assess Ulnar sided wrist pain? (5)
1) Standard X-rays => Perfect AP, Lat & oblique views (30 pro & 30 deg sup)
2) CT => To assess for nonunion, subtle joint subluxation
3) CT arthrogram => high sens & specificity for lig inj 4) Dynamic CT => Ideal for assessing DRUJ translation
MRI has largely replaced MR arthrogram for wrist pathology => due to high false +ve, pain related to inj, chemical synovitis 2ry to contrast. If suspecting non benigh tumor => MRI + gado.
Best radiological exam to order for ulnar sided pain
MR arthorgram
Benchmark for Dx of TFCC pathology
Wrist arthroscopy
Risk of rerupture achilles Op vs non Op (2)
6x higher non op with older rehab protocols
Comparable Op vs non op with early functional rehab
Describe a functional rehab protocol for acute achilles tendon rupture
0-2 wks: Initial immobilization + NWB
2-4 wks: CAM (control ankle motion) + 2 cm heel lift + Protected weight bearing + ROM to neutral
3 benefits of allowing early weight bearing for non op achilles tendon
Reduces ankle stiffness
Better health related QOL
Faster return to work
*No effect on rerupture rates
2 benefits of surgery vs functional rehab for acute achilles tendon tear
Lower time to return to work
Higher plantar flexion strength
List 4 complications of non operative management of achilles tendon tear
Rerupture
Ulcers: foot
Heel pain
DVT/PE
Name 1 absolute indication for surgical repair of achilles tendon tear
Avulsion from calc
Rate of wound complications from achilles tendon repair
7-8%
3 risk factors for wound complications after achilles tendon repair
Female
Smoking
Steroids
Weight bearing after achilles tendon repair 3 benefits
NO increase complications
Improved QOL at 6 weeks
Decreased activity limitations at 6 weeks
Evidence behind PRP and stem cells
No studies show superior vs control group
Ideal test for assesment of druj instability
Dynamic CT in pro/sup
Ulnar sided wrist pain use mr arthrogram if looking for
TFCC tear
Ligamentous injury
At risk population for HIV 3
IV drug users
Person who exchanges sex for drugs or money
Person who have had >3 partners since last HIV test
Rate of HIV transmission from needlestick
0.3%
Risk of transmission HIV from needlestick related to 6 factors
Deep injury
Visible blood on device
Needle that had been placed on artery or vein
Terminal HIV illness
Large gauge needle (<18)
Emergency procedure
HIV in THA - 3 things different from normal population
Major complications increased
Perioperative wound infection
Increased length of stay
HIV - molecule that is upregulated and effect on bone healing
Chronically elevated TNF-A > interferes with bone metabolism
HIV pre op work up
CXR if hx of PCP incfection: Risk spontaneaous pneumo
Regular blood including liver
CD4 counts + viral load
2 lab values that increase risk periop complications
Viral load >10 000-30 000
CD4 count <200
HIV increased post op complications 3
DVT
Being admitted to ICU for mechanical ventilation
Spontaneous pneumo
No differenc in outcome in (3)
THA
Spinal fusion
ORIF fracture
What type of reaction results in hypersensitivity to orthopedic implants
Type IV: delayed type > T cell mediated
7 risk factors for cutaneous metal hypersensitivity
Female
Piercings
History hand eczema
Metal allergy in first degree relative
History of smoking
Post well functioning arthroplasty
Post failed arthroplasty → even higher than well functioning arthro
Most common metal allergen in population
Nickel (14%)
What does ALVAL stand for
Aseptic lymphocytic vaculitis-associated lesions
MoM implant ion level that warrant additional imaging
5ppb: also may need joint aspiratons
Treatment for low,moderate, high risk patients with MoM implant
Low: Well fixed, good track record + no symptoms = annual follow up
Moderate: Well fixed/positioned + some symptoms or recalled implant = follow up 3-6 months. Revise if progressing symptoms,imaging abnormalities or elevated ion levels
High risk: severe symptomatic, high ion levels (>10 ppb), malpositioned components = revision
3 options for treatment of morel-lavallee
Observation
Percutaneous drainage: Some studies show high rate of reoccurence - esp if drainiage > 50cc initally
Open debridement and irrigation
Adjuvant treatment for morel-lavallee (4) to limit additional soft tissue injury
Serial aspiration
Compression bandage
Liposuction
Sclerosing agents
Describe an accepted technique for debridement of a morel lavalle lesion
2 2 cm incisions at proximal and distal end of the lesion
Simultaneous cavity access through these portals
Brushed and pulsed irrigation
Debride necrotic tissue
Placement of percutaneous drain
Drain removed after 2 weeks or when drain <30 cc per 24 hrs
Factors that affect recurrence of morel lavalle
Fluid aspirate >50 cc if done percutaneously
2 scelrotherapy agents used to treat chronic morel lavalle lesions
Talcum powder
Doxycycline
Definition Patellar tendinopathy
a clinical condition characterized by activity-related pain in the distal pole of the patella or in the proximal patellar tendon
Patellar tendonipathy is common in which sports?
D)
List risk factors for patellar tendonapathy (6):
1- high body mass index
2- a large abdominal circumference
3- limb-length discrepancy
4- flatfoot arch
5- weak quadriceps
6- low flexibility in quads and hamstrings
Q3: Where is the most common location of patellar tendonapathy?
Answer: Tendinosis typically is in the posterior portion of the proximal end of the patellar tendon adjacent to the inferior patellar pole.
Does patellar tendinopathy have inflammatory cells?
NO, progressive degeneration of the tendinous tissue, an inability of the tissue to repair itself, and the absence of inflammatory cells.
Names findings of patellar tendinopathy on physical exam:(2)
1- Decline squat test: pain at distal pole patella at 30degress of flexion of knee on single leg squat
2- Pain on palpation of distal pole pf patella
Q7: Give a DDx for patellar tendinopathy:(5)
Patellofemoral pain syndrome
Fat pad syndrome
Meniscal tears
Cartilage lesions
Referred pain
Q8: Name non surgical treatments of patellar tendinopathy:
1- Eccentric exercises – good data only one with good data
2- Extracorporeal Shock Wave Therapy – still experimental
3- Steroid injection – Not recommended
4- PRP – non consistent evidence in lit
5- Cell based therapy – experimental
6- Hyaluronic Acid – no evidence
7- Sclerosing Agents – experimental
Surgical treatment things to know for patellar tendinopathy
Arthroscopic and Open - The goals of these techniques are tenotomy of the patellar tendon, excision of abnormal tissue, and induction of the repair process through stimulation of the inferior pole of the patella by drilling and marginal resection.
Similar results in Open and arthroscopic technique: faster return to sports in arthroscopic treatment
What is the 1 year mortality for elderly patients w/ acetabular #?
- 8.1%-25%
How do the fracture patterns in acetabulum differ to young patents? (7)
- more likely anterior column
- fall onto posterior lateral hip à drives into ant column/ quad plate à protusio with intact posterior columno medial dome impaction --> poor outcomes-
If post column:-
More comminuted-
More marginal impaction-
Posterior wall, hip dislocation and medial dome impaction-
Poor outcomes with surgery
What effects does co-management (ortho/geri) of hip fractures have (6)
- reduced the LOS
- readmission rates
- time to surgery
- complication
- mortality rates
- hospital-acquired complications and improved outcomes in geri trauma
Describe a non op protocol for elderly acetabular #
- Therapy guided toe touch weight bearing for 6 weeks
- Radiographs at 2,6,12
- Often asymptomatic by 4-8 weeks
What is role of percutaneous screw fixation in acetabular # in the elderly (4 things)
- Column fractures with good bone coordiors
- Excellent knowledge of technique and anatomy required
- Conversion to THA similar to open fixation – ave 2.4 y
- EBL much lower
What sign is associated with inadequate reduction dn early loss of reduction + early joint space narrowing
Gull sign
superomedial dome impaction
What is the overall conversion rates to THA following ORIF of acetabular # in all comers
- 10-37%
- studies show signficnat patient hip score improvment for post traumatic OA from Acetabular fracture to THA
What type of Acetabular component for older pt with acetabular #
- uncememted superior survival to cememtned
What are the negative predictors of hip survival after ORIF of acetabular#?
- age 40 years
- nonanatomic reduction
- hip dislocation
- acetabular roof or posterior wall involvement
- acetabular articular impaction
- femoral head involvement- initial displacement more than 20 mm
When considering acute THA for acetabular # what is the goal of acetabular ORIF? (2)
- Reconstruction of stable columns
- Enough stability to accept press fit component
What factor can affect the Harris hip score for acute THA following acetabular ORIF
Need for dual approach do worse
How can you increase early mobility in acute THA following acetabular # (4)
- cement the stem
- place press fit cup into a position that affords max stability
- cement in liner into optimal anteversion
- patients ambulating within 7 days on average
What are outcomes of elderly with ORIF+ THA vs ORIF only? (systematic review) (6)
- ORIF and THA resulted in slightly less surgical time
- increased blood loss
- rate of coversion of ORIF to THA was 22% at 2 years
- mortaility same
- HARRIs hip scoresà higher in the ORIF
- SF-36 better in THA
What are the risk factors for developing lumbar spine stenosis (LSS)? (4)
Male
Caucasian
Obesity
Older
age
What is the most common site for lumbar spine stenosis?
L4-5
What are the 2 types of congenital LSS?
Achondroplastic and idiopathic
What are the criteria of the spinal canal in congenital LSS? (5)
Most commonly affects L3-L5
Decrease in Canal/VB ratio
Decrease in AP canal diameter to AP VB diameter ratio
Decrease in the transverse canal diameter to transverse VB diameter ratioShort pedicle
What causes central canal spine stenosis 2 anatomic structures
Disk-osteophyte complex
Ligamentum flavum hypertrophy
What causes lateral recess stenosis?
Facet hypertrophy associated with osteophytes
What causes Foraminal stenosis?
Loss of disk height leading to collapse in the foramen
Osteophytes
Disk protrusion
Angulation (scoliosis)
What causes extraforaminal stenosis?
Far lateral disk herniation
Radiographic classification of LSS based on cross sectional area
Moderate < 100 mm2
Severe < 75 mm2
Q) What is a worse prognostic factor in brachial plexus injuries?
A) Preganglionic nerve injuries (higher risk w/ subclavian Vs. axillary art. Inj) have limited healing potential, while postganglionic has greater nerve regenerative capability.
Q) What is the most common mechanism of injury in scapulothoracic dissociation?
A) Motorcycle accidents 44% followed by MVC 35% ((Distraction injury))
Q) What is the sequence of soft tissue injuries in scapulothoracic dissociation?
A) Musculoskeletal & vascular structures are 1st injured while Brachial plexus is injured last.
Q) What is the risk of limb-threatening ischemia in scapulothoracic dissociation?
DDx of ischemic limb (3)
A) 10%. Important to R/O other causes of absent pulses =>
1) Hypovolemic shock
2) Peripheral vasoconstriction
3) Ipsilateral extremity injury.
Q) What are things to look at on physical exam when assessing Scapulothoracis dissociation? 3
Asymmetric shoulderss
NV exam
Horner syndrome for pre ganglionic injury
Q) What is the initial radiological diagnostic modality to assess ST dissociation? And what are the findings? (3)
Non rotated CXR
Distraction clavicle #
Scapular index: distance between spinous process and medial border of scapula (+ve 1.29)
Distance btw spinous process and medial border of scapula (>1cm uninjured side)
Q) What is the role of Electrodiagnostic Testing (EMG & NCV) in ST dissociation? in scapulothoracic dissociation
Need to performed 3-4 weeks post injury
Can help locate lesion
Q) When is surgically managing a Scapulothoracic dissociation pt indication from Ortho standpoint?
Stabilization after vascular repair to stabilize or progressive neurologic deficit.
Otherwise, once pt. general condition stabilized
Q) What is the role of vascular repair in ST dissociation? (2)
ST injuries without active arterial hemorrhage or limb threatening ischemia could be managed with observation alone.
- If vascular repair is ever needed then UE fasciotomies + stabilization of clavicle, SC or AC fixation should be considered.
Q) What is the treatment plan for brachial plexus injury in ST dissociation
- Historically, complete preganglionic injury was managed with immediate above-elbow amputation +/- shoulder arthrodesis.
- Recently, increased interest in nerve transfer or neurotization in pts whom avulsion of nerve from the muscle.
Q) What are the main goals of nerve reconstruction in ST dissociation
A) Restore elbow function, shoulder stability, hand grip & sensation.
Q) What is the recommended timeline for surgical intervention for Neurological inj in ST dissociation
A) - No later than 6 months if no neuro recovery - Shorter time to Sx may have improved functional outcomes.
Q) What is the mortality rate in ST dissociation?
10% (Maybe even more since pt. die without knowing if they have it)
- Of pts who survive evaluation & Dx => 52% have flail extremity & 21% early above elbow amputation.
Q) What is Total wrist arthrodesis?
Fusion of the carpus to the radius. Eliminates wrist Flex/Ext & Uln/Rad deviation but preserves forearm rotation.
Q) Outcome of plating vs other techniques? wrist fusion
Fusion rate is significantly higher (98% Vs. 82%) in plating & complication rates significantly lower (51% Vs. 79%)
Q) What are the indications of wrist fusion?
Rheumatoid Arthritis (Significant decrease in numbers with DMARDs)
Post -traumatic arthritis
Osteoarthritis
SNAC & SLAC
End stage Keinbock Dx
Spastic wrist contracture
Distal radius non-union/malunion
Pseudoarthrosis of prev. Arthrodesis
Failed PRC
Failed SL reconstruction
Preiser osteonecrosis
Silicone synovitis
Failed wrist arthroplasty
Q) What are the complications of wrist arthrodesis? (6)
overall 29%
Nonunion 4.4%
Ulnocarpal impaction
Carpal tunnel syndrome
Extensor tenosynovitis
Deep infection
Implant-related problems (Persistent pain, prominent HW, # around implants)
Q) Which has better outcome ((Wrist Arthroplasty Or Fusion))?
In pts with Rheumatoid Arthritis
All pts were highly satisfied in both techniques BUT Fusion provided more reliable pain relief, lower rate of complications, less frequent need for revision than Arthroplasty.
Both procedures were extremely cost effective.
In pts with post-traumatic Arthritis
Both groups had similar DASH scores & comparable complication rates. However, Arthroplasty group had a significant better mean Patient-Rated Wrist Evaluation (PRWE) score.
Q) When would wrist Fusion be indicated over Arthroplasty? 5
< 50yrs
Manual laborers
Hx of infection
Use of Walking aids
Lack of active wrist motion
Q) How is the functional outcome of bilateral wrist arthrodesis?
Recent literature demonstrates high satisfaction rate in long term F/U.
((Historically, surgeons recommended preservation of motion in one wrist when other is fused))
Q) What are the contraindications to Total wrist arthrodesis? 2
Active infection
Lack of adequate soft tissue envelope
((Poor bone stock in RA is not a contraindication anymore with the use of Locking plates))
Q) What are the recommended surgical techniques in wrist fusion? 2
Precontoured stainless-steel locking plates (Majority of cases)
Steinmann rods (2 to control rotation) => for pt. Who require MCP arthroplasty & if forearm dissection can’t be performed
Q) What is the recommended wrist fusion position?
1) 10-15 degrees of extension + Slight ulnar deviation ((Shown to have significantly better grip strength))
2) Neutral position ((Thought to have better pro/supination + balanced flex/extensor forces))
Q1) List three Facet cyst contents:
Mucoid
Serous
Hemorrhagic
Where do lumbar facet cyst come from
Key point: Facet cysts arise from the zygapophyseal joints of the lumbar spine.
Q3) What are the most common location of facet cysts?
L4-L5 (68%),
L3-L4 (14%), and
L5-S1 (12%)
Q4) How often are facet cysts associated with spondylolythesis?
33% to 92% of LFCs were associated with spondylolisthesis
Q5) List treatment modalities for facet cysts: 3
NSAID's/supportive
Percucaneous drainage: 80% success...CT guided is better than U/S guided
Surgical decompression: 80% success
Q6) what is the recurrence rate of facet cysts after surgical excisison?
1.8%
Q7) What are common complications of facet cyst surgical treatment?
CSF leak (adherent to dura)
Spondylolisthesis
DVT
Death
Q8) What are the disadvantage of instrumentation versus decompression alone? (4)
Wound infection
Increased LOS
Increased blood loss
Increased CSF leak
Treatment algorithm for lumbar facet cyst
What is the most sensitive test for lumbar facet cyst
MRI GADO
What type of facet cyst have high recurrence 2
Mucoid
Hemorragic
List the Etiologies of pedicatric acute compartment syndrome
Trauma
Surgical positioning
Overexertion
Infection
Vascular insult
Snake or insect bites
What age people at risk for compartment and why
adolescent boys at particular risk due to rapid growing muscle in inelastic fascia
How long until tissue damage after ischemia from ACS
Begins 4hrs
Permanent 8hrs
Type of fracture that has highest rate compartment syndrome
>40% in tibias
11% of all tibia fractures
2 risk factors for compartment syndrome in tibia in peds
>14 yo
Tibial tubercle avulsion
3 factors that give you higher risk of compartment syndrome in pediatric tibia fracture
Patient >50 kg
Comminuted #
Neuro deficit prior to tx
Risk factors for peds ACS in forearm injuries 5
open fracture
longer surgical times
IM nailing
Neurologic injury
Displaced supracondylar and displaced forearm: 33% chance of ACS
How is Vascular PACS different to traumatic (4)
Often don’t present with pain (50%)
60% have vascular changes on exam
36% have motor deficit· poorer outcomes due to more rapid onset of ischemia
What surgery can risk ACS development (2)
High tibial osteotomites à advocate prophylactic anterior compartment fasciotomy
Prolonged dorsal lithotomy position
List the increasing 3 As for PACS
anxiety
agitation
analgesic requirement
What is the pressure tolerance in children
seem to tolerate >30mmhg with no problem as long as pressure not within 30mmHG of MAP
Initial management of at risk limb consists of? (4)
Maintain normotension
remove circumferential dressings
limb at heart height· supplemental oxygen
Should you extensively debride muscle in PACS?
Some evidence that children have more ability of spontaneous muscle recovery after myonecrosis
How does wound management differ to adults
Adults – wound that can not be closed at initial surgery likely needs skin graft
Kids à unlikely to need skin graft (12-21% of cases)
2 situations with poor outcome in PACS
UE worse than LE
Not associated with fracture
Q1: Form of inheritance of marfans (4)
D)
Q2: What’s the gene mutation in Marfan Syndrome?
A: Fibrillin-1 (FBN1) gene
Q3: How do marfans patients usually present?
A: Skeletal findings are usually the first manifestation.
Q3: What is the most common cause of death in marfan syndrome patients?
A: Aortic root dissection
Q4: What are the systemic manifestations of Marfan Syndrome?
Q6: What are the cardiovascular manifestations in Marfan Syndrome?
Aortic dissection
MV prolapse
Pulmonary artery enlargement
Left ventricular enlargement
Cardiovascular manifestations of Marfan Syndrome are usually managed with?
A: Beta blockers
Q8:Where is the lens dislocated in Marfan Syndrome?
A: Superolateral
Q9: What are the musckeloskeletal manifestations of Marfan Syndrome? 7
Pectus carinatum or excavatum
Abnormally long fingers (arachnodactyly)
Wrist sign (contralateral thumb overlaps the entire nail of the little finger when grasping the opposite wrist)
Thumb sign (when the entire distal phalanx of the clenched thumb protrudes beyond the ulnar border of the hand)
Scoliosis
Acetabular protrusio
Pes planus
Q10: How is scoliosis managed in patients with Marfan Syndrome?
Bracing, however less effective than in AIS. Usually in skeletally immature patients with 15-25 degrees curves
Surgery should be considered for curves > 45 degrees
Q11: Marfan syndrome patients with scoliosis are at higher risk of during surgery? (3)
Dural tears
Hardware failure
More revision procedures due to implant failure or fractures
What are the anatomical differences in the spine in Marfan Syndrome patients? (3)
Narrow pedicles
Wide transverse process
Vertebral scalloping
What is Dural ectasia and where is it most commonly found in Marfan Syndrome?
Definition: enlargement of the dural sac
Location: most common in the lumbosacral spine
Note: it is a highly specific finding of MFS and is present in more than ⅔ of patients with MFS
Q15: Pregnancy in patients with marfan syndrome:
Higher risk of aortic dissection because of hormonal and hemodynamic changes
Avoid hypertension throughout pregnancy
Beta blockers
What is the TMT joint complex composed of (3)
TMT joint
Intermetararsal joint
Proximal intermetatarsal joint
Describe the coronal plane anatomy of the TMT joint
3 cuneiforms + corresponding metatarsals have trapezoidal shape
MIddle cuneiform and 2nd metatarsal act as a keytone > inherent stability
How to find 2nd MT on lateral xray
Most recessed
What are the 4 main ligaments that stabilize the TMT complex
Transverse intermetatarsal ligaments: secure bases of second to fifth MT
Dorsal interosseous ligament: secure medial cuneiform to 2nd MT
Plantar oblique ligament: insert onto bases of second and third MT
Lisfranc ligament
In the TMT complex which ligaments are stronger
Plantar: that's why there is often dorsal subluxation
Name the 2 dynamic stabilizers of the TMT Joint
Tib ant
Peroneus longus
Name structure that often blocks reduction of space btw medial and middle cuneiform
Tib ant
Explain the concept of 3 columns of the TMT complex and its components
Medial column: Medial cuneiform and 1st metatarsal
Middle columb: Middle and lateral cuneiform + 2nd and 3rd MT
Lateral column: 4th + 5th MT + cuboid
In the TMT complex which column is most mobile
Lateral: shock absorber. Keep mobility when fixing injuries
Middle is least mobile
2 types of mechanisms for TMT joint complex injuries
Direct: crush injury to dorsal aspect of foot
Indirect: Axial and rotational force applied to plantar flexed/fixed foot
1 physical exam findinding highly associated with lisfranc injury
Echymosis plantar arch
X-ray findings of lisfranc injury (5)
On AP: Medial border of 2nd MT with medial border of middle cuneiform
On Oblique: Medial border of 4th MT with medial border of cuboid
On Lateral: No dorsal subluxation of MT
Fleck sign: avulsion # off base 2nd MT
Widening >2mm btw 1st MT/Cuneiform and 2nd MT
Best x ray to order if suspecting lisfranc + alternative
AP weight bearing of both feet on one cassette
Pronation abduction stress xray
Disruption of what ligament on MRI was indicative of TMT joint instability intraop
Plantar oblique
Describe classification of TMT injuries (based on 3 parameters) on x ray
Joint congruity
Location of involvement
Direction of instability
Describe indications for non op management for TMT complex injuries
Normal weight bearing x rays
If high suspicion need to do EUA to rule out instability
Describe fixation principle for Medial and lateral columns in complex TMT injuries
Medial: rigid fixation
Lateral: Flexible temporary fixation
Describe general principles of fixation for TMT complex injuries (what to fix first)
Start from proximal to distal
Continue from medial to lateral
Describe surgical approach for 3 column TMT complex injury (include mention of structures in danger)
2 INCISIONS
Dorsal-medial:
Btw 1st and 2nd rays
Dorsalis pedis and Deep peroneal nerve identified and mobilized lateral
Interval BTW EHL and EHB
Visualize 1st TMT and medial aspect of 2nd TMT
Protect branches of SPN in proximal part of incision
Dorsal Lateral
Centered over 4th MT
Visualize lateral 2nd MT + 3rd/4th TMT
Common extensor tendons mobilized medially
Muscle belly of EDB split in line with its fibers
Describe trick for better placement of retrograde lag screw from MT to tarsal bones in the foot
Burr a hole in dorsal cortex of MT to allow screw to countersink
Complex TMT complex injuries: what to look to for in the cuboid
Impaction of cuboid
Leads to shortening of lateral column > need to restore
Post operative protocol after TMT injury ORIF
NWB x 8 weeks
Supportive shoe wear at 3 months with arch support
Transarticular screw: very stable but violate joint
Dorsal bridging plate: equivalent biomechanical to screw
Tight rope: Cadaveric study show equivalent stability to screw
COmparable rates of complications and no difference in functional outcomes in small studies
Lisfranc injuries evidence behind ORIF vs Arthrodesis for ligamentous patterns (4 advantages
Arthrodesis group had
Improved functional outcome
Higher return to pre injury levels
Lower revision
Less pain
THIS IS THE QUOTED PAPER THATS SAYS ARTHRODESIS IS BETTER
Q) Explain the pathophysiology of muscle contractures
Reduced mobility = loss of muscle mass and length (decreased sarcomeres) = loss of elasticity in connective tissue = loss of ROM
Q) When are joint contractures considered irreversible and reversible based on animal models?
irreversible: 4 weeks
reversible: within 2 weeks if re-mobilise and muscles activated
Q) Name NON SURGICAL methods to treat joint contractures: (6)
Passive Stretching: lit conflicting results
Continuous Passive motion: no evidence in lit
Splinting: recommend static progressive splinting
Serial casting: TBI can be effective otherwise questionable
Neuromuscular Electrical Stimulation: Bad results
Botox: good short-term results in lit … l
What is the Strain Theory of Perren
Plating constructs must have mechanical characteristics that are neither too stiff nor too flexible to promote callus formation and avoid non union, malunion and implant failure
List the disadvantages of rigid compression plating popular in 70s-80s (2)
Excessive soft tissue dissection à reduces biology
Prolonged healing and porotic bone creation
List the 3 types of metals in fracture implants
316L stainless steel
Pure Titanium
Titanium alloy
Define Stiffness
Described as the Younges modulus of elasticity
Ability to resist deformation (strain) under certain stress
It is the slope of the Stress/strain curve
Define Fatigue strength
Also known as endurance strength
Ability of material to resist failure during cyclic loading at stresses under the ultimate tensile strength
What metal has the highest elastic modulus (stiffest)
Stainless steel
What metal has the best fatigue strength
Titanium: Can undergo repetitive high cycling at low stress as seen in ambulation
So what is main difference between steel and titanium? (2)
Steel can resist high stress but few cycles of it
Titanium can resist more cycles of low stress
How is stiffness related to the geometry of the implant
Rectangular implant – exponential of thickness
Nail – exponential to radius
What are the weakest areas of plates – stress risers? (3)
Reliefs – to preserve periosteal blood flow
Screw wholes
Sharp corners
What level of strain will promote primary bone healing
<2%
What level of strain (micromotion) will heal with callus
2-10%
If more will lead to fibrous tissue
What happens if construct is to stiff?
Not enough micro-motion and no callus
Eventual fatigue failure of implant
Why do you get more callus at far cortex in locked plates?
Bending of plate concentrates at fracture like a lever
creates more motion at far cortex than near cortex
2 advantages of hybrid screw fixation in a plate
Regular screw brings plate closer to the bone: better torsional strenght
Use of non locked screw at the end reduces stress at end of plate
List the variables that can change stiffness of construct (6)
Plate material
Plate thickness
Locking vs non locking
Screw density
Plate length
Working length
How long of a plate should be used?: simple #
Comminuted #
Simple: 7-8 times lenght
Comminuted: 2-3 lenght
What screw density should be used to maintain appropriate strain
should not exceed 40-50% of available holes
What is the working length
Distance between two closest screws or screw clusters to the fracture
What is a general rule for working length in #
At least 2 wholes should be left open on either side of fracture unless zone of commination is substantial
List the risk factures for distal femur non union (from 2 separate studies) (6)
Obesity
Open fracture
infection
Stainless steel plates
Short plate length
smoking
What plates have shown to have highest rate of healing in distal femur
Titanium locking plates (LISS)
Q2) What’s the sensitivity of the ADT and why?
50% when performed under GA
Due to the posterior horn of the medial meniscus acting as a secondary stabilizer
Pivot shift: why does the tibia reduce
Pull of the IT band as it passess posterior to the axis of the knee
Q7) True or False: a medial knee injury (i.e. MCL) can affect the reliability of the pivot shift test
True
Q9) What’s the most sensitive ACL test and what’s the most specific test in awake and anesthetized patients? (Meta-analysis)
The Lachman test was the most sensitive in both awake (81%) and anesthetized (91%) patients
All tests have similar specificity in awake patients, the pivot shift test is the most specific (98%) in anesthetized patients
Q10) How often is the ACL injured in patients with grade III MCL tear?
In one study, they found that in grade III MCL tears, the ACL was disrupted in 78% of the times
Q11) What is the primary restraint to valgus stress when the knee is at 0 degrees flexion and 30 degrees flexion?
At 0 degrees, the posterior oblique ligament and posteromedial corner complex
At 30 degrees, the superficial MCL
Q13) Which part of the MCL and LCL usually fail when injured?
MCL: the femoral side
LCL: the fibular side
Q14) What is injured if the knee opensm up in varus at 0 degrees flexion and 30 degrees flexion?
At 0 degrees: LCL, PLC, and or associated cruciates
At 30 degrees: LCL only
Q15) List 4 tests for the PLC?
Dial test
Posterolateral rotatory drawer
External rotation recurvatum
Standing apprehension test (Patient stands with both knees slightly flexed and internally rotates the torso away from the leg, if any feeling of instability or apprehension → +ve)
Q16) describe the external rotation recurvatum test and what does it indicate?
It tests the PLC
The examiner pulls on both big toes and hyperextension is then measured using a goniometer or heel to table distance is measured.
Q18) Describe the dial test, and what does it test at 30 and 90 degrees of flexion?
With the patient prone, both knees are flexed first to 30 , then to 90, with external rotation applied to the tibias at each position with the feet fully dorsiflexed
10 degrees difference At 30 degrees: PLC
10 degrees difference At 90 degrees: PLC + PCL
Q19) How often are PCL injuries associated with other ligamentous injuries in the knee?
97%
Q20) List 3 tests for the PCL and describe them?
Posterior sag sign: Supine, hips flexed 45 degrees, knees flexed 90 degrees. Loss of the normal anterior tibial step-off compared to the normal side
Posterior drawer test: in the same position the examiner pushes the proximal tibia posteriorly with the foot in neutral rotation to check for translation
Quadriceps active test: same position as in the PDT, patient is asked to contract their quads. If the tibia reduces then it’s positive
Q) What is the Sports hip triad?
1) Intra-articular hip injuries (FAI & labral tears)
2) Adductor & rectus abdominis muscles strain
3) Osteitis pubis/Athletic pubalgia
Q) What are the 4 broad categories of Athletic hip injuries?
Adductor strains
Osteitis pubis
Athletic pubalgia
FAI
Q) What does the Adductor complex include (5)? And which muscle is most frequently the source of pathology in athletes?
A) - Groin pain or medial thigh pain => exacerbated w/ resisted adduction & passive stretching.
Q) What is Osteitis Pubis?
A painful overuse stress inj. of the pubic symphysis that can cause lower abdominal pain or groin pain 2ry to excessive strain & motion of the joint.
Q) What causes Osteitis Pubis?
Injury to any of the muscles originating from pubic symphysis (Rectus abdominis “Elevates” Vs. Adductors “Depress”) => alters symphyseal biomechanics which could lead to cartilage degeneration.
Q) How to differentiate Osteitis pubis from Athletic pubalgia?
A) Spring test: pain with pressure on pubic ram >>pubic rami #
Q) What are the classic radiological findings in chronic osteitis pubis? (3)
Cystic change
sclerosis
pubic symphisis widening
Q) What is Athletic pubalgia (Also called sports hernia/core muscle inj)?
Unilateral or bil. Lower abdo pain that can radiate to perineum & prox adductors from repetitive pivoting & cutting (
Q) What are the clinical findings in Athletic pubalgia?
Pain with resisted sit ups
Q) What is the effect of cortisone inj. On Adductor strains?( in elite athletes)
Studies showed that in elite athletes with chronic Adductor strains w/out MRI findings of enthesopathy => Injections caused pain relief up to 1yr. Less improvement with MRI findings
Q) What are the surgical options to manage Athletic Pubalgia? (3)
) 1) Open repair with or without mesh reinforcement
2) Laparoscopic repair with mesh
3) Broad pelvic floor repair with possible adductor release/repair & neurectomy
Normal teardrop angle
What is it used for
70 degree: increased is worse
Assesment of volar rim displacement n DR #
Q) How to evaluate radiocarpal alignment in the sagittal plane?
A) By extending a line from the volar cortex of the intact radial shaft towards the carpus => should bisect the central axis of the capitate head
Q) What are the radiological goals of DRF surgical fixation? (5)
Radial shortening < 5mm
Radial inclination > 15 deg
Radiocarpal intra-articular step-off < 2mm
Sigmoid notch incongruity < 2mm
Tilt between 15 deg dorsal & 20 deg volar
Q) When is fragment specific fixation used in DR #
When unstable fracture fragments are too small of distal for adequate fixation w/ locking plates.
Q) What are the indications of Ex-Fix in DRF? (3)
1) Highly comminuted fractures when volar locking plate or fragment specific implant is not feasible.
2) Open fractures with moderate contamination
3) Medically unstable pts who cannot undergo a lengthy procedure
Risk factor for DR CRPS after surgery
Excessive distraction from Ex fix or bridge plate
Q) How do untreated & non-united ulnar styloid fractures with absence of DRUJ instability affect patients’ clinical outcome?
A) No effect on clinical outcome
Q) What position should the forearm be post op in cases of DRUJ instability?
A) Supination 60 deg for 6wks (Munster cast) => Followed by removable orthosis & starting ROM exercises.
Q) For how long should the Ex-fix or distraction bridge plate be kept?
Ex-Fix should not be kept more than 6-8wks then transitioned to cast or orthosis.
Bridge plate can be kept for 3-4 months till fracture healing
What is the average wear rate of high x-linked poly
0.024mm/year
How to assess poly wear on hip x ray
Hip AP: Distance from superior aspect of cup to superior aspect of head compared to distance from inferior aspect of cup to inferior part of head
Definition of a well fixed acetabular component
No radiolucent lines measuring > 1mm in any 2 zones on AP
Canadian study by beaule
Most sensitive way to measure Poly wear
RSA
What to use if exchanging head and find trunnion corrosion
Ceramic with sleeve adaptor
Revision THA for liner exchange..what structure do you release to improve exposure of acetabulum
Glut max insertion into femur: repair at the end
Wear rates of High x linked poly...compare thin (<7mm) to thick (>7mm) poly thickness
No difference
Main disadvantage of large femoral head
Increase the forces at the trunnion and are thought to contribute to corrosion
Size of cement mantle if planning to cement a liner into an existing cup
2mm
When cementing a poly into a cup...how can you increase the poly/cement interface
ROughen up the backside of the poly (from a study) and the cup with a burr
3 risk factors for failed revision using liner exchange only
Q1) List factors that are associated with better outcome in TKA revision for aseptic loosening: 3
Male
Low charlson comorbidity score
Higher pre op function
Q2) Name the classification of Bone loss of TKA and its categorie and treatment
Type 1
Minor and contained cancellous bony defects
Metal augments Type 2
2A: One femoral condyle or plateau
2B: Both femoral condyle or plateau
both treated with metal augments or structural allograft Type 3
Deficient metaphyseal segment, comprises major portion of the condyle
Metal augments, cones, sleeves, oncology procedure
Revision TKA what to use:Femoral defect <10mm
Femoral defect >10mm
Cement + screw
Cone, sleeve or augments
Q5) What is the indication to PS TKA for revision?
intact collateral ligaments without varus-valgus instability because the host soft tissue provides coronal stability
Q6) Unlinked constrained designs provide a taller and thicker polyethylene intercondylar post, which limits rotation, medial-lateral translation, and varus-valgus angulation. They experience what type of higher stress?
Torsional stress at fixation interface
Q8) What steps should be followed by Revision TKA - order?
Rebuild tibia platform
Reestablish flexion gap
Reconstituting extension gap
length of tibial stem in revision TKA that decreases the rate of loosening
30 mm
What decreases loosening rates in revision TKA, a tibial or femoral stem
Femoral stem
Q4) List some factors that may predispose patients to relapse of their clubfoot?
Clubfoot with arthrogryposis
Drop toe sign: big toe is plantarflexed in resting position with no active dorsiflexion
Q4) What position is the foot positioned in the foot abduction orthotics? for clubfoot
70 degrees for the affected side and 40 degrees for the unaffected side
The shoes are positioned at shoulder width
Q6) When is the best time to discontinue the denis brown bars (or any other FAO)? for clubfoot
Most centers recommend somewhere between 3-5 years of age. Never before 3 years
Q8) What is the first sign of relapse noticed by the parent? And how does it present? for clubfoot
Less dorsiflexion of the affected foot
Dynamic supination
Q11) When can a tibialis anterior tendon transfer be done? for recurrent clubfoot
In patients who experience a relapse at age >= 2.5 year. Full tendon transfer and not split
Q12) How can you manage a “bean shaped” foot?
A combined cuboid-cuneiform osteotomy (opening wedge for the cuneiform, and a closing wedge for the cuboid)
Usually done at the age of 4-9 years (mainly to have an ossified medial cuneiform)
Q13) What’s the rate of PMR after the Ponseti method has been implemented?
<5% require any release surgery after being treated with the posetti method
In recurrent clubfoot what is the indication for achilles tendon lenghtening
Unable to achieve 15 degrees of dorsiflexion
How much does blood flow to fetus change when in hypovolemic shock
o Reduces 10-20% which risk hypoxic injury/death
Is leukocytosis reliable in pregnancy?
o No – can increase to 18000cells/ul
Describe physiologic changes in pregnancy
When is transient osteoporosis the worst in pregnancy?
o Third trimester
o 80% of calcium required for fetal development is taken from mothers calcium deposits
o Resolves within few weeks after delivery
List the considerations in early evaluation of oregnant woman
o Treat like regular ATLS
o Mother life over fetal life at all times
o Maternal/fetal blood mixingo Prevention of aortocaval compression (past 20 weeks)
What is fetal-maternal hemorrhage?
o Development of RH D antibodies by mother to RH+ blood of fetus
o Test mother for RH D
o If negative give RhD immunogloblulin to mother after trauma within 72 hrs
Describe supine hypotension syndrome
o aortocaval compression from fetus
o Leads to decreased venous return and cardiac output
o Sympt: Dizziness, pallor, tachy, sweating, hypotension
What position should pregnant patient be placed in during evaluation
o 15-30 deg incline
o Angle to left
What are the indications for prolonged cardiotocographic fetal monitoring
Uterine tenderness
Abdominal pain
Vaginal bleeding
Sustained contractions
Ruptured membranes
Abnormal heart rate
Serum fibrinogen < 200
What are the guildlines for radiation during pregnancy
o Cumulative radiation should not exceed 5rad during preg
When is the effect of radiation worse to a fetus
3-15 weeks: teratogenic
Risk of DVT in pregnancy 3
5x regular patinet
if bmi >25 OR 62
common ortho antibiotics contraindicated in pregnant women
Genta/tobramycin
What are the main anesthetic concerns during 1st trimester (2)
o Fetus at largest risk
o GA is considered safe but try for local / regional if possible
Are local anesthetics safe? during pregnancy
yes at all standard doses
what is the best trimester to operate on during pregnancy
second trimester
best anticoagulation for pregnancy
LMWH
+ compression stockings
What are the indications in pelvic fixation in pregnancy
diasthasis >4cm
4 indications for cesarean delivery in pregnancy
Fetal HR <100
Prolonged decelerations
Recurrent late decelerations
Fetus older than 26 weeks
How long can the fetus grow with a supra-acetabular ex fix
34 weeks
what type of fracture gives a high rate of abruption and bad outcomes
Pelvic fractures
Q) What are the nerves & their branches that supply the wrist joints? 5
1) Ulnar => Dorsal sensory branch of the ulnar nerve (DSBUN) => Supplies Ulnar side of the wrist + 2nd-5th CMC joints
2) Median => AIN, Palmar cutaneous branch, thenar motor branch => Supplies carpal joints
3) Radial => PIN & radial sensory branch
4) Lat. ant brachial cut => 1st CMC joint & radial side of the wrist 5) Med. ant brachial cuy
Q) What are the indications of wrist denervation?
Chronic wrist pain > 6 months refractory to non-surgical management
Must be skeletally mature (No upper age limit)
Q) List the absolute contraindications to wrist denervation. (3)
Active infection
cognitive impairment
Poor patient compliance
What approaches are used for wrist denervation & which nerves involved for each approach?
Q3) Do NSAIDs increase the risk of nonunion post spinal fusion?
Retrospective study looked at ketorolac post spinal fusionHigher nonunion with ketorolac
Meta-analysis of 5 retrospective studies:Higher nonunion with high dose ketorolac (>120mg/dL)But no detrimental effects of short-term use of NSAIDs at normal doses
NSAIDs have a dose dependent effect on spinal fusion
Q3) Does the use of NSAIDs post-operatively decrease the use of opioids?
Yes!
What is the benefit of multimodal analgesia 3
Improve pain control
Reduce opioid consumption
Facilitate rehab
Q4) Should we be using NSAIDs in spinal fusion? Which type should be used?
Yes (level 1 evidence)COX-2 inhibitors or short term, low dose nonselective COX inhibitorsDoes not affect spinal fusion rates
Q5) What’s the MOA of neuromodulatory agents (Gabapentin, Pregabalin)?
They reduce neoronal excitability via inhibition of the alfa2-delta subunit of the Ca-gated channels on presynaptic axons
Q6) Do neuromodulatory agents help in post-operative pain management? 3 benefits
Reduction opioid use
Lower PCA use
Improved functional outcomes up to 3 months
Q7) is the use of an intraoperative epidural steroid injection beneficial after a lumbar discectomy surgery? outcomes 5
Yes (Level 1)
Reduced LOS
Reduce post-operative pain and opioid use
No difference in leg or back pain after 2 years
Similar revision rate
Q8) Is an intrathecal single dose opioid injection useful in spine surgery? 2 benefits and one disadvantage
Good post op pain control
41% less PCA use
Increased foley insertions
What test for herniations L1-L4
Femoral nerve stretch
Is the a role of oral corticosteroids in acute management of LDH (lumbar disc herniation)
Recent RCT showed no benefit over placebo
When to consider Sx for lumbar disc herniation in athletes
After 6 week course of non Sx management fails
What is the biggest risk factor for DDD
Genetics
Does athletic competition contribute to DDD?
Yes - elite athletes does add risk
What is the patholgensis of DDD
Loss of disk hydration → disk space narrowing → tears → eventual anklysosis + facet degeneration
What are modic changes?
Changes in end plates that correlate with positively to presence of LBP
What is indication for Sx for DDD in athletes
failed non op at least 6 months!
Contrast to disc herniation → 6 weeks
Should provocative diskography be used?
No - can lead to quicker degeneration
What are negative predictive factors of successful Sx of DDD (4)
Narcotic abuse
Smoking
Workers comp
Number of of fusion levels
What is the healing rate of an acute spondylolysis
90%
Is bracing effective for spondylodysis
not harmful but no difference in outcome
5 strategies to minimize blood loss and prevent allogenic transfusion in adolescent spine surgery
PreoperativePreoperative hgb level to 50 higher than your transfusion threshold
Iron, erythropoetin, autologous predonation
AnestheticControlled hypotension
Epidural blockade
acute normovolemic hemodilution
Pharmacologic
Antifibrinolytic agents: TXA
Desmopressin: not great evidence
SurgicalHemostatic products
Cell saver
Post opPhysiologic transfusion triggers
What are the risk factors for transfusion is adolescent spine surgery (6)
Surgical tine
>6 levels fused
Neuromuscular scoli
CObb angle >50
Use of ponte osteotomies
Low body weight
Q) What pre-operative blood test can help predict transfusion need?
A) A, preoperative fibrinogen level may predict increased blood loss and transfusion needs in AIS
Q) Name risk factors for post-op infection in neuromuscular patients having scoliosis surgery (2)
Low albumin levels: <35g/L
Total lymphocyte count (<1500)
Q) What is the main blood loss in AIS surgery from (MCQ)
C) – main blood loss from decortication which is venous … therefore lowering BP during case from anesthesia is not as effective as in other ortho cases
Q) Name physiological transfusion triggers 4
Tachycardia
Hypotension
Urine output <0.5
Acidosis
3 indications for operative management of chronic anterior pelvic instability
> 5mm vertical displacement on flamingo view
> 10 mm diastasis on AP
Acute symphysis rupture after birth
Operative mgmt of chronic anterior pelvic instability (2)
Symphyseal arthrodesis: use bone graft from iliac crest
Look for symptoms of posterior instability
How to do stress x rays of pubic symphysis
Single leg standing view or flamingo view
Normal vertical displacement is < 5mm
Normal distance btw pubic symphysis
4.4 mm
Normal amount of motion at pubic symphysis
<2mm translation
<3 degrees rotation
Strongest ligament in pubic symphysis
Arcuate ligament: located inferiorly
Q2) What’s the percentage of patients who are not satisfied after TKA?
20%
Q5) What’s Condylar liftoff and what does it signify?
The difference between medial and lateral condylar heights in relation to the tibial baseplateIt measured coronal stability
Q7) When placing the tibial tray, Where do you set the rotation of the tray?
Medial ⅓ of the tubercle
Q12) How does elevating the joint line affect patellofemoral biomechanics?
Every 1cm of joint elevation increases PF joint contact force by 60%
What is quadriceps avoidance gait?
patient bears weight with a knee locked in extension because of either a weakened extensor mechanism or pain.
List the functions of the meniscus (5)
load bearing
shock absorption
joint stability and congruity
joint lubrication
Proprioception
Q) What is the main challenge in oncologic resection in pediatrics when compared to adults?
LLD: bc of physis
Q) How to manage LLD post tumor resection?
- < 2cm => No intervention
- 2-5 cm => Contralateral hemiepiphysiodesis
- > 5cm => extendible prosthesis or 2ry limb lengthening
Q) What are the benefits of a vascularized autograft Vs. Allograft?
Less risk infection
Higher union
Less fracture
Q) What are the recon options for Intra-articular resection?
Endoprosthesis
Osteoarticullar allograft
Q) What are the advantages of compressive osseointegration implants? 2
1) Decrease periprosthetic osteolysis
2) Preserve bone stock as less bone needs to be cut & reamed
Q) What are implant options to address potential LLD post tumor resection? 2
Invasive expandable implants
Non-invasive expandable implants
Q) What is the percentage of peds MSK tumor pt. Requiring amputation?
10%
Q) What are the advantages of rotationplasty over the other options?
1) Less energy expenditure (When compared to AKA)
2) Avoids phantom pain
3) Decreases the need for further Sx for LLD, revision of loosening or #.
Q) What are the prerequisites of a rotationplasty & what structures need to be preserved intra-op? 2
Functional ankle joint
Functioning sciatic and its branches
Q) What did a cross-sectional, multicenter study comparing short term (1-5yrs) functional ability & physical activity comparing different limb salvage & ablative Sx show?
A) No functional difference on the basis of treatment type
Q) How is the QOL of a rotationplasty pt when compared to the general population?
A) Long term QOL after 17yrs were nearly the same
What are the anatomical differences in between medial and lateral menisci
Lateral → more circular and mobile, medial → more firmly attached to the capsule and medial collateral ligament and is subject to greater forces ( increased tear rate)
Describe the apltey test
ProneFlex knee to 90
Use 2 foot high table and place your knee on posterior theight
Grind knee in ext rotation ((for medial) and int rotation ( for lateral) in:
Compression → miniscus pain
Distraction → rotational ligament pain Neutral
What is the clark test?
Active patala grind test
Hold patella with webspace of thumb finger and ask to contract quads
What does patellar apprehension test tell us about patients?
Specific for patellar instability → + is strongly indicative of symptomatic instability
Describe the Wilson test for OCD
supine with the knee flexed to 90 and the tibia rotated internally. >>knee extended >>pain
+ test: patient reports pain as the tibial spine abuts the OCD lesion on the medial femoral condyle at approximately 30 short of full extension.
Q) what is the goal of ankle distraction arthroplasty?
A) optimize the body’s regenerative capacity and the function of the diseased joint. An external fixator is used to mechanically unload the ankle to relieve pain, preserve ROM, and potentially delay or even partially reverse the effects of arthritis.
A displacement of 1mm of the talus in the mortise accounts for how much joint contact area loss?
C)
Q) what is the minimum ROM of the ankle to consider distraction arthroplasty?
20 degrees
Q) Name factors associated with cartilage repair in distraction arthroplasty: 4
- decrease in joint reactive forces (shear)
- an increase in protoeglycan synthesis
- recruitment of mesenchymal stems cells
- optimization of the mechanical environment
Q) What is the average time to see benefits for joint distraction arthroplasty for patient
A) Can take up to 12-24 months
Q) List relative contraindications for ankle distraction arthroplasty: 6
- complex regional pain syndrome
- inflammatory arthritides
- previous infection
- neuropathic joint
- older patients with low demand
- flat top talus
What is second impact syndrome in the context of concussion
Closely timed repeat injury that can result in brain swelling and even death
What is Chronic Traumatic Encephalopathy
Neurodegerative disease associated with head trauma Diagnosed post mortum on histology Deposition of tau protein
What are the indications to transfer an athlete to emergency departments after sideline evaluation
GCS <15
Deteriorating mental status
Progressive worsening neuro signs
Potential spine injury
What co-morbilities have been associated with prolonged recovery in concussion (7)
Prior concussions
Loss of consciousness during injury
Female
ADHD
Depression
Migranes
Describe the treatment plan for a concussion
acute : physical and cognitive rest
Return to activity after patient is asymptomatic
Procedure through a graduated stepwise return
Must be symptom free for 24hr period before any advance in steps
Min time 1 week
If symptoms occur - go back to step where no symptoms
Describe return to play protocol for concussion 6
No activity
Light aerobic exercise
Sport specific exercise
Non-contact training drills
Full contact practice
Return to play
Q1) Do Bisphosphonates have any effect on vertebral morphology in OI?
Yes when started earlier in life
Have positive effect on morphology of vertebrae
3 benefits of bisphosphonates in spine OI
Better cortical bone
Improves pull out strenght
Decrease scoliosis progression in type 3
Scoliosis and PFT function if: <40 degrees
> 60 degrees
VC 78%
VC 41%
Incidence of craniocervical junction anomalies in OI
37%: basilar invagination or impression
Stringest predictor is predictor of OI
Q9) List some intraoperative considerations in patients with OI? (5)
Fractures while positioning and transferring the patient
Airway difficulties due to large head, large tongue and short neck
Poor pulmonary function due to chest wall deformities
The use of succinylcholine should be avoided because fasciculations can cause fractures
Blood loss: blood should be available, controlled hypotension, and TXA
Rate of infection in post TKA that required a return to the OR to manage wound complications (For example hematoma)
6-10%
Definition of persistent wound drainage post TKA
Continuous drainage >72hrs post op: Allows for early intervention
5 medical conditions that can be optimized prior to TKA to improve wound healing
DM: 5X risk infection
RA: 2-3X risk infection
Smoking: stop 6-8 weeks before TKA (2X rate)
Obesity: 6X risk of infection
Malnutrition
Describe the 3 stages of wound healing
Inflammation phase: Time of incision to 4-6 days post op >>Fibrin clot serves to attract cells and concentrate cytokines and growth factors
Proliferative phase: day 4-14. Epitheliazation, angiogenesis and granulation tissue formation.
Maturation phase: Day 8 to 1 year, collagen synthesis and organization
What is the percentage of wound strength at 1 week, 3 weeks, 3 months
3% at 1 week
30% at 3 weeks
80% at 3 months
Wound strength never back to normal because of les organized tissue
3 lab values that are associated with increased risk of wound complications
Albumin <3.5 g/dL
Lymphocyte count <1500cells/mm3
Transferrin < 200mg/dl
Blood supply to the anterior skin flat arises from?
Deep perforators from the medial side of knee
Blood supply to patella (5)
From patellar anastamoses of the following vessels
Supreme geniculate artery
Medial superior geniculate
Lateral superior geniculate
Anterior tibial recurrent
Branch from profunda femoris
Which incision to use in a TKA if there are multiple old incisions
The most lateral since the blood supply is medial
Limit of skin bridge between new and old incision
Ideal > 7cm
In a TKA how do you determine that the skin incision should be lengthened
If the apices have a U-shape instead of a V-shape
Q) What is C. difficile?
A) Gram-positive anaerobic bacillus that is ubiquitous in the environment, particularly in healthcare institutions.
What is the strongest risk factors from c diff on ortho
Deteriorated physical status & use of more than 1 Abx were the strongest risk factors
Rate of C diff infection is 8 times greater in orthopaedic trauma than in elective arthroplasty.
Risk factors for c diff 12
Age >65
Comorbidities
History of GI surgery
Antibiotic exposure
Medications
Tube feeding
ORTHO
Surgery >24 hrs after admission
Revision
Increased LOS
Q) When is it recommended to test hospitalized pts for C. diff?
A) 3 or more loose stools in 24hrs with no other explanation
Best test for C diff
Toxigenic culture
The one that is currently done is toxin immunoassay
How to treat c diff
MIld: Oral FLagyl TID x 14 days
Moderate: Oral vanco QiD x 14 days
Severe: Oral vanco + IV flagyl
Recurrent: Oral vanco in tapered regimen
3 antibiotics that predispose the most to c diff
Clinda
Fluoroquinilone
Vanco
DDx of pediatric foot mass 8
In order of most common
Ganglion cyst
Fibromatosis
Exostosis
GCT tendon sheath
Lipoma
NOF
Schwannoma
Enchondroma
list 5 malignant foot massess in peds
Synovial sarcoma
Liposarcoma
Fibrosarcoma
Osteosarcoma
Rhabdomyosarcoma
5 tumor features of foot mass that warrant investigation
Rapid growth
Fam hx
SIze >5cm
Deep fascial layer
incresed vascularity
Q9) What are the most common soft tissue tumours in children?
Hemangioma
Q11) What is the most common neurogenic tumor of the peripheral nerves in pediatrics?
NF
Q12) What is a target sign and which tumour gets it?
NF
Q13) What are the 2 types of osteochondroma?
Pedunculated
sessile
Q14) Indication to excise osteochondromas in a growing child?
When the lesion causes pain, deformity,or growth disturbance
Increases in size out of proportion to the child’s growth
Q15) What is Trevor’s disease?
Dysplasia epiphysealis hemimelica (exostosis)
Tarsal bones > distal femur > proximal tibia
Involve ½ of the epiphysis (medial)
Q16) What is the most common soft tissue sarcoma in the foot in pediatrics?
Synovial sarcoma
Q17) what is the most common bony sarcoma in the foot in pediatrics?
Osteosarcoma
Proposed benefits of cryotherapy (3)
Control local edema: promote local vasoconstriction (study in ankle trauma shown 74% effective)
Decrease pain: systematic review shows it improves pain in soft tissue trauma and short term pain reduction with acute back pain
Decrease muscular spasm
What is the use of contrast baths and is there evidence behind it
Tested in inflammatory conditions like CTS and RA: No difference is size of hand
Evidence behind whole body cryotherapy
2 studies show decrease in delayed onset soreness
Is it a good idea to cool a pitcher’s arm right before putting him back on
No: study showed decrease performance and accuracy after acute cryotherapy
What is the proposed mechanism of action of thermotherapy
Reduces muscle guarding because it increases muscle laxity and decreases viscosity of connective tissues
What has thermotherapy been shown to improve
Increased ROM when combined with stretching vs stretching alone
Acute low back pain: use hot or cold pack
RCT showed no difference btw hot and cold when pt also took NSAID
Mechanism of action of therapeutic ultrasonography
Raises soft tissue temperatures by transforming waves to mechanical energy in the soft tissues
Advantage of ultrasonography over heat packs
Depth of penetration: depends on frequency used
Name 3 proposed benefits of therapeutic US
Increases pain threshold
Reduces edema
Reduces inflammatory infiltrates and exudates
3 types of electrical stimulation used for rehab
Electrical stimulation of tissue
Electrical stimulation of muscle
Electrical stimlation of nerves
Theory behind TENS stimulator
Gate control theory: Increased activity of nociceptors in response to afferent input causes presynaptic inhibition. This diminishes sensation of pain
Mechanism of action of TENS
Delivery of pulsed current
Activate descending inhibitory pathway to reduce hyperalgesia
WHat is IFC and how does it work
Interferential current
Same principle as TENS but current comes from different angle so its effect is maximized in the deeper tissues
What has IFC been shown to help with
Post operative pain and swelling in the context of meniscectomy, ACL recon and chondroplasty
What is iontophoresis
Uses electrical current to deliver chemical or a drug to biologic tissue
Give a clinical application of iontophoresis
Delivery of dexamethasone onto tissue or lidocaine
SHown effective in athletes treated for lateral epicondylitis but only in the short term
What is the most common affected finger in FDS rupture (jersey finger)?
Ring finger
What is the overall management of FDS rupture and what should be considered for athlete?
All surgical
Long rehab: 4 months
Which type is most urgent (jersey finger)
Type 1: retracted to the hand
Leads to contraction
What is quadriga effect?
Decreased excursion to terminal flexion of the repaired digit limits excursion in the other digits
Occus if >1-1.5cm
Biomechanically what gives less gapping at repair site for tendon-bone repair in FDS rupture?
2 Suture achors placed at 45 deg distal to proximal with locking stich in tendon
WHat is the Crimp Grip position?
MPJ extension, PIP flexsion and DIP extension
WHat pully commonly injured in the hand?
A2 and A4 in climbers
Only A4 in pitchers
What is the management of pulley rupture in hand?
If no bow stringing can manage non op
Grade 4 → Op: bowstringing and multiple pulley ruptures
What is the combo that gives most pain? for ECU intability
wrist flexion, ulnar deviation, and supination against resistance
What is the primary stabilizer of the ECU?
ECU subsheath
What is the non op protocol? for ECU instability
Short arm or long arm splint in wrist extension and pronation x 2-4 weeks
What is done surgically? for ECU tendon instability
Direct repair of subsheeth vs reconstruction with extensor retinaculum
No need for groove deepening
What are the stablizaers to radial force on thumb?
UCL (primary) → 2 components: proper and accessory
Dorsal capsule
Volar plate
Osseous congruity
WHat is a stener lesion
Retraction of ligament resting proximally and superficially to the adductor aponeurosis
How do you examine UCL thumb injury
Valgus stess at 0 → aUCL
Valgus stress at 30 deg → pUCL
Laxity of > 35 deg oir 15 deg more than other side in both 0 and 30 of flexion = complete rupture (Grade 3)
Indications for Surgery? in UCL thumb
Any Grade 3 in high level athlete
Grade 3 with ligament displaced >3mm in anyone
Stener leison
Bony avulsions → often rotated and lead to chronic instability
What nerve is at risk durring approach? for UCL thumb
Superfical branch of radial nerve
What muscle is split to get to UCL thumb?
Adductor aponeurosis
Q) Name 6 different method to models orthopedic problems:
Static rigid body
Dynamic msk
Inverse dynamic
Forward dynamic
Finite element
Hybrid
Q) Name model methods that use a single vector to represent orthopedic forces involved:
Static rigid body
Dynamic MSK
Inverse dynamic
Q) What is the most widely used technique to model orthopedic problems?
Finite element model
Q) Which orthopedic modeling technique requires tracking system to estimate motion (motion analysis lab)?
A) - Inverse Dynamic
Q) Researchers use forward models to investigate?
C)
Q) What is the WHO classification of obesity?
class I: 30.0 to 34.9 kg/m2
class II: 35.0 to 39.9 kg/m2
class III: 40.0 kg/m2
Q) BMI affects?
A)
Q) Name the preoperative consideration of Obese patients requiring TKA:
A) - Comorbidities: many require attention, ex. DM
- Nutrition: most obese patients are malnourished (albumin ,3.5 g/dL, transferrin < 200 mg/dL and total lymphocyte count <1,500/mm3)
- Weight loss (no study showed significant (loosing 5% of body weight) weight loss prior to - surgery affects outcomes)
- Bariatric surgery prior to TKA (good literature results to decrease complications and re-operation)
Q) Name intraoperative consideration for TKA in obese patients:
- Exposure – advocate standard medial parapatellar approach
- Alignment – low threshold for navigation
- Blood loss – no obvious evidence of increase in blood in obese patients
- Fixation – No literature proof of loosening but suggest stemmed implants
- Anesthesia concerns – ventilation, spinals, blocks etc.