PANRE - Pulmonary XX

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How do the kidneys generate (adding in serum) new HCO₃^-?

By eliminating H⁺ from the body (one-for-one exchange).

(p. 123)
Where are all of the HCO₃^- virtually absorbed?

PCT

(p. 123)
Recall the differential diagnostic mnemonic for anion gap metabolic acidosis.
"MUDPILERS"
- Methanol
- Uremia
- DKA / Alcoholic KA
- Propylene glycol
- Isoniazid, Infection
- Lactic acidosis
- Ethylene glycol
- Rhabdo / Renal failure
- Salicylates
(p. 123)
Recall the differential diagnostic mnemonic for non-gap metabolic acidosis.
"HARDUPS"
- Hyperalimentation
- Acetazolamide
- Renal tubular acidosis
- Diarrhea
- Uretero-pelvic shunt
- Post-hypocapnia
- Spirinolactone
Too much acid or little bicarb.

(p. 123)
This is a chronic fibrotic lung disease secondary to inhalation of mineral dust.

Pneumoconiosis

- dust is ingested by alveolar macrophages --> inflammatory response --> parenchymal fibrosis ==> restrictive lung dz (decr. lung compliance)
What are the interventions involved in supportive management of pneumoconioses?
- 1. Bronchodilators
- 2. O2
- 3. Vaccinations (influenza, pneumococcal)
- 4. +/- corticosteroids (for silicosis alveolitis)
- 5. rehab
What is the only medical treatment to reduce mortality in COPD?

Oxygen

(p. 78)
Explain the pathophysiology of physiologic low V:Q ratio and give 3 conditions that exemplify this phenomenon.
- - Hypoxemic response to decr. PaO₂ is local hypoxic vasoconstriction --> pulmonary HTN.
- - RV works harder to pump blood into pulmonary arteries --> RA enlargement & RVH --> eventually leads to R HF (cor pulmonale).
ex. chronic bronchitis, asthma, acute pulmonary edema

(p.78)
__________ is the term used for areas with no perfusion & normal ventilation.

Dead space

(p. 78)

Author

NavyArmy

343457

Card Set

PANRE - Pulmonary XX

Description

Review of pulmonary disorders

Updated

2019-04-21T22:38:00Z