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What are chronotropes
drugs that impact impulse
- (+)hr ex. epinephrine
- (-) hr ex. digoxin
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P wave represents the
length
If the problem is in the P wave the problem is in the atria
should be less than 0.11
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QRS represents
length
QRS= ventricular depolarization ( stimulation)
should be less than 0.12 sec
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T represents
ventricular repolarization ( relax)
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U represents
the repolarization of Purkinje fibers
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PR represents
and the normal time
time needed for the SA node to be stimulated ( atrial depolarization) and the conduction to AV node before ventricular depolarization
.12-.20 ( if its longer = heart block)
really should be called PQ
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ST represents
ventricular repolarization
2+ blocks above= MI
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Normal Sinus Rhythm
- keep assessing
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NSR but 60bpm
Give atropene ( to speed up the hr)
0.5-1mg q3-5min 3mg total
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rate 100-150bpm
might lose a P wave
- cause: anxiety, fear, bleeding
- treatment: metoprolol ( beta blocker)
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What to do for dysrhythmias?
O- MI
oxygen, monitor, IV
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When to defib? what to make sure of?
- Defib the Vfib and pulseless V-tach
- - in an emergency ( no pulse)
start at 120J ( biphase)
- Not in sync mode
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What is cardioversion?
Sync with QRS
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Sinus arrhythmia
Normal except the PP, and RR intervals are irregular
if no hemodynamics are effects = dont treat
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Premature atrial contractions
- Rhythm=irregular the P can be early
- ( atria beats 2x before the ventricle picks up the contraction)
Need full minute strip
if its less than 6PAC/min dont treat
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Atrial fibrillation
- ALWAYS irregular ( NO PR)
- -atria is asynchronous with ventricle --> blood pools ( stroke risk)
Atrial rate 300-600
acute:
- - Ca+ channel blocker diltiazem
- - push 10-20mg
- -drip 5-10 mg/hr
- - Check TEE is (-) then cardiovert
- - give digoxin while waiting
chronic: give warfarin and monitor INR
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A flutter
Atrial Flutter
- 1) Md do vagal maneuver
- 2) adenosine ( slows AV node)
- - will cause asystole
- give 6mg, 12mg, 12mg then 20ml saline flush
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PVC
Premature ventricular contraction
Extra impulse in the ventricle
cause: dig toxicity(2+), nicotine, caffeine
More than 6PVC/min= get MD, on the T wave side
- amiodarone
- 150mg/10 min
- 1mg/min 6hr
- 0.5 mg/min 18 hr
monitor lungs, bp, thyroid
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Bigeminy
trigeminy
Bigeminy: every other beat is a PVC ---> emergency about to go to v tach
Trigeminy: every 3rd beat
- give lidocaine if amiodarone is not available
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Couplet + triplet
Couplet + triplet
- 2PVC in a row = couplet
- 3 PVC in a row = v tach--> triplet
Emergency give amiodarone
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Unifocal ,multifocal
unifocal : PVC 1 area of vent
multifocal PVC 2 area of the ventricle
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Junctional
Junctional Rhythm
- AV node takes over as the pacemaker
- - Absent/ inverted P wave
- Give atropene
- 0.5-1mg q3-5min 3mg total
If atropene doesnt work do defib
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SVT
- Supraventricular Tachycardia
- Sudden 150-200 bpm
- Narrow complex
- - No P wave, PR not measurable
cause: underlying rhythm afib, aflutter
Treat:
check hemodynamics if unstable cardiovert
stable: vagal , adenosine 6,12,12 , betablocker
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Vtach
Vtach
- wide complex >.12 sec
- P waves buried 100-200
1) check carotid, femoral
- Vtach + pulse, hemodynamically stable
- - amiodarone
- load 150 mg over 10 min
- 1mg/min 6hrs
- .5mg/min
- Lidocaine 1-1.5mg/kg IV 2-3min
- 1-2mg/min
toxicity SAMS ( slurred speech, seizure, muscle twitch)
- Hemodynamically unstable
- - cardiovert or defib?--look up
- -Epi q3-5min 1mg IVP
- -amiodarone
- 150mg over 10min 1mg/min 6 hr, .5/min 18hr
Check for reversible causes
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Drugs that can go down an ET tube if there is no IV access
NAVEL
- Narcan
- Atropine
- Vasopressin
- Epinephrine
- Lidocaine
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H's + T's
- Hypovolemia
- Hypoxia
- Hydrogen
- Hypothermia
- Hypokalemia
- Hyperkalemia ( 10U insulin, sodium bicarb, D50W)
- Tension pneumothorax- need compress 2nd mid
- Tamponade- pericardiocentesis
- Toxins
- Thrombosis pulmonary/coronary
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Ventricular Fibrillation
- Defibrilate q2min
- -Epi q3-5min 1 mg
- -Amiodarone - load 150mg/10min 1mg/min 6hr .5mg/min 18hr
Look for Hs, Ts
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Asystole
Asystole
CPR NOT Defib
- Epi: 1mg IVP q3-5min
- Must confirm asystole in 2+ leads= death
Reversible causes H's+T's
-
no pulse
PEA
- Mimics NSR but no pulse ( hearts not pumping but the electrical work is)
- CPR
- epi 1mg IVP q3-5min
- NO atropine
H's + T's ( usually hypovolemia)
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Torsades
Torsades de Pointes
Mg deficiency --< give 1-2g Mg IVP
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ROSC
ROSC
Pulse is back post CPR
- low bp: Ise 1-2L of lactage ringers, or NS
- if it doesnt work start vasopressor
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Idioventricular or Ventricular escape rhythm
idioventricular or ventricular escape rhythm
- impulse starts below the AV node
- vent 20-40
- 40+ = accelerated junctional rhythm
Treat
- CPR
- Epi 1mg q3-5min
- NO atropine
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v standstill
Ventricular standstill
atria is firing but no ventricle response
CPR no pulse + Epi
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1st degree
- 1st degree AV Block
- PR prolonged
atropene .5-1mg
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2nd degree AV Block, Wenckebach, Mobitz Type 1
- 2nd degree AV Block, Wenckebach, Mobitz Type 1
- irregular
- More P than QRS
- progressive PR it widens until its drops
- atropene
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2nd Degree AV block Mobitz type 2
2nd Degree AV block Mobitz type 2
more P> QRS , QRS=abnormal
- about to get pacemaker
- no atropene
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3rd degree AV block , complete heart block
3rd degree AV block , complete heart block
must get pacemaker
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Whats failure to capture?
fired at the right time
hearts not responding
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Failure to sense
fired spikes at the wrong time
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3 stages of shock
1) Compensatory : HR goes up, BP norm
- cool skin
2) Progressive: Bp goes down, organs suffer
- 3) Irreversible
- bp down, hr up MOD= 2+ org fail
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