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Atrial fibrilation
- - the most common chronic arrhythmia
- -incidence and prevalence increase with age
- - it is low amplitude fibrillary waves without discrete p-waves and an irregularly irregular pattern of QRS complexes
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what can a-fib lead to?
- a significant decrease in cardiac output and is the most common cause of embolic cerebrovascular accidents
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what is also called "holiday heart" ?
-a fib, when caused by excessive alcohol use or withdrawal
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a fib sx
- asymptomatic, SOB, chest pain, palpitations
- +/- ashmans phenomonon
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what is ashcans phenomenon?
- in a-fib
- occasional aberrantly conducted beats (wide QRS) after short R-R cycles
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Etiology of A-fib?
- "PIRATES"
- P - pulmonary disease
- I - ischemia
- R - rheumatic heart disease
- A - anemia/atrial myxoma
- T - thyotoxicosis
- E - ethanol
- S - sepsis
- Chronic a fib = HTN, CHF
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types of atrial fibrillation?
- paroxysmal
- persistant
- permanent
- lone
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paroxysmal a fib
self-terminating within 7 days (usually <24 hours)
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persistant A fib
- fails to self terminate, lasts > 7 days
- requires termination; medical or electrical
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permanent a fib
persistent a fib >1 year
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lone a fib
paroxysmal, persistant or permanent WITHOUT evidence of heart disease
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a fib tx
- - rate control with beta blockers, CCB, or digoxin
- - anticoagulant with warfarin
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atrial flutter
regular sawtooth pattern, an atrial rate of 250-300 bpm, and a narrow QRS complex
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who does atrial flutter usually occur in?
puts with COPD, HF, atrial septal defect or CAD
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atrial flutter sx
usually asymptomatic but can present with palpitations, syncope, and lightheadedness
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atrial flutter tx
- -anticoagulation
- - rate control
- - beta blockers or CCB
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atrioventricular block
characterized by refractory conduction of impulses from the atria to the ventricles through the AV node and/or bundle of His and is divided into: 1st degree, 2nd degree (mobitz 1 & 2) and 3rd degree
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first degree AV block
- - all atrial beats are conducted to the ventricles but the PR interval is >.2 seconds
- (constant prolonged PR interval)
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who can first degree blocks occur in?
- normal individuals associated with:
- - increased vagal tone and with CCB or beta blocker use
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first degree block tx
none, observation
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second degree av block
not all atrial beats are conducted to the ventricles
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second degree av block mobitz 1
- "longer, longer, longer, drop now you've got a wenckebach"
- - progressive PR interval lengthening, then a dropped QRS
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2nd degree av block mobitz 1 tx
- - stop the offending drug
- - Atropine (most commonly given), or epinephrine
- - +/- a pacemaker
- - observation
- - cardiac consult
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2nd degree av block type 2
- intermittently non conducted atrial beats
- - " some get dropped, some get through, now you've got a mobitz 2"
- - constant prolonged PR interval then a dropped QRS
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what is 2nd degree type 2 av block caused by
a block within the his bundle system
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mobitz 2 sx
occasionally: syncope, frequent progression to 3rd degree av block
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mobitz 2 tx
- - atropine or temporary pacing
- - pacemaker
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3rd degree av block
- - also called complete heart block
- - a complete dissociation between atria and ventricles
- - " P's and Q's don't agree now the block is 3rd degree"
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3rd degree av block sx
syncope, dizziness, acute heart failure, hypotension, cannon A waves
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3rd degree av block tx
pacemaker
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paroxysmal supraventricular tachycardia
- - the most common paroxysmal tachycardia and usually occurs in persons without structural problems
- - a SVT with abrupt onset and offset
- - 2 main types:
- - AV nodal reentry tachycardia (AVNRT)
- - AV reciprocating tachycardia (AVRT)
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what does PSVT look like
- - HR > 100 bpm
- - rhythm is usually regular with narrow QRS
- - p waves are hard to discern due to the rapid heart rate
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what do pt's usually complain of with PSVT?
a racing heart
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AVNRT
- - 2 Pathways both within the AV node (slow and fast)
- - the most common type
- - any tachydysrythmia arising from above the level of the bundle of his
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AVRT
- - 1 pathway within the AV node and a second assessor pathway outside the AV node
- - examples: wolf-parkinson, white down-ganong-levine syndrome
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wolff parkinson white
caused by the presence of abnormal assessor conduction pathway between the atria and the ventricles
- EKG shows - shortened PR interval, widened QRS, and delta waves
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PSVT conduction patterns
- - orthodromic (95%)
- - antidromic (5%)
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orthodromic pattern
narrow complex tachycardia
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antidromic pattern
wide complex tachycardia
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SVT tx
- - vagal maneuvers
- - adenosine = first line
- - beta blockers ot CCB
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PVC (premature ventricular complexes)
- ectopic beats arise from ventricular foci
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PVC risks
-hypoxia, electrolyte abnormalities, and hyperthyroidism
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PVC sx
asymptomatic, may lead to palpitations
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PVC EKG signs
- - early, wide QRS not preceded by P wave
- - usually followed by a compensatory pause
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PVC tx
- - treat underlying cause
- - if symptomatic give beta blockers or other antiarythmias
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PAC
abnormally shaped p wave
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PJC
- - the QRS complex will be narrow, usually measured at 0.10 seconds or less
- - no p wave or inverted p wave
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sick sinus syndrome
- - inappropriate bradycardia, sinus pause, sinus arrest, or episodes of alternating sinus tachycardia and bradycardia
- - collective term to describe dysfunction in the sinus node's automaticity and impulse generation
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who is SSS most often found in and caused by
- - the elderly
- - caused by scaring of the hearts conduction system
- - may occur in infants who have had heart surgery
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what kind of disorder is SSS
Heterozygous
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what causes or exacerbates SSS
- - Digitais
- - CCB
- - beta blockers
- - sympatholytic agents
- - antiarrythmic drugs
- - aerosal propellant abuse
- - underlying collagen vascular or metastatic disease, surgical injury
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when is SSS reversible
if caused by: digitalis, quinidine, beta blockers, or aerosol propellants
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SSS sx
- - asymptomatic
- - syncope, dizziness, confusion, HF, palpitations, decreased exersize tolerance
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sinus pause
pause < 3 seconds
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sinus arrest
pause > 3 seconds
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ventricular tachycardia
- - three or more consecutive ventricular premature beats
- - may be sustained or unsustained (both are associated with electrolyte abnormalities)
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what is v-tach a frequent complication of
acute MI and dialed cardiomyopathy
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V-tach on an EKG
- 3 or more PVCs, wide QRS in regular rapid rhythm
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v-tach tx
- - stable -- amioderone, lidocane, procainamide (in that order!!)
- - unstable -- CPR, defibrillation
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ventricular fibrilation
- no effective pumping action exists, without intevention --> death
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V fib EKG
- - no discernible heart contractions
- - wide erratic complex tracing
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v fib tx
CPR, defiibrilation
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torsades de pointes
- a v-tach in which the QRS complex twists around the baseline
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torsades de pointes EKG
- continuously changing axis, polymorphic ventricular contraction
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when can torsades de pointes happen
- may occur spontaneously or when the patient has hypokalemia or hypomagnesemia or following administration of drugs that prolong QT interval
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torsades de pointes tx
- magnesium sulfate
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