Cardio quiz week 1

  1. Atrial fibrilation
    • - the most common chronic arrhythmia 
    • -incidence and prevalence increase with age
    • - it is low amplitude fibrillary waves without discrete p-waves and an irregularly irregular pattern of QRS complexes
  2. what can a-fib lead to?
    - a significant decrease in cardiac output and is the most common cause of embolic cerebrovascular accidents
  3. what is also called "holiday heart" ?
    -a fib, when caused by excessive alcohol use or withdrawal
  4. a fib sx
    • asymptomatic, SOB, chest pain, palpitations
    • +/- ashmans phenomonon
  5. what is ashcans phenomenon?
    • in a-fib
    • occasional aberrantly conducted beats (wide QRS) after short R-R cycles
  6. Etiology of A-fib?
    • "PIRATES"
    • P - pulmonary disease
    • I - ischemia
    • R - rheumatic heart disease
    • A - anemia/atrial myxoma
    • T - thyotoxicosis
    • E - ethanol
    • S - sepsis
    • Chronic a fib = HTN, CHF
  7. A-fib dx?
    EKG
  8. types of atrial fibrillation?
    • paroxysmal 
    • persistant
    • permanent
    • lone
  9. paroxysmal a fib
    self-terminating within 7 days (usually <24 hours)
  10. persistant A fib
    • fails to self terminate, lasts > 7 days
    • requires termination; medical or electrical
  11. permanent a fib
    persistent a fib >1 year
  12. lone a fib
    paroxysmal, persistant or permanent WITHOUT evidence of heart disease
  13. a fib tx
    • - rate control with beta blockers, CCB, or digoxin
    • - anticoagulant with warfarin
  14. atrial flutter
    regular sawtooth pattern, an atrial rate of 250-300 bpm, and a narrow QRS complex
  15. who does atrial flutter usually occur in?
    puts with COPD, HF, atrial septal defect or CAD
  16. atrial flutter sx
    usually asymptomatic but can present with palpitations, syncope, and lightheadedness
  17. atrial flutter tx
    • -anticoagulation
    • - rate control
    • - beta blockers or CCB
  18. atrioventricular block
    characterized by refractory conduction of impulses from the atria to the ventricles through the AV node and/or bundle of His and is divided into: 1st degree, 2nd degree (mobitz 1 & 2) and 3rd degree
  19. first degree AV block
    • - all atrial beats are conducted to the ventricles but the PR interval is >.2 seconds
    • (constant prolonged PR interval)
  20. who can first degree blocks occur in?
    • normal individuals associated with:
    • - increased vagal tone and with CCB or beta blocker use
  21. first degree block tx
    none, observation
  22. second degree av block
    not all atrial beats are conducted to the ventricles
  23. second degree av block mobitz 1
    • "longer, longer, longer, drop now you've got a wenckebach"
    •  - progressive PR interval lengthening, then a dropped QRS
  24. 2nd degree av block mobitz 1 tx
    • - stop the offending drug
    • - Atropine (most commonly given), or epinephrine 
    • - +/- a pacemaker
    • - observation
    • - cardiac consult
  25. 2nd degree av block type 2
    • intermittently non conducted atrial beats
    • - " some get dropped, some get through, now you've got a mobitz 2"
    • - constant prolonged PR interval then a dropped QRS
  26. what is 2nd degree type 2 av block caused by
    a block within the his bundle system
  27. mobitz 2 sx
    occasionally: syncope, frequent progression to 3rd degree av block
  28. mobitz 2 tx
    • - atropine or temporary pacing
    • - pacemaker
  29. 3rd degree av block
    • - also called complete heart block
    • - a complete dissociation between atria and ventricles 
    • - " P's and Q's don't agree now the block is 3rd degree"
  30. 3rd degree av block sx
    syncope, dizziness, acute heart failure, hypotension, cannon A waves
  31. 3rd degree av block tx
    pacemaker
  32. paroxysmal supraventricular tachycardia
    • - the most common paroxysmal tachycardia and usually occurs in persons without structural problems 
    • - a SVT with abrupt onset and offset
    • - 2 main types: 
    •           - AV nodal reentry tachycardia (AVNRT)
    •           - AV reciprocating tachycardia (AVRT)
  33. what does PSVT look like
    • - HR > 100 bpm
    • - rhythm is usually regular with narrow QRS 
    • - p waves are hard to discern due to the rapid heart rate
  34. what do pt's usually complain of with PSVT?
    a racing heart
  35. AVNRT
    • - 2 Pathways both within the AV node (slow and fast)
    • - the most common type
    • - any tachydysrythmia arising from above the level of the bundle of his
  36. AVRT
    • - 1 pathway within the AV node and a second assessor pathway outside the AV node 
    • - examples: wolf-parkinson, white  down-ganong-levine syndrome
  37. wolff parkinson white
    caused by the presence of abnormal assessor conduction pathway between the atria and the ventricles 

    - EKG shows - shortened PR interval, widened QRS, and delta waves
  38. PSVT conduction patterns
    • - orthodromic (95%)
    • - antidromic (5%)
  39. orthodromic pattern
    narrow complex tachycardia
  40. antidromic pattern
    wide complex tachycardia
  41. SVT tx
    • - vagal maneuvers
    • - adenosine = first line 
    • - beta blockers ot CCB
  42. premature beats
    • - PVC
    • - PAC
    • - PJC
  43. PVC (premature ventricular complexes)
    - ectopic beats arise from ventricular foci
  44. PVC risks
    -hypoxia, electrolyte abnormalities, and hyperthyroidism
  45. PVC sx
    asymptomatic, may lead to palpitations
  46. PVC EKG signs
    • - early, wide QRS not preceded by P wave
    • - usually followed by a compensatory pause
  47. PVC tx
    • - treat underlying cause 
    • - if symptomatic give beta blockers or other antiarythmias
  48. PAC
    abnormally shaped p wave
  49. PJC
    • - the QRS complex will be narrow, usually measured at 0.10 seconds or less
    • - no p wave or inverted p wave
  50. sick sinus syndrome
    • - inappropriate bradycardia, sinus pause, sinus arrest, or episodes of alternating sinus tachycardia and bradycardia
    • - collective term to describe dysfunction in the sinus node's automaticity and impulse generation
  51. who is SSS most often found in and caused by
    • - the elderly
    • - caused by scaring of the hearts conduction system
    • - may occur in infants who have had heart surgery
  52. what kind of disorder is SSS
    Heterozygous
  53. what causes or exacerbates SSS
    • - Digitais
    • - CCB
    • - beta blockers
    • - sympatholytic agents
    • - antiarrythmic drugs
    • - aerosal propellant abuse 
    • - underlying collagen vascular or metastatic disease, surgical injury
  54. when is SSS reversible
    if caused by: digitalis, quinidine, beta blockers, or aerosol propellants
  55. SSS sx
    • - asymptomatic 
    • - syncope, dizziness, confusion, HF, palpitations, decreased exersize tolerance
  56. SSS tx
    pacemaker
  57. sinus pause
    pause < 3 seconds
  58. sinus arrest
    pause > 3 seconds
  59. ventricular tachycardia
    • - three or more consecutive ventricular premature beats
    • - may be sustained or unsustained (both are associated with electrolyte abnormalities)
  60. what is v-tach a frequent complication of
    acute MI and dialed cardiomyopathy
  61. V-tach on an EKG
    - 3 or more PVCs, wide QRS in regular rapid rhythm
  62. v-tach tx
    • - stable -- amioderone, lidocane, procainamide (in that order!!)
    • - unstable -- CPR, defibrillation
  63. ventricular fibrilation
    - no effective pumping action exists, without intevention --> death
  64. V fib EKG
    • - no discernible heart contractions 
    • - wide erratic complex tracing
  65. v fib sx
    • - no pulse
    • - syncope
  66. v fib tx
    CPR, defiibrilation
  67. torsades de pointes
    - a v-tach in which the QRS complex twists around the baseline
  68. torsades de pointes EKG
    - continuously changing axis, polymorphic ventricular contraction
  69. when can torsades de pointes happen
    - may occur spontaneously or when the patient has hypokalemia or hypomagnesemia or following administration of drugs that prolong QT interval
  70. torsades de pointes tx
    - magnesium sulfate
Author
Kaylasrice
ID
339822
Card Set
Cardio quiz week 1
Description
cardio
Updated