-
hypokalemia: assessment
- less than 3.5 mEq/L (3.5 mmol/L)
- Anorexia, nausea, and vomiting
- Muscle weakness, paresthesias
- Dysrhythmias, increased sensitivity to digitalis
-
hypokalemia: diagnose
- Potassium—main intracellular ion; involved in cardiac rhythm, nerve transmission
- Normal level—3.5–5.0 mEq/L (3.5-5 mmol/L)
- Causes—vomiting, gastric suction, diarrhea, diuretics and steroids, inadequate intake
-
hypokalemia: plan/implentation
- Administration of oral potassium supplements—dilute in juice to avoid gastric irritation
- Increase dietary intake—raisins, bananas, apricots, oranges, beans, potatoes, carrots, celery
- IV supplements—40 mEq/L usual concentration; cannot give concentration greater than 40 mEq/L into peripheral IV or without cardiac monitor
- Increases risk of digoxin toxicity
- Protect from injury
- Assess renal function prior to administration
-
hyperkalemia: assessment
- greater than 5.0 mEq/L (5 mmol/L)
- EKG changes, dysrhythmias, cardiac arrest
- Muscle weakness
- Paralysis
- Nausea
- Diarrhea
-
hyperkalemia: diagnose
Causes—renal failure, use of potassium supplements, burns, crushing injuries
-
hyperkalemia: plan/implementation
- Restrict dietary potassium and potassium-containing medications
- Sodium polystyrene sulfonate—cation exchange resin (causes diarrhea)
- Orally—dilute to make more palatable
- Rectally—give in conjunction with sorbitol to avoid fecal impaction
- In emergency situation, calcium gluconate given IV, sodium bicarbonate given IV, regular insulin and dextrose
- IV administration of regular insulin and dextrose shifts potassium into the cells
- Peritoneal or hemodialysis
- Diuretics
-
hyponatremia: assessment
- Less than 135 mEq/L (135 mmol/L)
- Nausea
- Muscle cramps
- Increased intracranial pressure, confusion, muscular twitching, convulsions
-
hyponatremia: diagnose
- Sodium—main extracellular ion; responsible for water balance
- Normal—135–145 mEq/L (135–145 mmol/L)
- Causes—vomiting, diuretics, excessive administration of dextrose and water IVs, prolonged low-sodium diet, excessive water intake
-
hyponatremia: plan/implementation
- Oral administration of sodium-rich foods—beef broth, tomato juice
- IV lactated Ringer’s or 0.9% NaCl
- Water restriction (safer method)
- I and O
- Daily weight
-
hypernatremia: assessment
- greater than 145 mEq/L (145 mmol/L)
- Elevated temperature
- Weakness
- Disorientation
- Delusion and hallucinations
- Thirst, dry swollen tongue, sticky mucous membranes
- Postural hypotension with decreased ECF
- Hypertension with normal or increased ECF
- Tachycardia
-
hypernatremia: diagnose
Causes—hypertonic tube feedings without water supplements, diarrhea, hyperventilation, diabetes insipidus, ingestion of OTC drugs such as Alka-Seltzer, inhaling large amounts of salt water (near-drowning), inadequate water ingestion
-
hypernatremia: plan/implementation
- IV administration of hypotonic solution—0.3%
- NaCl or 0.45% NaCl; 5% dextrose in water
- Offer fluids at regular intervals
- Decrease sodium in diet
- Daily weight
-
hypocalcemia: assessment
- Ionized serum calcium less than 4.5 mg/dL or total serum calcium less than 8.6 mg/dL (2.2 mmol/L) (total serum calcium levels used)
- Nervous system becomes increasingly excitable
- Tetany
- Trousseau’s sign—inflate BP cuff on upper arm to 20 mm Hg above systolic pressure; carpal spasms within 2–5 minutes indicate tetany
- Chvostek’s sign—tap facial nerve 2 cm anterior to the earlobe just below the zygomatic arch; twitching of facial muscles indicates tetany
- Seizures
- Confusion
- Paresthesia
- Irritability
-
hypocalcemia: diagnose
- Calcium—needed for blood clotting, skeletal muscle contraction
- Ionized serum calcium less than 4.5 mg/dL or total serum calcium less than 8.6 mg/dL (2.2 mmol/L)
- Regulated by parathyroid hormone and vitamin D, which facilitates reabsorption of calcium from bone and enhances absorption from the GI tract
- Causes—hypoparathyroidism, pancreatitis, renal failure, steroids and loop diuretics, inadequate intake, post-thyroid surgery
-
hypocalcemia: plan/implementation
- Orally—calcium gluconate (less concentrated) or calcium chloride; administer with orange juice to maximize absorption
- Parenterally—calcium gluconate--Effect is transitory and additional doses may be necessary
- Use caution with digitalized clients because both are cardiac depressants
- Calcium may cause vessel irritation and should be administered through a long, stable intravenous line
- Avoid infiltration since tissue can become necrotic and slough
- Administer at a slow rate to avoid high serum concentrations and cardiac depression
- Seizure precautions
- Maintain airway because laryngeal stridor can occur
- Maintain safety needs since confusion is often present
- Increase dietary intake of calcium, calcium
- supplements
- Regular exercise
- Administer phosphate-binding antacids, calcitriol, vitamin D
-
hypercalcemia: assessment
- Ionized serum calcium greater than 5.1 mg/L or total serum calcium greater than 10.2 mg/dL (2.5 mmol/L)(total serum calcium levels used)
- Sedative effect on central and peripheral nervous system
- Muscle weakness, lack of coordination, constipation, abdominal pain, and distension
- Confusion
- Depressed or absent tendon reflexes
- Dysrhythmias
-
hypercalcemia: diagnose
Causes—malignant neoplastic diseases, hyperparathyroidism, excessive intake, immobility, excessive intake of calcium carbonate antacids
-
hypercalcemia: plan/implementation
- IV administration of 0.45% NaCl or 0.9% NaCl
- Encourage fluids
- furosemide
- Calcitonin—decreases calcium level
- Mobilize the client
- Restrict dietary calcium
- Prevent development of renal calculi
- Increase fluid intake
- Maintain acidic urine
- Prevent UTI
- Surgical intervention may be indicated in hyperparathyroidism
- Injury prevention
- Limit intake of calcium carbonate antacids
-
hypomagnesemia: assessment
- less than 1.3 mEq/L (0.53 mmol/L)
- Magnesium acts as a depressant
- Increased neuromuscular irritability
- Tremors, tetany, seizures
- Dysrhythmias
- Depression, confusion
- Dysphagia
-
hypomagnesemia: diagnose
- Normal—1.3–2.3 mEq/L (0.53-0.95 mmol/L)
- Causes—alcoholism, GI suction, diarrhea, intestinal fistulas, abuse of diuretics or laxatives
- Usually seen with other electrolyte deficits
-
hypomagnesemia: plan/implementation
- Increased intake of dietary Mg—green vegetables, nuts, bananas, oranges, peanut butter, chocolate
- Parenteral administration of supplements magnesium sulfate
- Monitor cardiac rhythm and reflexes to detect depressive effects of magnesium Keep self-inflating breathing bag, Monitor respiratory status airway and oxygen at bedside, in case of respiratory emergency
- Calcium preparations may be given to counteract the potential danger of myocardial dysfunction that may result from magnesium intoxication secondary to rapid infusions
- Oral—long-term maintenance with oral magnesium
- IV administration—assess renal function
- Monitor for digitalis toxicity
- Maintain seizure precautions
- Provide safety measures because confusion is often present
- Test ability to swallow before PO fluids/ food because of dysphagia
-
hypermagnesemia: assessment
- greater than 2.3 mEq/L (0.95 mmol/L)
- Depresses the CNS
- Depresses cardiac impulse transmission
- Hypotension
- Facial flushing, muscle weakness
- Absent deep tendon reflexes, paralysis
- Shallow respirations
-
hypermagnesemia: diagnose
- Causes—renal failure, excessive magnesium
- administration (antacids, cathartics)
-
hypermagnesemia: plan/implementation
- Discontinue oral and IV Mg
- Emergency
- Support ventilation
- IV calcium gluconate
- Hemodialysis
- Monitor reflexes
- Teach about OTC drugs containing Mg
- Monitor cardiac rhythm; have calcium preparations available to antagonize cardiac depression
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