Camelids- Medicine

  1. Why do camelids have a small genetic pool today?
    camelid populations were nearly eliminated in South America to provide land for grazing sheep (during the Spanish invasion of the 1500s)
  2. What are the different types of alpacas?
    • Huacaya (most of them)- Fuzzy ones
    • Suri (not many people have these)- dread locks, silky
  3. How do you tell the difference between a llama and an alpaca?
    llamas have short, sharp ears; alpacas have banana ears
  4. Describe normal camelid behavior in regards to body position.
    • sternal- common position to relax and sleep, also default position when stressed
    • rolling- normal behavior
    • laying in lateral- NOT NORMAL
  5. Describe the digestive physiology of camelids. (5)
    • continuous growth of incisors
    • chew cud, but not true ruminants
    • 3 compartment stomach: C1= rumen, C2= reticulum, C3= abomasum; no rumen papillae
    • all compartments have glandular mucosal areas
    • last 5th of C3 contains gastric glands
  6. Why do camelids do better than true ruminants with lower quality feeds?
    • they are more effective fermenters, slow transit
    • need >25% roughage
  7. What's normal camelid body temperature?
    99-102
  8. What is a normal HR and RR for camelids? What is a normal number of C1 gut contractions/ minute?
    • 40-60 HR
    • 10-30 RR
    • 2-5 contractions/ min
  9. What are important things to palpate during a camelid physical exam? (4)
    • lymph nodes
    • mandible (mandibular abscesses common)
    • udder
    • abdomen
  10. Describe camelid lung auscultation.
    lungs should b easily ausculted in normal animals
  11. Camelids are stoic so...
    assume abnormal animals are sicker than they appear
  12. Describe important aspects of fecals on camelids.
    • saturated sugar solution (SG >1.025)- LA parasites
    • repeat negative fecals- intermittent shedders, Eimeria disease occurs 2-5 days before patency, Nematodirus and Trichuris shed very few eggs
  13. Pneumonia in crias and adults is commonly caused by...
    • adenovirus
    • coronavirus
  14. Respiratory difficulty in a newborn cria...think...
    choanal atresia
  15. What are common causes of pleuritis+ peritonitis?
    Streptococcus zooepidemicus, and streptococcal septicemia
  16. What is the most common liver disease in camelids?
    • hepatic lipidosis
    • high metabolic demand during pregnancy + lactation, prolonged illness, stress
    • hepatic lipidosis occurs after period of anorexia
  17. What are causes of anemia in camelids?(6)
    • GI parasites- Haemonchus
    • Mycoplasma haemolamae- RBC parasite
    • Chronic Disease (renal dz, neoplasia)
    • Nutritional deficiencies (iron)
    • Generalized immunosuppression
    • Juvenile Llama Immune Deficiency Syndrome (JLIDS)
  18. How do you know the tube is in the esophagus when trying to perform a transfaunation?
    • feel the tube on the left side of neck
    • blow air into tube while someone auscults C1
    • aspirate, esophagus should collapse
    • give water first, no cough
  19. What are causes of choke?
    • [management problem]
    • gang feeding
    • pelletized concentrates
  20. What are clinical signs of choke?
    • feed material coming from nares and mouth
    • discomfort depression
    • lowered head and neck and swinging back and forth
  21. What are presentation and signs of megaesophagus? What are some likely causes?
    • 3-5 month old, more common in llamas
    • failure to thrive- poor growth, low BCS
    • coughing, regurgitation, esophageal waves
    • in young animals- vascular malformation around esophagus; older animals- idiopathic
  22. Describe the presentation of Mycoplasma haemolamae.
    • Cria- hemolysis and anemia (intra-uterine infection)
    • Adults- either subclinical (asymptomatic chronic carrier) or anemic animal
    • Immunosuppression may lead to clinical signs
    • often associated with marked parasitemia
  23. What are clinical signs of M. haemolamae infection?
    • failure to thrive, eight loss
    • depression, lethargy
    • cria- seizures, anemia
    • adult- mild anemia, subclinical
  24. How do you diagnose Mycoplasma haemolamae?
    • Blood smear- small cocci and rings with rods apparent along margins of RBCs; negative smear does not rule out
    • PCR- highly sensitive, EDTA or citrated blood
  25. How do you treat Mycoplasma haemolamae infection?
    IV oxytet
  26. What are some common disorders of C1?
    • C1 overdistention: undigestible content (fiber consumption too high), diaphragmatic paralysis, grain/ pellet engorgement
    • C1 ulcers: anorexia, distended abdomen, feces, normal C1 floria with dead microbes
    • C1 gastroliths: usually incidental finding, concretions of salt over straw, sand, etc; found at necropsy
  27. What are some common disorders of C3?
    • ulceration: stress induced ulceration, perforation/ leakage--> peritonitis
    • obstruction: pyloric or duodenal obstruction, usually trichobezoars
    • C3 or spiral colon obstruction: distended bowel loops, palpate or observed after shearing/ shaving
  28. What are some initiating factors of GI ulcers?
    • recent changes in social structure
    • transport/ showing
    • recent illness in herd
    • dietary issues
  29. What are ways to mitigate/ manage and treat GI ulceration?
    • reduce stressful situations
    • GI protectants (pepto, sucralfate)
    • pantoprazole IV or SQ
    • cimetadine/ ranitidine SQ or IV
    • [gastrogard oral DOES NOT WORK]
  30. What are causes of enteritis in camelids?
    • intestinal parasites*****
    • Johne's disease
  31. What are causes of colic in camelids?
    • ileus
    • obstruction
  32. What are causes of pyloric obstructions in crias?
    • dam's milk production insufficient, orphaned/ bottle fed
    • cria will then nurse on dams fiber or pica because still hungry--> trichobezoar
  33. Causes of trichophytobezoars.
    • grooming behavior
    • ectoparasites (adults)
    • confinement
    • weaning
    • feeding practices
    • dietary deficiencies
    • raising animals by bottle (flank sucking, nursing dams fiber)
  34. How do you sample for chorioptic mange?
    surface mite- superficial scrape
  35. How do you sample for sarcoptic and psorioptic mange?
    burrowing mites- deep skin scrape
  36. How do you treat psorioptic and sarcoptic mange?
    ivermectin injectable twice at a 2 week interval
  37. How do you treat chorioptic mange?
    • fipronil topically, permethrin-type topical, lime sulfur topical
    • clean environment (put animal outside)
  38. Where is the most likely location of an intestinal obstruction?
    • spiral colon
    • once a bezoar gets past the pylorus, the next stopping spot is central flexure of spiral colon
  39. What history and PE findings are commonly associated with spiral colon obstructions?
    • not growing well
    • off feed
    • "respiratory disease"
    • "regurgitation"
    • PE: normal TPR, depressed, uncomfortable, abdominal palpation- turgid loops of bowel, NO FECES
  40. What is the treatment for bezoars?
    • Medical mgmt: fluids, laxatives
    • Surgical mgmt: force bezoar anterograde to larger diameter colon for enterotomy; flank approach- paracostal right flank for pyloric regions, mid-paralumbar left fossa for spiral colon
  41. What are inciting causes of hepatic lipidosis?
    • any disease that produces anorexia, stress, including pregnancy
    • feed restriction
    • insulin resistance or lack of adequate insulin
    • ketosis/ preg tox
  42. What is a way to differentiate natural hepatic lipidosis from feed restriction?
    • natural disease is much more severe
    • insulin:cortisol ratio is lower in natural disease
  43. Camelids have a ________ capacity to mobilize fat from the liver than cows.
    greater
  44. What is the most common neoplasia of camelids? What is the second most common?
    • lymphoma #1
    • intestinal adenocarcinoma #2
  45. Signs of Johnes in a camelid herd.
    • chronic diarrhea
    • lethargy
    • weight loss
    • enlarged lymph nodes
    • non-responsive to long term treatments (antibiotics, ivermectin, FBZ, sulfas, etc etc)
  46. How do you diagnose Johnes disease?
    • modification of a bovine ELISA to detect camelids antibodies (anti-llama IgG conjugated to horseradish peroxidase)
    • culture of feces for organisms
    • PCR of feces
  47. Respiratory disease in a young cria; what are some differentials?
    • choanal atresia
    • milk aspiration (esp if bottle fed, old nipples, etc)
  48. What is the therapy for uterine/ vaginal infections?
    broad-spectrum antibiotics- Ticarcillin, Ceftiofur
  49. What are viral diseases affecting the respiratory tract of camelids? (2)
    • Adenovirus
    • Coronavirus
  50. What is "Alpaca Fever"?
    • Strep equi subsp zooepidemicus
    • Spreads rapidly to most body compartments- Blood, milk, uterine d/c, peritoneal exudates, abscesses
    • Pneumonia, mastitis, metritis- FEVER, ANOREXIA, SIGNS OF DEPRESSION
    • Strep zoo IDed by culture of body fluids or tissue specimens
    • Always include coverage for Gram + cocci (fluoroquinolones alone are never sufficient b/c not great gram + cocci coverage)
  51. What is the therapy for rickets?
    • Vit D injection 1000-3000IU/kg
    • Usually recommend Vit D prophylaxis in Nov and Feb, or every 3-6 weeks during the winter
  52. Describe cria prostatitis.
    • Somewhat unknown cause- suspect development of steroidal hormone secretion in males--> prostatic hypertrophy/ hyperplasia--> compression of urethra
    • Most common sign- straining young male cria
    • Anti-microbials (for UTI) and NSAIDs for 2-3 weeks
  53. Describe caseous lymphadenitis.
    • outbreaks associated with shearing
    • excision helpful for treatment, exudate is contagious material, treat as biohazard
    • Dx by SHIT test
    • Antibiotics- Ampicillin, Pen G, Doxy
  54. What diseases should we vaccinate for?
    • Clostridial enteritis CDT or 7-way
    • Killed EHV vaccine
    • Rabies
    • WNV
    • killed EEE vaccine
  55. How is Listeria dignosed and treated in camelids?
    • CSF mononuclear pleocytosis, localizing unilateral brain lesions
    • Oxytet, procaine pen G, or florfenicol, NSAIDs, thiamine, supportive care
  56. What are causes of cerebrocortical necrosis? (4)
    • young animals on high conc diets, abrupt changes in feed
    • animals consuming bacterial thiaminases
    • animals consuming diets high in sulfur
    • animals receiving Corid for coccidiosis
  57. What are manifestations of cerebrocortical necrosis?
    • global brain swelling/ edema--> non-localizing CNS signs
    • central blindness, opisthptonus, bruxism, incoordination, head pressing, recumbency, convulsions, death
    • DORSOMEDIAL STRABISMUS
  58. What is the treatment for cerebrocortical necrosis?
    • thiamine
    • NSAIDs or steroids
    • supportive care
  59. How do you Dx meningeal worms?
    • compatible Hx (deer in pasture)
    • neuro exam- localizing neuro lesions
    • CSF- eosinophilic pleocytosis highly suggestive
    • rule out other common neuro diseases (Listeria, polio)
  60. What is the treatment for meningeal worms?
    • Fenbendazole (injectable ivermectin can cause severe reactions in camelids) [moxidectin may also work]
    • flunixin and Dexamethasone
    • Naxcel or Excenel or Oxytet
    • anti-ulcer therapy (pantoprazole, cimetidine)
    • supportive care**
  61. What are precipitating factors of heat stress in camelids?
    • llamas>> alpacas
    • adults
    • unshorn
    • stressed
    • --> death, abortion, or infertility common sequelae
  62. How do you treat heat stress?
    • cooling with ice pack,fans, shearing, alcohol
    • fluid therapy
    • flunixin
    • vit E
    • floating in float tank
Author
Mawad
ID
330178
Card Set
Camelids- Medicine
Description
vetmed camelids
Updated