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Exam 4 - Dysrhythmias - Chapter 30
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What is the pathophysiology of dysrhythmias?
abnormalities of electrical conduction
may be harmless or life threatening
Name s/s of dysrhythmia
abnormal rhythm on EKG
tachycardia or bradycardia
abnormal VS
dizziness, fainting
weakness
SOB
palpitations
↓exercise tolerance
what electrolytes are associated with normal electrical heart conduction?
Na+
Ca+
K+
In anti-dysrhythmic drugs what does the acronym SBPC stand for?
Sinus-rhythm (sodium-ion chan blockers) class I
Best (Beta-adrenergic blockers) class II
Possible (Potassium-ion chan blockers) class III
Condition (Calcium-ion chan blockers) class IV
what is the mechanism of action of propranolol (Inderal)
block beta receptors
reduces automaticity
slows conduction velocity across myocardium
what are the adverse effects of propranolol (Inderal)?
bradycardia
hypotension, dizziness, fainting
bronchospasm
hypoglycemia
↓ libido
what is the mechanism of action of amiodarone (Cordarone)?
block K+ channels in myocardial cells
lengthens refractory period
stabilizes dysrhythmias
what is the primary use of amiodarone (Cordarone)?
serious dysrhythmias
what are the adverse effects of amiodarone (Cordarone)?
bradycardia
hypotension
blurred vision
pneumonia-like syndrome
photosensitivity
hepatotoxicity, thyroid dysfunction
can create new dysrhythmia or worsen one
can ↑ digoxin levels by 70%
what is the mechanism of action of verapamil (Calan)?
block Ca+-ion channels
reduces automaticity
slows myocardial (AV) conduction velocity
what are the adverse effects of verapamil (Calan)?
bradycardia
hypotension
constipation
headache
*well tolerated and safe for most patients
Inderal (propranolol) is what type of beta-blocker?
What type of pt should it NEVER be used on?
non-selective beta blocker (beta2 affects lungs!)
COPD patients
Author
cbennett
ID
330039
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Exam 4 - Dysrhythmias - Chapter 30
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Exam 4 - Dysrhythmias - Chapter 30
Updated
2017-04-07T02:41:09Z
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