CCDM18 Control of communicable diseases in man

  1. What is the likelihood of an epidemic of Filariasis?
    Because of low infectivity and long incubation period, epidemics of filariasis are almost unlikely.
  2. What are the steps to take control of patients, contacts, and the immediate environment?
    • 1) report to local health authority
    • 2) isolation
    • 3) concurrent disinfection
    • 4) quarantine
    • 5) immunization
    • 6) investigation of contacts
    • 7) specific treatment
  3. What are the preventative measures for Filariasis?
    • 1) educate
    • 2) identify vectors
    • 3) long term vector control
    • 4) mass treatment with diethylcarbamazine citrate (DEC)
  4. B. timori is transmitted by?
    An. barbirostris
  5. In the female mosquito, ingested microfilariae?
    Penetrate the stomach wall and develop in the thoracic muscles into elongated, infective filariform larvae
  6. What is the susceptibility to infection of Filariasis universally?
    It is propable.
  7. What is the incubation period of filariasis?
    microfilariae may not appear in the blood until 3-6 months in B. malayi or 6-12 months in W. bancrofti
  8. What is the period of communicability in filariasis?
    microfilaraemia may persist for 5-10 years or longer after initial infection.
  9. Infectious agents of filariasis?
    Wuchereria bancrofti, Brugia malayi, and B. timori; long threadlike worms.
  10. Which is the most common prevalent of the 3 parasites responsible for 90% of the lymphatic filariases?
    W. bancrofti
  11. B. timori infections have been described on?
    Timor and on southeastern islands of Indonesia.
  12. Clinical manifestations of B timori are comparable to those seen in?
    B. malayi infections
  13. What are the reservoirs for filariasis?
    Humans with microfilariae in the blood for W. bancrofti, periodic B. malayi and B. timori.
  14. The mode of transmission of filariasis?
    Bite of a mosquito harbouring infective larvae.
  15. The three kinds of filariasis are?
    Filariasis due to Brugia Timori, Brugia Malayi, or Wuchereria Bancrofti.
  16. Filariasis?
    the lymphatic-dwelling filariae we have previously listed.
  17. W. Bancrofti usually resides in?
    The lymphatics in infected people.
  18. The nocturnally periodic form of B Malayi occurs when?
    living in open rice growing areas throughout much of Southeastern Asia.
  19. The subperiodic form of B. Malayi infects?
    Humans, monkeys, and carnivores in the forests of Malaysia and Indonesia.
  20. Preventative measures of Trypanosomiasis are?
    • 1) educate
    • 2) attack vectors
    • 3) construct of repair living areas
    • 4)bednets
    • 5) screen blood and organ donors
  21. What are the controls of patients, contacts, and the immediate environment of Trypanosomiasis?
    • 1) Report
    • 2) isolation
    • 3) disinfect
    • 4) quarantine
    • 5) immunization
    • 6) investigation of contacts and sources of infection.
    • 7) specific treatment.
  22. What is the incubation period of Trypanosomiasis?
    About 5-14 days after bite of insect vector, 30-40 days if infected through blood transfusion.
  23. What is the period of communicability of Trypanosomiasis?
    The vector becomes infective 10-30 days after biting an infected host
  24. What ages are susceptible to Trypanosomiasis?
    All ages.
  25. Who can be reservoirs for Trypanosomiasis?
    Humans and over 150 domestic and wild mammals species
  26. Parasitemia is most intense during?
    febrile episodes early in the course of infection.
  27. An inflammatory response at the site of infection (chagoma) may last up to?
    8 weeks
  28. Trypanosomiasis: In 20-30% of infections?
    Irreversible chronic manifestations generally appear later in life.
  29. Trypanosomiasis?
    The acute disease, with variable fever, lymphadenopathy, malaise, and hepatosplenomegaly generally occurs in children.
  30. Trypanosomiasis is confined to the?
    Western hemisphere, also in rural Mexico and central and south America
  31. Reactivated infection in what kind of patients may cause meningoencephalitis?
  32. Trypanosomiasis: To 5 reported acute vector borne human infections acquired within the USA, how many were acquired by blood transfusions?
  33. What is the mode of transmission for Trypanosomiasis?
    Infected vectors, ie. bloodsucking species of Reduviidae (cone nosed bugs or kissing bugs).
  34. No clinical manifestations of what have been noted?
    T. Rangeli
  35. T cruzi flagellates can share reservoir hosts with?
    T. Rangeli
  36. Trypanosoma Rangeli occurs in foci of endemic?
    Chagas disease extending from Central America to Columbia and Venezuela.
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CCDM18 Control of communicable diseases in man
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