-
Contrast refers to:
1.
2.
-
Definity is technically used when...
2 consecutive segments are not visualized well.
-
DO NOT give definity when...
patient has RT to LT shunting. (Perform bubble study prior).
-
Adverse events due to definity:
1.
2.
3.
4.
5.
6.
- 1. headache
- 2. back pain
- 3. nausea
- 4. chest pain
- 5. dizziness
- 6. injection site reaction
-
Equipment needed for TEE:
1.
2.
3.
4.
5.
6.
7.
8.
9.
- 1. US system with TEE capability.
- 2. Patient monitoring equipment- BP, O2 Saturation, and Suctioning.
- 3. Three way stop cock.
- 4. 2 or 3 -12cc syringes with Saline/ Preservative for bubbles.
- 5. Bite block.
- 6. Cetacaine spray.
- 7. Tongue depressor.
- 8. Clean towels and washcloths.
- 9. Gloves for physician/sonographer.
-
TEE Probe Cleaning:
- Check to make sure the Cidex has been tested for the day. Look on chart. Cidex changed every two weeks.
- Soak and scrub the endoscope and bite block in Endozyme to remove saliva and blood. (Far left sink or the "dirty sink").
- Soak endoscope and bite block in Cidex solution for 12 minutes.
- Rinse well with running tap water or in tub for 1 x3. Tub must be dumped and refilled three different times. This is the "clean sink" and is far right.
- Hang probe in storage unit.
-
If they have an arterial sheath on the Rt side can they roll onto the Lt side?
Yes.
-
Etiology of congenital heart disease:
1.
2.
3.
4.
5.
- 1. Single gene defects. (Hereditary)
- 2. Environmental factors. (Stress/ Unhealthy lifestyle)
- 3. Maternal ingestion of toxic substances. (Substance abuse)
- 4. Viral exposures
- 5. Unknown
- 6. ? (Premature)
-
The hear is the firest organ to complete its development.
- Single tube = _____ days
- Complete heart = _____ days
-
What are the two exceptions to a normal fetal heart versus a normal adult heart?
- Formen Ovale
- Ductus Arteriosus
-
Heart Tube Segments:
1.
2.
3.
4.
5.
- 1. Truncus Arteriosus
- 2. Bulbus Cordis OR Conus Cordis
- 3. "Common" Primitive Ventricle
- 4. "Common" Primitive Atrium
- 5. Sinus Venosus
-
1st Heart Tube Segment:
- Truncus Arteriosus
- - Divides to form the roots of the Aorta and Pulmonic A.
-
2nd Heart Tube Segment:
- Bulbus Cordis OR Conus Cordis
- - Future RVOT and LVOT. (Outlet of Ventricles)
-
3rd Heart Tube Segment:
- "Common" Primitive Ventricle
- - (Inlet of Ventricles)
-
4th Heart Tube Segment:
- "Common" Primitive Atrium
- - Right & Left Atria
-
5th Heart Tube Segment:
- Sinus Venosus
- - Proximal Vena Cava & part of Right Atrium.
-
RVSP =
4(v)2 + IVC pressure
-
Mean PA Pressure =
- Antegrade PV flow:
- 80 - (AT/2)
OR
-
Diastolic PA Pressure =
4(v)2 + IVC pressure
-
What is the RVSP when given a VSD velocity and a BP?
4(v)2 = _____ mmHg
Take systolic BP - _____ mmHg
-
These IVC pressures are categorized as what?
5 mmHg
10 mmHg
15 mmHg
20 mmHg
- 5 mmHg - Normal and reactive.
- 10 mmHg - Normal and partial reactivity.
- 15 mmHg - Dialated but reactive.
- 20 mmHg - Dialated and not reactive.
-
Pulmonary Hypertension:
Normal ___ mmHg
Mild ___ mmHg
Moderate ___ mmHg
Severe ___ mmHg
- Normal 18-25 mmHg
- Mild 30-40 mmHg
- Moderate 40-70 mmHg
- Severe >70 mmHg
-
Tricuspid Stenosis
- A narrowing of the orifice of the TV.
- Auscultation reveals an opening snap and diastolic rumble, best heard at the Lt sternal border. (Gets louder with inspiration).
- DIASTOLIC MURMUR
-
Causes of TS:
(6)
- Rheumatic Heart Disease
- Systemic Lupus Erythematosus
- Carcinoid Disease
- Loeffler's Endocaritis
- Metastatic Melanoma
- Congenital Defects
-
2-D findings of TS
- thickened leaflets
- dilated IVC
- doming of the TV leaflets in diastole
- enlarged RA
-
M-Mode findings of TS
- a decreased E-F slope (due to slower filling and higher RA pressures).
- decreased D-E excursion
- enlarged RA
-
Doppler findings of TS:
- a decrease E-F slope
- peak velocity is >1m/s
- spectral broadening of diastolic signal
- aliasing
- possible absence of "a wave"
-
Quantitation of TS:
Pressure half-time: is the time it takes for initial velocity divided by the square root of 2 OR time for trans-tricuspid flow to fall 1/2 its initial value.
P1/2T isn't dependant on cardiac output.
-
Tricuspid Regurgitation
- Leakage from RV into the RA during systole.
- Murmur is described as a holosystolic, high-pitched blowing sound.
- SYSTOLIC MURMUR
-
Causes of TR:
- Dilatation of the annulus preventing the leaflets from closing completely.
- Enlargement of RV caused by a volume overload (RVVO)
- Aortic and Mitral Valve Disease
- Pulmonary Hypertension
- RV Infarction
- Pacemaker wires
- Heart transplant
-
Less common causes of TR:
- Congenital Ebsteins anomaly
- Rheumatic disease
- Carcinoid Disease
- trauma
- tumors
- Endocarditis
- Chordal rupture
-
Physiology of TR:
- RA enlargement
- RV enlargement
- A FIB
- Enlarged IVC and dilatation of the other vessels coming off of the IVC.
- Leg and abdominal swelling, liver enlargement, and portal HTN.
-
Echo findings of TR:
- RVVO
- Paradoxical septal motion
- thickened leaflets
- dilated RA and RV
- dilated IVC
- reversed flowin Hepatic V
-
Pulmonary hypertension is caused by:
(3)
- Mitral Valve disease
- Congenital lesions
- Cor pulmonale
-
Pulmonic Stenosis
- the obstruction of blood flow from RV to main Pulmonary A
- rare in adults, more common in children/infants
- flow in Pulmonary A in adults = 0.9m/s, children = 1.1m/s
- auscultation- harsh systolic ejection murmur heard best in pulmonic area. thrill may also be present, splittin of S2.
-
Physiology of PS
- prominent jugular venous "a wave"
- RVH - increased pressure = thickened walls
- post stenotic dilatation of the PA
-
Causes of PS:
- congenital - most common
- carcinoid heart disease
- rheumatic heart disease
- sinus of valsalva aneurysm
- Ross Procedure
-
Treatment of PS
surgery if the systolic gradient in >50 mmHg across PV OR >70 mmHg in the RV
-
M-Mode findings of PS
- RVH
- RV failure in later stages
- systolic doming
- flattening of the IVS
- increased depth of the "a wave"
-
2-D findings of PI
- dilated RV from RVVO
- pancake septum
-
M-Mode findings for PI
diastolic flutter on the TV
-
Causes of PI:
- Pulmonary HTN
- endocarditis
- valvotomy
- congenital defects
- carcinoid heart disease
- trauma
|
|