Derm3- Bovine Derm

  1. What are the 2 categories of photodermatitis?
    • UV-A- spectrum associated with photosensitization
    • UV-B- spectrum associated with sunburn
  2. Describe the process of photosensitization.
    not direct- photodynamic compounds deposited in skin, absorb light, and produce free radicals, which cause damage to the skin and skin necrosis
  3. Where does photosensitization occur?
    lightly pigmented or non-pigmented areas of skin
  4. What are the different types of photosensitization? (4)
    • Type I: ingestion of photodynamic compounds
    • Type II: congenital errors in metabolism of pigments
    • Type III: hepatogenous- liver no longer conjugates phylloerythrin
    • Type IV: idiopathic
  5. How is type III photosensitization diagnosed? (3)
    GGT (Cholestasis), SDH (direct cellular damage), AST/SGOT
  6. Porphyria is a ________ photosensitization; it is...
    type II; genetic defect in hemoglobin production due to a deficiency in uroporphyrinogen III cosynthetase, leading to accumulation of photodyanmic substances in tissues (skin, teeth, bones, urine)
  7. What causes contact photosensitivity?
    contact with certain compounds, plants, or sap can result in a localized photosensivity
  8. What is the treatment for photosensitization? (4)
    remove from sunlight, remove underlying cause, steroid if contact photosensitivity, topical zinc
  9. What is the prognosis for photosensitization?
    • primary- good
    • hepatogenous- poor
  10. What is soremouth?
    • orf, ovine contagious pustular dermatitis, contagious ecthyma- poxvirus
    • sheet, goats, rabbits, humans
  11. How is orf transmitted? (3)
    direct animal to animal contact, fomites, environment (can survive for years off host- less in direct sunlight)
  12. Describe clinical disease associated with orf. (5)
    short incubation, young animals, immunity after infection, proliferative lesions around mouth/ muzzle (teats on nursing ewes), gone in 2-3 weeks
  13. What lesions are present with orf?
    papule--> vesicle--> pustule--> scab
  14. What is the treatment and prevention for orf?
    • palliative treatment only, nutritional support
    • vaccine: decide when they get infected
  15. What is unique about atypical soremouth?
    lesions do not regress (unlike typical, in which lesions are gone in 2-3 weeks)
  16. What is the cause of papillomatosis? In what animals does it occur?
    • bovine papilloma virus (strain determines area of body effected)
    • mostly young animals- cattle predisposed
  17. Describe the treatment of papillomatosis.
    • cosmetic problem unless affecting the penis
    • resection is curative
  18. What are the most common strains of BPV?
    1&2 usually on face/ neck
  19. What is unique about BPV-4?
    association b/w BPV-4 and ingestion of bracken fern can lad to the malignant transformation of the papilloma--> bladder tumors
  20. What is unique about BPV-5-10?
    papillomas on teats- must be removed
  21. What causes ulcerative dermatosis? Describe the clinical course.
    • viral
    • ulcerative lesions, duration is 2-8 weeks, supportive care
  22. What is the cause of ulcerative mammilitis? What are signs of disease?
    • BHV-2 and 4
    • sudden onset teat swelling and tenderness, vesicles--> ulcers
    • course lasts from 3-10 weeks
  23. How is ulcerative mammilitis managed? (3)
    ID affected cattle, segregate and milk last, recrudescence will occur
  24. Vesicular stomatitis in cattle is _________ and looks similar to __________.
    reportable; herpes mammilitis
  25. All vesicular diseases in swine are ______________.
  26. What are the signs and treatment of Erysipelas in swine?
    • Signs: fever, arthritis, diamond skin lesions, death
    • Txt: beta-lactams, anti-inflammatories
  27. What causes greasy pig disease? What is the result?
    Staph hyicus--> produces exfoliative toxin
  28. What are the signs and treatment of greasy pig disease?
    • Signs: exudative skin, crusts
    • Txt: beta-lactams, eryhtromycin
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Derm3- Bovine Derm
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