GI3- Peritonitis

  1. What are the layers of the peritoneum? (3)
    subserosa (deeper layer), serosa (single layer of mesothelial cells), lymphatic collection on surface of diaphragm
  2. Where does lymphatic collection occur in the abdomen?
    small stomata on surface of diaphragm
  3. What ways can the peritoneum become inflammed? (2)
    trauma, bacteria
  4. How can we characterize peritonitis? (2)
    localized peritonitis (GI contamination, ulcers), diffuse peritonitis (failure to control localized infection)
  5. How does pain arise with peritonitis?
    afferent nerve endings in the parietal serous membrane of the peritoneum [visceral peritoneum does not have a lot of nerve endings....in the abdomen during a DA and they don't react]
  6. How does healing of the peritoneum occur?
    by regeneration or by adhesion formation- fibrin deposition or omental adhesions
  7. Contrast fibrinious from fibrous adhesions.
    fibrinous are acute and can be dissolved by fibrinolysis; fibrinous become fibrous in 7-10 days and that is permanent
  8. Describe primary peritonitis.
    no identifiable peritoneal source (hematogenous? lymphatic?), usually monomicrobial
  9. Primary peritonitis is more common in _________ [species].
    cats
  10. Describe secondary peritonitis.
    usually GI in origin from perforated ulcer, GI foreign body, ischemic intestine, dehisced anastomosis, rectal tear; usually polymicrobial (E. coli is common),
  11. What are causes of secondary peritonitis? (5)
    GI, urogenital, abdominal abscess, bile, iatrogenic
  12. What are clinical signs of peritonitis? (7)
    fever, inappetence, ileus/decreased feces, abdominal discomfort, vomiting (SA), diarrhea, SIRS/sepsis
  13. What clinicopathologic abnormalities may be present with peritonitis? (4)
    WBC normal/increased/decreased, hyperfibrinogenemia, hypovolemia (increased PCV), hypoproteinemia (loss into peritoneal space)/ maybe high globulins
  14. What are findings on abdominal US with peritonitis? (3)
    free echogenic fluid in peritoneum, intestinal abnormalities (thickened walls, poor motility/ileus), +/- abdominal abscess (if that's the cause)
  15. What radiographic findings might be present with peritonitis in SA? (3)
    loss of serosa detail (b/c of fluid), pneumoperitoneum, +/- foreign body (if cause)
  16. Describe normal abdominal fluid as taken on abdominocentesis. (4)
    straw colored, clear, WBC< 5000/μL, TP< 2.5 g/dL
  17. What might you find on abdominocentesis with septic peritonitis? (7)
    cloudy, increased TP, increased cell count (neutrophils that may be degenerate), increased lactate, decreased glucose, +/- organic debris, +/- bacteria
  18. What are causes for non-spectic peritonitis? (2)
    common after abdominal surgery, sterile uroabdomen
  19. How can you tell the difference between GI rupture and enterocentesis on abdominocentesis?
    may see plant material and mixed bacterial pop with both; BUT enterocentesis will not have an inflammatory response; GI rupture will have degenerate neuts and intracellular bacteria and these animals will have signs of septic shock
  20. Describe medical treatment of peritonitis. (4)
    supportive care (fluids, nutrition), pain management, NSAIDs, antibiotics (broad spectrum, culture)
  21. Indications for surgical treatment of peritonitis. (4)
    toxic/degenerate neutrophils on cytology, intracellular bacteria (esp polymicrobial), suspect GI leakage (first 2 point to this), peritoneal drainage and lavage
  22. Describe adhesion prevention in horses after abdominal surgery. (8)
    anti-inflammatories, good surgical technique, antibiotics,  peritoneal lavage, sodium carboxymethycellulose (when manipulating intestine at surgery), fucoidan (Peridan- intraperitoneal barrier), omentectomy, heparin
  23. What are the most common causes of peritonitis in SA? (2)
    GI perf, pyometra
  24. What empiric antibiotics are commonly used in SA with peritonitis? (4)
    Ampicillin, Sulbactam, Enrofloxacin, Metronidazole
  25. What are the most common causes of peritonitis in cattle? (3)
    traumatic reticuloperitonitis, intra-abdominal abscess, abomasal ulcer perforation
  26. Cattle can often _________ peritonitis by __________.
    wall off; local fibrin formation
  27. What is empiric treatment for peritonitis in cattle?
    third generation cephalosporin
  28. What are the most common causes of peritonitis in horses? (3)
    intra-abdominal abscess, uterine tear during foaling, GI perf or ischemia
  29. Horses with primary peritonitis caused by __________ have an excellent prognosis.
    Actinobacillus equuli
  30. What empiric antibiotics are used in horses with peritonitis? (4)
    penicillin, gentamicin, enrofloxacin, metronidazole
Author
Mawad
ID
318105
Card Set
GI3- Peritonitis
Description
vetmed GI3
Updated