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AOS
- can exist without clinically apparent impairments in the speech muscles for volitional production of speech
- -disturbance in higher level planning and programming
- -can be comorbid with broca's
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AOS vs Aphasia
- AOS can exist independent of problems with reading and verbal comprehension and writing as well as spoken errors unrelated to artic and prosody.
- -AOS has more predictable errors
- -more initial sound errors
- -stress and intonation are off
- -more groping
- -worse SMRs than AMRs
- -the person can RECOGNIZE when they have made an error
- - more distortions and non english phonemes
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motor speech planning
- is the goal or destination
- -movements goals of the articulators
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motor speech programming
- how you are going to get to your goal or destination
- -selects, sequences, activates and controls preprogrammed sequences of movements. Includes left posterior frontal lobe, insula and basal ganglia as well as parietal lobe
- -specifies which muscles
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AOS etiologies
- Tumours, trauma=most commonly vascular
- less common=toxic or metabolic, infection
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AOS patient perspectives
- surprised by errors
- talk more slowly
- know what they want to say but cant say it
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AOS: no difficulties with..
- chewing, swallowing, or drooling
- no difficulties with comprehension
- Caveat: high comorbidity with aphasia
- normal gag
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AOS Difficulties
- oral sensory deficits
- limb apraxia
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Complex speech tasks for AOS
- -conversation and reading difficulties in intelligibility
- -complex multisyllabic word and sentence repetition
- -SMRs- as fast and as steady as they can
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Simple speech tasks for AOS
- AMRs
- isolated sounds or single words
- -when do we see irregularities
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Perceptual Signs of AOS
- groping or trial and error movements
- -dysprosody
- -difficulties initiating utterances
- -articulatory inconsistencies
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AOS vs Dysarthria
- AOS has more substitutions, more additions, more groping and less consistency(can have segments of error free speech)
- -Normal CN exam= the movement is POSSIBLE they are just not happening
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CAS
- occurs in early child hood. Origin is often unknown but can be from CP. Could be caused by trauma, infection etc
- Hallmarks: Speech sound production and prosody errors
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CAS Theoretical viewpoints: Motor theory
a disruption in speech timing, not a language component. Disruption in feedback system
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CAS Theoretical viewpoints:ling theory
not motor planning but is rooted in language and the impaired ability to develop and use phonetic/ phono heirarchy
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CAS Theoretical viewpoints: Motolinguistic
a moto deficit would affect the child's ability to learn language
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CAS: non speech observations
- normal receptive lang skills
- clumsy or awkward
- difficulty in performing volitional oral movements
- messy eaters
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CAS: most common characteristics (6)
- -difficulty sequencing sounds or syllables correctly
- -groping
- -silent posturing
- -incorrect use of prosody
- -hypernasality
- -novel utterances more difficult to produce
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Diagnosis of CAS
- feeding history
- artic and phono
- motor functioning
- intelligibility
- social behaviour
- fine motor
- cognitive development
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CAS therapy
few functional words, songs, repitition, AACs
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Dysarthria
Caused by damage to the CNS and or PNS, characterized by paralysis, weakness and or incoordination of the muscles during movement
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Variables to take into consideration when trying to diagnose Dysarthria
- age of onset
- etiology
- natural course
- site of lesion
- speech components
- perceptual characteristics
- severity
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