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Cranial Nerves
- 1. olfactory: smell
- 2. optic: vision
- 3. oculomotor: eye movement, opening of eyelid, pupil constriction, focusing
- 4. trochlear: inferior and lateral movement of eye
- 5. trigeminal: senssation to the face, mastication
- 6. Abducens: lateral movement of eye
- 7. Facial: motor nerve of facial expression, taste, control of tear, nasal, sublingual salivary and submaxillary glands
- 8. Vestibulocochlear: hearing and equilibrium
- 9. Glossopharyngeal: swallowing, salivation, gag feflex, sensation from tongue and ear
- 10. Vagus: swallowing, speech, regultaion of pulmonary, cardiovascular and gastrointestinal functions
- 11. Accessory: swallowing, innervation of sternocleidomastoid
- 12. Hypoglossal: tongue movement, speech, swallowing
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Skull Fx's-Etiology
MOI- blunt trauma
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Skull Fx's-S&S
- severe headache and nausea
- unconscious
- may have palpable deformity
- racoon's eye, battle's sign
- cerebral spinal fluid in ear or nose
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Skull Fx's-Mgmt
- intracranial bleeding
- bony fragments
- infection
- hospitalization
- neurosurgeon
- refer to dr
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Cerebral Concussions (Mild Traumatic Brain Injuries)-Etiology
- major public health concern, with return to play decisions remaining the most challenging task for any sports medicine clinician
- result of direct blow, acceleration/deceleration forces producing shaking of the brain
- coup (impact) and counter coup (opposite side)
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Cerebral Concussions (Mild Traumatic Brain Injuries)-S&S
- changes in level of consciousness (10% do lose consciousness, less than 1% are longer than 1 minute)
- post traumatic amnesia
- glasgow coma scale
- concentration deficits and attention span difficulties
- balance and coordination problems
- must monitor duration of signs and symptoms
- 2 primary symptoms: loss of consciousness (LOC) and post traumatic amnesia
- variety of scales and return to play criteria have been examined
- *typically involve LOC or amnesia
- recent classification systems have included concentration deficits, attention span difficulties, and balance and coordination in addition to LOC and amnesia
- *placing more emphasis on all S&S's may be a more logical approach
- usuing S&S's immediate post injury and 15 minutes post injury to provide an estimate of injury severity has also been suggested
- 3rd approach involves recovery of symptoms, neuropsychological testing, postural stability testing
- focus on patient symptomatology
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Cerebral Concussions (Mild Traumatic Brain Injuries)-Mgmt
- the decision to return any patient to competition following a brain injury is a difficult one that take a great deal of consideration
- if any LOC occurs the AT must remove the patient from competition
- wth and LOC a cervical spine injury should be assumed
- objective measures (BESS and SAC) should be used to determine readiness to play
- a # of guideline have been established in an effot to aid clinicians in their decisions
- return to normal baseline requires approx. 3-5 days
- all post-concussive symptoms should be resolved prior to returning to play- any return to play should be gradual
- recurrent concussions can produce cumulative traumatic injury to the brain
- following an initial concussion the chances of a 2nd episode are 3-6x greater
- must be able to determine need for dr.referral and be able to decide when patient should return home v. being admitted to hospital
- a system should be in place that allows for supervision and monitoring of patient when at home following concussive episode
- in the past, rest was deemed best treatment
- efficacy of dual task rehab is being explored
- *involves postural stability and cognitive tasks
- *little evidence available
- involves divided attention tasks
- *balance training
- *neurocognitive tasks
- *simultaneously performed
- more research is necessary to establish efficiacy of treatment method
- *which patients are best candidate
- *how soon should technique be introduced
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2nd Impact Syndrome-Etiology
- result of rapid swelling and herniation of brain after a 2nd head injury before symptoms of initial injury have resolved
- 2nd impact may be relatively minimal and not involve contact with the cranium
- impact disrupts the brain's blood auto-regulatory system leading to swelling, increasing intracranial pressure
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2nd Impact Syndrome-S&S
- often patient doesn't have LOC and may look stunned
- within 15 seconds to several minutes of injury patients condition degrades rapidly
- *dilated pupils, loss of eye movement, LOC leading to coma and respiratory failure
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2nd Impact Syndrome-Mgmt
- life threatening injury that must be addressed within 5 minutes with live saving measures performed at an emergency facility
- best mgmt is prevention from AT's perspective
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Cerebral Contusion-Etiology
- involves small hemorrhage and/or intercranial bleeding
- it is within the cortex, stem or cerebellum
- usually from an impact injury with an immovable object
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Cerebral Contusion-S&S
- may vary significantly
- LOC, but will become very alert and talkative quickly
- normal neurological exam
- headaches, dizziness, and nausea will persist
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Cerebral Contusion-Mgmt
- hospitilization w/ CT or MRI are standard
- *rule out intracranial hematomas
- *epidural *subdural *intracerebral
- treat like a concussion
- return to play when CT is normal and asymptomatic
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Epidural Hematoma-Etiology
- a blow to the head or a skull fx can cause a tear of the meningeal arteries
- blood will accumulate because of arterial pressure very quickly
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Epidural Hematoma-S&S
- LOC
- once consciousness is regained the patiet will be lucid and show few signs of a serious head injury
- symptoms will gradually worsen
- *head pains, dizziness, dilation of 1 pupil (usually on same side as the injury), sleepiness
- *later stages include deteriorating consciousness level, neck rigidity, depression of pulse and respiration with convulsions
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Epidural Hematoma-Mgmt
- life threatening
- CT is necessary to diagnose
- pressure must be surgically removed ASAP
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Subdural Hematoma-Etiology
- acute occur much more frequently than epidural
- most common cause of death in athletes
- acceleration/deceleration forces tear blood vessels thta connect the duramater and the brain
- 3 kinds of subdural:
- 1. acute- progresses rapidly and acts as an epidural hematoma (arterial bleeding)
- 2. associatied with other brain injuries (contusions) and skull injuries
- 3. chronic- due to venous bleeding
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Subdural Hematoma-S&S
- simple: unconscious unlikely
- complicated: almost always unconscious and dilation of 1 pupil (usually on same side)
- headache, dizziness, sleepiness and nausea
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Subdural Hematoma-Mgmt
- life threatening
- immediate medical attention
- Ct or MRI required to determine extent and location
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Mandible Injuries-Etiology
- direct blow (generally fx's frontal angle)
- 2nd most common fx'd facial bone
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Mandible Injuries-S&S
deformity, loss of occlusion, pain w/ biting, bleeding around teeth, lower lip anesthesia
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Mandible Injuries-Mgmt
temporary immobilization with elastic wrap followed by reduction and fixation
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Mandibular Luxation-Etiology
- involves TMJ
- generally a blow to an open mouth from the side
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Mandibular Luxation-S&S
dislocated jaw presents in locked-open position with ROM minimal with poor occlusion
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Mandibular Luxation-Mgmt
- cold application, elastic wrap immobilization and reduction
- follow-up with soft diet, NSAIDs, and analgesics with a gradual return to activity 7-10 days following acute period
- can be recurrent or result in malocclusion or TMJ dysfunction
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Zygomatic Complex (cheek bone) fx-Etiology
- MOI- direct blow
- 3rd most commonly fx'd facial bone
- LeFort fx: a fx of 1+ facial bones including zygomatic, maxillary, orbital and nasal bone
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Zygomatic Complex (cheek bone) fx-S&S
deformity or bony discrepancy, epistaxis (nose bleed), diplopia (double vision) and numbness in cheek
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Zygomatic Complex (cheek bone) fx-Mgmt
- cold application to control edema and immediate referral to dr
- healing will take 6-8 weeks and proper protective gear will be required upon return to play
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Maxillary fx-Etiology
- MOI- blow to upper jaw
- 4th most commonly fx'd facial bone
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Maxillary fx-S&S
pain w/ chewing, malocclusion, noseblee, double vision, bumbness of lip and cheek region
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Maxillary fx-Mgmt
- due to severe bleeding, maintain airway
- must be aware of possible brain injury (concussion)
- be aware of possible cervical injury
- transport hospital immediately upright and leaning forward if conscious
- allows for drainage of saliva and blood
- fx reduction, fixation and immobilization by dr.
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Tooth Fx-Etiology
impact to the mandible or maxilla, direct trauma
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Tooth Fx-S&S
- uncomplicated fx: produce fragments w/o bleeding (chip)
- complicated fx: produce bleeding with the thooth chamber being exposed w/ a great deal of pain
- root fx's: are difficult to determine and require follow up with xray
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Tooth Fx-Mgmt
- uncomplicated and complicated crown fx's do not require immediate attention
- fx'd pieces can be placed in a bag if not senstive to air or cold, follow up can wait 24-48 hours
- bleeding can be controlled via gauze
- cosmetic reconstruction of tooth
- in instances of root rx's the patient can continue to play but must follow up immediately following competition
- tooth repositioning may be required along with bracing and the use of mouth pieces in the future
- mandibular fx's and concussions must also be ruled out
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Tooth Subluxation, Luxation, and Avulsion-Etiology
direct blow
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Tooth Subluxation, Luxation, and Avulsion-S&S
- tooth may be slightly loosened, dislodged
- when subluxed tooth may be loose within socket with little or no pain
- w/ luxations, no fx has occurred however there is displacement
- with an avulsion, the thooth is completely knocked from oral cavity
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Tooth Subluxation, Luxation, and Avulsion-Mgmt
- subluxed tooth: referral should occur within the 1st 48 hours
- luxated tooth: repositioning should be attempted along with immediate follow up
- avulsed tooth: tooth should be re implanted if it can be easily done, if not use a save a tooth kid, milk, or saline
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Nasal Fx-Etiology
- direct blow
- most commonly fx'd facial bone
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Nasal Fx-S&S
- separation of frontal process of maxilla of lateral cartilage or combo
- profuse bleeding and hemorrhaging, immediate swelling and deformity
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Nasal Fx-Mgmt
- control bleeding and refer to dr. for xray, examination and reduction
- uncomplicated and simple fx's will pose little problem for athlete's quick return
- splinting may be necessary
- uncomplicated fx's may return to play within a few days
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Deviated Septum-Etiology
compression or lateral trauma
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Deviated Septum-S&S
- bleeding and in some instances a septal hemotoma will form
- patient will complain of nasal pain
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Deviated Septum-Mgmt
- at the site of hematoma compression will be required (and if present, drained immediately)
- following drainage, a wick is inserted to allow for further drainage
- packing will be necessary to prevent a return of hematoma
- a neglected hematoma will result in formation of an abscess along with bone and cartilage loss and deformity
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Auricular (Pinna) Hematoma (Cauliflower ear)-Etiology
occurs from either compression or shear injury to the ear (single or repeated)
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Auricular (Pinna) Hematoma (Cauliflower ear)-S&S
- tearing of overlying tissue away from cartilage
- hemorrhaging and fluid accumulation
- if unattended- coagulation, organization and fibrosis occurs
- appears elevated, white, rounded, nodular formation that is firm and resembles cauliflower
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Auricular (Pinna) Hematoma (Cauliflower ear)-Mgmt
- to prevent, wear proper ear protection
- cold application will minimize hemorrhaging
- if swelling occurs, measures must be taken to prevent fluid solidification
- dr. aspiration, packing, pressure
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Preventing Eye Injuries
- protective devices must provide protection from front and lateral blows
- goggles with high impact-resistant polycarbonate lenses for refraction
- unfortunately googles may distort peripheral vision and or become fogged under certain conditions
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Assessment of the eye
- must utilize extreme caution in evaluating and caring for eye injuries
- multiple conditions require immediate referral for additional care to be provided
- transportation to hospital should take place with patient in recumbent position
- eyes should be covered together
- *movement of unaffected eye will cause movement in affected eye
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Special Tests
- Special Test
- Pupillary response
- *dilation and accomodation
- Visual Acuity
- *clarity, blurred vision, diplopia, floating black spots, flashes of light
- Ophthalmascope
- *instrument used for observing the interior of the eye (retina)
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Orbital Fx-Etiology
blow to the eyeball forcing it posteriorly, compressing the orbital fat until a blowout rupture occurs to the floor of the orbit (muscle and fat can herniate)
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Orbital Fx-S&S
- diplopia, restricted eye movement, downward displacement of the eye, soft-tissue swelling and hemorrhaging
- numbness associated with infraorbital nerve on the floor of the orbit
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Orbital Fx-Mgmt
- x ray necessary to confirm fx
- antibiotics to decrease risk of infection (due to proximity of maxillary sinus and bacteria)
- treat surgically or allow to resolve spontaneously
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Foreign Bodies in the Eye-Etiology
frequent occurrence in sports and can be dangerous
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Foreign Bodies in the Eye-S&S
- foreign object produces considerable pain and disability
- no attempt should be made to remove by rubbing or by recovering with fingers
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Foreign Bodies in the Eye-Mgmt
- close eye and determine location (upper or lower lid)
- pull upper lid over lower lid to cause tearing
- wash eyes with saline, use protrolleum jelly to relieve soreness
- if object is embedded, close and patch eye and refer to dr.
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