Bio Ch18a

Blood does not
A. transport a variety of nutrients.
B. participate in the initiation of blood clotting.
C. help to stabilize the pH of extracellular fluids.
D. help to regulate body temperature.
E. produce plasma hormones

The buffy coat does not contain
A. erythrocytes.
B. agranulocytes.
C. lymphocytes.
D. platelets.
E. granulocytes.

The hematocrit is ____________ of the total blood volume.
A. 47% to 63%
B. 42% to 45%
C. less than 1%
D. 37% to 52%
E. 25% to 37%

_____________ is not found in plasma.
A. Fibrinogen
B. Glucose
C. Glycogen
D. Urea
E. Albumin

___________ is a protein not commonly found in plasma.
A. Prothrombin
B. Hemoglobin
C. Transferrin
D. Fibrinogen
E. Albumin

_____________ is the most abundant protein in plasma.
A. Albumin
B. Insulin
C. Creatine
D. Bilirubin
E. Creatinine

_____________ would not decrease colloid osmotic pressure (COP) in blood.
A. Hypoproteinemia
B. Starvation
C. Severe liver failure
D. A diet predominantly based on red meat
E. An extremely low-protein diet

Serum is essentially identical to plasma except for the absence of __________.
A. platelets
B. nitrogenous wastes
C. glucose
D. fibrinogen
E. albumin

Tissues can become edematous (swollen) when
A. the concentration of sodium and proteins in blood is high.
B. there is hyperproteinemia.
C. there is a dietary protein deficiency.
D. colloid osmotic pressure (COP) is high.
E. hematocrit is high.

Myeloid hemopoiesis in adults happens in the
A. spleen.
B. yellow bone marrow.
C. red bone marrow.
D. liver.
E. thymus.

The viscosity of blood is due more to _____________ than to any other factor.
A. fibrin
B. erythrocytes
C. sodium
D. albumin
E. nitrogenous wastes

Erythrocytes transport oxygen and
A. initiate blood clotting.
B. regulate erythropoiesis.
C. defend the body against pathogens.
D. transport nutrients.
E. transport some carbon dioxide.

Most oxygen is transported bound to
A. heme groups in hemoglobin.
B. the plasma membrane of erythrocytes.
C. delta chains in hemoglobin.
D. beta chains in hemoglobin.
E. alpha chains in hemoglobin.

If all the molecules of hemoglobin contained in RBCs were free in the plasma,
A. it would facilitate delivery of oxygen into tissues supplied by small capillaries.
B. it would drastically increase osmotic colloid pressure.
C. it would considerably increase blood oxygen carrying capacity.
D. it would significantly increase blood osmolarity.
E. it would facilitate oxygen diffusion into cells distant from blood capillaries.

An increased erythropoietin (EPO) output by the kidneys would lead to all of the following except
A. increased hematocrit.
B. increased blood viscosity.
C. increased blood osmolarity.
D. increased hypoxemia.
E. increased RBC production.

Many RBCs die in the
A. red bone marrow.
B. stomach and small intestine.
C. spleen and liver.
D. lymph nodes and thymus.
E. stomach and liver.

All these can lead to polycythemia except
A. iron deficiency.
B. emphysema.
C. cancer of the erythropoietic line of the red bone marrow.
D. excessive aerobic exercise.
E. dehydration.

______________ is more likely to cause anemia than any of the other factors below.
A. Any factor that creates a state of hypoxemia
B. Air pollution other than by carbon monoxide
C. Smoking
D. Renal disease
E. High altitude

Anemia has these potential consequences except
A. the individual is lethargic.
B. blood viscosity is increased.
C. blood osmolarity is reduced.
D. more fluid transfers from the bloodstream to the intercellular spaces.
E. blood resistance to flow is reduced.

What is the final product of the breakdown of the organic nonprotein moiety of hemoglobin?
A. bilirubin
B. iron
C. heme
D. globin
E. biliverdin

Correction of hypoxemia is regulated by
A. a positive feedback loop
B. a negative feedback loop.
C. a self-amplifying mechanism.
D. an enzymatic amplification
E. a cascade effect.

A deficiency of _____________ can cause pernicious anemia.
A. EPO secretion
B. iron
C. vitamin C
D. vitamin B12
E. folic acid

Sickle-cell disease is not
A. a hereditary hemoglobin defect.
B. caused by a recessive allele that modifies the structure of hemoglobin.
C. a cause of anemia.
D. a cause of malaria.
E. advantageous to protect carriers against malaria.

The ABO blood group is determined by _____________ in the plasma membrane of RBCs.
A. glycolipids
B. antigen-antibody complexes
C. antibodies
D. glycoproteins
E. agglutinins

Type A blood can safely donate RBCs to _____________ and can receive RBCs of type___________.
A. AB; O
B. O; AB
C. B; A
D. A; B
E. O; O

Type AB blood has ____________ RBC antigen(s).
A. anti-A
B. no
C. anti-A and anti-B
D. anti-B
E. A and B

RhoGAM is an antibody given to Rh- women who give birth to Rh+ children. RhoGAM is what type of plasma protein?
A. beta (β)globulin
B. gamma (γ)globulin
C. albumin
D. alpha (α)globulin
E. fibrinogen

The universal donor of RBCs, but not necessarily plasma, is
A. O, Rh-negative.
B. ABO, Rh-negative.
C. AB, Rh-negative.
D. O, Rh-positive.
E. AB, Rh-positive.

An individual has type B, Rh-positive blood. The individual has _____ antigen(s) and can produce anti-_____ antibody(ies).
A. A; B and D
B. D; A and B
C. B and D; A
D. B; A and D
E. A and D; B

The main reason why an individual AB, Rh-negative cannot donate blood to an individual A, Rh-positive is because
A. anti-D antibodies in the donor will agglutinate RBC of the recipient.
B. anti-B antibodies in the donor will agglutinate RBCs of the recipient.
C. anti-A antibodies in the recipient will agglutinate RBCs of the donor.
D. anti-A antibodies in the donor will agglutinate RBCs of the recipient.
E. anti-B antibodies in the recipient will agglutinate RBCs of the donor.