-
automaticity
ability to spontaneously depolarize without external stimulus
-
excitability
ease or difficulty to invoke depolarization
-
conductivity
ability to transmit impulse to other cells
-
contractility
ability of cardiac cells to shorten in response to electrical simulation; squeeze of the heart
-
rhythm analysis
- normal QRS = .12
- normal PR = .12 - .20
-
dysrhythmia risk factors
- electrolytes
- fluid volume
- hypoxemia/myocardial ischemia
- mechanical factors (cardiomegaly; myocardial scar)
- body temperature
-
sinus bradycardia
- HR<60 bpm
- causes:
- 1. sinus node disease
- 2. meds
- 3. hypoxia
- 4. well conditioned athletes
- treatment (if pt. is symptomatic):
- 1. O2
- 2. IV access
- 3. consider temporary pacing
- 4. meds - atropine sulfate : anticholinergic; 0.5mg IV Q3-5min max3mg
-
sinus tachycardia
- HR>100bpm (usually 100-150)
- always has a cause :anxiety, pain, fever, activity, dehydration, heart failure, anemia
- fix the cause and the rhythm will be fixed
- meds - beta blockers & CCB
- O2
-
atrial fibrillation
- irregularly irregular (QRS comes irregularly) with no meaningful P waves
- loss of atrial kick (20-30% decrease in CO)
- blood pools and clots in the atria, so high risk for embolus
-
atrial flutter
- atrial rate>250bpm
- sawtooth P wave
-
treatment for a fib/ a flutter
- cardioversion
- CCB
- beta blockers
- digoxin
- amiodarone
- diltiazem
- anticoagulate
-
superventricular tachycardia (SVT)
- HR 150-250
- always regular
- paroxysmal (abrupt start and stop)
- may or may not see p waves
- treatment for stable SVT:
- 1. valsalva maneuver
- 2. adenosine - drug of choice
- 3. carotid massage
- 4. O2
- treatment for unstable SVT:
- 1. adenosine
- 2. CCB
- 3. beta blocker
- 4 cardioversion
-
premature atrial contraction
- caused by early atrial depolarization (P wave) before next scheduled sinus beat
- causes: emotion, stress, anxiety, fatigue, infection, lack of sleep, meds, caffeine, heart failure, MI, electrolyte imbalance
- treat the cause if become symptomatic
-
premature ventricular contraction
- irritable ectopic site in ventricle
- QRS wide and bizarre
- t wave deflected opposite to the QRS
- cause: increased age, hypoxia, MI, CHF, stress, meds, electrolyte imbalance, caffeine, alcohol, tobacco
- treatment: eliminate cause; consider antiarrhythmic (amiodarone, lidocaine)
-
ventricular tachycardia
- cause: significant cardiac disease, K or Mag imbalance, artifacts (loose leads, brushing teeth); be sure to check the patient
- treatment for stable with a pulse:
- 1. amiodarone
- 2. lidocaine
- 3. magnesium
- 4. potassium
- 5. cardioversion
- treatment for unstable with no pulse:
- 1. CPR
- 2. defibrillate
- 3. epinephrine
- 4. vasopressin
- 5. amiodarone
- 6. lidocaine
- 7. resuscitation
-
ventricular fibrillation
- electrical chaos; no perfusion
- always fatal unless terminated
- treatment : immediate electrical shock
- causes:
- 1. heart disease, CAD, cardiomyopathy
- 2. hypovolemia, hypoxia
- 3. acidosis
- 4. hypokalemia, glycemia, thermia
- 5. hyperkalemia
- 6. toxins
- 7. cardiac tamponade
- 8. trauma, tension pneumothorax, thrombus
-
asystole
- flat line; no electrical impulses
- treatment :
- 1. CPR
- 2. epinephrine and vasopressin
- 3. treat the cause
-
1st degree AV block
- PR interval > .20
- usually asymptomatic
-
2nd degree AV Block Mobitz I (wenkebach)
- PR lengthens progressively until QRS is dropped
- treat symptoms
-
2nd degree AV block Mobitz II
- PR interval is constant
- some beats are conducted = p wave then QRS
- some beats nonconducted = p wave then no QRS
- unstable and usually requires pacemaker
- cause :
- 1. MI; AV node disease
- treatment :
- 1. atropine, dopamine, epinephrine
- 2. pacemaker
-
3r degree AV block
- no communication between atria and ventricles
- no relationship between P waves and QRS
- CO is decreased
- P waves are a constant distance no matter what
- Treat as symptomatic bradycardia
- cause :
- 1. meds (dig toxicity)
- 2. acute MI; myocarditis
- 3. degenerative heart disease
- treatment :
- 1. treat symptoms
- 2. temporary pacing
- 3. atropine, epi, dopamine
-
defibrillation
- asynchronous
- high energy level
- for pulseless V tach and V fib
-
cardioversion
- low energy shock
- for unstable SVT, a fib, a flutter, V tach with a pulse
- premedicate with sedation and pain meds
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