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Heart failure
inability of the heart to pump sufficient blood to meet the needs of the tissues for O2 and nutrients
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Heart failure
chronic or acute; fluid overload; myocardial disease (contraction, systolic or diastolic); some cases are reversible; progressive and lifelong; poor tissue perfusion
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Causes of left sided heart failure
HTN, CAD, valvular disease involving the mitral or aortic valve; pulmonary congestion from increased pressure in pulmonary vessels
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Systolic heart failure
when heart cannot contract forcefully enough during systole to eject adequate amounts of blood into the circulation; preload increases and afterload increases as a result of increased peripheral resistence; ejection fraction drops from 50-70% to below 40%
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Systolic HF
fluid backs up into the pulmonary system
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Patients with an ejection fraction of less than 30%
are candidates for implantable ICD
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Diastolic heart failure
left ventricle cannot relax adequately during diastole; inadequate relaxation or stiffening prevents ventricle from filling with sufficient blood to ensure adequate cardiac output
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Right sided heart failure
caused by left ventricular failure, right ventricular MI or pulmonary HTN; right ventricle cannot empty completely, increased volume and pressure develop in the venous system and peripheral edema results
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High-output heart failure
can occur when CO remains normal or above normal; caused by increased metabolic needs or hyperkinetic conditions like septicemia, high fever anemia and hyperthyroidism
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What compensatory mechanisms work to improve cardiac output
sympathetic nervous system stimulation, renin angiotension system, other chemical responses, myocardial hypertrophy
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BNP
a peptide produced and released by the ventricles when the patient has fluid overload as a result of HF
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Signs of left sided HF
decreased CO, fatigue, weakness, oliguria, angina, confusion, dizziness, tachycardia, pallor, weak peripheral pulses, cool extremities, pulmonary congestion, hacking cough, dyspnea, crackles, wheezing, frothy pink tinged sputum, tachypnea, S3/S4 gallop
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Assessment of left HF
pulse strength and auscultate AP for 1 minute; respiratory-rate, rhythm, character, O2 sats, auscultate lungs, A&O X 3
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Signs of right sided HF
systemic congestion
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Assessment of right sided HF
neck veins, ABD girth, dependent edema
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Labs for rt sided HF
electrolytes, HGB, HCT, BNP, UA, ABGs, echo, chest xray, ECG, pulmonary artery cath
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Assessment for HF
health history, sleep and activity, knowledge and coping, physical exam, assess responses to meds
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Problems or complications of HF
cardiogenic shock, dysrhythmias, thromboembolism, pericardial effusion, cardiac tamponade
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Goals for teaching HF
promote, relieve, decrease, encourage, teach
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Activity intolerance for HF
bed rest, physical activity, exercise, pacing self, wait after eating, conserve energy, avoid weather conditions, positioning of self
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How to help with fluid volume excess
assessment, daily weight, I&O, timing of meds, fluid intake, sodium restructions
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Paroxysmal nocturnal dyspnea
sudden awakening with a feeling of breathlessness 2-5 hours after falling asleep; sitting upright and danging feet usually helps
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Crackles
usually start low in lung and work their way up
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Benefits of dig
increased contractility, reduced HR, slowing of conduction through AV node, inhibit sympat while enhancing parasym
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PVCs are most common with
Dig and potassium levels can cause problems with dig
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Classes of HF
I: no symptoms with activity; II: symptoms with ordinary exertion; III: Symptoms with minimal exertion; IV: symptoms ar rest
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What med decreases preload
diuretics
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What meds decrease afterload
ACE inhibitors (pril), AR II blockers (Cozaar), cal channel blockers (Cardizem), phophop 3 (Primacor)
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Inotropic agents
dig, dopamine used to increase contractility and thereby improve CO
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What to watch for with dig
count pulse before taking med, take at same time of day, dont take with antacids, toxicity includes fatigue and muscle weakness and confusion and loss of appetite; have dig and K levels checked
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Vasodilators can cause
orthostatic hypotension and headache is common side effect; sit and lie slowly
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hBNP cause
natriureses (loss of Na and vasodilation)
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If someone has HF and SOB, give O2 then do what
put in high fowlers-decrease venous return to the heart (preload) and help decrease lung congestion
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Pulmonary artery catheter
multi-lumen catheter with the capacity to measure right atrial and indirect left atrial pressures or pulmonary artery wedge pressure
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Increased RA pressures may occur
with right ventricular failure whereas low RA pressures usually indicate hypovolemia
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Normal PAP is from
15-26 systolic vs 5-15 diastolic
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Transducer needs to be at
phlebostatic axis (4th intercostal space and 1/2way between front and back)
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Central venous catheter
through vena cava or right atrium; normal CVP is 2-6; lower than 2 is reduced rt ventricular preload caused by hypovolemia, vomiting or overdiuresis and higher than 6 indicates right ventricular preload
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Complications of central venous catheter
blood clot, infection
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Pulmonary artery catheter
assess pulmonary function
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Intraarterial BP monitoring
can use blood from this for ABGs, check pulses (may be tingling, usually in radial area)
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Complications with PAC
pulmonary infarction, air embolism, ventricular dysrhythmias if catheter slips into right ventricle, infection, bleeding
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Complications of antraarterial BP monitoring
air embolism, blood loss, pain, arteriospasm, tingling in arm, hold pressure for 10 minutes (NO PEEKING)
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