3 different joint by movement allowed classifications
syarthoses-no movement
ampiarthoses-slight movement
diarthroses-freely moveable
3 material joint classifications
fibrous-fibers connecting to bone
cartilagenous-cartilage between
synovial-fluid between-most abundant
3 types of fibrous material
gomphoses (peg in joint)
sutures
syndesmoses
2 types of carilagenous material
synchadroses-hyaline
symhyses-fibro
1 type of synovial material
diarthroses
4 characteristics of synovial
membrane that produces lubrication
joint capsule around joint
ligaments connecting bones
articular(hyaline) cartilage on ends of bones
4 other random things in joints
fibrocartilage pads-meniscus
fat pads
tendon sheaths-tube bursa"around tendon"
bursa-fluid sac
6 synovial joint types and the type of movement
plantar/gliding- biaxial
hinge-uniaxial
pivot-uniaxial
ellipsoidal/condyloid-biaxial
saddle(only in thumb)-biaxial
ball n socket-multiaxial
4 ways of joint stability
way bones articulate with each other
ligaments that connect
muscles n tendons across joints
external bone support
6 different knee joints
lateral (fibular) collateral ligament-LCL
medial (tibial) collateral ligament-MCL
anterior crucate ligament-ACL
posterior crucate ligament-PCL
lateral meniscus
medial meniscus
3 Groupings of General ROM
flexion-extension-hyperextension
abduction-adduction
rotation-circumdiction
flexion-extension-hyperextension
reduce angle
increase angle
increase beyond 180
abduction-adduction
away from midline
towards midline
rotation-circumduction
circle one plane
circle many
7 Groupings of Special ROM
supination-pronation
inversion-eversion
dorsiflexion-plantarflexion
opposition
protraction-retraction
elevation-depression
gliding
supination-pronation
palms forward
palms backwards
inversion-eversion
curl inward
curl outward
dorsiflexion-plantarflexion
toes up kicking
toes down point
opposition
thumb to our hand
protraction-retraction
chin forward
chin backward
elevation-depression
mouth closed-mouth open
gliding
jaw side to side
Bursitis
inflamed bursa on elbow
tendonitis
inflamed tendon sheaths
3 types of arthritis
osteoarthritis-old age/wear n tear
rheumatoid-autoimmune
gouty-uric acid
dislocation
bones pop out
sprain
stretched ligament or joint capsule
2 types of living cells in bones
osteoblasts-build up bones
osteoclast-breaks down thebones
2 types of non living matrix in bones
collagen fibers-give flexibility
calcium salts-give strenght (phosphate n carbonate)
2 major types of bones
flat n long
Flat bones made....
made by intermembranous ossification
ossification centers
bone grows out from them
fontanels
soft spot that takes 18 months to use
long bones made....
made by replacement of cartilage/endochodrial ossification
Epiphysis ends in long bones(features)
spongy bone
red bone marrow-blood cells
has articular cartilage
diaphysis in long bones (features)
hollow compact bone
has yellow bone marrow-adipose
laid bone in circular layers around vessels
membranes
-periosteum-ostioblasts-outside
-endosteum-osteoclasts-inside
gradient diffusion in spongy bone
no blood vessels
5 steps to formation of bone
make crude bone from hyaline cartilage
form collar of bone on outside
do primary ossification of the diaphisis
begin secondary ossification of the epiphysis
bones grow in length at epiphyseal/growth plate
3 ways of bone maintenance
minerals
vitamins
hormones
3 minerals in bone maintenance
calcium
phosphorus
fluoride
4 vitamins in bone maintenance and their use
A-control of osteoblast/clast
C-collagen fibers
D-calcium uptake
Folic Acid- development
5 hormones in bone maintenance and their use
calcitonin(thyroid) removes CA from blood into bones
PTH(parathyroid) increases blood CA levels-breakdown bone matrix
HgH(Antipituitary)bone growth n development
Testosterone n Estrogen-growth spurts n changes puberty
Thyroxin(Thyroid) metabolism control
3 random factors in bone maintenance
genetics
activity
not smoking
Remodelling
changes in our bone overtime
involves local action of the osteoblasts/clasts
3 major things can cause remodelling
bone growth-keep bones strong but light-make sure they are proper shape-make sure they get together properyly
Stress on bone activity
bone repair
3 fracture types
complete-incomplete
open (compound)-closed(simple)
direction of fracture
-transverse-across
-linem-long acess
-oblique-angle
5 bone healing steps
fracture
hematoma
bony callus
repaired bone
remodeling
typical fracture lasts...
lasts 8 weeks
weight bearing take longer
osteomalacia
soft bones
osteoporosis
porous bones
pagets disease
excessive spongy bone
3 abnormal types of spine curvatures
lordosis-sway back lumbar to far forward
kyphosis-abnormal thorasic curve-hunchback
scolliosis-abnormal side to side curve
5 Muscle Functions
movement
posture maintenance
joint stability
heat production
body size n shape
Fasicles-bundle of muscle fibers help to determine (4 things)
shape of muscle
function of muscle
strength of contraction
degree of shortening-how small they can get parallel to how much they contract
origin
attached to the non moving bone-bigger end of muscle with more mass
insertion
attached to the moving bone, across joint
Agonist
prime mover
antagonist
moves opposite of agonist
synergist
aids the agonist
fixator
anchors the origin
lever action of muscles (3)
muscle-force effort
joint-fulcrum/pivot point
bone/body part-load resistance
levers provide (2 things)
mechanical advantage-movement with loss of force
provides speed
9 ways in muscle naming
direc
bone attached to
location
shape
size of muscle
function of the muscle
direction
number of origins
origin/insertion
directional terms
all flexors are on the...
on the anterior side (except knee)
all extensors are on the....
on the posterior side
abductors are on the...while adductors are on the....
on the lateral side while...are on the medial
muscles are _____ of what they are moving
proximal
most muscle is connected to bones by tendons because (4 reasons)
tendons make stronger connections
more durable
save space
connect to muscle by connective tissue
muscles are bundles of bundles which (3 reasons)
allow better control
helps with durability
space 4 blood vessels and nerves
6 muscle structural levels
muscle
fasicle
myofibers
myofibril
sarcomeres
myofilaments
muscle
bundles of fasicles surrounded by epimysium
fasicle
bundles of myofibers surrounded by perimysium filled with endomysium
myofibers
bundle of myofibrils
muscle cells-long cylinder striated surrounded by sarcolemma
8 features of myofibers
multinucleated
3 types
(slow twitch) have lots of mitochondria
(fast twitch) have glycogen
myofibrils-contractile
T tubule system-get contraction signals
sarcoplasmic reticulum- stores CA
contain myoglobin-stores Oxygen
myofibril
bundle of myofilaments n strings of sarcomeres
sarcomeres
contractile unites
produce striation of muscles formed by overlap of dark n light alternating
when muscle contracts....
slide filaments together
shortens sarcomere which shortens mucle
myofilaments
protein of fiber group
thick filament
composed of myosin
forms stubby club
has protein titan springs on ends
thin filaments
2 strings of actin beads twisted
strands of tropomysin in beads
typroponin pieces on tropomysin
myogram
graph of contraction
4 periods on myogram
latent-time from stimulation to contraction
contraction-muscle shortens force increases
relaxation
refractory-recovery
3 types of muscle fibers
fast
intermediate
slow
2 contraction types
isotonic
isometric
isotonic
muscle length changes
force constant
increase endurance n efficiency
typical of aerobic
isometric
develops force
length doesn't change
cause increase in myofibril mass and number
increase contractile mass=increase strength
typical of anerobic resistance activities
cardiac muscle
branched striated
woven into bands of our heart
cells have interculated discs on the ends which hold cells tightly n have gap junctions
cardiac muscle features
lots of mitochondria
large T tubules
less sarcoplasmic reticulum
respond to both neural n chemical signals involuntary
as muscle stretches it develops tension increases contractile force
long refractory period prevents tetanus(long sustained contractions)
Smooth Muscle
no striation but some proteins
fiber not on surface pulls together
single nucleus per cell
responds to neural n chem signals
involuntary
muscle stretch doesn't always develop tension
usually found in layers of organs
muscle contraction steps1-3
-muscle divided up into fiber groups called motor units-all fibers of the group controlled by the same motor neuron-they all contract together and to the same extent-make stronger by activating more groups
-motor neurons make a connection (synapse) with muscle fibers
-motor neuron releases acetylcholine (neurotransmitter) by exocytosis
muscle contraction steps 4-6
-acetylcholine binds to the receptors on the fibers-when enough are bout the muscle cell is stimulated
-contraction signal enters the cells via the T tubules
-signal causes release of CA from the sarcoplasmic reticulum
muscle contraction steps 7-9
-CA binds to the thin filament removing block-thick n thin may now connect
-thick filament head bend n pull thin across thick like tug o war
-go through alternating connect-pull
muscle contraction steps 10 n 11
-go through process until it can't shorten anymore or stop contraction signal or no more energy (ATP)
-ATP used to reset system-muscles are always ready