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Atrophy
- Translucent, dry, paper-like, wrinkled skin surface resulting from thinning/ wasting of the skin due to loss of collagen/ elastin
- - striae, aged skin
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Erosion
- Wearing away from superficial epid. causing moist, shallow depressions. Doesn't go to dermus, heal w/o scaring
- ex.) scratchmarks/ ruptured vessicles
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Lichenification
Rough, thick, hardened area of epi. resulting from chronic irritation like scrat/ rubbing
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Scales
- shedding flakes of greasy, keratinized skin tissue: color- gray/white/silver ex.) dry skin, dandruff, psoriasis and eczema
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Pallor
- Resulting of inadequate circulation blood/ hemoglobin and subsequent reduction of tissue oxygenation
- Dark skin- no red tones, buccal mucosa Brown skin- yellowish/ browing tinge black - ashen gray
- - Most evident in areas w/ least pigmentation
- Ex.) conjuction, oral mucus membrane nail beds, palms, soles of feet
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Cyanosis
- Blueish tinge nail beds, lips, and buccal mucosa
- - Dark~ look palpebral conjuctiva, palms,sole
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Jaundice
Yellowish tinge 1st seen in solera of the eyes and then in mucosse membrane and skin
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Erythema
- Skin redness ( associated with lots of rashes and other candit)
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Hypopigmentation
~ decreased pigmentation (vitiligo)
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Hyperpigmentation
- Increased pilgrim ( birthmark)
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Vitiligo
~ Patches of hypopigmented skin caused by destruction of melanocytes (albinism)
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Edema
- ~ Excess interstitial fluid
- appears: swollen
- - shiny
- - taut and tends to blanck sking color
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Skin lesion
- Alteration in skin appearence
- Primary- appear initially in response to a change in ext/int. envirionment if slan
- Secondary- result from modifications like chronicity/ trauma/infection if 1 lesion
- Ex) vessicle/ bluster = Primary
- -> ruptures causes erosion= secondary.
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- describe lesion in~ 1. location
- 2. distributions
- 3. Configuration
- - shape
- - size
- - firmness
- - texture
- - characteristics
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