Secondary skin lesions

• Translucent, dry, paper-like, wrinkled skin surface resulting from thinning/ wasting of the skin due to loss of collagen/ elastin 
•     - striae, aged skin

• Wearing away from superficial epid. causing moist, shallow depressions. Doesn't go to dermus, heal w/o scaring
•    ex.) scratchmarks/ ruptured vessicles

Rough, thick, hardened area of epi. resulting from chronic irritation like scrat/ rubbing

- shedding flakes of greasy, keratinized skin tissue: color- gray/white/silver   ex.) dry skin, dandruff, psoriasis and eczema

• Resulting of inadequate circulation blood/ hemoglobin and subsequent reduction of tissue oxygenation
•       Dark skin- no red tones, buccal mucosa
•       Brown skin- yellowish/ browing tinge
•       black - ashen gray
• - Most evident in areas w/ least pigmentation
•   Ex.) conjuction, oral mucus membrane nail beds, palms, soles of feet

• Blueish tinge nail beds, lips, and buccal mucosa
• - Dark~ look palpebral conjuctiva, palms,sole

Yellowish tinge 1st seen in solera of the eyes and then in mucosse membrane and skin

- Skin redness ( associated with lots of rashes and other candit)

~ decreased pigmentation (vitiligo)

- Increased pilgrim ( birthmark)

~ Patches of hypopigmented skin caused by destruction of melanocytes (albinism)

• ~ Excess interstitial fluid
• appears: swollen
• - shiny
• - taut and tends to blanck sking color

• Alteration in skin appearence
•   Primary- appear initially in response to a change in ext/int. envirionment if slan
•   Secondary- result from modifications like chronicity/ trauma/infection if 1 lesion
•        Ex) vessicle/ bluster = Primary
•                  -> ruptures causes erosion= secondary.
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• describe lesion in
• ~ 1. location
•     2. distributions
•    3. Configuration
•         - shape
•         - size
•         - firmness
•         - texture
•         -  characteristics
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