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Effects of Sensory Impairment
- Independence
- Self-Esteem
- Safety Issues
- Interaction with others and environment
- Overall quality of life
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Population at Risk for Neurosensory Impairment
- **Leading Causes Associated with Aging
- 2/3 of affected population over 65 years
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What makes up the 1st Outer Layer of the Eye
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White protective outer coat
Sclera
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Transparent structure that allows for the entry of light
Cornea
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What makes up the 2nd Middle Layer of the Eye
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Rich,vascular coat lines posterior segment of eye – nourishes retina
Choroid
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Produces aqueous humor – Maintain IOP – Controls shape of lens (focuses light onto retina)
Ciliary Body
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What does the 3rd back lining of the eye do
Forms retina – complex nervous tissue layer – converts light rays into elec. Signals/relays via optic nerve to brain/interpretation of vision
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Eye Assessment: Ocular History
- Visual disturbance?
- * Pain?
- *Are both eyes affected?
- * Discharge? (how long has this been going on)
- * Duration
- * Other disease processes
- * Visual & Cranial history (cranial nerves)
- * Medications – can affect intraocular pressure
- * Family history
- * Nutrition
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What is checked during the Eye Assessment
- •Pupillary Function
- •Assessment of EOM -- 6 fields of gaze
- •Visual Acuity
- •Color Vision
- •Stereopsis Testing --depth perception, ability to see 3D
- •Tonometry
- •Slit Lamp Microscopy
- •Opthalmoscopy
- •Inspection
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Visual Acuity Test
Snellen Chart
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Normal IOP (tonometry)
- 10-22 mmHg
- important with glaucoma
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Inability of the eye to bend light rays
Refraction Errors
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Causes of Refraction
- –Irregularities of corneal curvature
- –Focusing power of lens
- –Length of eye
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Symptoms of Refraction Errors
- –Blurred vision
- –Headache
- –Eye strain
- –Ocular discomfort
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Near Sightedness-- Light Rays focus IN FRONT of the retina
Myopia
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Correction for Myopia (nearsightedness)
Concave Lens
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Hyperopia
Far-Sightedness-- Light rays focus BEHIND the retina
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Correction for Hyperopia
Convex Lens
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Age Related change of lens- becomes less elastic
Presbyopia
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Difference between Hyperopia and Presbyopia
Presbyopia-- loss of accomodation
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Correction of Presbyopia
- Reading Glasses
- Bifocal Lens
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Corneal surface irregularly curved/unevenly shaped
No single focus of light
Astigmatism
-
-
Causes of Aphakia
–Congenital defect
–Surgical Removal- IOL implant
–Trauma causing dislocation
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Correction of Aphakia
- Thick glasses
- Aphakic glasses can magnify 25% (not used freq because of surgery
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Nursing Care for Contact Lens
- Know pattern of wear
- Daily or extended wear
- Remove in emergency/surgery
- Teach detection of problems
- Remove immediately if problems (RSVP)
- Teach prevention of infection/complications
- Redness, Sensitivity,Vision problems, Pain
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Laser to reshape central corneal surface
Photorefractive keratectomy
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Fold back corneal flap
Laser removes some of internal layers
Earlier visual stability
LASIK (Laser assisted in-situ keratomileusis)
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2 plastic rings placed b/w corneal layers, removable
(IOL implants)
Implants (Intracorneal ring segments)
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BCVA
Best Corrected Visual Acuity
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Assessment for Visual Acuity
- - Length of impairment? (acute- glycoma v. chronic)
- - Effects on ADL functioning?
- - Coping Strategies?
- - Support Systems?
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Interventions for Visual problems: Resources
- –American Foundation for the Blind
- –Center for the Visually Impaired
•www.cviatlanta.org
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Optical Assisstive Devices
•Braille or Audio books
•Cane or guide dog
•Closed circuit TV
•High powered magnification
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NonOptical Assisstive Devices
- •Hold book closer to eyes. Sit closer to TV
- •Contrast enhancement – white on black, black felt-tip marker
- •Increased lighting
- •Large type print
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Assessment Findings for Eye Trauma
Pain, photophobia, redness, swelling, ecchymosis, tearing, absent eye movements, drainage, abnormal vision, prolapsed globe, abnormal IOP
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Eye Trauma Interventions
- •ABC’s
- •Determine mechanism of injury
- •Assess for other injuries
- •Chemical exposure- flush eye
- •Do NOT put pressure on eye!
- •Instruct Not to blow nose!
- •Stabilize foreign object
- •Cover eye with dry sterile patch & protective shield (if not chemical)
- •Keep NPO, Elevate HOB 45 degrees
- •No eye meds. unless dr. order
- •Analgesia, Reassurance, Monitor pain.
- •Anticipate surgical repair for penetrating, globe rupture, or avulsion
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Types of Extraocular Inflammation/Infection
- Hordeolum (sty)
- Chalazion
- Blepharitis
- Conjunctivitis
- Keratitis
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Tender – Infx sebaceous glands in lid – Warm moist compresses (aka...sty)
Hordeolum
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May evolve from hordeolum – Inflammation of seb gland – nonpainful – more chronic – mary need removal
Chalazion
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Bilat inflammation lid margins – red/scales/crusts – C/O Itching/burning/irritation – Simultaneous with conjunctivitis, Seborrhea Hair/Eyebrows related - Conscientious hygiene (hair, too) – gentle cleansing of lid margins
Blepharitis
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Inflam/infx of cornea – Bact/Viral (herpes simplex)
Keratitis
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Types of Conjunctivitis Infections
- –Bacteria – Staph. aureus --pink eye
- –Viral
- -Chlamydial -- blindness
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Signs/Symptoms of Conjunctivitis
redness, tearing, foreign body sensation, drainage, photophobia
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Treatment of Conjunctivitis
Strict handwashing, individual towels/wipes, antibiotic drops, assess for STD
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Infection/Inflammation of the Cornea
Keratitis
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S/S of Keratitis
Pain, photophobia
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Treatment of Keratitis
- No corticosteroids for HSV!
- –Antibiotics
- –(viral) Acyclovir
- –Good hygiene – contact lens, soiled drsg, direct contact
- –Discard possibly contaminated products
- –Hot/cold packs, dark glasses, analgesics
- –Eye drops (stagger for max absorption)
- –Corneal Transplant
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Leading Cause of Blindness in Adults within the US
- Intraocular Disorders
- (ie: Cataracts, Retinal Detachment, Age-Related Macular Degeneration, Glaucoma)
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Causes of Cataracts
age, congenital (maternal rubella), radiation/UV exposure, corticosteroids, trauma
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S/S of Cataracts
gradual decline in vision, abn. color perception, glare
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Collaborative Care for Cataracts
- •No non-surgical cure
- •Visual aids
- •Mydriatics to dilate pupil
- •Cycloplegic- paralysis of accommodation
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Surgical Management of Cataract
- Phacoemulsification
- Lens Replacement
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Pre-op Cateract Removal Care
- –Eye drops – Mydriatics, Cycloplegics, NSAIDs, antibiotics
- –Antianxiety meds
- –NPO X 6-8 hrs
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Post-op Cataract Removal Care
- –Topical antibiotics & corticosteroids
- –Mild analgesia prn
- –Eyeshield & activity restrictions per MD
- –Home within few hrs
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Patient/Family Teaching
- •Pre-op - Expect photophobia and transient stinging/burning from eye drops
- •Will not have depth perception until patch removed
- •Expect minimal pain/scratchiness in operative eye
- •Call MD for pain, redness, purulent drainage, or visual
- acuity
- •Environmental safety measures
- •Administration of eye drops
- •Avoid stooping, bending, coughing, lifting
- •Wear eye shield at night
- •Health promotion – wear sunglasses, good nutrition (Vit. C & E)
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Retinal Detachment
•Separation of sensory retina and pigment epithelium• •Fluid accumulation between layers •1 out of 10,000 people yearly•Untreated usually results in blindness
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Risk Factors for Retinal Detachment
- •Severe Myopia
- •Aphakia
- •Proliferative Diabetic Retinopathy
- •Ocular Trauma
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Detaching Retina
- - Photopsia –light flashes
- - Altered field of vision – floaters/cobweb/hairnet
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Detached Retina
- “Curtain coming down”
- - Small detachment may not be noticed, cobweb
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Collaborative Care for Retinal Break
- - Laser photocoagulation
- - Cryopexy
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Collaborative Care for Detachment
- - Scleral Buckling
- - Vitrectomy
- - Pneumatic retinopexy
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Intense light beam – inflammatory response – Scar seals break
Laser Photocoagulation
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Freezes area to seal
Cryopexy
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Removal of vitreous (jellylike substance fills space between lens and retina)
Vitrectomy
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Injection of gas to form bubble/close retinal break
Retinopexy
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Post Operative (Retina Detachment Procedures) Nursing Management
- - Positioning
- - Activity
- - Topical medications as ordered
- - Pain Assessment
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ARMD
Age-Related Macular Disorder
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Risk Factors for ARMD
- –Increasing Age
- –Smoking History
- –Hypertension
- –Overweight
- –Hyperopia
- –Family History
- –Wet ARMD
- –Use of thyroid hormones/HCTZ’s
- –Arthritis
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Most Common ARMD
Central Vision Loss after age 60
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Retinal Pigment Epithelium
- - Nourish Macula
- - Remove metabolic waste
- - Aging decreases efficiency
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ARMD dry form starts with the _______ and results in _____
- drusen-
- results in atrophy and degeneration of macular cells;
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Distortion of Vision
Metamorphopsia
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Atrophy of macular cells
Decreased functioning
Gradual blurring of vision
Nonexudative ARMD (dry)
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Development of abnormal blood vessels in/near macula
- May have abrupt onset
- Progress rapidly over days/weeks
- Distinct area of blurred/darkened/distorted vision
(if left untreated, will cause permanent blindness b/c blood vessels leak and create scar tissue)
Exudative ARMD (wet)
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Appearance of drusen
Opthalmoscopy
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Test that defines the involved area of the eye
Amsler Grid Test
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Defines the extent and type of disease
Fluorescein Angiography
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Collaborative Care: Photodynamic Therapy (PDT)
- -Wet ARMD
- - Seals leaking blood vessels
- - IV injection of verteporfin dye to visualize vessels
- -“Cold” Laser activates dye to close vessels
- -Prevents further vision loss
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Collaborative Care: Intraocular injections
- Lucentis-
- -preserves vision, monthly, side effects- conjunctival hemorrhage, eye pain, inflammation, and floaters
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Collaborative Care that has very slow progression
High Dose Vitamin Therapy
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Nursing Management: POST PDT
Avoid Direct Sunlight Exposure
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Nursing Management: Psychologican Implications
- Encourage that remaining vision can be retained
- Arrange social supports
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Nursing Management: Patient Education
- Information for independent ADL functioning
- - Organize aids for low vision
- - Reduce glare when outside
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2nd Leading Cause of Blindness in the US
Glaucoma
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Increased Intraocular Pressure (IOP)
* Congestion of aqueous humor
- flows between iris & lens
- nourishes cornea and lens
*Leads to optic nerve damage/atrophy
*Peripheral visual field loss
Glaucoma
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What is Outflow Dependent on?
- intact drainage system - Trabecular meshwork
- (Open angle (45 degrees) between iris and cornea)
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Risk Factors for Glaucoma
- •Family History
- •African American
- •Age
- •Diabetes
- •Cardiovascular disease
- •Migraine Syndromes
- •Myopia
- •Eye Trauma
- •Prolonged use of topical or systemic corticosteroids
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90% of cases
Develops slowly
No Pain or pressure
Decreased outflow through trabecular meshwork
Loss of Peripheral Vision
“Tunnel Vision”
IOP = 22-32 mmHg
**in most cases the patient may not know they have this until significant damage has already occurred
Open Angle Glaucoma
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IOP for primary open angle glaucoma
22-32 mmHg
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Symptoms of primary angle closure glaucoma
- Definite symptoms-Sudden severe pain, N&V, colored halos around lights, blurred vision, ocular redness, “frosted cornea”
- Outflow decreased due to decreased angle between Iris and Cornea
- - Bulging Lens - age
- - Pupil Dilation
- - Drug Induced
- - Trauma, neoplasms
- **VERY SERIOUS...PATIENT CAN LOSE EYE
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IOP level for primary angle CLOSURE glaucoma
50 mmHg or greater
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Collaborative Care for Glaucoma
- •Prevention of Optic Nerve damage
- •Damage control/not cure
- •Pharmacologic Therapy
- •Surgical Management
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Initial Treatment in POAG- ocular
- Beta-Adrenergic Blockers
- - Timolol (Timoptic)
- - Decrease aqueous humor (AH) production
- - Contraindicated in bradycardia/asthma/COPD
- Alpha Adrenergic Agonists
- - Propine
- - Decreased AH production/Increased Outflow
- - Systemic effects
- - Punctal Occlusion
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S/E of Beta-Adrenergic Blockers
- Discomfort/blurred vision/low blood pressure -
- ***Watch for postsystemic absorption
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S/E of Alpha Adrenergic Agonists
Redness/tachycardia/hypertension
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Cholinergic Agent (miotics)
- Pilocarpine
- - Iris sphincter contraction opens meshwork
- - Increase AH outflow
- - Decreased visual acuity
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Carbonic Anhydrase Inhibitors
- Diamox (systemic)
- - Decreases AH production – Diuretic effect
- - Extremity tingling/tinnitus/GI disturbances
- - ? Sulfa allergy/electrolytes/high dose ASA therapy
- Azopt (topical)
- Stinging/redness/blurred vision (SE)
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S/E of Cholinergics
- Decreased visual acuity due to miosis –
- S/E headaches/vomiting/hypotension
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Because Carbonic Anyhdrase Inhibitors act as a diuretic, you should monitor ______ closely
Electrolytes
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Types of Surgical Intervention for POAG
- •Argon Laser Trabeculoplasty (ALT)
- - Outpatient/ only Topical Anesthetic
- - Laser to meshwork – Opens spaces – Increase outflow
- •Trabeculectomy
- - Filtering procedure
- - Removal of part of iris/meshwork
- - Increase AH outflow
- •Cyclocryotherapy
- - Freezes parts of ciliary body
- - Decreased production of AH
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Medications given for the Eye
Miotics (cholinergic agents)
- Hyperosmotics – Glycerin liquid/Mannitol
- - Increases extracellular osmolarity
- - Reduces IOP
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Creates opening in iris to eliminate blocking of pupil
Laser iridotomy/Surgical Iridectomy
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Nursing Management: Eye Assessment
- - Compliance with treatment
- - Psychologic reaction
- - Support systems
- - Changes in visual acuity/visual fields/Opthalmoscopy
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Nursing Management: Planning
- - No progression of disease
- - Understand disease
- - Compliance
- - No Postoperative complication
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Nursing Management: Implementation
- Teaching
- - Risks
- - Prevention
- - Medication administration / Other meds?
- - Safety at home / referrals
- PACG
- - Requires acute intervention – medication
- - Comfort measures
- - Postoperative teaching (also POAG)
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Nursing Management: Evaluation
- Expected Outcomes
- – No further loss of vision/compliance/safely function/relief from pain
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Auditory Assessment: Symptoms
Pain, dizziness, vertigo, tinnitus, hearing deficit
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Auditory Assessment: Past History
- –Childhood problems
- –Head trauma
- –Surgeries
- –Allergies
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Ototoxic Medications
Aspirin, Loop Diuretics, Aminoglycoside antibiotics, Antimalarial, Neoplastic agents
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External Ear Disorders
- Trauma
- –Hematoma
- –aspirate, antibiotics
- –Assess hearing
- External Otitis
- –Pain w/ movement of auricle/tragus
- –Drainage, Fever, Dizziness, Impaired Hearing
- *Collaborative & Nursing Care
- •C&S of drainage
- •Warm compresses
- •Antibiotic drops/systemic
- •Mild Analgesics
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Administration of Ear Drops
- •Ear drops at room temp.
- •Do Not touch dropper to ear
- •Position ear so drops can run down canal – hold position X 2 minutes after given
- •Handle material saturated with drainage carefully
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Cerum Impaction
- •Discomfort, decreased hearing (conductive hearing loss), otalgia, tinnitus, vertigo, cough
- •Bradycardia (Vagal)
- •Irrigation of ear canal
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Acute Otitis Media
–Pain, fever, malaise, h/a, decreased hearing
- Collaborative Care:
- •Antibiotics
- •Myringotomy
- •Antihistamines - adults
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Chronic Otitis Media
- •Drainage
- •Hearing loss
- –Destruction of ossicles
- –TM perforation
- –Fluid in middle ear
- •Ear pain, nausea, dizziness
- Complication:
- **Cholesteatoma
- –Skin overgrowth that destroys adjacent bone & tissue
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Diagnoses of Chronic Otitis Media/auditory problems
Otoscopy, C&S, audiogram, sinus xrays, MRI, CT
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Collaborative Care for Auditory Prob
- •Antibiotics (systemic)
- •2% acetic acid drops
- •Tympanoplasty
- –Reconstruction of TM &/or ossicles
- •Mastoidectomy
- Removal of diseased tissue/infection source
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Nursing Management: Auditory Prob
- •Pre-op teaching
- •Avoid complications
- –Avoid blowing nose, coughing, sneezing
- •If must, leave mouth open
- –Assist with getting up first time
- •Dizziness & loss of balance
- –Cotton ball dressing – endaural incision
- –Mastoid drain w/ post-auricular incision
- –Monitor tightness of dressing over ear to prevent necrosis
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Chronic Otitis Media with Effusion
•Inflammation/Accumulation of fluid
• Etiology: Eustachian tube malfunction, chronic otitis media, URI, barotrauma, overgrowth of adenoids
- •Collaborative Care:
- –Decongestants, Antihistamines, Corticosteroids, Antibiotics
- –Chewing gum, swallowing, Valsalva
- –Myringotomy w/ ventilating tube placement
- •Teach
- – No H2O in ear or swimming
- –Adenoidectomy
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Insertion of Tubes into the Ears
Myringotomy
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Otosclerosis
- •Spongy bone overgrowth from labyrinth causing fixation of stapes
- •Conductive hearing loss – common, young adults.
- •Reddish blush of TM - from vascular/bone change
- •Collaborative Care:
- –Stapedectomy – stapes removed & prosthesis implant
- –Na Fluoride w/ Vit. D & Ca Carbonate – retard bone resorption & encourage calcification of bony lesions
- •Post-Op Nursing Care:
- –Avoid sudden movements
- –Avoid coughing, sneezing, lifting, bending, straining with BM
-
Inner ear problem, cause unknown, excessive accumulation of endolymph until the membranous labyrinth ruptures, mixing high potassium endolymph with low potassium perilymph; ANS- pallor, sweat, N/V, glycerol test po with audiograms. Improvement = dx
Meniere's Disease
-
Collaborative Care for Meniere's Disease
- –Acute: Antihistamines, anticholinergics, antiemetics, benzodiazepines -Valium & Meclizine(Antivert)
- –B/T attacks: vasodilation, diuretics, antihistamines, low Na diet, no caffeine or nicotine
- –Vestibular nerve resection – alleviate vertigo & preserve hearing
- –Labyrinth Ablation – loss of vestibular & cochlear hearing function
-
Nursing Care: Meniere's Disease
- Goals: Minimize vertigo & ensure safety•Reassure not life threatening
- •Bedrest; Quiet, dark room; comfortable position
- •Avoid sudden head movements or position changes
- •Keep emesis basin within reach
- •Side rails up, bed in low position
- •Assist when getting OOB
- •IV meds & fluids. Monitor I&O.
- •After attack, assist w/ ambulation – unsteady.
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Inflammation/infection of inner ear affecting cochlear &/or vestibular portion of labyrinth
Labrynthitis
-
•Complete destruction causes permanent hearing loss & extreme unsteadiness
*Nystagmus
•Viral – Lasts 7-10ccd. No tinnitus or hearing loss. Usually due to acute otitis media.
Manifestations of Labrynthitis
-
Conductive Hearing Loss
- –Impacted cerumen
- –Otitis externa
- –Trauma
- –Otitis media
- –Otosclerosis Tumors
-
Sensorineural Hearing Loss
- –Trauma
- –CNS infections
- –Presbycusis
- –Vascular
- –Ototoxic drugs
- –Tumors
- –Meniere’s disease
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Mixed Hearing Loss
Barotrauma, Cholesteatoma, Otosclerosis, Temporal bone fractures
-
–May speak softly b/c own voice sounds loud
–Hears better in noisy environment
–Treat underlying cause
–Hearing aids unnecessary usually
Conductive Hearing Loss
-
–Hear sound but cannot understand speech
–Decreased hearing of high-pitched sounds (consonants)
–Lack of understanding of problem by others
–Hearing aids help, but sounds still muffled
Sensorineural Hearing Loss
-
Manifestations of Hearing Loss
Asking others to speak up
Answering questions inappropriately
Not responding when not looking at speaker
Straining to hear
Cupping hand around ear
Showing irritability when others don’t speak up
Behaviors: Irritability, Denial, Withdrawal, Suspicion, Loss of self esteem, Insecurity
-
Health Promotion for Hearing Loss
•Environmental Noise Control
•Immunizations - MMR
- •Ototoxic Drugs
- Salicylates
- Loop diuretics
- Aminoglycoside antibiotics
- Antineoplastics
- Antimalaria
- Manifestations- dizziness, tinnitus, hearing loss
-
Hearing Aid Care
- Keep in cool, dry place
- Disconnect/remove battery
- Clean ear molds weekly
-
Communication with Hearing Impaired
- –Draw attention with hand movements
- –Have speaker’s face in good light
- –Avoid covering mouth or face with hands
- –Avoid chewing gum or food
- –Maintain eye contact
- –Avoid distracting environment
- –Move closer to better ear
-
Communicating with Verbal Aids
- –Speak normally and slowly!
- –Do not over exaggerate facial expressions
- –Do not over enunciate
- –Use simple sentences
- –Rephrase sentence; use different words
- –Write name or difficult words
- –Avoid shouting
- –Speak in normal voice into better ear
-
Gerontologic Considerations for Hearing Impaired
- •Unwillingness to use aid
- •Difficulty with care and handling
- •Acceptance of loss with aging
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