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Lecture 26
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Frank Pathogen
Cause a disease in host
Opportunistic Pathogen
live in body normally and cause a disease when immunocompromised
Subclinical Infections
Too few or not Virulent enough to cause an infection
Clinical Infections
Cause signs and symptoms
Infectious Dose
Amount of bacteria that causes an infection; dependent on person
Signs
apparent upon examination
Symptoms
effects patient notices; ex. nausea, pain
Balanced Pathogenicity
pathogen becomes less virulent while host becomes more resistant
Localized Infection
affect the immediate area of replication
Disseminated Infection
affects area outside the area of replication
Systematic Infection
affects the whole body
Bacteremia
Bacteria that infects the blood
Viremia
Viruses that infect blood
Toxemia
Toxins that infect blood
Septicemia
acute illness caused by pathogens in blood
Koch's Postulate
used to determine the specific organism that causes a disease
Infection
Multiplication of organism causes the disease
Intoxication
Toxin produced by organism causes the disease
Fimbrial adhesions
proteins at the tip of a pili
IgA Protease
destroys IgA
Siderophore
steal iron from the host
type III effector
secrete molecules from bacterium into host cell
Membrane Ruffles
rearrangement of host cell due to type III effector that allows bacteria to hide from host
Horizontal Gene Transfer
Swapping of Genes between bacteria
Pathogenicity Island
"block" of genes for virulence factors passed between bacteria
M-Cells
specialized for transit; passes bacteria immune cells
MALT
mucousal associated lymphoid tissue containing M cells
Activated Macrophage
has enhanced oxidative burst and digestive capabilities
Actin "Rockets"
Propel bacteria from one cell to another
Serum Resistance
Block C3b activity to avoid complement pathway
C3b Regulatory Protein
Prevents C3b from activating the compliment cascade
C5a Peptidase
prevents Neutrophil recruitment
Lysin
kills phagocyte
Fc Binding Protein
Bacteria bind the wrong part of antibody so attack does not happen
Staphylococcal Protein A
????
Pilus Switching
switching surface proteins so it isn't recognized
Hyaluronic Acid
bacteria coat in a self antigen so they aren't recognized
Author
epaynich
ID
16448
Card Set
Lecture 26 notecards.txt
Description
Micro 221 Lecture 26
Updated
2010-04-27T19:17:34Z
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