Pharmacology

This slows conduction through AV node, interrupts AV nodal reentry pathways & restores NSR in PSVT. Half life is < 10sec due to red blood cell sequestering

Sympathomimetic that is relatively selective for Beta 2 adrenergic receptors.
Bronchodilator with few cardiovascular effects
Duration 4-6 hrs

Class IIb Antidysrhythmic for ventricular dysrhythmias.
Prolongs action potential duration in all cardiac tissues

NSAID that inhibits platelet Aggregation

Parasympatholytic
Enhances sinus node automatacity and AV conduction via vagolytic action

Dextrose is fuel for cellular metabolism and is regulated by insulin

Benzodiazepine sedative
Acts as a tranquilizer, anticonvulsant, and skeletal muscle relaxant (via CNS)

Reverses effects of phenothiazines and has anticholinergic and antiemetic effects
Antihistamine

Beta 1 receptor with positive inotropic and negative chronotropic effects

Beta stimulator with positive chronotropic and inotropic effects.

Relaxes smooth muscle of the bronchi and is therefore effective in treating bronchoconstriction associated with asthma and anaphylaxis

Increases blood flow to heart during CPR

50-100 times more potent than Morphine with shorter duration of action. Principal therapeutic values are analgesic and sedative
Resp rate and alveolar ventilation changes might last longer than analgesic effect.
Onset is immediate

Inhibits reabsorbtion of sodium and chloride in kidney, producing peak diuresis. Rapidly reduces extracellular fluid, decreases venous return and ventricular output by causing venous dilation (3-4min).

Sympathomimetic that is selective for Beta 2 adrenergic receptors.
Causes relaxation of bronchial smooth muscle, thus decreases airway resistance and increases vital capacity.
Greater affinity for B2 adrenergic receptors than its chemical variant

Increases Fibrillation threshold of myocardium and decreases automatacity, supressing ventricular ectopy.
Decreases stimulation of CNS during intubation

Has selective Beta 1 adrenergic receptors.
Decreases HR, systolic BP, and Cardiac output
Effective in reducing incidence of V-Fib & Chest pain in MI patients.

Benzodiazepine sedative
Acts as a amnesic, hypnotic, antiolytoc, and anticonvlsant
3-4 times more potent than other benzodiazepine

Potent analgesic
Reduces anxiety associated with pain (important in cardiac related pain)
CNS depressant, increases peripheral venous capacity
Decreases venous return, decreases myocardial demand

Narcotic antagonist that binds with narcotic receptor sites

Nasal decongestant that causes vasoconstriction of superficial blood vessels in nasal mucosa

Smooth muscle relaxant causing vasodilation.
Decreases cardiac preload.
May improve perfusion t ischemic myocardium by dilating coronary arteries.
May provide a significant degree of pain control in patients with cardiac chest pain

Reduces the workload on the hear and lungs to maintain proper perfusion.
Facilitates breakdown of glucose into a usable energy for to maintain aerobic metabolism

Alkalizing agent that binds with excess ions to correct metabolic acidosis

Synthetic adrenal corticosteroid used as an antiinflammatory in spinal injury and in the management of acute and chronic inflammatory diseases.
Inhibits inflammatory response by interfering with many of the substances that cause inflammation (cytokines, interleukin, interferon)

This is required for the conversion of pyruvic acid to acetyl coenzyme A.
With out this a significant amount of energy available in glucose cannot be obtained.

A naturally occuring anti-diuretic hormone which becomes a powerful vasoconstrictor when used at a higher dose than normal.
Exerts a greater vasoconstrictive effect under conditions of hypoxia and acidosis than its co-drug. It also lasts longer, has greater arterial tone, and therefore, has an effect that correlates with greater myocardial perfusion.
It does not increase myocardial oxygen consumption and lactate production in the arrested heart.

This has mild sedative properties, is tolerated well in pediatric populations, is safe in pregnancy, and does not produce extrapyramidal effects of phenothiazines.
Serotonin 5-HT3 receptor antagonist antiemetic