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THE STOMACH
- Holding tank for food
- Food is exposed to stomach acids and digestive effects of pepsin
- Saturates food with gastric juices
- Excretes HCL (hydrochloric acid)
- Ph 2.0
- Absorbs H20, alcohol, sugars, salt, electrolytes & some drugs
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GASTROESPHAGEAL REFLUX DISEASE (GERD)
- Caused by gastric acid flowing upward into the esophagus
- Incompetent lower esophageal sphincter
- Acid becomes an irritant destroying esophageal lining
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GERD Degree of Reaction
- Heartburn
- •Most common symptom
- •Burning chest pain behind breast bone
- •Moves upward toward throat
- •Worse after eating, lying down or bending down
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GERD – LIFESTYLE VARIABLES
- – Relaxed lower esophageal sphincter (LES)
- – Being overweight
- – Overeating
- – Caffeine / alcohol (increases amount of HCA)
- – Smoking
- – Gastritis
- – Ulcer disease
- – Stress
- – Nonsteroidal antiinflammatory drugs (NSAID’s) (over use of)
- • Aspirin & Ibuprofen
- – Certain foods
- • Citrus
- • Peppermint
- • Chocolate
- • Fatty & Spicy food
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GRED Diagnosis
Upper GI Series (Barium Swallow)
- – Ingestion of barium followed by x-rays
- > shows how well the sphincter is closing
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GERD Diagnosis
Esophagogastroduodenoscopy (EGD)
- – Endoscope used
- – Direct visualization of stomach and stomach wall
- – Can perform biopsy
- – Oral anesthetic (spray in mouth to decrease gag reflex)
- – Observe for return of “Gag Reflex” (can not eat or drink anything until the gag reflex has returned
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GERD Diagnosis
Esophageal Manometry
- – Determines the strength of the muscles in the esophagus
- – Small nasal tube
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GERD Diagnosis
PH Monitoring
- – Small nasal tube
- – Rest above LES
- – Last 12-24 hours
check to see the presents of acid over a period of time
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GERD Diagnosis
Bernstein Test
– Mild acid placed in the esophagus
creating mild signs of GERD
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GERD Diagnosis
BRAVO PH Monitoring
- - Places capsule next to LES
- - Detaches after 24 - 48 hours
- - Then download the results
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GERD – TREATMENT
- Diet and lifestyle changes
- Medications
- Quit smoking
- Observe food intake and food types
- Eat smaller portions
- Avoid overeating
- Watch alcohol consumption
- Do not lie down or go to bed right after eating
- Decrease fluid intake
- Lie on LEFT side, elevate HOB 30°
- Lose excess weight
- SURGICAL CORRECTION
- – Nissen Fundoplication NON-SURGICAL CORRECTION
- –Stretta Procedure
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GERD – Treatment determined by:
- –Age, overall health and medical history
- –Extent of condition
- –Tolerance to specific meds, procedures & therapies
- –Expectation for the course of the condition
- –Patient opinion or preference
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Heartburn mimics an MI
- must rule out MI before treating the heartburn
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GERD – TREATMENT
STRETTA PROCEDURE
- –Done on the LES
- –Use of Radiofrequency (radio waves)
- –Tiny cuts leading to scar tissue
- > which can help close/narrow the esophagal sphincter and relieve some of the symptoms

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GERD – MEDICATIONS
ANTACIDS
- Excessive use of these leads to an ion imbalance
- • Sodium bicarbonate
- • Calcium carbonate
- • Aluminum hydroxide
- • Magnesium hydroxide
- –Neutralize stomach acid
- –OTC
- –Tablet or liquid forms
- – Fast pain relief
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GERD – MEDICATIONS
H2-RECEPTOR BLOCKERS
- • Zantac (ranitidine)
- • Pepcid (famotidine)
- • Tagment (cimetidine)
- • Axid (nizatidine)
- - OTC or by prescription
- - Blocks histamine (in the stomach which produces acid)
- - Reduces acid and pain
- - Do not promote healing
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GERD – MEDICATIONS
PROTON PUMP INHIBITORS
- • Prevacid (lansoprazole)
- • Aciphex (rabeprazole)
- • Prilocec (omeprazole)
- • Protonix (pantoprazole)
- • Nexium (esomeprazole)
- - Blocks the enzyme in the stomach that produces acid
- - Promotes healing of the stomach and esophagus
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GERD – MEDICATIONS
PROKINETIC AGENTS
- • Reglan (metoclopramide)
- - Assists the stomach to empty more rapidly
- - May help tighten the LES
- - Prescription
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GERD – MEDICATIONS
ANTISPASMOTICS
- • Bentyl, Dibent (dicyclomine)
- • Levsin, Cystospaz (hyoscyamine)
- - Relaxes smooth muscles of intestine
- - Works to decrease digestion
- - Prescription
- - Makes the food move faster through the system
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GERD – MEDICATIONS
CYTOPROTECTIVE AGENTS
- - Prescription only
- • Carafate (sucralfate)
- • Cytotec (misoprostol)
- - OTC
- • Pepto-Bismol (bismuth subsalicylate) [black,tarry stool]
- - Protects lining of stomach & intestine
- - Does not decrease the amount of acid
- - Used to prevent ulcer formation
- - Must give an hour before meals and at bedtime
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GERD – COMPLICATIONS
- ESOPHAGITIS (inflammation of esophagus)
- ESOPHAGEAL STRICTURE (food can no enter the stomach -projective vomit)
- BARRETT’S ESOPHAGUS (changes the epithelium and is precancerous, can not return to normal)
- – Considered Precancerous
- HIATAL HERNIA
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NURSING DIAGNOSES for GERD
- Altered nutrition
- Pain (acute versus chronic)
- Altered sleep pattern
- Knowledge deficit
- Risk for fluid volume imbalance
- Risk for impaired swallowing
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GERD – Patient / family teaching
- – Foods
- –Smoking cessation
- –Stress avoidance
- –Medications and side effects
- – Importance of following medical regime
- –S&S to report to physician
- –Possible pre and post operative care
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HIATAL HERNIA CLASSIFICATIONS
- Type 1 –SLIDING: Stomach moves back and forth through hiatus of the diaphragm
- Type 2 –PARAESHOPHAGEAL or ROLLING: greater curvature of the stomach move above diaphragm forming a pocket
- > Food may accumulate in the pocket and cause bacterial changes and physical changes

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HIATAL HERNIA PRIMARY PREVENTION
- – unknown
- – Weakening of diaphragm muscles
- – Increased intra-abdomimal pressure
- – Increased age
- – Trauma
- – Poor nutrition
- – Forced recumbent positioning (if they have had a C1 or C2 fracture were they must lay on back)
- – Congenital
- – Obesity
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HIATAL HERNIA DEGREE OF REACTION
- – May be asymptomatic
- – Heartburn
- – Nocturnal heartburn
- – Dysphagia
- – Mimics gallbladder disease
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HIATAL HERNIA PRECIPITATING FACTORS
- – Large meals
- – Alcohol
- – Smoking
- - no tight clothes or tight belts
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HIATAL HERNIA COMPLICATIONS:
- – GERD (primary problem)
- – Hemorrhage (by ulcer formation)
- – Esophageal stenosis
- – Ulceration
- – Strangulation
- – Regurgitation with aspiration
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HIATAL HERNIA DIAGNOSIS:
– EGD and Barium Swallow
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HIATAL HERNIA SECONDARY PREVENTION–
- Conservative Therapy
- •Lifestyle Modifications
- •Medications
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HIATAL HERNIA SECONDARY PREVENTION–
Surgical Therapy
- •Nissen Fundoplication

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PEPTIC ULCER DISEASE (PUD)
- Erosion of the GI mucosa from the action of HCL and pepsin
- Includes gastric & duodenal ulcers
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PHASES OF PUD
- Erosion
- Acute Ulcer
- Perforated Ulcer (eats through all the layers of the stomach wall)
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PUD - ETIOLOGY
HELICOBACTER PYLORI (H. PYLORI)
- •80-90% OF ALL ULCERS
- •Bacterium infection
- •Weakens the stomach’s protective mucus
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PUD - ETIOLOGY
- Lifestyle Overactive acid & pepsin secretion
- Smoking
- Caffeine
- Alcohol
- Vagal nerve stimulation
- Stress
- Physical stress
- – “Stress Ulcers” (every pt prone)
- NSAIDs
- Corticosteroids
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PUD – DEGREE OF REACTION
- May be asymptomatic until serious complications occur.
- – Heartburn
- – Gnawing / burning pain
- – Acid, bitter, slimy taste in mouth
- – Belching / indigestion
- – Nausea / vomiting [presence of blood means need for HCP]
- – Weight loss & poor appetite
- – Feeling tired and weak
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PUD - COMPLICATIONS
HEMORRHAGE
- ALL ARE EMERGENCIES!
- - Most common
- - Black, tarry stools (MELENA)
- - Occult blood [in the beginning]
- - Emesis (coffee ground or fresh)
- - Have blood in the stomach (possibility of multiple bleeding sites [can loose LOTS OF BLOOD])
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PUD - COMPLICATIONS
PERFORATION
- ALL ARE EMERGENCIES!
- - Most lethal complication
- - Requires surgery
- - Causes peritonitis
- - S&S onset sudden and dramatic
- • Sudden, severe upper abdominal pain
- • Abdomen muscles contract - rigid & “board-like”
- • Respirations shallow & rapid
- • Absent bowel sounds
- • Tachycardiac
They need to open and wash the abdomen (may take some time to completly clean all the organs)
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PUD - COMPLICATIONS
GASTRIC OUTLET OBSTRUCTION
- ALL ARE EMERGENCIES!
- – Narrowing of pylorus
- • Scar tissue
- • Pylorospasm
- • Edema / Inflammation
- – Vomiting projectile (nothing getting to the stomach [smells really bad])
- – Contains food particles
- – Offensive odor
Immeadiate NG tube to decompress the stomach
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PUD – SECONDARY PREVENTION
DIAGNOSTIC PROCEDURES
- –Endoscopy: direct visualization (done 1st) [NPO]
- –H. pylori testing:
- sputum, urine, blood, tissue, breath
- • Urea breath shows active infection
- –Occult blood
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PUD – TREATMENTS
LIFESTYLE MODIFICATIONS
- – Bland diet & 6 small meals per day
- – Protein neutralizes but stimulates gastric secretions
- – Adequate physical / emotional rest
- – Stop ASA & NSAID’s (not always possible)
- – Strict adherence to prescribed meds
- – Antibiotic therapy for H. pylori
- • May use two or more antibiotics [long term]
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PUD – TREATMENTS DRUG THERAPY
– CARAFATE (Sucralfate)
- • Slurry (mix it up)
- • Give on empty stomach 1 hour before meals and bedtime.
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PUD – TREATMENTS DRUG THERAPY
– PEPTO-BISMOL
- • Promotes healing
- • Partially effective against H. pylori
- • May blacken stools
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PUD – TREATMENTS DRUG THERAPY
– CYTOTEC (Misoprostol)
- • For pts. taking ASA or NSAIDs who can not stop taking them
- • Prevents gastric ulcers induced by the above
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PUD – TREATMENTS
SURGICAL THERAPY
- –20% of ulcer patients
- – Indications:
- • Obstruction
- • Perforation
- • Hemorrhage
- • Ulcers unresponsive to treatment
- • Multiple ulcer sites
- • Possible malignancy
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BILLROTH I
- is an operation in which the pylorus is removed and the distal stomach is connected directly to the duodenum

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BILLROTH II
- is an operation in which the greater curvature of the stomach is connected to the first part of the jejunum in a side-to-side manner.
- The surgical procedure is called gastrojejunostomy
- – For gastric outlet obstruction
- – Food bypasses the obstruction

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VAGOTOMY
- –Truncal (total)
- –Cut all or part of the Vagus Nerve
- –Selective
- –Reduces acid
- –↓ gastric motility
- –Often combined with Billroth I & II

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PYLOROPLASTY
- –Surgical enlargement
- –Aids gastric emptying
- –Can do balloon angioplasty
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POST-OP COMPLICATIONS
DUMPING SYNDROME
- – Biggest issue
- – Result of large portion of stomach and pyloric sphincter removal
- – The food enters the small intestines before it can be properly digested/broken down
- – Do not drink liquids with meal
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POST-OP COMPLICATIONS
POSTPRANDIAL HYPOGLYCEMIA
- – Form of dumping syndrome
- – Large bolus of carbohydrates dumps into small intestine resulting in ↓BS (Increased carb diet will cause the pancrease to release insulin causing the blood sugar to drop)
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POST-OP COMPLICATIONS
BILE REFLUX GASTRITIS
– Related to surgery on pyloric sphincter
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PREOPERATIVE TEACHING
- NPO status (after midnight)
- The procedure itself
- C & DB, IS use, incisional splinting
- IV therapy (supplement IV therapy)
- NG tube (placed by OR sutured to the nose [DO NOT TOUCH])
- Pain relief
- Answering all patient questions
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POSTPERATIVE PATIENT CARE
- Promote comfort
- Promote effective airway management and gas exchange Monitor I & O
- – NG drainage: amount, color, odor
- – Bright red in beginning (normal), then coffeeground
- – Becomes yellow-green after 36-48 hours
- –DO NOT IRRIGATE OR REPOSITION
- Abdominal dressingdrainage, bleeding, odor
- ALWAYS AT RISK FOR ULCER REDEVELOPMENT
- Adequate rest, nutrition with avoidance of stressors
- EMPHASIZE avoidance of meds not prescribed by MD, alcohol and smoking
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GASTRIC CANCER
can migrate to the esophogus, duedoneum, or may mirgrate throught the stomach wall and spread to other organs and to distal lymph nodes
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GASTRIC CANCER PRIMARY PREVENTION
- – Twice as often in men (depends on diet)
- • Especially from lower socioeconomic class in urban areas
- – More often occurrence over age 55
- – Higher in African Americans than Caucasians
- – Highest incidence in Japan, South America, Eastern Europe and part of the Middle East.
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GASTRIC CANCER ETIOLOGY:
- UNKNOWN
- DIET
- – High in nitrates, salt and spiced foods
- FOOD PREPARATION– Preserved by salt-curing, smoking, pickling or drying
- PREPARATION ENVIROMENT– Poor drinking water or lack of refrigeration
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GASTRIC CANCER
GENETICS
- – Hx of GI cancer
- – Type A blood has higher incidence
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GASTRIC CANCER
MEDICAL CONDITIONS
– Pernicious anemia, chronic gastritis, intestinal polyps, GERD
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GASTRIC CANCER
ACQUIRED
- - Obesity
- - H. Pylori
- - Smoking
- - Occupational (rubber plants and coal mines)
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GASTRIC CANCER RISK FACTORS
- Gastritis
- Nitrites
- Poor nutrition
- Poor sanitation
- H. Pylori
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GASTRIC CANCER
S&S INDICATE ADVANCED DISEASE
- Abdominal pain Stool guiac +, GI bleeding
- Bloating after meals, indigestion, heartburn
- Diarrhea or constipation
- Fatigue, weak
- Anorexia, nausea, vomiting
- Weight loss
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GASTRIC CANCER DIAGNOSTIC STUDIES
- –Fecal occult blood
- –Complete blood count
- –Upper GI series
- –Endoscopic gastroscopy (EGD)
- •Permits biopsy
- –CT scan +/or PET scan (assessing to see if metestasis)
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GASTRIC CANCER SURGICAL INTERVENTION
- –Total Gastrectomy (Esophojejunostomy)
- • Stomach completely removed
- • Anastomosis of lower esophagus to the jejunum
- –Lymph node involvement
- –Followed by Chemo & Radiation
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GASTRIC CANCER
TERTIARY PREVENTION
- Compliance of prescribed dietary & drug regimes
- Keep appointments for chemo &/or radiation
- Referral to home care (TPN, nurse needs to hang)
- Long term follow-up stressed
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