cardiac meds

  1. Adrenergics



  2. Catecholamines

    dopamine, epinephrine, dobutamine
    Constrict - peripheral blood vessels

    Increase - heart rate

    Dialate - bronchi

    makes the heart contract more forcefully
  3. Non Catecholamines

    albuterol, isoetharine, alupent
    local / systmeic

    constriction - blood vessels
  4. Adrengergic Blockers

    disrupt sympathetic nervous system function

    Dilate - blood vessels

    Decrease - Blood pressure

    Increase - blood flow
  5. Beta-Adrenergic Blockers

    Beta 1 (heart)

    Beta 2 (bronchi, blood vessels, uterus)

    Selective - reduce stimulation to heart

    Non- constrict bronchioles of the lungs
    increase - peripheral vascular resistance

    decrease - blood pressure

    decrease - force of contractions

    decrease - oxygen consumption

    slow - conduction of impulses

    decrease - cardiac output
  6. Anti-Anginals
    chest pain from low o2 causing heart muscles to become ischemic
    Nitrates - accute

    Beta-Adrenergic blockers - long term

    Calcium channel blockers - when other meds fail
  7. Nitrates

    isodril, imdur, nitroglycerin
    dialte - veins / arteries

    decrease - blood return / amount in the ventricles at the end of diastole
  8. Beta-Adrenergic blockers

    metoprolol, lopressor, nadolom propranolol
    decrease - blood pressure

    decrease - heart rate

    force - heart contractions
  9. Calcium Channel blockers

    amlodipine, diltiazem, verapamil
    dilates - coronary / peripheral arteries

    decrease - heart contractions

    decrease - heart rate

    slows - conduction through SA and AV nodes
  10. Class 1 antiarrrhythmics

    norpace, procanbid,
    sodium channel blockers

    decrease - heart rate

    increase - conduction rate of AV node

    interfere with myocardial cell membrane, parasympathetic stimulation of SA nodes
  11. Class 1B antiarrhythmics

    accute ventricular arrhythmics

    lidocaine, xylocaine
    blocks - rapid influx of sodium ions during depolarization which decreases refractory period
  12. Class 1C antiarrhythmics

    flecainide, moricizine
    slow - conduction along heart

    decrease - inward flow of sodium ions which depresses the depolarization rate
  13. Class 2 antiarrhythmics

    propranolol, esmolol
    slow - SA node spontaneous firing

    decrease - strength of heart contractions
  14. Class 3 antiarrhythmics

    converts - unidirectional block to a bidirectional block, no effect on depolarization
  15. Class 4 antiarrhythmics
    slow - conduction / refractory period of calcium channel tissues

    Adenosine -

    reduces - heart rate

    depresses - SA node activity
  16. Anticoagulants
    reduce - blood to clot

    antiplatelet - prevent arterial thomboembolism

    clopidogrel - prevents clumping of platelets
  17. Heparin

    enoxaparin, lovenox
    prevents - deep vein thombosis

    monitor for decrease in platelets
  18. Oral Anticoagulants

    warfain, coumadin
    alters - liver to synthesize vitamin K
  19. Antihypertensives

    reduce blood pressure

    dialate - blood vessels

    dercrease - cardiac output
    central acting - clonidine, guanabenz

    alpha blockers - doxazosin, terazosin

    alpha / beta - labetalol

    norepinephrine - guanadrel
  20. Vasodilators

    hydralazine, minoxidil

    calcium channel blockers (arteriolar relaxiation)

    direct (vein,arteries)
    dilate - blood vessels

    decrease - blood pressure

    reduce - peripheral resistance
  21. ACE inhibitors

    captopril, lisinopril, ramipril
    interrupts - renin angiotensin - aldosterone system

    promotes retention of sodium and water

    increases - volume of blood
  22. Cardiac glycosides / PDE inhibitors

    digoxin, inocar
    increase - force of heart contractions

    reduce - heart rate

    reduce - conduction through AV node
  23. Diuretics
    excrete - water and electrolytes by the kidneys
  24. Loop Diuretics

    furosemide, lasix, bumex
    greatest - amount of volume removed

    inhibits - sodium, chloride, and water reabsorption
  25. Potassium-sparing Diuretics

    amiloride, triamterene
    conserves - potassium

    decrease - blood pressure

    increrase - serum potassium levels
  26. Thiazide Diuretics

    chlorthalidone, lozol
    increase - excretes of chloride, potassium, bicarbonate, which results in electrolyte imbalances

    decrease - circulating blood volume

    decrease - cardiac output
  27. Thrombolytics

    activase, reteplase
    dissolve - preexisting clot or thrombus
Card Set
cardiac meds
cardiac meds