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kidneys after birth
- cortex grows through childhood and pyramids become smaller
- not a large amount of cortical fat
- cortex fairly thin equal or greater echogen than the liver
- 4-6 months decreases to less than liver
- arcuate vessles-specular echos btwn cortex and medulla
- contour lobulated from fetal lobulation
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arcuate arteries
at base of pyramids
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adrenal glands
- larger in neonate than older infant and young child
- superior to upper pole of kidney
- left is more medial
- looks like inverted V in long, linear in short
- if kidney is absent adrenal gland is still in renal fossa
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bladder
- 3-5mm wall depending on distention
- color can be used to see distal ureter entering posterior wall of bladder
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hydronephrosis
- dilitation of collecting system
- common causes: obstruction, reflux, abn muscle development
- check other side and ureter and bladder for dilation
- grade hydro
-
ureteropelvic junction obstruction
- most common obstruction of urinary tract
- caused by intrinsic narrowing or extrinsic vascular compression
- hydro in kidney with normal ureter
- coronal scan plane best
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ureteral obstruction
- ureter obstructed anywhere- can be ureterovesicle junction
- can be caused by abcess or lymphnode
- may see diminished ureteral jet
-
bladder outlet obstruction
- bilateral hydro usually bladder or outlet
- can be neurogenic bladder, pelvic mass, or congen. anom. like uretheral valves
- thick bladder wall, urinary ascites, urinoma at kidney
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posterior urethral valves
- most common cause of bladder outlet obstruction in neonate male
- can be pelvic mass or tumor
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ectopic ureterocele
- females, left side
- ectopic insertion and dilitation of distal ureter in completely duplicated collecting system
- fluid mass within bladder inferiomedially to urethral insertion of lower pole ureter
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prune belly syndrome
- abdominal deficiency syndrome
- three things: hypoplasia or deficency of abd musculature, cryptorchidism, urinary tract anomolies
- dilated tortuous ureters, large bladder
- reflux common problem
- can be very severe with fatal pulmonary hypoplasia, dysplastic echogenic kidneys
- wrinkled prune like abdomen physically
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multicystic dysplastic kidney
- MCDK
- most common cause of renal cystic disease in neonate
- congenital- interference with ureteral bud function in first trimester
- ureteral atresia
- unilateral mass resembling a cluster of grapes
- bilateral is fatal
- may resemble ureteropelvic juncion obst
- involute and disappear completely or are small echogenic kidney
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medullary cystic disease
- indistinguishable from juvinile nephronophthisis
- small echogenic kidney variable sized cysts at corticomedullary junction
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autosomal recessive polycystic kidney disease ARPKD
- aka infantile polycystic disease
- not common
- female
- diffuse enlargement
- associated with biliary ectasia and hepatic fibrosis
- most severe in neonatal- less in infant/juv
- third trimester-kidneys protrude in abd
- hyperechoic enlarged with hypo outter rim (cortex)
- juv form- later on hepatic fibrosis and portal htn, gi bleeding
- overall bilateral enlarge echogenic kidneys with loss of definintion btw parts
- oligohydraminos 16-18wks inutero
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autosomal dominant polycystic kidney disease ADPKD
- adult form usually appears during middle ages
- 40s- htn, hematuria, enlarged kidneys
- cysts can be in liver, spleen, panc
- increased rcc
- well defined cysts on both kidneys
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renal cysts
like with adults can be associated with various syndromes (tuberous sclerosis- 40% incidence of cysts- and vonhippel lindau)
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acute pyelonephritis
- fever flank pain tenderness
- usually begins with bladder and ascends
- thickening of pelvis and ureter
- abcess forms as infection walls itself off
- can follow pts after antibiotic therapy
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chronic pyelonephritis
- repeated episodes of acute
- kidneys become scarred and decrease size
- cortex becomes more echogenic than liver
- pyramids difficult to separate from parenchyma
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renal vein thrombosis
- most likely in dehydrated or septic infant
- in infants of diabetic mothers
- renal enlargement, low platelet, proteinuria, hematuria
- thrombus starts in intrarenal venous branches if reaches renal vein or ivc may be visualized in vascular structures
- calcification may result
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congenital mesoblastic nephroma
- most common renal tumor of neonate
- aka fetal renal hamartoma or congential Wilms tumor
- rare
- connective tissue replaces parenchyma
- benign but looks like wilms, nephrectomy
- can extend through capsule into retroper.
- children less than 1 yr
-
nephroblastoma
wilms tumor
- most common intraabdominal malignant renal tumor in children
- 2-5 yrs
- unilateral usually
- distortion and displacement of collecting system
- can be monitored during chemo- surgery best
- liquification when necrosis and hemorrhage
- tumor may spread through renal veins, ivc, and into right atrium of heart. Also to other kidney
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neuroblastoma
- malignant tumor in adrenal medulla
- second most common tumor 2mo-2yr
- highly echogenic
- may have calcifications
- increased vascularity
- many pts have mets. check liver
- neuroblastoma is poorly defined whereas wilms is well encapsulated
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adrenal hemorrhage
- difficult delivery, large size, diabetic mother, stress/hypoxia at delivery, septicemia, predispose to adrenal hemorrhage
- abd mass, jaundice, anemia
- usually secondary to other complications
- ovoid enlargement of gland
- blunting of sup pole of kidney with inf placement
- looks like neuroblastoma
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