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Cardio Physio and Pathology Sem1 Mini3
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S1 heart sound
Mitral and tricuspid valve closure
splitting in 40% of normal but hard to hear or right bundle block
position near tricuspid valve left lateral to intercostal space 5
S2 heart sound
aortic and pulmonic valve closure
physiologial splitting during inspiration due to increased venous return
best heard left and right side of 2nd intercostal space
S3
Ventricular filling sound
Physiological in babies and thin (athelets)
Pathological in increased ventricular filling due to mitral valve reguirgitation (and dialated cardio myopathy)
Does not dissapear with atrial fibrilation (unlike S4)
S4 heart sound
Always pathological
associated with left ventricular hypertrophy(decreased ventricular distentability)
Dissapears with atrial fibrilation (which is why mitral valve reguirg will not cause S3 even when ventricular distentability is present)??
Aortic stenosis, hypertension, sever mitral and aortic reguirg
Aortic stenosis
Age and calification
or bicuspid genetic defect
Concentric left ventricular hypertrophy (S4)
Weakend pulse
Systolic murmur and S4 characteristic
Pulmonic stenosis
almost always congenital
high diasltic pressure and system edema?
Mitral Stenosis
Old age and rhmatic heard disease
causes Right atrial hypertrophy --> atrial fibrilation
Diastolic murmur
Mitral valve prolase
Typically due to connective tissue disorder
Myxomatous degeneration
secondary to chordae tendinae rupture and papillar muscle rupture from MI
S1 - click - shhh - S2
mid systolic click with late systolic murmur
Mitral valve insufficency /reguirg
Calcification and papillary muscle rupture
Systolic murmur and often S3 (rapid ventricular filling)
Atrial hypertrophy??
Atrial fibrilation ??
Acyantoic heart defects
Ventricular septal defect (most commmon)
atrial septal defect
patent ductus ateriosus
Atrioventricular setpal defect
Cyonotic Heart defect
Tetrology of Fallot (most common cyonotic)
transposition of the great arteries
persistant truncus arteriosus
Ventricular septal defect
most common heart malformation
Holostolic murmur (whoosh is all you hear)
chronic defect may result in eisenmengers syndrom
turning left --> right to right --> left due to high pulmonic pressure
Eisenmenger's syndrom
long term left to right shunting increases right ventricular size
increase pulmonary hypertension resulting in a reverse of flow
from right to left shunting when pulmonary pressure exceeds systematic pressure
ventricular septal defect and patent ductus arteriousis
Author
pandachowmein
ID
118678
Card Set
Cardio Physio and Pathology Sem1 Mini3
Description
Semester 1 mini 3 cardio
Updated
2011-11-23T16:17:05Z
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