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BEYOND MENDAL
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WILD TRAITS
SYMBOLIZED USING THE INITAL LETTER OR COMBINATION OF TWO OR 3 LETTERS WITH A +
DOMINATE TRAITS CAPITALIZED
RECESSIVE TRAI LOWER CASE
MUTANTS TRAIS ARE SYMBOLIZED USINT THE SAME LETTERS WITHOUT A +
INCOMPLETE DOMINANCE
NEITHER ALLELE DOMINTATES
THE HETEROZYGOUS INDICIDUAL HAVE AN APPEARANCE IN BETWEEN THE PHENOTYPES OF THE HOMOZYGOTES
CODOMINANCE
HETEROZYGOTE HAS JOINT OR EQUAL EXPRESSSION OF BOTH ALLELES
MULTIPLE ALLELE INHERITANCE
HAPPENS WHEN 3 OR MORE ALLELES EXIST FOR ONE GENE
2 TYPES OF MULTIPLE GENE INHERITANCE
POLYGENIC INHERITANCE
EPISTASIS
POLYGENIC INHERITANCE
ADDITIVE EFFECTS OF 2 OR MORE GENS ON SINGLE PHENOTYPE
SEVERAL GENS REGULATE SKIN COLOR
EPISTASIS
OCCURS WHEN THE ALLES OF ONE GENE COVER UP OR ALTER THE EXPRESSION OF ALLELES OF ANOTHER GENE
SEX CHROMOSOMES
CARY MORE GENES THAN THOSE INVOLVED IN SEX DETERMINATION
DISTICT PATTERN OF INHERITANCE CALLED X LINKED OR SEX LINKED INHERITANCE
X LINKED
GENES DETERMINING THE PHENOTYPE ARE CARRIED ON THE X CHROMOSOMES AS THE RESULT THE SEX EXPERIENCE VARYING PATTERN OF INHERITANCE
X LINKED
HEMOPHILI IN HUMANS IS LINKED TO THE X CHROMOSOME
FEMALES HAVE 2 X CHROMOROMES AS RESULTS THEY HAVE A BACK UP
SEX LIMITED
TRAITS THAT ARE EXPRESSED IN ONLY ONE SEX EVEN THO ITS NOT CARRIED ON THE SEX CHROMOSOMES
SEX INFLUENCED
PHENOTYPIC EXPRESSION IS CONDITIONED BY SEX OF THE INDIVIDUAL
HETEROZYGOTE MAY EXPRESS ONE PHENOTYPE IN ONE SEX AND THE ALTERNATE PHENOTYPE IN THE OPPOSITE SEX
PLEIOTROPY
IMPPACT OF A SINGLE GENE ON MORE THAN ONE CHARACTERISTIC
ONE ALLELE THAT IS EXPRESSED IN VARIOUS PHENOTYPIC FORMS
SICKLE CELL DISEASE
CHARACTERIZED BY A DIVERSITY OF SYMPTOMS
CYTOPLASMIC INHERITANCE
MITOCHONDRIA POSSESS A SMALL AMOUT OF DNA
600,000GENS IN THE NUCLEUS AND 37 GEIN IN MITOCHONDRIA
13 PROTEINS FOR CELLULAR RESPIRATION
22 TRANSFER RNA (TRNA) FOR TRANSLATION
2 RIBOSOMAL RNAS (RRNA) FOR TRANSLATION
CYTOPLASMIC INHERITANCE
MITOCHONDRIA DNA PASSED ON BY MOTHER ONLY
MITOCHONDRIA IN SPERM DO NOT TAKE PART IN GAMETE UNION
AS EMBRYO DIVIDES SO DOES THE MITOCHONDRIA
MITOCHONDRIAL DEFECTS
ARE INHERITEED THROUGH THE MATERNAL LINE
GENETIC DISEASES RESULT FROM HETEROPLASMY
MYOCLONIC EPILEPSY/RAGGED RED FIBER DISEASE (LACK OF MUSCLE COORDINATION, DEAFNESS,DEMENTIA, EPILEPTIC SEIZURES)
LEBERS OPTIC NEUROPATHY( BILATERAL BLINDNESS)
KEARNS(BLINDNESS DEAFNESS AND HEART CONDITIONS)
LINKED GENES
GENS LOCARED CLOSE TO EACH OTHER ON THE SAME CHROMOSOME
TYPICALLY INHERITED TOGETHER
DURING CROSSING OVER GENES CLOSE TO ONE ANOTHER DO NOT GET SEPERATED
CROSSING OVER
THE FATHER APART ON THE SAME CHROMO GENE ARE MORE LIKELY THEY WILL SEPARATE DURING RECOMBINATION
EXTREMES OF CROSSING OVER
INDEPENDENT ASSORTMENT & COMPLETE
ABSOLUTE LINKAGE
RECOMBINANT FREQUENCY
GREATER RECOMBINATION FREQUENCIES ARE OBSERVED FOR GENES THAT ARE FARTHER APART ON THE CHROMOSOMES
GENETIC SUPPRESION
SUPPRESSOR GENE CAUSES THE COMPLETE REVERSAL OF THE EXPECTED PHENOTYPE
POSITIONAL EFFECT
PHYSICAL LOCATION OF A GENE IN A GENOME MAY EFFECTITS EXPRESSION
TEMPERATURE SENSITIVE
MUTATIONS ARE TEMPERATURE SENSITIVE
NUTRITIONAL MUTATIONS
LACK OF NUTRIENT VITAMINS MINERALS OR ORGANICE MOLECURE PREVENT THE COMPLETE EXPRESSION OF THE GENE
SOME DISEASE IN HUMANS CAN BE DUE TO A NUTRIENT THAT GENES ARE INCAPABLE OF PROCESSING
GENOMIC IMPRINTING
DURING MEIOSIS SOME GENS ARE IMPRINTED
METHYL GROUPS ADDE ON CYTOSINES
CAUSE GENE TO INACTIVATE OR ACTIVATED
PHENOTYPE IN OFFSRPING DEPEND ON ALLELE FROM MOTHER OR FATHER
Author
datgrl_honey
ID
115757
Card Set
BEYOND MENDAL
Description
BEYOND MENDAL
Updated
2011-11-09T23:21:23Z
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