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Normal EKG
- P wave- atrial Depolarization
- PR - interval - time required for conduction to go from SA node to AV node
- QRS complex - atrail repolarization and ventricle depolarization
- ST segment - delay before repolarization of ventricle, useful in assessing mycardial ischemia
- T wave - ventricular repolarization
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Cardiac Output
- The amount of blood pumped out of the heart through the aorta each min
- Normal adult 5.6 (female 10% less)
- CO_ SV x HR
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Stroke Volume
Amount of blood ejected from the ventricles each contraction
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Venous Return
The amount of blood returning to the right atrium each minute similar in volume to cardiac output
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Primary Hypertension
- Unknown cause
- approximately 90-95%
-
Secondary Hypertension
- Related to another medical Problem
- Kidney disease
- Kidney failure
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Pathophysiology of HNT
- An increase prssure load fon the left ventricle leading to left ventricular hypertropy
- Increased dependence on ative "atrial kick" for left ventricle filling. IF inadequate L ventricle filling, stroke volume is decreased, leading to symptoms of decreased CO and pumonary congrestion
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Conplication to HNT
CVA, CHF, ASHD, renal failure, aneurysm, PVD or retinopaty
- Generally Asymptomatic
- Adults do not know what their BP usually is
-
Treatment for HNT
- weight reduction
- salt restriction
- aerobic exericse - at least 20mins 3-5 times per week has been shown to decrease BP
- Stress reduction/relaxation training
- Medication "anit-pyertensive" - beta blockers, diuretics, alpha adregergic blockers, calcium channel blockers. ACE inhibitors
-
Implication for Rehab for Pt with HNT
- Many Pts are undiagnosed
- Need to monitor BP at rest and exercise - terminate exercise if SBP 220-250 or DBP 110
- Be aware of side effects of anit - hypertensive medicatons
- AVOID HOLDING BREATH and VALSALVA
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Coronary Artery Disease (CAD) Pathology
- Atheroclerotic process
- Vasospasm
- Otehr disease processes taht affect coronary arteries
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Clinical Presentation of CAD
- 1. myocardiao ischemia (angina)
- 2. Myocardial Infarction
- 3. CHF
- 4. Sudden Death
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Myocardial Ischemia Pathology and Manifestations
- Patho - mayocardial O2 demand exceeds O2 supply
- Manifextations -
- Angina Pectoris
- Chest pain described as pressure, heaviness, tightness,
- last for minutes, relieve by rest and use of nitroglycerin
- Precipiated by exertion, stress, emotions, large meal
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Myocardial Infarction Manifestations
- Severes crushing pain with possible radiatin to shoulder, throat, jaw, arms or back
- lasts more than 30mins not relieved by rest or nitro
- Sweating, dyspnea, nausea/ vomiting, lightneaded, syncope, apprehension, weak, fatigues, DENIAL
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MI Diagnosis
- The Pt has classic S&S
- EKG changes - ST elevation with transural MI
- May see St depression with subendocradail MI
- Serum Enzymes
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Conservative Treatment of CAD
- Lifestyle Modifications - weight reduction, salt restriciton, aerobic exercise, stress reduction / relaxation training
- Medications - anti - angianl, anti- platelet, Anti -arthythmia, Thrombolytic Therapy
-
Surgial Treatment of CAD
- PTCA - precutaneous transluminal coronary angioplasty (balloon)
- CABG
- Pacemaker
- AICD: automatic implantable cardiac defibillator
- Intracoronary atents
- Arhrectomy
-
Implications for Rehab with CAD
- UE activities may cuase more symptoms than LE
- Use rest periods to decrease intensity of strenuous activites
- Use energy conservation techniques
-
Sternal Precautions
- NO not lift more than 8 lbs (a gallon of milk)
- Do not push of pull with your armswhen moving in bed and getting out of bed
- DO not flex of extend your shoulder over 90
- Avoid reaching too far across your body
- Avoid twisting or deep bending
- Do not hold your breath during activity
- Brace your chest when caughing or sneezing
- No driving
- Avoid long periods of over the shoulder activity
- If you feel any pulling or stretching in your chest, stop what you are doing
- Report any clicking or popping noise around your chest bone to your surgeon right away
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Conservative Treatment for CHF
- Lifestyle modifications
- rest
- Sodium restriction
- Phlebotomy if HCT. 55-65%
- Thoracentese - for pleural effusions
- Exercise training to improve peripheral efficiency
- Meds - diuretics, vasodiliators
- Supplemental O2
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Surgical Options for CHF
- Cardiomyoplasty - wrap a portion of latismus around heart to increase contractility
- Organ Transplants
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Implications for Rehab
- Physiological responses to activity need to be carefully monitored
- Side effects of med may cuase problems
- Activity modification may be indicated
- Exercise low level and slow progression
- Frequent rest breaks
- Use energy conservation techniques
- GRADUAL exercise training
- NEED TO WARM UP
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Valves of the Heart
- Aortic - between the left ventricle and the aorta
- Mitral - between the left atrium and left ventricle
- Pulmonary - between the right ventricle and pulmonary artery
- Tricuspid - bwtween the right atrium and right ventricle
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Valvular Stenosis
A restriced valve opening that creates a pressure load on the chamber preceding the abnoral valve which then develops compensatory hypertrophy
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Valvual Insufficiency
- Incomplete valve closer creates a volume load on the chambers or vessels on both sides of the affected valve
- Compensatory Hypertrophy
-
Treatment for Valve Disease
- Meds - digitalis, diurestics
- Salt resistriction
- Cardioversion - if arrhythmia is a problem
- Surgery - to repair or replacement
-
Clinical S&S for Valve disease
- Asymptomatic for years
- Aortic and mitral valve disease can produce S&S of left heart failure (pulmonary)
- Pulmonary and tricuspids valve disease can produce S&S of right side heart failure
-
Implications for Rehab for a person with Valve disease
- Activity and exercise precautions may be indicated
- Start Slow and Progress gradually
- Exercise training can improve functional status
-
What do abnormal rhythms cause
- Increaed HR
- Decreased HR and possible decreased CO
- lack of effective atrial contractions
- Loss of effective ventricular contraction
-
Clinical S&S of Arrhythmias
- May be asymptomatic
- Palpitations skipped beats, fluttering
- Symptoms of decreased CO - lightheaded, weak, dyspnea, syncope, confusion
- Symptoms of MI or cardiac arrest
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Treatment of Arhythmia
- No treatment may be necessary
- Treat the underlying cause
- Antiarrhyghmic drugs
- Cardioversion
- Pacemaker
- Automatic implantable cardioverter
- Defibrillatory
- Chemical or surgical ablation
- Surgical excision
-
Cardiac Rehab INdications
- Stable ANgina
- MI, CABG
- Compensated heart failure
- Cardiac surgery
- High risk for coronary heart disease
- High risk for hypertension
- End stage renal disease
- Status post pacemaker insertion
- Cardiomyopathy
- PVD
- Heart transplant
- High Risk for Diabetes
-
Contraindicationf for Cardiac Rehab
- Uncontrolled arrhythmias, unstable angina
- Recent diagnosis of emmbolis
- Resking DPB > 110 or SBP > 200
- Thrombophlebities
- Orthostatic BP
- Acute Infection
- Resting ST segment displacement >2mm
- Uncompensated CHF
-
Cardiac Rehab inpatient sessions
- inital assessment of vitals
- warm up
- Exercise (ambulate or bike)
- Cool down
- Monitor vitals S&S
- Pt Ed
- Rest -vitals, warm -up, training -vitals, cooldown -vitals
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STOP EXERCISE IF
- Abnormal HR taht increase 50BPM with low level eercise
- SBP > 210 pr DBP 110
- Decrease in SBP with low level exercise
- any ST segment changes
- Severe LE claudication
- Angina, mental confusion, extreme fatigues
- Ventricular gallop
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Cardiac Rehab Phase 1
- Referred to cardiac rehab when medically stable
- Initiate risk factor education and need for lifestyle modification
- Initiate self care activities progress from sitting to standing
- Provide an orthostatic challenge to the pt
- Begin a supervied ambulation program
- AROM
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Exercise Intensity Goals for Phase 1
- Target HR 18-24 bpm increase with ex
- RPE scale
- Monitoring the S&S of exercise intolerance
- Last 3-5 days while inpatient
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Pt Educations topics for phase 1
- Risk factors & lifestyle modification
- How to take own HR and monitor exercise intensity
- Graded ambulation program
- Normal vs. adverse response and what to do if they have an adverse response
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Phase 2 goals
- Increasing function capacity through eercise
- Continued patient education about risk factors, modification and self monitorying
- Beings immediately after hospitalization
- Lasts 2-12 weeks
- Frequency 2-3 times per week
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Phase 3
- Last 6-8 weeks and once a week
- Exercise training, physical fitness, level of endurance and risk factor modivication are typical goals
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Phase 4
- last thoughout the patients lifetime
- Designed to promote optimal health
- Maintencance activities
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